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1 nes that are mutated for the alpha1 chain of collagen type IV.
2 to collagen type VI and, to a lesser extent, collagen type IV.
3 rmation, and the adhesion of A. baumannii to collagen type IV.
4 t injury for 24 hours induced an increase in collagen type IV.
5 y be required for increased cell adhesion to collagen type IV.
6 egulating adhesion of breast cancer cells to collagen type IV.
7 egulatory elements within matrix-immobilized collagen type IV.
8 d arachidonic acid-mediated cell adhesion to collagen type IV.
9 in and collagen type I (P < 0.01) but not on collagen type IV.
10 otease specificity is under the influence of collagen type IV (1).
11 of SMA (9-fold), fibronectin (2.5-fold), and collagen type IV (2-fold).
12 aging to investigate the thermal response of collagen type IV, a key component of basement membranes.
13             Surviving podocytes up-regulated collagen type IV a5, causing reactive changes in integri
14 with diminished MMP-2 activity and increased collagen type IV accumulation.
15 /K+-ATPase, beta3-integrin, fibronectin, and collagen type IV achieved their mature localization patt
16 tronectin, RGD peptide, collagen type I, and collagen type IV) adsorbed to tissue culture plastic was
17 fluence of BL proteins-laminin, fibronectin, collagen type IV, agrin, and perlecan-on adhesion and TE
18                    Heterotrimers composed of collagen type IV alpha 1 (COL4A1) and alpha 2 (COL4A2) c
19                                              Collagen type IV alpha 1 and alpha 2 chains form heterot
20 ctor 10, retinoblastoma susceptibility gene, collagen type IV alpha 1), and one additional marker was
21 f Alport syndrome gene COL4A3, which encodes collagen type IV alpha 3, and we present immunohistochem
22                                 Mutations in Collagen type IV alpha-1 (COL4A1) and Beta-1,3-galactosy
23                                              Collagen, type IV, alpha 1 (COL4A1) and alpha 2 (COL4A2)
24 inactivation of fumarate hydratase (FH), and collagen, type IV, alpha 5 and collagen, type IV, alpha
25 ase (FH), and collagen, type IV, alpha 5 and collagen, type IV, alpha 6 (COL4A5-COL4A6) deletions.
26                                              Collagen type IV alpha1 (COL4A1) and alpha2 (COL4A2) for
27          Tyrosine kinase receptor 1 (Tie-1), collagen type IV alpha1 (Col4a1), complement component 1
28 itope pCol(28-40) of noncollagen domain 1 of collagen type IV alpha3 chain not only uniformly induced
29  antibodies against the matrix constituents, collagen type IV and fibronectin (FN).
30  of extracellular matrix components, such as collagen type IV and fibronectin, and suggests an import
31 erstitial accumulation of collagen type III, collagen type IV and fibronectin.
32 ular matrix, particularly the degradation of collagen type IV and increased elastin expression, were
33 ees C-grown E. faecalis OG1RF to immobilized collagen type IV and laminin as well as collagen type I,
34  conditional binding of E. faecalis OG1RF to collagen type IV and laminin in addition to collagen typ
35               The decreased interaction with collagen type IV and laminin was consistent with a reduc
36 ng to these changes, including expression of collagen type IV and laminin, transforming growth factor
37 dothelial cell monolayers as well as through collagen type IV and laminin-coated BD BIOCOAT inserts,
38 32 would be crucial for its interaction with collagen type IV and plasma proteins fibronectin and pla
39 n of whole A. baumannii cells to immobilized collagen type IV and played a role in the survival of A.
40 ltration rate, mesangial cell expansion, and collagen type IV and transforming growth factor-beta exp
41 iance in the level of serum antibody binding collagen types IV and V.
42 ons to varying extents and is most common in collagen types IV and V.
43 y borne gene cluster and mediates binding to collagen types IV and V.
44 U87MG, U251, and LN229 induces expression of collagen types IV and VI and the collagen crosslinking e
45 ions, included distinct structural proteins (collagen types IV and VI) and cellular components (actin
46 iating the adhesion of A. baumannii cells to collagen type IV, and contributing to the survival of A.
47 B proteins, including laminin, entactin, and collagen type IV, are elevated in the cerebrospinal flui
48 eratin, the junctional complex protein ZO-1, collagen type IV, as well as UB and collecting duct mark
49                   The ECM targeting peptide, collagen type IV-binding peptide (C4BP), was chosen from
50 ct that PDLFs adhere well to fibronectin and collagen type IV bound to plastic, and express integrins
51 B-435 cells to the basement membrane protein collagen type IV can be activated by treatment with arac
52                     Six genetically distinct collagen type IV chains have been identified and are dis
53                                          The collagen type IV cleavage fragment tumstatin and its act
54 hanced adhesion, and reduced detachment from collagen type IV-coated plates but also, with decreased
55 ltured beta-cells adhered to and migrated on collagen type IV (Col-IV), and these responses were medi
56 es to the self-antigens fibronectin (FN) and collagen type-IV (Col-IV).
57 d tumor epithelial cells, but also to type I collagen, type IV collagen, and laminin.
58 eaved gelatin but was inactive toward type I collagen, type IV collagen, fibronectin, and laminin.
59 NA amounts in cells grown in the presence of collagen type IV compared to the controls.
60 lasma membrane polarity; and fibronectin and collagen type IV, constituents of Descemet's membrane.
