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1 each successive generation born after 1951 (cohort effect).
2 enced by year of birth in the United States (cohort effect).
3 fects) or individuals in successive cohorts (cohort effect).
4 the year of birth of either partner (a birth cohort effect).
5 ar-associated differences in susceptibility (cohort effects).
6 ispersion of the residuals, ameliorating any cohort effects).
7 westernization was associated with the birth cohort effect.
8 nic interaction," given that there is no age cohort effect.
9 whether this is an aging or a year-of-birth cohort effect.
10 ore educated, this did not fully explain the cohort effect.
11 growth trajectories after accounting for the cohort effect.
12 ct of normal aging and not primarily a birth cohort effect.
13 o culling of seropositive donors and a birth cohort effect.
14 ession models were used to examine the birth cohort effect.
15 ated using linear mixed models with a random cohort effect.
16 younger participants, suggesting a possible cohort effect.
17 sk factor adjustment for analyzing the birth cohort effect.
18 itudinal changes observed, there was a birth cohort effect.
19 y during 1985 and 1994 was explained by this cohort effect.
20 and declined thereafter, suggesting a birth cohort effect.
21 carefully interpreted in light of the birth cohort effect.
22 and suicide method-specific age, period, and cohort effects.
23 se findings were not discriminable from pure cohort effects.
24 values, especially in scenarios dominated by cohort effects.
25 he cohorts, indicating possible age or birth cohort effects.
26 ous scenarios influenced by age, period, and cohort effects.
27 an age period cohort model to estimate birth cohort effects.
28 n different age groups, representing age and cohort effects.
29 idate genes of aging and longevity and their cohort effects.
30 cohort analysis was used to isolate age and cohort effects.
31 r the WHI report, adjusted for age and birth cohort effects.
32 ider the possible explanations of period and cohort effects.
33 tched and do not include cross-generation or cohort effects.
34 butable to calendar period rather than birth cohort effects.
36 Health Survey results indicated a decreasing cohort effect among those born in 1922-1925 through 1935
37 ge-specific rates were consistent with birth cohort effects among both American Indians and Hispanics
41 riable analyses incorporating random RCB and cohort effects and adjustments for age and pretreatment
42 Our empirical approach separates age from cohort effects and corrects for measurement error from r
44 nds over time requires separation of age and cohort effects, and few prior studies have used this app
45 n, repeat vaccination, birth (immunological) cohort effects, and potential within-season waning of va
47 to pregnancy, gene-environment interactions, cohort effects, and time trends in patients with allergi
50 y (N = 27,572), we find strong evidence that cohort effects are driving the increase in population-le
52 st on the basis of constant age, period, and cohort effects at 2012 values, as is most commonly done
54 ere was substantial heterogeneity in age and cohort effects by method, sex, and race, with a first pe
55 le, control for calendar-period and/or birth cohort effects can be achieved by stratifying the model
56 endar period of diagnosis effects, and birth cohort effects, can help guide resource allocation and d
59 difficult to disentangle period effects from cohort effects, demographers, epidemiologists, actuaries
60 tant signal, in sharp contrast to the fit-to-cohort effect, disappointing findings to date, and limit
62 5, -0.27) and then a continuously increasing cohort effect during the remainder of the 20th century t
66 nce of germ cell cancer is linked to a birth cohort effect; evidence in support of the importance of
69 o), Kramer et al. find evidence that age and cohort effects figure more prominently than do period ef
70 strated that this rise was visible as a male cohort effect for both TTP and contraceptive failure.
72 lier-born cohorts and indicated an increased cohort effect for the earliest born (for 1912-1914, beta
74 rmine and incorporate prior age, period, and cohort effects from 1979 to 2012, stratified by age, sex
76 to estimate the impact that age, period, and cohort effects have had on trends in black-white inequal
77 agnostics and clinical awareness), and birth cohort effects (ie, environmental risk factors) over tim
78 of ALS incidence is attributable to a birth cohort effect in women, with a peak in the 1930 cohort.
79 Study (n ~ 390,000), we estimated age-period-cohort effects in adolescent internalizing symptoms (e.g
80 We sought to disentangle age, period, and cohort effects in chewing ability between 2007 and 2018.
83 his variation can be explained by litter and cohort effects, individual host genotype had a measurabl
87 older female individuals, suggesting a birth cohort effect may have been associated with previously o
88 sible unmeasured risk factors that cause the cohort effect may help us understand the etiology of the
89 ive data on recent trends and how period and cohort effects may affect these trends among young women
90 ression in all contexts, which may be due to cohort effects, modes of transmission, viral clade, or o
93 two alternatives better explains the data: a cohort effect of changing prevalence by decade or a long
102 inding of this study was discovering a birth cohort effect on axial length, especially in persons bor
105 retations of the impacts of age, period, and cohort effects on racial inequalities in heart disease m
106 s assessed the influence of age, period, and cohort effects on rates of preterm delivery in the Unite
107 ck girls and women, by estimating age-period-cohort effects on suicide rates among decedents coded as
110 nor the direction of a linear trend in birth cohort effects or calendar period effects can be determi
111 ly attrition from care was due to a "healthy cohort" effect or to overcrowding as programs expanded t
114 omparisons have been complicated by regional cohort effects, phenotypic differences in sex ratio and
115 carcinoma in recent years is largely a birth-cohort effect presumably associated with greater exposur
116 ed, including viral genomic variation, birth cohort effects, prior vaccination, and epidemic period.
118 ation to infection ratios and explored birth cohort effects referencing the pandemic years (1957; 196
120 owed a substantial increase over time, while cohort effects remained stable throughout the study peri
123 om age-period-cohort models indicate a birth cohort effect starting with the 1912 cohort in American
124 ntribution of sociodemographic status to the cohort effect that may be the antecedent of the current
126 els with the incorporation of random RCB and cohort effects to account for between-study heterogeneit
128 We multiplied the absolute risks by the age cohort effects to provide absolute risks of cervical can
135 Asian countries/regions, virtually no birth cohort effect was identified in the Philippines (a Spani
142 ding recent improvements in chewing ability, cohort effects were somewhat more important than period
144 as suppressing the SES-telomere association; cohort effects with regard different experiences of SES;
145 asing across time for most ages, and (iii) a cohort effect, with a clustering of increased suicide ra