ライフサイエンスコーパス: cloacae

1 of M. morganii, C. freundii, E. coli, and E. cloacae).

2 n of their endophytic bacteria (Enterobacter cloacae).

3 o case patients yielded S. marcescens and E. cloacae.

4 most laboratories recognized as Enterobacter cloacae.

5 injection of prepupae with live Enterobacter cloacae.

6 ctive behaviors of the bacteria Enterobacter cloacae.

7 ) are Klebsiella pneumoniae and Enterobacter cloacae.

8 the type VI secretion system of Enterobacter cloacae.

9 rAB is dispensable for CAMP resistance in E. cloacae.

10 ical to plasmid pNDM-HF727 from Enterobacter cloacae.

11 segment (D-limited mice), with Enterobacter cloacae.

12 -1,3 dextran (DEX) expressed on Enterobacter cloacae.

13 r spp., P. aeruginosa, K. pneumoniae, and E. cloacae.

14 revealed that the organism was Enterobacter cloacae.

15 or treatment of infections with Enterobacter cloacae.

16 ri but 97.0% similar to that of Enterobacter cloacae.

17 eding, restored the immunity to Enterobacter cloacae.

18 /1; Klebsiella pneumoniae, 2/4; Enterobacter cloacae 1/4; and Stenotrophomonas maltophilia 2/8.

19 ebsiella pneumoniae (10.9%) and Enterobacter cloacae (16.3%) were the main MDRE species isolated.

20 Pseudomonas aeruginosa (27.5%), Enterobacter cloacae (16.8%), and Enterobacter asburiae (10.7%) were

21 re Pseudomonas aeruginosa (22), Enterobacter cloacae (21), Acinetobacter spp. (13), Enterobacter aero

22 herichia coli (14 patients) and Enterobacter cloacae (7 patients).

23 Klebsiella pneumoniae (14.2%), Enterobacter cloacae (9.1%), Acinetobacter spp. (6.2%), Serratia marc

24 nterpretive criteria, excluding Enterobacter cloacae (98.3% S) and E. faecalis (86.0% S), and MIC(90)

25 hia coli, Klebsiella pneumonia, Enterobacter cloacae, Acinetobacter baumannii, and methicillin-resist

26 Colonization of E. cloacae also reduced HuNoV titers in intestinal tissues

27 on cluster of blaKPC-2-positive Enterobacter cloacae among patients treated in a highly frequented ou

28 ortant enzymes CTX-M-15, KPC-2, Enterobacter cloacae AmpC, Pseudomonas aeruginosa AmpC, OXA-10, and O

29 ed for the class C enzymes from Enterobacter cloacae and Citrobacter freundii.

30 this study involving clinical isolates of E. cloacae and E. aerogenes, susceptibility testing methods

31 l biosynthetic enzyme MurA from Enterobacter cloacae and Escherichia coli in vitro.

32 vivo, we colonized gnotobiotic pigs with E. cloacae and inoculated pigs with 2.74 x 10(4) genome cop

33 n and skin-structure infections caused by E. cloacae and K. pneumoniae, and for treatment of communit

34 A case of ventriculitis due to Enterobacter cloacae and Pseudomonas fulva following placement of an

35 teremia, and seven of these had Enterobacter cloacae and S. marcescens in the same culture.

36 , approximately 10,000 nM), and Enterobacter cloacae and Serratia marcescens were highly resistant (I

37 t is, Klebsiella pneumoniae and Enterobacter cloacae) and their corresponding antimicrobial resistanc

38 ichia coli, Citrobacter koseri, Enterobacter cloacae, and clinical isolates of non-typhoidal Salmonel

39 Serratia marcescens, E. cloacae, and Enterobacter kobei presented high rates of

40 la pneumoniae, one (<1%) of 890 Enterobacter cloacae, and one (1%) of 162 Enterobacter aerogenes.

41 trains), Klebsiella pneumoniae, Enterobacter cloacae, and Pseudomonas aeruginosa.

42 a coli, Pseudomonas aeruginosa, Enterobacter cloacae, and Yersinia enterocolitica.

43 An outbreak of S. marcescens and E. cloacae bacteremia in a surgical intensive care unit was

44 ETEST FO should not be used for Enterobacter cloacae, because of low EA and a high VME rate.

45 s were chemically conjugated to Enterobacter cloacae beta-lactamase (bL), and their abilities to effe

46 wever, in our institution the MIC(90) for E. cloacae bloodstream isolates is 16 microg/ml.