61  vs SOC alone, -24% vs SOC, P < .05) and the collagen type IV degradation product C4M (-26% vs -11%,
62                                              Collagen type IV degradation results in disruption and b
63 P resulting in collagenase IV activation and collagen type IV degradation.
64                                     Although collagen type IV demonstrated no effect on the associati
65  (highly exposed in the liver sinusoids) and collagen type IV-dependent activation of focal adhesion
66 f human metastatic breast carcinoma cells to collagen type IV depends on the protein kinase C (PKC) p
67 iated with a moderately increased glomerular collagen type IV deposition compared with NS/Pla mice.
68 th tissues is accompanied by a discontinuous collagen type IV expression pattern and decreased lamini
69 eins, including fibrinogen, collagen type I, collagen type IV, fibronectin, and laminin.
70 scle cell adhesion to vitronectin but not to collagen type IV, fibronectin, or laminin, whereas selec
71 on hensin or laminin, but not fibronectin or collagen type IV, formed hemispheric epithelial structur
72 nalysis has determined that viking encodes a collagen type IV gene, alpha2(IV).
73          Integrin alpha2-mediated binding to collagen type IV (highly exposed in the liver sinusoids)
74                     The delicate lacework of collagen type IV immunoreactivity was replaced by bundle
75 lular TIGR immunoreactivity colocalized with collagen type IV immunoreactivity.
76 h as B16F10 melanoma interact with denatured collagen type IV in part by recognizing the HUIV26 crypt
77 cular defects, we examined the deposition of collagen type IV in the basement membrane, and found it
78 sed gelatinolytic activity and low levels of collagen type IV in the basement membranes of TSP-2-null
79 hXA85 was increased to 200 nM, the amount of collagen type IV in the cell layer extract increased by
80 , alpha3(IV), and alpha6(IV) chains of human collagen type IV in the regulation of angiogenesis and t
81 sence of otherwise active substrates such as collagen type IV, indicative of active inhibition.
82 ain of the alpha3 chain of basement membrane collagen (type IV) inhibits the activation of polymorpho
83                                              Collagen type IV is a major component of the basal lamin
84 lation of PN1 target protease specificity by collagen type IV is a result of the purification protoco
85                                              Collagen type IV is the major structural component of th
86 ared with fibronectin (FN), laminin (LN), or collagen type IV (IV).
87 on of Roscovitine reduced immunostaining for collagen type IV, laminin, and fibronectin at days 5 and
88  JCT were confirmed to be made up chiefly of collagen type IV, laminin, and fibronectin.
89 and on several distinct substrates including collagen type IV, laminin, fibronectin, and plastic.
90 indings suggest that specific NC1 domains of collagen type IV may represent an important new class of
91  cell interactions with structurally altered collagen type IV may suppress the expression of insulin-
92 on for a more complete genetic dissection of collagen type IV molecules and their developmental funct
93 eases in protein levels but had no effect on collagen type IV mRNA or protein levels in vitro.
94 -smooth muscle actin (SMA), fibronectin, and collagen type IV mRNA or protein levels.
95 ted the increased expression of TGF-beta and collagen type IV mRNAs and proteins.
96 ase, including Alport Syndrome, is linked to collagen type IV mutations, lipid dysmetabolism, and alt
97 clinical study investigates a novel targeted collagen type IV nanoparticle formulation, Ac2-26 coated
98  the common laminin contaminants entactin or collagen type IV, neither of these proteins are likely t
99 fibronectin, nidogens, laminin isoforms, and collagen type IV, occurs in EC-pericyte cocultures, but
100  quiescent A1N4 cells to fibronectin, and to collagen types IV or I, induces the expression of c-Myc
101 ytosis of beads coated with collagen type I, collagen type IV, or thrombospondin or uncoated controls
102                                  Laminin and collagen type IV, other basal lamina proteins commonly f
103 ciency allows an increase in fibronectin and collagen type IV receptor activities.
104              Integrin alpha1beta1, the major collagen type IV receptor, is expressed by endothelial c
105 abolism of the most abundant protein in pBM, collagen Type IV, requires prolidase, an exopeptidase cl
106 nce is provided that proteolytic cleavage of collagen type IV results in the exposure of a functional
107 at hBVR is a regulator of the TNF-alpha-GPBP-collagen type IV signaling cascade and uncover a novel b
108                                 The TGFalpha/collagen type IV signaling system that induces LAMP expr
109 n an in vitro assay (laminin > fibronectin > collagen type IV), suggesting that the parasitic cell su
110 I were deposited in a diffuse manner whereas collagen type IV surrounded the clusters of the epitheli
111 overexpression of CCN3 increased adhesion to collagen type IV, the major component of the basement me
112 dental basement membrane (BM) is composed of collagen types IV, VI, VII, and XVII, fibronectin, and l
113 ction of Ace after growth in the presence of collagen type IV was demonstrated by immunofluorescence
114             We show here that Ata binding to collagen type IV was inhibited by antibodies to Ata.
115  arachidonic acid; however, cell adhesion to collagen type IV was not highly sensitive to PD98059, an
116 that mediate the allosteric interaction with collagen type IV, we found that protease specificity was
117 ms of CS meshwork, fibronectin, laminin, and collagen type IV were associated with basement membranes
118 lecular phenotype if grown with TGFalpha and collagen type IV, while other signals fail to induce LAM
119       We also showed in vitro interaction of collagen type IV with Ace from OG1RF mutanolysin extract

 
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