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1 on of release-ready synaptic vesicles in the cochlear hair cell.
2 two shorter rows of stereocilia in mammalian cochlear hair cells.
3 tion of the hair bundle is lost in mammalian cochlear hair cells.
4 rons and mechanosensory stereocilia of mouse cochlear hair cells.
5 lly in retinal photoreceptors and developing cochlear hair cells.
6 al photoreceptors and for the development of cochlear hair cells.
7 lification of the mechanical stimulus by the cochlear hair cells.
8 lfactory receptor neurons and vestibular and cochlear hair cells.
9 myosin XVa mRNA and protein are expressed in cochlear hair cells.
10 rters and may explain observed properties of cochlear hair cells.
11 wth of stereocilia bundles in vestibular and cochlear hair cells.
12 constant-frequency neurons and contralateral cochlear hair cells.
13 rgans and is required for normal function of cochlear hair cells.
14 26SB), on mechano-electrical transduction in cochlear hair cells.
15 technique to quantify this deflection in rat cochlear hair cells.
16 inetic range needed for electrical tuning of cochlear hair cells.
17 ganisation and has a role in the function of cochlear hair cells.
18 re ototoxic and cause irreversible damage to cochlear hair cells.
19 ng from central neurons to spinal ganglia to cochlear hair cells.
20  perhaps because of early disorganization of cochlear hair cells.
21 lication of photonic force to vestibular and cochlear hair cells.
22 sts that TMC1 is a better isoform for use in cochlear hair cells.
23 ysis found that Crmp1 is highly expressed in cochlear hair cells.
24  mechanical deflection of the hair bundle of cochlear hair cells.
25 cochlear tissues makes it difficult to study cochlear hair cells.
26 deafness, and MET is drastically impaired in cochlear hair cells.
27 ccount for the sole use of TMC1 in the adult cochlear hair cells.
28  synergistically in USH2 complex assembly in cochlear hair cells.
29 e pore of the mechanotransduction channel of cochlear hair cells.
30 nsors, or pumps to control calcium levels in cochlear hair cells.
31  the transducing stereocilia in mature mouse cochlear hair cells.
32 described the loss of afferent contacts from cochlear hair cells.
33 rophages following the selective ablation of cochlear hair cells.
34 nce of the transducing stereocilia in mature cochlear hair cells.
35 he USH2 complex at ankle links in developing cochlear hair cells.
36 oration to express cDNAs and shRNAs in mouse cochlear hair cells.
37 s, resulting in a toxic microenvironment for cochlear hair cells.
38 ter rows of transducing stereocilia in mouse cochlear hair cells.
39 reocilia of teleost vestibular and mammalian cochlear hair cells.
40  the improper development or degeneration of cochlear hair cells.
41 ngth of the cochlear duct, and the number of cochlear hair cells.
42 oustic signal acquisition and propagation in cochlear hair cells.
43 erlie the late acquisition of BK currents in cochlear hair cells.
44 hat regulates synaptic vesicle exocytosis in cochlear hair cells.
45  and temporally precise neurotransmission by cochlear hair cells.
46                                 Here, we use cochlear hair cell ablation to isolate the effects of SN
47                                           In cochlear hair cells, AC6 distribution relies on an adhes
48                                           In cochlear hair cells, ACF7 localization is graded, with t
49 everse-polarity" current was investigated in cochlear hair cells after tip-link destruction with BAPT
50 pment, but little is known about its role in cochlear hair cell and Deiter's support cell development
51                                              Cochlear hair cell and neuronal function was assessed vi
52 specific protease domain and is expressed in cochlear hair cells and a subset of supporting cells.
53 posure destroys synaptic connections between cochlear hair cells and auditory nerve fibers; however,
54 tion of the mechanotransduction machinery of cochlear hair cells and how this machinery contributes t
55 chano-electrical transducer (MET) channel in cochlear hair cells and is subject to numerous mutations
56                              For survival of cochlear hair cells and preservation of hearing, NO-medi
57 l ganglion neurons (SGNs) receive input from cochlear hair cells and project from the cochlea to the
58 sts of spontaneous activity are generated in cochlear hair cells and propagated along central auditor
59  efferent system, which provides feedback to cochlear hair cells and sensory neurons.
60                               We report that cochlear hair cells and supporting cells in transcriptio
61  vector for inner ear gene therapy targeting cochlear hair cells and supporting cells, and it will li
62  Tmc2 were expressed in mouse vestibular and cochlear hair cells and that GFP-tagged TMC proteins loc
63           GATA2 regulates the development of cochlear hair cells and the inferior colliculus (IC), wh
64  loading zinc into vesicles, is expressed in cochlear hair cells and the spiral limbus, with labile z
65 diversity of splice variants exist in rodent cochlear hair cells and this diversity is similar to tha
66 ring loss associated with mild damage to the cochlear hair cells and, strikingly, significant degener
67 lear duct, a decrease in the total number of cochlear hair cells, and defects in the formation of the
68 sion has been reported in photoreceptors and cochlear hair cells, and mutations in whirlin cause Ushe
69 ecific for vestibular hair cells, others for cochlear hair cells, and some are expressed just before
70  roles in the actin cytoskeleton dynamics in cochlear hair cells, and thus relate to hearing impairme
71 ner ear phenotype in that the vestibular and cochlear hair cells are differentially affected.