47 genes revealed that one-third (15/45) of E. cloacae bloodstream isolates produced SHV-type extended-

48 several Gram-negative bacteria including E. cloacae but not against Gram-positive bacteria.

49 o effectively blocked adhesion of pili or E. cloacae, but no effect was observed with nonspecific ant

50 However, E. cloacae carries a chromosomally encoded ampC conferring

51 Here, we discover Enterobacter cloacae CD-NTase-associated protein 4 (Cap4) as a foundi

52 ly significant AmpC production (Enterobacter cloacae, Citrobacter freundii, and Klebsiella aerogenes

53 ndicates extensive dissemination of these E. cloacae clones across the UK and Ireland.

54 uNoV in gnotobiotic pigs, with or without E. cloacae colonization.

55 s, reduced HuNoV shedding was observed in E. cloacae colonized pigs, characterized by significantly s

56 In both control and E. cloacae colonized pigs, HuNoV infection of enterocytes w

57 pneumoniae (10/11 [90.9%]), and Enterobacter cloacae complex (2/4 [50%]).

58 e 3 most prevalent species were Enterobacter cloacae complex (42%), Klebsiella pneumoniae (18%), and

59 d patients with K. aerogenes or Enterobacter cloacae complex (Ecc) BSI from 2002 to 2015.

60 The Enterobacter cloacae complex (ECC) consists of closely related bacter

61 , Escherichia coli (n = 75) and Enterobacter cloacae complex (n = 57) also detected.

62 terminant was highly conserved within the E. cloacae complex and mediated resistance to up to 600 muM

63 sing clinical importance of the Enterobacter cloacae complex have often been discussed.

64 All E. cloacae complex strains, 89% of K. pneumoniae, and half

65 i, 55 Klebsiella pneumoniae, 21 Enterobacter cloacae complex, 18 Serratia marcescens, 12 Proteus mira

66 3 nonduplicate Enterobacter isolates (102 E. cloacae complex, 41 E. aerogenes) were tested, including

67 lent pathogens causing EOS in Bukavu were E. cloacae complex, K. pneumoniae, and S. marcescens.

68 a coli, Enterobacter aerogenes, Enterobacter cloacae complex, Klebsiella pneumoniae, or Klebsiella ox

69 Pseudomonas aeruginosa, n = 17; Enterobacter cloacae complex, n = 9; and Acinetobacter baumannii, n =

70 vast majority of clinical isolates of the E. cloacae complex, predominantly in (sub)species that freq

71 iella pneumoniae and Enterobacter cloacae/E. cloacae complex, the most commonly found isolates, were

72 . coli, Serratia marcescens and Enterobacter cloacae complex.

73 inically most prevalent subspecies of the E. cloacae complex.

74 cus aureus, and aac(3)-VIa from Enterobacter cloacae (conferring resistance to kanamycin, spectinomyc

75 nduction of the SOS response in Enterobacter cloacae decreased the amount of DNA measurable in the su

76 ates of Serratia marcescens and Enterobacter cloacae, demonstrating the presence of in-frame translat

77 coli, Klebsiella pneumoniae and Enterobacter cloacae/E. cloacae complex, the most commonly found isol

78 etion system (T6SS) activity in Enterobacter cloacae (ECL).

79 alternative nitroreductase from Enterobacter cloacae enables the tailoring of a photoenzymatic system

80 clinical isolates representing Enterobacter cloacae, Enterobacter bugandensis, Enterobacter kobei, E

81 freundii, Clostridium species, Enterobacter cloacae, Enterococcus faecalis, Klebsiella oxytoca, Kleb

82 nterobacteria Escherichia coli, Enterobacter cloacae, Erwinia herbicola, and Salmonella typhimurium.

83 r pathogenic bacteria including Enterobacter cloacae, Escherichia coli J96, Pseudomonas aeruginosa, p

84 important Gram-negative species-Enterobacter cloacae, Escherichia coli, Klebsiella pneumoniae, and Ps

85 As in other bacterial species, Enterobacter cloacae form macroscopic aggregates.

86 The Enterobacter cloacae GC1 enzyme is an example of a class C ES beta-la

87 ystallographic structure of the Enterobacter cloacae GC1 extended-spectrum class C beta-lactamase, in

88 The beta-lactamase of E. cloacae GC1, an extended spectrum mutant of the P99 enzy

89 erobacterales (K. pneumoniae, E. coli and E. cloacae) harboured multiple cephalosporin and carbapenem

90 the nitroreductase enzyme from Enterobacter cloacae has been determined for the oxidized form in sep

91 Enterobacter cloacae has been implicated as one of the causative agen

92 tly, enteric commensal bacteria Enterobacter cloacae has been recognized as a helper in HuNoV infecti

93 After E. cloacae immunization, MZ B cells were responsible for th

94 plement-mediated killing of H. pylori and E. cloacae, indicating its potential as a cross-species ant

95 raocular infection secondary to Enterobacter cloacae infection is a devastating disease which, despit

96 bleb-related endophthalmitis secondary to E. cloacae infection.

