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1 ation and progression of both murine IGC and colitis-associated colon cancer.
2 n this paper, we consider the progression of colitis-associated colon cancer.
3 mice were highly susceptible to colitis and colitis-associated colon cancer.
4 chronic inflammation and its progression to colitis-associated colon cancer.
5 amily history of colon cancer and ulcerative colitis-associated colon cancer.
6 umor immune responses play critical roles in colitis-associated colon cancer.
8 -based network describing the development of colitis-associated colon cancer (CAC) by integrating the
9 showed that miR-19a can promote colitis and colitis-associated colon cancer (CAC) development using
11 protect against intestinal inflammation and colitis-associated colon cancer (CAC) in animal models.
12 term exposure to chronic inflammation drives colitis-associated colon cancer (CAC) in patients with i
13 velopment in most tissues, but their role in colitis-associated colon cancer (CAC) remains unclear.
14 us carcinogenesis for the pathophysiology of colitis-associated colon cancer (CAC) was unknown and th
20 sh that p38gamma and p38delta are central to colitis-associated colon cancer formation through regula
21 n the immunobiology and pathology of IBD and colitis-associated colon cancer have been done in variou
25 oxymethane/dextran sodium sulphate (AOM/DSS) colitis-associated colon cancer model in wild-type (WT),
30 polyunsaturated fatty acids on inflammation/colitis-associated colon cancer risk is still largely ob