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1 he neutrophil chemokine, KC was decreased in colitic A(2B)AR(-/-) mice.
2 lammatory response to luminally administered colitic agents in the face of intact systemic immune res
3 ation, 25.4 years) resected 70 polyps (60 in colitic and 10 in noncolitic mucosa).
4                                           In colitic and inflamed diverticular mucosa p27 was express
5                                         Both colitic and noncolitic T5KO exhibited transiently unstab
6 ring both the dextran sodium sulfate-induced colitic and the recovery phase.
7 opria mononuclear cells (LPMC) isolated from colitic animals blocked OVA IFN-gamma/IL-17 responses in
8                                  Adrenals of colitic animals were enlarged and hypervascularized.
9 ases in both IFN-gamma and TNF-alpha mRNA in colitic animals, independent of genotype.
10 consistency when administered to chronically colitic animals.
11 itis and inhibits intestinal inflammation in colitic animals.
12  purified from the mesenteric lymph nodes of colitic BM-->tg epsilon26 mice were adoptively transferr
13                        Serum Ab responses of colitic C3H/HeJBir and noncolitic parental C3H/HeJ mice
14 acterial-antigen-activated CD4+ T cells from colitic C3H/HeJBir but not from control C3H/HeJ mice int
15 omain with integrin alpha9, overexpressed on colitic CD103(-) DCs, suppressed the inflammatory potent
16 -1 blocked recruitment of lymphocytes to the colitic colon, and more importantly, these Abs significa
17 f BM-derived vascular smooth muscle cells in colitic compared with noninflamed regions.
18 amed areas, with striking upregulation under colitic conditions that correlated with increased expres
19 is of colonic inflammatory disease using the colitic cotton-top tamarin, an animal model of human ulc
20                                  Chronically colitic cotton-top tamarins were given either a cross-re
21 el of colitis revealed a correlation between colitic disease severity and tissue ITGB1 expression (R(
22 myeloid-derived IL-33 functionally restrains colitic disease, whereas intestinal epithelial cell-deri
23 ly provide a protective adaptation to murine colitic disease.
24 ophages in recipient mice abrogated the anti-colitic effect of HdBc(7(d)).
25 imal Mlh1 promoter in hypoxic YAMC cells and colitic Gialpha2-/- crypts.
26 uced by several cytokines present within the colitic gut, including IL-22 and IFN-gamma.
27 udies with the DNBS model, revealed the anti-colitic HdBc(7(d)) was within the follicular B cell popu
28                        Treatment of infected colitic IL-10 KO mice with anti-IL-12 p40 resulted in ma
29                              Colonization of colitic IL-10-deficient mice with H. polygyrus suppresse
30 s and SIECs as well as CECs from healthy and colitic IL-2(-/-) mice to present antigen to T cells.
31 sodium sulfate exposure abolished the excess colitic inflammation and reduced colonic IL-5 and eosino
32 of colonic and anal epithelium, that resists colitic injury.
33   The effects of chemically blocking PI3K in colitic interleukin-10(-/-) mice were examined.
34                                              Colitic lesions are much more severe in C3H/HeJBir (C3H)
35 significantly smaller subset of T cells from colitic lesions expresses this ligand (24 +/- 2%).
36 ma and interleukin-10 from H. bilis-infected colitic mdr1a-/- mice.
37  of NF-kappaB p65 in lamina propria cells of colitic mice and in activated macrophages.
38  colonic, and fecal samples from healthy and colitic mice and to uncover differences that would aid i
39       IL-1alpha was detected in the stool of colitic mice before IL-1beta.
40                                              Colitic mice developed hypothalamic astrogliosis that co
41                          Multiple strains of colitic mice had elevated serum anti-flagellin IgG2a res
42 icantly more alpha-V particles accumulate in colitic mice relative to (i) control mice (i.e., selecti
43 on of EGCG to dextran sodium sulfate-induced colitic mice significantly reduced the colonic myelopero
44 els and apoptosis were reduced in IL-10(-/-) colitic mice treated with neutralizing TNF mAb and the i
45  cremaster muscle microvessels of normal and colitic mice using a light/dye injury model.
46 roducing, bacterial-reactive CD4+ T cells in colitic mice was 1 out of 2,000 compared to 1 out of 20,
47                                 Treatment of colitic mice with SM3, but not its mutants, enriched ben
48 s detected in cultured colonic explants from colitic mice, GSDMD deficiency substantially attenuates
49  from the blood, the MLNs, and the spleen of colitic mice.
50  healthy and dextran sodium sulphate-treated colitic mice.
51 njury, and blunted thrombus formation in DSS colitic mice.
52 e monitored in untreated and muTF-Ab-treated colitic mice.
53 ke growth factor 1 expression was reduced in colitic mice.
54 ich synthesizes CA, was induced in livers of colitic mice.
55 A was similarly induced in WT and STAT6(-/-) colitic mice; however, we observed increased mRNA expres
56 serum insulin-like growth factor 1 levels in colitic mice; this preceded improvements in weight gain
57 he gut epithelium were a striking feature of colitic microbiota.
58  T cells were selectively accumulated in the colitic microenvironment and associated colon carcinoma.
59 ine the immune reactivity of a spontaneously colitic mouse strain, C3H/HeJBir, to epithelial, food, a
60 splasia (2), or carcinoma (1) in polyps from colitic mucosa.
61 xenograft formation will be useful to survey colitic patients at risk of developing cancer.
62 ifying potential tumor-initiating cells from colitic patients, suggesting that sphere and/or xenograf
63 n II into wild-type mice mimicked the severe colitic phenotype of RenTgMK mice, and treatment with lo
64  that CD4(+) T cells infiltrate the brain of colitic Rag1 (-/-) mice in proportional levels to coliti
65  we assessed the putative generation of anti-colitic regulatory B cells following H. diminuta infecti
66 tibility to, and clinical manifestations of, colitic spondylarthritis.
67 es of AS associated with IBD (referred to as colitic spondylarthritis; comprising 78 with CD and 119
68 ble microbiotas, with lasting differences in colitic T5KO, while their noncolitic siblings stabilized
69 e, conditioned media from colonic tissues of colitic Th17 cell recipients induced IFN-gamma productio
70 osis of IECs occurred in the lower crypts of colitic tissue from humans and mice.
71        Serine proteinase levels increased in colitic tissue, with major species of 23 kd, 30 kd, and
72 rigins of sporadic colon cancer, the primary colitic tissues did not express any histologic evidence
73 tological and functional features of primary colitic tissues, including the absence of acidic mucus s
74 ated inflammatory cytokine production in the colitic tissues.
75 imary FOXP3(+) T cells in blood, tumors, and colitic tissues.
76  MMP-2 and MMP-9 activation, was elevated in colitic tissues.
77                                              Colitic Tyk2(-/-) mice have less p-STAT3 in colon tissue
78 et genes is reduced in IECs from healthy and colitic Tyk2(-/-) mice.
79 igand (alpha-V) exhibit enhanced adhesion to colitic vasculature.
80   Activated protein C (APC), administered to colitic WT mice immediately prior to photoactivation, al
81 sed p-STAT6(+) IL-13-producing NKT cells, in colitic WT mice.