97 population underlies multidrug-resistant E. cloacae infections within hospitals.

98 een described in the context of Enterobacter cloacae infections.

99 In summary, E. cloacae inhibited HuNoV infectivity, and B cells were no

100 H2O2 in the leaves of plants 3h after the E. cloacae inoculation, according to a mechanism involving

101 Enterobacter cloacae is a clinically important Gram-negative member o

102 Enterobacter cloacae is a Gram-negative nosocomial human pathogen tha

103 We describe an Enterobacter cloacae isolate harbouring a minor subpopulation that is

104 tematic collection of multidrug-resistant E. cloacae isolated between 2001 and 2011 from bloodstream

105 ion) was achieved in only 76/114 (67%) of E. cloacae isolates (65 susceptible, 11 resistant).

106 and 7 previously determined PB-resistant E. cloacae isolates from JMI Laboratories.

107 mid was present in K. oxytoca,E. coli and E. cloacae isolates from unlinked patients within the same

108 oxytoca, Escherichia coli, and Enterobacter cloacae isolates from unlinked patients within the same

109 enes isolates and 6.0% of the results for E. cloacae isolates tested with the Vitek system.

110 Enterobacter cloacae isolates were resistant to all first-line antibi

111 3 Escherichia coli isolates, 5 Enterobacter cloacae isolates, 2 S. marcescens isolates, 1 Proteus mi

112 All but two of the ESBL-producing E. cloacae isolates, but none of the non-ESBL-producing str

113 hods were generally unreliable with these E. cloacae isolates.

114 This protein was termed Enterobacter cloacae lectin A (EclA) and found to bind l-fucose.

115 We demonstrate that E. cloacae lipid A is modified with l-Ara4N to induce CAMP

116 reevaluation of cefepime breakpoints for E. cloacae may be prudent.

117 ructures of the D305A mutant of Enterobacter cloacae MurA and the D313A mutant of Escherichia coli Ar

118 icted the Cys-115-PEP adduct of Enterobacter cloacae MurA in various reaction states by X-ray crystal

119 Cys115 of Escherichia coli and Enterobacter cloacae MurA is the active site nucleophile alkylated by

120 ined to be 8.3, by titration of Enterobacter cloacae MurA with the alkylating agent iodoacetamide as

121 tructure of the C115S mutant of Enterobacter cloacae MurA, which was crystallized in the presence of

122 Escherichia coli (n = 149), and Enterobacter cloacae (n = 110).

123 36), Escherichia coli (n = 22), Enterobacter cloacae (n = 23), Klebsiella oxytoca (n = 8), Serratia m

124 coccus epidermidis (n = 100) or Enterobacter cloacae (n = 60).

125 ichia coli (n = 129, 30.0%) and Enterobacter cloacae (n = 62, 14.4%) were the main Enterobacteriaceae

126 avin mononucleotide cofactor of Enterobacter cloacae nitroreductase (NR), determined under a variety

127 Thus E. cloacae NR is not a good candidate for enzymatic product

128 insensitive nitroreductase from Enterobacter cloacae (NR) catalyzes two-electron reduction of nitroar

129 Catheter infection with E. cloacae occurred in 50% of roll plates and 80% of broth

130 To test the influences of E. cloacae on HuNoV infectivity and to determine whether Hu

131 icroarray analysis of Aag2 challenged with E.cloacae or infected with Dengue virus revealed high tran

132 ation of either wound model with E. coli, E. cloacae, or A. baumannii, produces the typical 'S'-shape

133 Unlike wild-type (WT) forms, such as the E. cloacae P99 and Citrobacter freundii enzymes, the ES GC1

134 A library of Enterobacter cloacae P99 beta-lactamase mutants was produced to inves

135 cephalosporin hydrolysis by the Enterobacter cloacae P99 cephalosporinase (beta-lactam hydrolase, EC

136 tomyces sp. R61, a PBP, and the Enterobacter cloacae P99 cephalosporinase, a class C beta-lactamase.

137 f the class C beta-lactamase of Enterobacter cloacae P99 determined.

