コーパス検索結果 (１語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 rain, providing a basis for the seizures and cognitive disorder.
2 ence of attentional exhaustion in functional cognitive disorder.
3 orbidities prohibiting exercise or diagnosed cognitive disorder.
4 was associated with higher risk of incident cognitive disorder.
5 n mutations in PDZD8 in a neurodevelopmental cognitive disorder.
6 ural networks is a key mechanism of vascular cognitive disorder.
7 uirements of patients with neurodegenerative cognitive disorders.
8 s of BAF complexes in human intellectual and cognitive disorders.
9 roach for the treatment of schizophrenia and cognitive disorders.
10 rain development and pathogenesis of diverse cognitive disorders.
11 s to treat infectious diseases, cancers, and cognitive disorders.
12 f fibrinogen in infection, inflammation, and cognitive disorders.
13 y are especially dosage-sensitive targets of cognitive disorders.
14 tanding of the epigenetic changes underlying cognitive disorders.
15 compounds for the treatment of affective and cognitive disorders.
16 vulnerability or resilience to emotional and cognitive disorders.
17 increases the risk for anxiety, immune, and cognitive disorders.
18 ies can induce mood, anxiety, psychotic, and cognitive disorders.
19 icity have been linked to drug addiction and cognitive disorders.
20 lation may inform therapeutic strategies for cognitive disorders.
21 uting genetic factor in Chr22qter-associated cognitive disorders.
22 mphasis on the development of treatments for cognitive disorders.
23 resents a promising therapeutic strategy for cognitive disorders.
24 ns in SV recycling have been associated with cognitive disorders.
25 e sleepiness (EDS) as well as other sleep or cognitive disorders.
26 ibute to the pathogenesis of SYNGAP1-related cognitive disorders.
27 test the disconnection mechanism directly in cognitive disorders.
28 e possible involvement of Notch signaling in cognitive disorders.
29 ment of schizophrenia, and potentially other cognitive disorders.
30 rbations in Rho GTPase signaling may lead to cognitive disorders.
31 tors such as dopamine have a central role in cognitive disorders.
32 alleviate symptoms associated with numerous cognitive disorders.
33 te for further evaluation as a treatment for cognitive disorders.
34 neration, demyelinating and aging-associated cognitive disorders.
35 ote screening, prevention, and management of cognitive disorders.
36 d mice attenuates AD pathologies and rescues cognitive disorders.
37 cing our understanding of the role of WMH in cognitive disorders.
38 e the identification of subgroups at risk of cognitive disorders.
39 ulmonary disease, nerve agent exposures, and cognitive disorders.
40 (NRGs) are EGF-like ligands associated with cognitive disorders.
41 g B2M in NMDAR dysfunction in DS and related cognitive disorders.
42 f CSF1R inhibitor for MIA-related social and cognitive disorders.
43 ve medicine for patients with MTLE and other cognitive disorders.
44 higher burden of cardiometabolic, mood, and cognitive disorders.
45 dysfunction in a wide range of movement and cognitive disorders.
46 CNA1C were associated with increased risk of cognitive disorders.
47 implicated in multiple neuropsychiatric and cognitive disorders.
48 tal cortex that are especially vulnerable in cognitive disorders.
49 e the complex links between bone biology and cognitive disorders.
50 s a promising candidate for the treatment of cognitive disorders.
51 lucidating neurodevelopmental and behavioral-cognitive disorders.
52 and as a therapeutic intervention in various cognitive disorders.
53 h comorbid rheumatological, neurological, or cognitive disorders.
54 mer's disease, Parkinson's disease and other cognitive disorders.
55 ts clinical application in neurodegenerative cognitive disorders.
56 lopment and their dysfunction leads to human cognitive disorders.
57 e for GRASP1 in the pathophysiology of human cognitive disorders.
58 linical candidate for potential treatment of cognitive disorders.
59 triatum and cortex that results in motor and cognitive disorders.
60 o predicted to contribute to other motor and cognitive disorders.
61 rranean diet score with the incident risk of cognitive disorders.
62 ined by its association with common and rare cognitive disorders.
63 the pathophysiology of a variety of mood and cognitive disorders.
64 ntributes to multiple neurodevelopmental and cognitive disorders.
65 erns of Mediterranean diet score on incident cognitive disorders.
66 neurobiological predisposition to later-life cognitive disorders.
67 tegory was not significantly associated with cognitive disorders.
68 tertiary research clinic that specialized in cognitive disorders.
69 systems and potential avenues for correcting cognitive disorders.
70 neurodevelopment and may underlie postnatal cognitive disorders.
71 uman brain, increasing the risk for mood and cognitive disorders.
72 restore learning and memory in a variety of cognitive disorders.
