戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 sed by mutations in the gene COL7A1 encoding collagen VII.
2  trimerization of collagen VI, as well as in collagen VII.
3 5 and part of collagen IV, and disassembling collagen VII.
4 ment mediate antiparallel dimer formation of collagen VII.
5  each representing one antiparallel dimer of collagen VII.
6 ysis bullosa caused by genetic deficiency of collagen VII.
7 utoantigen, followed by laminin 332 (21.7%), collagen VII (13%), and BP230 IgG (11.6%).
8 posed gel for treatment of wounds to restore collagen VII abundance in human RDEB skin.
9 velop in RDEB patients lacking expression of collagen VII altogether.
10 ullosa skin, they are directly visible where collagen VII anchoring fibrils are ablated.
11 fts provide basal keratinocytes coexpressing collagen VII and basal stem cell marker keratin 15.
12                                        Bulky collagen VII and endogenous collagen I were collected at
13 emphigoid (BP) autoantigens BP180 and BP230, collagen VII, and laminin 332 were evaluated.
14 vitro at 2 wk of incubation for collagen IV, collagen VII, and laminin 5, and through 5 wk for epider
15 marked reductions in procollagens I and III, collagen VII, and the fibrillin-rich microfibrillar appa
16         Laminin-332 (formerly laminin-5) and collagen VII are basement membrane proteins expressed at
17                                 We show that collagen VII binds TSP1, which could potentially limit T
18 t showed negative staining for laminin 5 and collagen VII, but interrupted linear basal staining for
19 shown that it is required for the loading of collagen VII, but not collagen I, into COPII-coated tran
20 A1 encoding the extracellular matrix protein collagen VII (C7), which is necessary for epidermal-derm
21 ene encoding the anchoring fibril component, collagen VII (C7).
22               Mutations in the gene encoding collagen VII cause the devastating blistering disease re
23 ss the role of the basement membrane protein collagen VII (COL7A1) in wound healing.
24                  These findings suggest that collagen VII could be involved in the process of abnorma
25                              Cells devoid of collagen VII did not form tumors in mice, whereas those
26 rovided direct evidence for the existence of collagen VII dimers, but the dynamic process of dimer fo
27                                              Collagen VII expressed by intradermally injected mesench
28 py and immunohistochemistry for collagen IV, collagen VII, fibronectin, and laminin.
29  in mice, whereas those retaining a specific collagen VII fragment (the amino-terminal noncollagenous
30 tumor progression, which led us to subject a collagen VII hypomorphic mouse model of RDEB to chemical
31 on and therapeutic introduction of exogenous collagen VII in a preclinical model.
32 creased TGF-beta signaling in the absence of collagen VII in RDEB patient skin.
33  basement membrane, and diffuse staining for collagen VII in the superior stroma subjacent to the bas
34                         To study the role of collagen VII in these cancers, we examined Ras-driven tu
35 tracellular cues, or whether laminin-332 and collagen VII interact together in this process remains u
36 sis in transformed cells lacking laminin-332/collagen VII interaction in a manner independent of cell
37 ion in our studies suggests that laminin-332/collagen VII interaction promotes epidermal carcinogenes
38                                              Collagen VII is an essential anchoring protein in the ba
39                    The collagenous domain of collagen VII is encoded by 82 in-frame exons, which make
40                                     However, collagen VII is largely unexplored in normal lungs and i
41 ed NC1 expression restored tumorigenicity to collagen VII-null epidermis in a non-cell-autonomous fas
42 n had significant effect on the abundance of collagen VII or its mRNA.
43                         Expression of either collagen VII or the noncollagenous (NC1) fragment derive
44 at we define as an intermediate half-life of collagen VII, our study challenges the view of the derma
45 enic properties, but whether laminin-332 and collagen VII promote SCC tumors by providing adhesion or
46                                  Analysis of collagen VII protein and mRNA encoded by COL7A1 gene in
47 enodermatosis characterized by dysfunctional collagen VII protein resulting in epithelial blistering
48 imary fibroblasts, whereas overexpression of collagen VII reduced phosphorylation of smad3.
49        Tumor-stroma interactions mediated by collagen VII thus promote neoplasia, and retention of NC
50                            In the IPF lungs, collagen VII was abundant in pathologically remodeled ai
51           In contrast, in the control lungs, collagen VII was mainly localized in larger airways.
52               A similar in vivo stability of collagen VII was observed in the skin, tongue, and esoph
53 on of laminin, collagen IV, fibronectin, and collagen VII was performed.
54 t on the stromal surface in all samples, and collagen VII was present in four of five samples.
55 binding of all analyzed mutants to wild type collagen VII were different from those for binding betwe
56                    Laminin, collagen IV, and collagen VII were usually either not present or were pre
57 C carcinogenesis/invasion through binding to collagen VII, which in turn, led to phosphoinositol-3-ki
58  with interrupted triple helices, as well as collagens VII, XIII, XXIII, and XXV, were found to conta