72                           The stereocilia of cochlear hair cells are disorganized and splayed in muta
73                                       Turtle cochlear hair cells are electrically tuned by a voltage-
74                                              Cochlear hair cells are epithelial cells that are not re
75 anoelectrical transduction (MET) channels in cochlear hair cells are gated by tip links through a mec
76                                              Cochlear hair cells are inhibited by cholinergic efferen
77     Notably, mechanotransduction channels of cochlear hair cells are not only critical for auditory p
78                                  In mammals, cochlear hair cells are only produced during development
79                                              Cochlear hair cells are vulnerable to a variety of insul
80 ssion was transient in early postnatal mouse cochlear hair cells but persisted in vestibular hair cel
81  for mechanoelectrical transduction (MET) by cochlear hair cells, but not by vestibular hair cells th
82 e for Barhl1 in the long-term maintenance of cochlear hair cells, but not in the determination or dif
83 ediate the intrinsic frequency tuning of the cochlear hair cell by an electrical resonance mechanism.
84 d the effective delivery of RNPs by EVs into cochlear hair cells by cross-sectional and whole-mount c
85 assium currents were characterized in turtle cochlear hair cells by whole-cell voltage clamp during s
86 inoglycoside antibiotics are known toxins to cochlear hair cells, causing permanent hearing loss.
87                        Here, we show that in cochlear hair cells, CIB3 can functionally substitute fo
88             The sensory bundle of vertebrate cochlear hair cells consists of actin-containing stereoc
89                                              Cochlear hair cells convert sound stimuli into electrica
90                                              Cochlear hair cells convert sound-induced vibration into
91          Our goals were to determine whether cochlear hair cells could survive the trauma and whether
92 splatin-induced ototoxicity is the result of cochlear hair cell damage through a mechanism that is po
93 s Cx26 and Cx30 gene knockout mice show that cochlear hair cells degenerate after the onset of hearin
94 , partial pontine nucleus migration defects, cochlear hair cell degeneration, and profound hearing lo
95  mouse model that allows temporal control of cochlear hair cell deletion.
96                          Recordings from rat cochlear hair cells demonstrate that altering Ca(2+) ent
97 e describe the effects of three mutations on cochlear hair cell development and function.
98 egulating epithelial polarity, ion channels, cochlear hair cell development, vesicular sorting, and n
99 e indicated retinoic acid's critical role in cochlear hair cell development.
100 postnatal maturation of the mouse inner ear, cochlear hair cells display at least two types of mechan
101   However, SGNs degenerate following loss of cochlear hair cells, due at least in part to a reduction
102 associated with these mutations is caused by cochlear hair cell dysfunction, as indicated by conspicu
103 ssociated with the mutation may be caused by cochlear hair cells dysfunction, which manifests with sh
104                                              Cochlear hair cells each possess an exquisite bundle of
105                    After substantial loss of cochlear hair cells, exogenous neurotrophins prevent deg
106                                For mammalian cochlear hair cells, fate determination is normally comp
107                                              Cochlear hair cells form ribbon synapses with terminals
108 ti-functional small molecule drug, protected cochlear hair cells from gentamicin damage.
109 opment in the 1970s of objective measures of cochlear hair cell function (cochlear microphonics, otoa
110 iary localization of TRPML3 and investigated cochlear hair cell function in varitint-waddler (Va(J))
111 m by which the corticofugal system modulates cochlear hair cells has been unexplored.
112                                              Cochlear hair cells (HCs) are mechanosensory receptors t
113       Alpha9 AChR mRNA was first observed in cochlear hair cells (HCs) at embryonic day 18 (E18), inc
114 ness associated with a rapid degeneration of cochlear hair cells (HCs) but have a normal endocochlear
115                             Mature mammalian cochlear hair cells (HCs) do not spontaneously regenerat
116 ns (SGNs) extend their neurites to innervate cochlear hair cells (HCs) with their soma aggregated int
117 nsporters that regulate platinum uptake into cochlear hair cells, if conducted appropriately, may all
118 eurons via the corticofugal system modulates cochlear hair cells in a highly specific way according t
119 tes both the hearing loss and destruction of cochlear hair cells in guinea pigs treated with aminogly
120                                      Loss of cochlear hair cells in mammals is currently believed to
121 transducer (MET) currents were recorded from cochlear hair cells in mice with mutations of transmembr
122                             We recorded from cochlear hair cells in SK2 knockout mice to evaluate fur
123                                              Cochlear hair cells in the av mutants show abnormal ster
124 quired for the maintenance of vestibular and cochlear hair cells in vivo.
125  genetic strategies to ablate neonatal mouse cochlear hair cells in vivo.
126 (2+)-activated K(+) channel of the chicken's cochlear hair cell interacts with beta-catenin.