73 elated complications, diabetes mellitus, and cognitive disorders.
74 t strategies for improving working memory in cognitive disorders.
75 outcomes for the fetus, including postnatal cognitive disorders.
76 nd reduce neuropathology in rodent models of cognitive disorders.
77 tween orchiectomy and GnRHa for diabetes and cognitive disorders.
78 host of neurodegenerative and developmental cognitive disorders.
79 tribute to the pathogenesis of developmental cognitive disorders.
80 ay be of therapeutic benefit in AD and other cognitive disorders.
81 ic plasticity and are implicated in multiple cognitive disorders.
82 a novel therapeutic target for treatment of cognitive disorders.
83 al connectivity measurements, especially for cognitive disorders.
84 loss may hold promise for new treatments for cognitive disorders.
85 g pathology in many symptomatically distinct cognitive disorders.
86 f value in the treatment of mGluR-LTD-linked cognitive disorders.
87 ribute to our understanding and treatment of cognitive disorders.
88 and with it, the prevalence of postoperative cognitive disorders.
89 several human intellectual disabilities and cognitive disorders.
90 ty, sleep apnea, cardiovascular disease, and cognitive disorders, a broad spectrum of collateral oral
91 f these people are likely to have functional cognitive disorders, a subtype of functional neurologica
92 ischarge premature death was associated with cognitive disorders (adjusted hazard ratio [AHR], 2.89 [
94 lexia is one of the most prevalent childhood cognitive disorders, affecting approximately 5% of schoo
96 We also discuss the concept of pain as a cognitive disorder and new strategies for treating chron
98 iscuss the prevalence and characteristics of cognitive disorders and dementia in patients with CKD, b
101 tential as a drug target in the treatment of cognitive disorders and learning impairments is primed f
102 to the National Institute of Neurologic and Cognitive Disorders and Stroke (NINCDS) compared with 14
103 ion might help to identify people at risk of cognitive disorders and to facilitate the development of
104 g of the genetic influences on cognition and cognitive disorders and will direct future therapeutics.
105 identify effective treatments for functional cognitive disorders, and better understanding these phen
107 ence and clinical associations of functional cognitive disorders, and related phenotypes, within the
109 inversely associated with the developing of cognitive disorders, and the pooled RR (95% CI) was 0.79
110 biquitin ligase that is mutated in the human cognitive disorder Angelman syndrome and duplicated in s
111 rates and mechanisms of neuropathic pain and cognitive disorders are increasingly recognized, yet no
115 d promising diagnostic accuracy for vascular cognitive disorder (area under the curve, 0.82 [95% conf
116 a valuable model to study heritable risk for cognitive disorders arising from cholinergic dysfunction
117 B-cell proliferative disorders, depression, cognitive disorders, arthritis and Sjogren's syndrome).
118 ociations with LD including chronic pain and cognitive disorders, as well as particular comorbidities
119 cognitive impairment refers to all forms of cognitive disorder associated with cerebrovascular disea
120 llnesses such as autism, mental retardation, cognitive disorders associated with Alzheimer and Parkin
122 n and circadian disruption underlie mood and cognitive disorders associated with irregular light sche
123 implications for understanding the origin of cognitive disorders associated with Reelin deficiency.
124 sticity in the hippocampus may contribute to cognitive disorders associated with TBI (traumatic brain
125 veral neurodevelopmental disorders including cognitive disorders, autism, juvenile-onset schizophreni
126 imals but cannot be used in the treatment of cognitive disorders because of anxiogenic and convulsant
127 ry importance in the design of therapies for cognitive disorders, because this type of memory is impa
128 We show that Phf6, a gene mutated in the cognitive disorder Borjeson-Forssman-Lehmann syndrome, i
129 ked with schizophrenia, a neurodevelopmental cognitive disorder characterized by imbalances in glutam
130 serves as a new target for the treatment of cognitive disorders characterized by aberrant neuronal m
132 in 111 patients who presented to the Oxford Cognitive Disorders Clinic, Oxford, UK, including 29 amn
134 hway upregulation, microglia activation, and cognitive disorders compared to mice receiving healthy d
136 a history of low thyrotropin (TSH) level or cognitive disorder diagnoses within 6 months of their fi
137 is was significantly associated with risk of cognitive disorder diagnosis (adjusted hazard ratio, 1.3
138 l prefrontal cortex especially vulnerable in cognitive disorders differentially express calbindin and
139 ry early after infection, yet HIV-associated cognitive disorders do not typically manifest until the
140 d worldwide for the symptomatic treatment of cognitive disorders due to its neuroprotective effects.
