戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 served mainly in tubular epithelial cells in CO-treated, but not air-exposed, kidney grafts at 3 hr a
2 rate that SFN reverses the effects of PMI in co-treated cells by reducing the accumulation of p62 in
3 s enhancer by nuclear proteins isolated from CO-treated cells, although HIF-1 protein levels were una
4 gher mineral to matrix ratios of DEX/rhBMP-2 co-treated cultures compared with control.
5 suppressing TNF-alpha at micromolar doses in co-treated glial cells with LPS.
6                             These changes in CO-treated grafts correlated with well-preserved CD31(+)
7 owed Ngb distribution in normal controls and CO treated groups.
8 monstrate interesting activity profiles when co-treated in a panel of metal(II) cations in MIC assays
9 ted females for chlamydial infection, and to co-treat individuals with gonorrhea for chlamydia.
10 d upregulation of HIF-1alpha and VEGF in the CO-treated kidney grafts as early as 1 hr after reperfus
11                         We demonstrated that co-treated mice experienced wasting and increased AHR ac
12 )types were different between TCDD alone and co-treated mice including important mediators of NAFLD p
13                        In a lethal 85% PHTx, CO-treated mice showed a greater survival rate compared
14 d more then 9 hours earlier in the livers of CO-treated mice.
15                                         When co-treating normal STBEVs with HbF, the miRNA deposition
16 s to beta-amyloid (Abeta) peptides, we first co-treated primary cultured microglia with a tyrosine ph
17 ding produced by TNF-alpha were abrogated by co-treating the cells with dexamethasone.
18 otably, 10, 1 and 0.1 microM 3Me-H TRH, when co-treated with 500 microM Glu, protected fetal neurons
19 10 cells were treated with an FA mixture and co-treated with 8-Br-cAMP to stimulate the steroidogenes
20 induction of hsp72 mRNA was blocked in cells co-treated with actinomycin D. hsp72 mRNA levels were af
21      Thus, when BFA-treated HepG2 cells were co-treated with ALLN, which protects apoB but does not f
22 ved GBM cells cultured in 3D microwells were co-treated with BAY 11-7082 and TMZ or BAY 11-7082 and T
23 g7 or Atg5 prolonged the survival of neurons co-treated with BDNF and rapamycin, suggesting that supp
24 tion of K-Ras was highly resistant even when co-treated with both inhibitors.
25 a dramatic decrease in the survival of cells co-treated with Casodex and ATM inhibitor as compared wi
26 e in contrast to what we saw when cells were co-treated with cisplatin plus an EGFR tyrosine kinase i
27                               However, cells co-treated with CPT-11 and Apo2L/TRAIL, or pretreated wi
28 nover as similar findings were seen in cells co-treated with cycloheximide.
29 so were studied in MG-63 cells that had been co-treated with either sterol and transforming growth fa
30  reactive oxygen species (ROS) scavenger, is co-treated with ETO, it inhibits an ETO-induced increase
31         After characterization, PL-MSCs were co-treated with fucoxanthin and H(2)O(2) for 24 h (co-tr
32 17beta-estradiol (estrogen) for 1 h and then co-treated with glutamate, apoptotic death was significa
33  mitigates phosphorylation of STAT5 in cells co-treated with hGH, reducing intracellular STAT5 phosph
34 ellular matrix (ECM) degradation, cells were co-treated with interleukin-1 (IL-1).
35 served when the GBM-derived U87MG cells were co-treated with LPS and Temozolomide (TMZ) in comparison
36           Fractionation of pol II from cells co-treated with MG132 and cisplatin indicated that the u
37 excitability is not present when animals are co-treated with NaBut suggesting gut derived mediators m
38 ment with recombinant MMP-9, either alone or co-treated with native TIMP-1, TIMP-3, or the engineered
39         (13)C flux analysis of H4IIEC3 cells co-treated with palmitate and the pan-transaminase inhib
40 ted in response to thapsigargin if they were co-treated with PMA, while adherent cells secreted in re
41                                       M-SMCs co-treated with polyI:C and a polyclonal antibody to Ial
42 er 2 weeks and by 60% (P < 0.01) in dko mice co-treated with prednisolone over an 8 week treatment pe
43 iated degradation is reversed when cells are co-treated with TCDD and Erk kinase inhibitors.
44 deficient HL-60/MX2 cells and in HL-60 cells co-treated with the Top II catalytic inhibitor ICRF-193.
45 anel was tested in mice bearing U87-MG cells co-treated with TMZ.
46 proviral DNA synthesis as compared with MDMs co-treated with Vpx+ VLP and HIV vector.