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1  the other modulates the excitability of the cochlear nerve.
2 s form ribbon synapses with terminals of the cochlear nerve.
3 ve terminals and delayed degeneration of the cochlear nerve.
4 unrecognized embryologic malformation of the cochlear nerve.
5 ts NM neurons by preventing formation of the cochlear nerve.
6 cues by direct electrical stimulation of the cochlear nerve.
7  potentially safe therapeutic pathway to the cochlear nerve.
8 or the delivery of therapeutic agents to the cochlear nerve.
9 hearing loss by electrically stimulating the cochlear nerve.
10 rally during formation of the vestibular and cochlear nerves.
11                            Lesioning >95% of cochlear nerve afferent synapses, while sparing hair cel
12         Type I neurons make up 90-95% of the cochlear nerve and contact single inner hair cells to pr
13  Nav1.6 and Nav1.2 in the cochlear ganglion, cochlear nerve, and organ of Corti, including the type I
14                 Individuals with compromised cochlear nerves are ineligible for cochlear implants and
15 cits, likely originating at the level of the cochlear nerve, are part of "normal hearing."
16 the basilar papilla and NM is established as cochlear nerve axons arrive in the NM prior to the onset
17                                         When cochlear nerve axons arrive in the NM, they are already
18                                              Cochlear nerve axons first penetrate NM between stages 2
19 nts examined the arrival and organization of cochlear nerve axons in the primary auditory brainstem n
20           DiI and DiD were injected into the cochlear nerve, cochlear ganglion, and basilar papilla (
21                                     Notably, cochlear nerve deficiency (n = 20) detected by IAC-MRI t
22 dibility or cognition may reflect underlying cochlear nerve degeneration (CND).
23 f cochlear afferent synapses and progressive cochlear nerve degeneration in noise-exposed ears with r
24 nts who demonstrate abnormal function of the cochlear nerve despite typical function of sensory cells
25  relationship between stimulus intensity and cochlear nerve discharge rate (the rate-intensity functi
26  density of neurons and denser clustering of cochlear nerve fascicles(7-13).
27 ional model of a single mammalian myelinated cochlear nerve fiber coupled to a stimulator-electrode-t
28 ses spontaneous and sound-evoked activity in cochlear nerve fibers and helps control noise-induced ex
29 ral reduction in spontaneous activity in the cochlear nerve fibers caused by the acoustic injury to t
30 ory epithelium, where functional subtypes of cochlear nerve fibers differ in threshold sensitivity, a
31 opy to count synapses between hair cells and cochlear nerve fibers, and using measurement of auditory
32 pses between hair cells and the terminals of cochlear nerve fibers, as seen in confocal analysis of t
33    D1 and D2 immunolabeling was localized to cochlear nerve fibers, near the first nodes of Ranvier (
34          Traditional approaches to the human cochlear nerve have been impeded by its bony encasement
35           The hypoplastic bony canal for the cochlear nerve in patients with SNHL may be indicative o
36 e length and width of the bony canal for the cochlear nerve in two groups of patients.
37                             We conclude that cochlear nerve input is not necessary for expression of
38 nd post-synaptic elements at the hair cell / cochlear nerve interface.
39                                              Cochlear nerve loss parallels the synaptic loss, after a
40            The high-frequency portion of the cochlear nerve of mice (Mus musculus) generates a robust
41 ment factor staining showed axon loss in the cochlear nerves of Nhe6 KO mice compared to WT mice.
42   SNHL indicates dysfunction of the cochlea, cochlear nerve, or brain.
43                                          But cochlear-nerve-recipient neurons in the brainstem demons
44                           The changes in the cochlear nerve response in exposed chicks may be due to
45 as significantly associated with (1) reduced cochlear nerve responses, (2) weaker middle-ear muscle r
46                     The cell clusters in the cochlear nerve root of the chinchilla provide the simple
47  superficial dorsal cochlear nucleus and the cochlear nerve root on the ablated side.
48  mice demonstrate a significant reduction in cochlear nerve sound-evoked activity compared with wild-
49 o determine an energy-efficient waveform for cochlear nerve stimulation, we used a genetic algorithm
50 hanges, there is a dramatic reduction of the cochlear nerve supply.
51 erented animals, there was up to 40% loss of cochlear nerve synapses and a corresponding decline in t
52 auditory processing compensate for a loss of cochlear nerve synapses by increasing the gain on remain
53 ate excitotoxicity has been characterized in cochlear nerve terminals, but much less is known about w
54 s postnatal day 3 (P3) in the portion of the cochlear nerve that innervates the base of the modiolus.
55 ent likely contributes to the differences in cochlear nerve threshold and SR seen on the two sides of
56 s, used to predict the response of the human cochlear nerve to vowels coded by a cochlear implant.
57 e length and width of the bony canal for the cochlear nerve were significantly smaller in patients wi
58                        Central fibers of the cochlear nerve were stained for neuronal nitric oxide sy