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1  hepatitis C virus + serology, donor age and cold ischemic time.
2 ric transplantation and cases with prolonged cold ischemic time.
3 t on portal venous lactate concentration and cold ischemic time.
4  point of retrieval, and so does not include cold ischemic time.
5 diatric transplants and cases with prolonged cold ischemic times.
6  transplants (1.556, P<0.01), with prolonged cold ischemic time (1.097, P=0.03), for black recipients
7 1 vs. 59, P < 0.001) and reduction in median cold ischemic time (10 vs. 6 h, P = 0.001).
8  index (1.83 vs. 1.32, p < .001), and longer cold ischemic time (16.5 vs. 14.8 h, p = .03).
9 rm ischemia time greater than 30 minutes and cold ischemic time also occurred over the same period.
10 0009), anastomotic time (P=0.0012), combined cold ischemic time and anastomotic time (P=0.00018), and
11 riate analyses accounting for the effects of cold ischemic time and donor age, Treg suppressive funct
12 s associated with first transplants, shorter cold ischemic time and operative time, and less intraope
13                                    Prolonged cold ischemic time and recipient life support were predi
14 e of edema, donor/recipient HLA-DR mismatch, cold ischemic time, and donor age were independently ass
15 fferences in the first warm ischemic period, cold ischemic time, and donor age.
16                              Donor age, sex, cold ischemic time, and NMP time did not correlate with
17         Increased transfusion burden, longer cold ischemic time, and non-White recipients were associ
18           In contrast, donor asystolic time, cold ischemic time, and reperfusion time were independen
19 BC included older age at transplant, shorter cold ischemic time, and single strictures.
20 phics, blood product use, primary diagnosis, cold ischemic time, and surgeon were similar between the
21 r disease, diabetes status, body mass index, cold ischemic time, and UNOS status.
22 aphics, primary diagnosis, surgeon, warm and cold ischemic times, and blood product use were recorded
23 Kidney Donor Profile Index strata, increased cold ischemic times, and shorter distance traveled.
24 quency of poor ex vivo perfusion, had longer cold ischemic times, and were transplanted into older re
25 after circulatory death livers with extended cold ischemic time at 10 degrees C demonstrates superior
26                                    Increased cold ischemic time but neither donor age nor intensity o
27 yte antigen mismatches (chi-square 3.06) and cold ischemic time (chi-square 3.23).
28 l, 149 renal transplants were performed with cold ischemic times (CI) greater than 16 hr (UW 87, HTK
29 median 30% versus 35%, P < 0.001) but longer cold ischemic time (CIT) (median 21.0 h versus 18.6 h, P
30    We also examined the relationship between cold ischemic time (CIT) and likelihood of surgical comp
31                                        Total cold ischemic time (CIT) consisted of the time from retr
32          Multivariate analysis revealed that cold ischemic time (CIT) greater than 8 hours (HR: 2.46;
33                    We stratified on basis of cold ischemic time (CIT) to determine the interaction of
34                                         Mean cold ischemic time (CIT) was 5.4 hours in O and 7.3 hour
35              The degree of HLA matching, the cold ischemic time (CIT), the balance of exchange, and g
36                                         Long cold ischemic time (CIT), with or without delayed graft
37 of pulsatile perfusion (PP) across different cold ischemic times (CIT) within different donor groups
38    The heart transplant was completed first (cold ischemic time [CIT]: 131 minutes), followed by the
39  of diabetes, body mass index, waiting time, cold ischemic time, delayed graft function, and coronary
40 , but not long-term, graft survival, whereas cold ischemic time did not have statistically significan
41 tivariate logistic regression accounting for cold ischemic time, donor age, previous transplant, and
42 nes the association between transit time and cold ischemic time for adult, deceased kidney transplant
43 erence between the two groups with regard to cold ischemic time for organ storage, donor age, recipie
44                                     The mean cold ischemic times for the kidney and the pancreas were
45 egionally and shared nationally, livers with cold ischemic time &gt;12 hours, livers from hepatitis C vi
46 tive antibodies (PRAs) (>20%), and prolonged cold ischemic times (&gt;24 hours) in each group.
47 ant and graft loss, cause of graft loss, and cold ischemic time in the geriatric population.
48 y was to examine whether older donor age and cold ischemic time interact to produce inferior allograf
49 slet isolations (donor age 41-59, BMI 26-38, cold ischemic time &lt; 10 h).
50 y mass index <35, non-status 1 registration, cold ischemic time &lt;8 hours, and either hepatocellular c
51 ause they are typically young and have short cold ischemic times, may be advantageous for HCV-infecte
52 DPI (median 30% vs. 35%, p<0.001) but longer cold ischemic time (median 21.0 hours vs. 18.6, p<0.001)
53  42.0%, P < .001), despite a slightly longer cold ischemic time (median: 14.8 vs 14.1 hours, P < .001
54 schemia/reperfusion injury by prolonging the cold ischemic time of the allograft did not affect the s
55  FEV1 achieved, age and gender of the donor, cold ischemic time of the graft, and matching of CMV ser
56                                              Cold ischemic times of both pancreas and kidneys were lo
57 rshall's solution was associated with longer cold ischemic time, older donors, kidney-only donors, do
58                      We see little effect of cold ischemic time on cell yield, total number of reads
59  cytomegalovirus (CMV) status or blood type, cold ischemic times, or the incidence of outflow obstruc
60 virus infection (4.9%, P = 0.001) and longer cold ischemic time (P = 0.001).
61 entration (p =.029) and inversely with graft cold ischemic time (p =.007).
62 erative transfusion requirements (P=0.0001), cold ischemic time (P<0.0001), use of roux-en-Y biliary
63 e of death (P=0.0053), donor age (P=0.0017), cold ischemic time (P=0.0009), anastomotic time (P=0.001
64 c artery thrombosis (P=0.0018) and prolonged cold ischemic time (P=0.034), were independent risk fact
65  of type (DCD vs DBD), donor age, steatosis, cold ischemic time, peak aspartate transaminase, day 5 b
66 er terminal creatinines and shorter pancreas cold ischemic times than DBD donors.
67                                           If cold ischemic time was > or =24 hr, there was a 2.19-fol
68                                   The median cold ischemic time was 1.6 hours (IQR: 1.0-2.3 hours) wi
69                                     The mean cold ischemic time was 16.5 hr in the two-layer group ve
70                                          The cold ischemic time was 219 minutes and warm ischemic tim
71                                         Mean cold ischemic time was longer in DKT (22.2+/-9.7 hr), bu
72                                              Cold ischemic time was significantly longer in patients
73                                              Cold ischemic times were similar between groups, but mor
74                                     Warm and cold ischemic times were typically <45 min and <90 min,