127                       Efferent inhibition of cochlear hair cells is mediated by 'nicotinic' cholinerg
128 ly believed that mechanical amplification by cochlear hair cells is necessary to enhance the sensitiv
129                           The hair bundle of cochlear hair cells is the site of auditory mechanoelect
130       Furthermore, Gfi1 mutant mice lose all cochlear hair cells just prior to and soon after birth t
131 GPR98 and WHRN, to ankle links in developing cochlear hair cells, likely through its direct interacti
132 rin-1 protein expressed in transfected mouse cochlear hair cells localized to the bundle; however, th
133 toacoustic emissions, and was accompanied by cochlear hair cell loss and ribbon synapse degeneration.
134 ry brainstem responses (ABRs) and determined cochlear hair cell loss in 13-month-old 129/CD-1 mice wi
135                                              Cochlear hair cell loss results in secondary regression
136 fe, CBA/J (CBA), with one that suffers early cochlear hair cell loss, C57BL/6 (BL/6).
137 c gradients underlying sound transduction in cochlear hair cells necessary for hearing.
138                                              Cochlear hair cells normally detect positive deflections
139 lls of the cochlea and vestibule and also to cochlear hair cell nuclei and stereocilia.
140   We identified >600 enriched transcripts in cochlear hair cells, of which 90% have not been previous
141 pendent hearing loss is not due to a loss of cochlear hair cells or spiral ganglion neurons, both of
142 for Notch-mediated lateral inhibition during cochlear hair cell patterning, we also detected abnormal
143                                 In contrast, cochlear hair cell polarity is disorganized.
144                       CaBP2 was expressed by cochlear hair cells, preferentially in inner hair cells
145         The mechanotransduction machinery of cochlear hair cells provides a particularly striking exa
146                       Electron microscopy of cochlear hair cells revealed loss of some tall stereocil
147                          Using RT-PCR of rat cochlear hair cell-specific cDNA libraries, we detect an
148 lls, and HCN1 protein was immunolocalized to cochlear hair cell stereocilia.
149 ids, open neural tube, and misorientation of cochlear hair cell stereociliary bundles, indicative of
150 ortant in sites such as the gamma-actin-rich cochlear hair cell stereocilium where local mm calcium c
151 astest adaptation differs with the method of cochlear hair cell stimulation.
152 -CreER activity was also detected in >50% of cochlear hair cells, suggesting that Sox2-CreER may not
153 fi1, two transcription factors implicated in cochlear hair cell survival, depends upon continued Atoh
154           Due to higher Pou4f3 expression in cochlear hair cells than vestibular hair cells, administ
155 rming subunit of the transduction channel of cochlear hair cells that is mechanically gated by tensio
156                                           In cochlear hair cells, the activation of mechano-electrica
157                                           In cochlear hair cells, the assembly and maintenance of the
158 g has been established, including the heart, cochlear hair cells, the brain and spinal cord, and dens
159 hearing loss have mutations which affect the cochlear hair cells, the mechanosensory cells which allo
160 2, is a critical gene for the development of cochlear hair cells, the receptor cells for hearing, but
161 mal mechanical activation of MET channels in cochlear hair cells thereby affecting the hair bundle se
162 eurons of the medial olivary complex inhibit cochlear hair cells through the activation of alpha9alph
163 n neurons communicate sound information from cochlear hair cells to auditory brainstem neurons throug
164 y of actin-based stereocilia is critical for cochlear hair cells to detect sound.
165 lion neurons (SGNs) relay acoustic code from cochlear hair cells to the brainstem, and their stimulat
166                                              Cochlear hair cells transduce mechanical stimuli into el
167 ding neuronal firing, synaptic transmission, cochlear hair cell tuning, insulin release, and smooth m
168 lia organization in the cochlea, and rescues cochlear hair cells, vestibular function and low-frequen
169 l synapses mediate cholinergic inhibition of cochlear hair cells via alpha9alpha10-containing ionotro
170     Otoferlin, the proposed Ca(2+) sensor in cochlear hair cells, was similarly distributed in both c
171                                              Cochlear hair cells were conditionally and selectively a
172                           We found that when cochlear hair cells were depolarized near the Ca(2+) rev
173 toxicity, changes in lysosomal activities in cochlear hair cells were evaluated during a repeated adm
174 chanoelectrical transducer (MET) currents in cochlear hair cells were made in mice with mutations of
175 hlear function was rapidly abolished and all cochlear hair cells were missing within 24 h after treat
176 g loss accompanied by loss of mechanosensory cochlear hair cells, while the endocochlear potential an
177 rapy for patients with hereditary absence of cochlear hair cells, who have severe or profound deafnes
178 mice resulted in nearly complete ablation of cochlear hair cells, with no evident pathology among sup
179 for specific Usher protein variants in mouse cochlear hair cells, with the apical variants playing a
180  cannot form mechanotransduction channels in cochlear hair cells without TMIE.
181        Given that Myo7a is also expressed in cochlear hair cells, Yy1 shows the appropriate localizat

 
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