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1 s in the 170, 186, and 220 loops to those of coagulation factor Xa.
2 asured in terms of its inactivation of blood coagulation factor Xa.
3 ontaining the m-cyano group was observed for coagulation factor Xa and for the factor VIIa.tissue fac
4 ne protease inhibitor that directly inhibits coagulation factor Xa and regulates blood coagulation vi
5 Kunitz-type protease inhibitor that inhibits coagulation factor Xa and, in a factor Xa-dependent mann
6 o enhance thrombin generation due to binding coagulation factors Xa and Va and accelerating prothromb
9 otease inhibitor, enhances the inhibition of coagulation factor Xa, and protein Z-dependent protease
10 es of porcine trypsin, human thrombin, human coagulation factor Xa, and the Bacillus subtilis dibasic
11 es of water transferred during inhibition of coagulation factor Xa are compared to water-permeable vo
12 uronate containing pentasaccharide inhibited coagulation factor Xa by 50% relative to the parent comp
13 ) serves as a cofactor for the inhibition of coagulation factor Xa by a plasma protein called PZ-depe
14 or to dramatically enhance the inhibition of coagulation factor Xa by the serpin, protein Z-dependent
15 receptor, hepatocyte growth factor receptor, coagulation factor Xa, contactin 4, kynureninase, neurog
16 o using a conformationally pliant variant of coagulation factor Xa (FXa(I16L)) rendered partially ina
18 nstrate synergistic interactions between the coagulation factor Xa (fXa) and the proinflammatory cyto
19 otent tick salivary anticoagulant that binds coagulation factor Xa (FXa) and zymogen FX, with formati
20 or VIIa) pathways in the coagulation system, coagulation factor Xa (FXa) has been a theoretically int
23 ly inhibited the catalytic activity of blood coagulation factor Xa (fXa), while a third form (AcAPc2)
31 human trypsin and plasmin but not thrombin, coagulation factor Xa, or urokinase-type plasminogen act