1 n, with Deltapgp1DeltaamiA producing minimal
coccoids.
2 .50-10.71%), Eubacterium rectale/Clostridium
coccoides (
1.37-3.70%), and C. histolyticum (0.91-2.30%)
3 of shapes that are Pareto-optimal, including
coccoids,
all straight rods, and a range of curvatures.
4 ussed ion beam microscopy (FIB-SEM) revealed
coccoid and filamentous-like structures within subsurfac
5 Neisseriaceae, which includes Gram-negative
coccoid and rod-shaped species.
6 nd temporary suppression of both Clostridium
coccoides and Clostridium leptum group organisms.
7 tant show a striking morphology of irregular
coccoids and aberrant DNA replication.
8 how cellular clusters of unexpectedly large
coccoids and tubular sheath-like envelopes were trapped
9 Coccoids are observed in patients and during in vitro gr
10 ngle perpendicular to their long axis, while
coccoid bacteria like Staphylococcus aureus rotate plane
11 ganized biofilm-like community consisting of
coccoid bacteria that ultimately filled most of the cyto
12 Here, we elucidate why
coccoid bacteria, such as Staphylococcus aureus, also po
13 In most non-
coccoid bacteria, this shape is also determined by an in
14 try as a general organizational principle in
coccoid bacterial cell division.
15 Strain 195, a
coccoid bacterium that dechlorinates tetrachloroethene t
16 to as Bacteroidetes), Clostridium leptum, C.
coccoides,
bifidobacteria, Escherichia coli and Archaea
17 the inability of the immune system to detect
coccoid C. jejuni may be significant in its pathogenesis
18 ted in differences in pathogenic properties;
coccoid C. jejuni were non-motile and non-infectious, wi
19 Clone libraries targeting Clostridium
coccoides (
C. coccoides) in sewage samples demonstrated
20 M. verstraetei are
coccoid cells with archaella and chemoreceptor arrays, a
21 rmation from spiral-shaped bacteria to round
coccoid cells.
22 , the submucosal microbiota was dominated by
coccoid cells.
23 rized by increased proportion of Clostridium
coccoides (
cluster XIVa), C coccoides-Eubacterium rectal
24 th SS and HC (P = 0.006) and higher fecal C.
coccoides compared to those with SS (P = 0.04).
25 ion resulted in a revised growth model where
coccoid cyanobacteria predominate in mat communities for
26 ommunities develop, which include endolithic
coccoid cyanobacteria.
27 r time, but only strains capable of becoming
coccoid displayed tripeptide changes.
28 Thus, in common with
coccoids,
Drosophila is capable of generating an imprint
29 n of Clostridium coccoides (cluster XIVa), C
coccoides-
Eubacterium rectale (cluster XIVab), Bacteroid
30 es uniformis, Eggerthella lenta, and Blautia
coccoides-
Eubacterium rectale groups (P < 0.05).
31 against Bacteroides, Prevotella and Blautia
coccoides-
Eubacterium rectale.
32 Here we show that the
coccoid form of H. pylori, in contrast to the spiral for
33 environment transforms into a nonculturable,
coccoid form, which frequently results in the failure to
34 ake and a morphological shift from spiral to
coccoid form.
35 iral form to the nonreplicating, but viable,
coccoid form.
36 The organism has the propensity to become a
coccoid form.
37 nd a nonculturable, but viable, metabolizing
coccoid form.
38 Oxidative stress induces
coccoid formation and is associated with repression of t
39 Mutants in pgp1 and amiA showed reduced
coccoid formation, with Deltapgp1DeltaamiA producing min
40 .05) and in an increase in the proportion of
coccoid forms (P <0.0001) relative to baseline.
41 Because only spiral organisms-and not
coccoid forms-are capable of inducing interleukin-8 secr
42 so adopt straight rod, elongated helical and
coccoid forms.
43 the planktic cyanobacterium, Microcystis, a
coccoid genus that at the present-day commonly forms blo
44 otags encompassed the previously reported C.
coccoides group.
45 Coccoid H. pylori, which are thought to be terminally di
46 ne, but reduced the abundance of Clostridium
coccoides in the caecum.
47 ibraries targeting Clostridium coccoides (C.
coccoides)
in sewage samples demonstrated that Lachnospi
48 The peptidoglycan composition of
coccoids is modified with respect to spiral bacteria.
49 Strain SL100 is a gram-positive
coccoid isolate prototype with an adhesin specific for g
50 These
coccoids modify the sediment, forming thicker lithified
51 demonstrate here that iron depletion induces
coccoid morphology in strains lacking cagA.
52 at the role of this endopeptidase in forming
coccoid morphology may be critical for pathogenesis.
53 Concomitantly, changing to a
coccoid morphology resulted in differences in pathogenic
54 their activity must be balanced to maintain
coccoid morphology.
55 nd undergoes a premature transformation to a
coccoid morphology.
56 d the normal transition to a densely packed,
coccoid morphology.
57 ced long axis of rod-shaped bacteria, in the
coccoid N. gonorrhoeae, ParB segregates along this long
58 res from which the poles disappear, yielding
coccoid or lemon-shaped forms.
59 dult human gut also known as the Clostridium
coccoides or Eubacterium rectale group, contains species
60 However, for Neisseria gonorrhoeae, a
coccoid organism that most commonly exists as a diplococ
61 ry and division rotation in the understudied
coccoid pathogen Neisseria gonorrhoeae.
62 Coccoid peptidoglycan exhibited reduced activation of in
63 As C. jejuni transformed from helical to
coccoid,
peptidoglycan dipeptides increased and tri- and
64 s zoonotic disease caused by a Gram-negative
coccoid rod bacterium, Francisella tularensis.
65 ere, in some species, most notably among the
coccoids (
scale insects and allies), the differential ma
66 During this period, cells adopt an almost
coccoid shape and become tolerant to antibiotics.
67 strains treated with CBR-4830 transition to
coccoid shape, consistent with MreB inhibition or deplet
68 , we hypothesise that most major lineages of
coccoids shifted from gymnosperms onto angiosperms when
69 aled endocarditis with small silver positive
coccoid structures in endothelial cells.
70 In
coccoids,
such as the human pathogen, Staphylococcus aur
71 The AapA1 toxin, first molecular effector of
coccoids to be identified, targets H. pylori inner membr
72 lanyl amidase AmiA were both involved in the
coccoid transition.
73 ogen that changes morphology from helical to
coccoid under unfavorable conditions.
74 ion result from AapA1 expression, suggesting
coccoid viability.
75 The difference in C.
coccoides was no longer significant after adjusting for
76 Our data support the hypothesis of viable
coccoids with characteristics of dormant bacteria that m
77 C. jejuni also transitioned to
coccoid within epithelial cells, so the inability of the