144 portion of patients referred with functional cognitive disorder (FCD), that is, non-progressive cogni
148 rovide an explanation for lifelong disabling cognitive disorder, guide prognosis, and highlight medic
150 neuropathic pain, and neurodegenerative and cognitive disorders has been exhibited clinically for se
152 connect them to behavioral endophenotypes in cognitive disorders has proven to be an effective approa
153 osure to some pollutants and higher risk for cognitive disorders (higher incidence of dementia with h
154 triggering Alzheimer's disease pathology and cognitive disorder in a viral overexpression mouse model
159 onkeys has already led to new treatments for cognitive disorders in humans, encouraging future resear
164 s in IL1RAPL1-regulated pathways can lead to cognitive disorders in humans.SIGNIFICANCE STATEMENT Abn
168 re of the changes resulting in the motor and cognitive disorders in the chronic stage is unknown.
171 f VCI has evolved to describe a continuum of cognitive disorders in which vascular brain injury plays
174 herapeutic approaches toward psychiatric and cognitive disorders, including cognitive impairment asso
176 ive circuitry; however, the role of HDACs in cognitive disorders, including SCZ, remains unknown in h
177 art cerebrovascular disease plays in several cognitive disorders, including the hereditary vascular d
179 We previously hypothesized that functional cognitive disorder is characterized by heightened subjec
181 e neuronal damage seen in AIDS patients with cognitive disorders is caused indirectly via viral and c
183 volvement of organophosphate insecticides in cognitive disorders is supported by epidemiologic and bi
185 h studies have implicated SYNGAP1 in several cognitive disorders, it is not clear which SynGAP isofor
186 ndent kinase 5 (Cdk5) has been implicated in cognitive disorders, its role in learning has been obscu
188 g disability (RD), or dyslexia, is a complex cognitive disorder manifested by difficulties in learnin
191 ticipants with ICD10 diagnosis of AD or mild cognitive disorder (MCD) in a single-center cross-sectio
192 st current Alzheimer's disease (AD) and mild cognitive disorders (MCI) studies use single data modali
194 pathogenesis of complex diseases, including cognitive disorders, neurodegeneration, cancer, diabetes
195 Our results support the hypothesis that cognitive disorders observed in vine workers may be asso
199 n axon tracts, which could contribute to the cognitive disorders of these patients, has not been expl
200 herapeutic agent for preventing and treating cognitive disorders of various origins, preferably in pa
202 ry of delirium, dementia, amnestic and other cognitive disorders (OR = 1.90, 95% CI: 1.24-2.90).
204 normal brain function to the early stages of cognitive disorders, or how changes in immune signaling
205 range of pathological conditions, including cognitive disorders, Parkinson's disease, and neuropathi
208 clinical observations in the modern era that cognitive disorders present relatively infrequently.
210 ositive selection in lncRNAs associated with cognitive disorder provides a set of candidate genes for
214 erceptual postural dizziness, and functional cognitive disorder-show similarities in aetiology and pa
215 perspective on the association of TCF4 with cognitive disorders.SIGNIFICANCE STATEMENT The importanc
216 definitions used, and the presence of these cognitive disorders significantly impacts the overall we
219 tion cortices are particularly vulnerable in cognitive disorders such as schizophrenia and Alzheimer'
220 mechanisms underlying memory impairments in cognitive disorders, such as Alzheimer's disease (AD), a
222 plasticity in Coffin-Lowry Syndrome (CLS), a cognitive disorder that is caused by mutations in rsk2 a
223 berrant epigenetic modifications may lead to cognitive disorders that affect learning and memory, and
224 dissecting the molecular pathways underlying cognitive disorders that disrupt speech, language, and v
225 en the cortex and hippocampus, especially in cognitive disorders that involve both structures, such a
226 schizophrenia, Parkinson's disease, autism, cognitive disorders) that are linked with abnormalities
228 AR) hypofunction as an underlying factor for cognitive disorders, the precise roles of various NMDAR
229 t of Mediterranean diet in the occurrence of cognitive disorders; therefore, we performed an updated
231 g from cardiometabolic diseases, cancer, and cognitive disorders to respiratory pathologies and condi
234 ination antiretroviral therapy (ART) for HIV cognitive disorders vary substantially between individua
236 rticipants with no history of psychiatric or cognitive disorders were randomized to receive daily ora
237 .1%], classified as having probable vascular cognitive disorder) were older than nonimpaired patients
238 ognitive abilities may make us vulnerable to cognitive disorders when we lose essential regulation.
239 Auts2, a gene linked to human evolution and cognitive disorders, which displays strong clustering of
240 gene expression program are mutated in human cognitive disorders, which suggest that this program is
241 at might indicate the presence of functional cognitive disorders, which were associated with affectiv