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1 1.6 mg/kg and dose-dependent efficacy in rat collagen-induced arthritis.
2 on of arthritis in the experimental model of collagen-induced arthritis.
3 mechanical hyperalgesia and inflammation in collagen-induced arthritis.
4 h atRA suppressed progression of established collagen-induced arthritis.
5 odels of arthritis: Ab-induced arthritis and collagen-induced arthritis.
6 ining of joint tissue derived from mice with collagen-induced arthritis.
7 ts in a mouse model of autoimmune arthritis, collagen-induced arthritis.
8 llular and humoral immunity and the onset of collagen-induced arthritis.
9 re resistant to LPS-induced septic shock and collagen-induced arthritis.
10 and caspase-1(-/-) mice were susceptible to collagen-induced arthritis.
11 c T cells at the site of inflammation during collagen-induced arthritis.
12 ental autoimmune encephalomyelitis (EAE) and collagen-induced arthritis.
13 c (Tg) mice expressing HLA-DRB1*0401 develop collagen-induced arthritis.
14 ed animals in a systemic model of RA disease collagen-induced arthritis.
15 as attenuated disease in MRL/lpr mice and in collagen-induced arthritis.
16 Salmonella-CFA/I can protect DBA/1 mice from collagen-induced arthritis.
17 d its impact on the clinical model of bovine collagen-induced arthritis.
18 a T cells have also been found to exacerbate collagen-induced arthritis.
19 ptor family has a role in the progression of collagen-induced arthritis.
20 reduced the clinical and histologic signs of collagen-induced arthritis.
21 mmune mice, including T cells from mice with collagen-induced arthritis.
22 for B cells during the elicitation phase of collagen-induced arthritis.
23 use anti-mouse CD20 mAbs in a mouse model of collagen-induced arthritis.
24 ively correlated with paw swelling in murine collagen-induced arthritis.
25 optosis in thymocytes and pre-disposition to collagen-induced arthritis.
26 reduce the incidence and severity of murine collagen-induced arthritis.
27 efficacy in some animal models, most notably collagen-induced arthritis.
28 tic effect of sST2-Fc in the murine model of collagen-induced arthritis.
29 ansgenic mice that display susceptibility to collagen-induced arthritis.
30 of arthritis and in its ability to suppress collagen-induced arthritis.
31 therapeutic evaluation in a murine model of collagen-induced arthritis.
32 rotective, whereas loss of IL-12 exacerbates collagen-induced arthritis.
33 ice susceptible to porcine and human type II collagen-induced arthritis.
34 xperimental autoimmune encephalomyelitis and collagen-induced arthritis.
35 ad the capacity to prevent and/or ameliorate collagen-induced arthritis.
36 tage and IgG2a Ab's at the effector phase of collagen-induced arthritis.
37 pared following induction of monoclonal anti-collagen-induced arthritis.
38 of experimental models of arthritis, such as collagen-induced arthritis.
39 are normally susceptible to the induction of collagen-induced arthritis.
40 d particles inhibited development of type II collagen-induced arthritis.
41 combined anti-TNF-alpha/anti-CD4 therapy in collagen-induced arthritis.
42 nase 9 on the development and progression of collagen-induced arthritis.
43 o strongly alleviated disease development in collagen-induced arthritis.
44 exposure-dependent fashion in a rat model of collagen-induced arthritis.
45 immunocytokine F8-IL4 in the mouse model of collagen-induced arthritis.
46 ria significantly aggravated the severity of collagen-induced arthritis.
47 8-IL4 was able to cure mice with established collagen-induced arthritis.
48 hase that contributes to the pathogenesis of collagen-induced arthritis, a disease model of human rhe
50 (EGFR), reduces the severity of established collagen-induced arthritis, a mouse model of RA, and tha
51 affords protection against joint disease in collagen-induced arthritis, a mouse model of rheumatoid
52 e the contribution of NK cells to regulating collagen-induced arthritis, a well-characterized preclin
53 60(p216) but not Qa-1-Qdm strongly inhibited collagen-induced arthritis, an animal model of human rhe
54 ed asthma-like allergic lung inflammation, a collagen-induced arthritis, an induced ulcerative coliti
55 close analog, SNX-4414, was evaluated in rat collagen-induced arthritis and adjuvant-induced arthriti
57 s of T15-NAb also efficiently inhibited both collagen-induced arthritis and anti-collagen II Ab-media
58 gen-specific CD4(+) T cell expansion in both collagen-induced arthritis and autoimmune diabetes mouse
59 BA/1 background were completely resistant to collagen-induced arthritis and exhibited absence of join
60 rity of the inflammatory autoimmune diseases collagen-induced arthritis and experimental autoimmune e
61 imal models of inflammatory diseases such as collagen-induced arthritis and experimental autoimmune e
63 in experimental allergic encephalomyelitis, collagen-induced arthritis and inflammatory bowel diseas
64 ved dendritic cells and in vivo in models of collagen-induced arthritis and inflammatory bowel diseas
67 ese data indicate that ICOS is essential for collagen-induced arthritis and may suggest novel means f
70 ed up-regulation, mice developed more severe collagen-induced arthritis and spontaneous glomerular im