138 s the class C beta-lactamase of Enterobacter cloacae P99 in a covalent fashion.

139 h the class C beta-lactamase of Enterobacter cloacae P99 in two ways, by acylation and by phosphylati

140 drolysis by beta-lactamase from Enterobacter cloacae P99 indicated kcat values of 476 +/- 170 and 248

141 The class C beta-lactamase of Enterobacter cloacae P99 is closely similar in structure to the DD-pe

142 The class C beta-lactamase of Enterobacter cloacae P99 is competitively inhibited by low concentrat

143 lass C serine beta-lactamase of Enterobacter cloacae P99 is irreversibly inhibited by O-aryloxycarbon

144 r the class C beta-lactamase of Enterobacter cloacae P99 suggest an acyl-transfer transition state fo

145 The class C beta-lactamase of Enterobacter cloacae P99 was employed.

146 al class A (TEM-2) and class C (Enterobacter cloacae P99) beta-lactamases in a time-dependent fashion

147 y the class C beta-lactamase of Enterobacter cloacae P99, have been studied in order to more firmly e

148 etected Escherichia coli CMY-2, Enterobacter cloacae P99, Klebsiella pneumoniae ACT-1, and the AmpC b

149 d the class C beta-lactamase of Enterobacter cloacae P99.

150 milar class C beta-lactamase of Enterobacter cloacae P99.

151 f the class C beta-lactamase of Enterobacter cloacae P99.

152 ta-lactamases such as that from Enterobacter cloacae P99.

153 eumoniae, Proteus mirabilis, or Enterobacter cloacae promoted greater recruitment of neutrophils to t

154 negative bacteria, specifically Enterobacter cloacae, Pseudomonas aeruginosa, and Pantoea agglomerans

155 solubilizing bacterial strains Enterobacter cloacae, Pseudomonas pseudoalcaligenes, and Bacillus thu

156 production of DEX-specific Ab titers upon E. cloacae rechallenge.

157 (e.g., Helicobacter pylori and Enterobacter cloacae), revealing it to be a promising probe for detec

158 robacter lignolyticus (formerly Enterobacter cloacae) SCF1 (enterolysin [ELY]).

159 pergillosis (20%, 3 of 15), and Enterobacter cloacae, Serratia marcescens, Pneumocystis carinii pneum

160 O157:H7, Klebsiella pneumoniae, Enterobacter cloacae, Shigella dysenteriae, Salmonella enterica, S. p

161 only Klebsiella pneumoniae and Enterobacter cloacae) showed the following performance: EA = 97.9% (4

162 ulates LDs when challenged with Enterobacter cloacae, Sindbis, and Dengue viruses.

163 ane-bound selenate reductase of Enterobacter cloacae SLD1a-1 is purified in low yield and has relativ

164 he membrane-bound selenate reductase from E. cloacae SLD1a-1.

165 such as Klebsiella pneumoniae, Enterobacter cloacae, Stenotrophomonas maltophilia, and the Burkholde

166 Enterobacter cloacae strain G6809 with reduced susceptibility to carb

167 mase from a clinical isolate of Enterobacter cloacae strain GC1 with improved hydrolytic activity for

168 Shiga toxin 1 (Stx1)-producing Enterobacter cloacae strain, M12X01451, from a human clinical specime

169 erogenes and 13,954 isolates of Enterobacter cloacae tested using a Vitek system; for the same specie

170 iose-H(+) symporter (MelY) from Enterobacter cloacae that had enhanced fermentation on 1% maltose Mac

171 The adhesion of radiolabeled E. cloacae to HT-29 cells was concentration and temperature

172 es of endophthalmitis caused by Enterobacter cloacae: two in patients with acute postoperative endoph

173 The bacterial isolate was identified as E. cloacae using a polyphasic approach that included phenot

174 than the totals were noted with Enterobacter cloacae versus ampicillin-sulbactam, aztreonam, ticarcil

175 and shows that MDR plasmid acquisition in E. cloacae was not indicative of success in the hospital.

176 This contrasted E. cloacae where there was no temporally changing pattern,

177 0(6) colony-forming units/mL of Enterobacter cloacae with the third feeding.

178 eria (Klebsiella pneumoniae and Enterobacter cloacae) with a detection limit of 10 3 bacteria/mL, whi

179 ng of particles and bacteria (E. coli and E. cloacae) with developmental age and exposure to NEC stre

180 used by carbapenemase-producing Enterobacter cloacae within 21 days of cefiderocol therapy.