71 of disease severity and early onset in both collagen-induced arthritis and spontaneous lupus nephrit
72 IL-deficient mice are also hypersensitive to collagen-induced arthritis and streptozotocin-induced di
73 itis and its neutralization inhibited murine collagen-induced arthritis and suppressed IFN-gamma and
74 n that is up-regulated in the early stage of collagen-induced arthritis and that exacerbates arthriti
75 exhibited significantly reduced severity of collagen-induced arthritis and this was accompanied by a
76 7A in a mouse model of rheumatoid arthritis (collagen-induced arthritis) and the concomitant suscepti
78 essed LPS-induced TNF production, alleviated collagen-induced arthritis, and blocked gout formation i
80 odent models of polyarthritis: rat and mouse collagen-induced arthritis, and mouse collagen antibody-
81 sitivity model, suppress the onset on murine collagen-induced arthritis, and reduce the severity of e
82 mmune disease: systemic lupus erythematosus, collagen-induced arthritis, and serum-transfer arthritis
83 ted intravenously into mice with established collagen-induced arthritis, and the effects on leukocyte
84 essfully but had increased susceptibility to collagen-induced arthritis, and the levels of huTLR8 cor
85 osomes was able suppress the onset of murine collagen-induced arthritis as well as reduce severity of
86 iency of mPGES-1 inhibits the development of collagen-induced arthritis, at least in part, by blockin
87 their ability to prevent the development of collagen induced arthritis, both the engineered DR1-CII-
88 luorescence in arthritic joints of mice with collagen-induced arthritis by both noninvasive fluoresce
89 I vaccination of DBA/1 mice protects against collagen-induced arthritis by stimulating TGF-beta- and
90 of EC144 blocked disease development in rat collagen-induced arthritis by suppressing the inflammato
92 s study was to investigate the regulation of collagen-induced arthritis by tryptophan catabolism medi
93 iator for PGRN-mediated anti-inflammation in collagen-induced arthritis by using PGRN and IL-10 genet
94 models of autoimmune disease; in particular, collagen induced arthritis (CIA) and experimental autoim
99 y modified DC to suppress established murine collagen-induced arthritis (CIA) after i.v. delivery.
100 nd growth of bony spurs was performed in rat collagen-induced arthritis (CIA) and adjuvant-induced ar
101 level was dramatically elevated during mouse collagen-induced arthritis (CIA) and blocking TWEAK by a
102 complementary experimental RA mouse models, collagen-induced arthritis (CIA) and collagen antibody-i
103 letely halts and reverses the development of collagen-induced arthritis (CIA) and discover cellular a
104 SSRIs, fluoxetine and citalopram, in murine collagen-induced arthritis (CIA) and in a human ex vivo
105 n vivo mechanism of action was determined in collagen-induced arthritis (CIA) and local joint TLR5 li
107 3 receptor alpha-chain, developed attenuated collagen-induced arthritis (CIA) and reduced ex vivo col
108 the development of pain in the rat model of collagen-induced arthritis (CIA) and to evaluate the con
109 her mucosal administration of EtxB can block collagen-induced arthritis (CIA) and to investigate the
110 perimental autoimmune encephalomyelitis, and collagen-induced arthritis (CIA) are associated with typ
111 actor Ag I (CFA/I) fimbriae protects against collagen-induced arthritis (CIA) by eliciting two regula
112 keyhole limpet hemocyanin (KLH) study and a collagen-induced arthritis (CIA) disease model of rheuma
114 dy to test the hypothesis that resistance to collagen-induced arthritis (CIA) in C57BL/6 (B6) mice is
115 he objective of these studies was to examine collagen-induced arthritis (CIA) in C57BL/6 mice transge
116 stress protein, BiP, prevented induction of collagen-induced arthritis (CIA) in HLA-DRB*0101+/+ (HLA
118 he IL-15R on the prevention and treatment of collagen-induced arthritis (CIA) in mice and probed the
119 of a soluble ligand of CD200R in established collagen-induced arthritis (CIA) in mice and to analyze
121 A major difference between RA in humans and collagen-induced arthritis (CIA) in mice is the lack of
123 te the significance of these autoantibodies, collagen-induced arthritis (CIA) in mice was used to est
128 ll depletion was further studied in a murine collagen-induced arthritis (CIA) model and a respiratory
131 s (PADs), are known to develop in the murine collagen-induced arthritis (CIA) model of inflammatory a
146 r (uPA) reduces arthritis progression in the collagen-induced arthritis (CIA) mouse model to an exten
149 of wild type (WT) versus CRIg(-/-) mice with collagen-induced arthritis (CIA) or K/BxN serum transfer
151 nvadosomal structures by synovial cells from collagen-induced arthritis (CIA) rats and RA patients.
154 of the MEK/ERK MAP kinase pathway in murine collagen-induced arthritis (CIA) using the selective MEK
157 t and to identify their mechanism of action, collagen-induced arthritis (CIA) was therapeutically tre
160 h carrageenan-induced acute inflammation and collagen-induced arthritis (CIA) were used to assess eff
161 the ability of these cells to control murine collagen-induced arthritis (CIA), a model for rheumatoid
162 e such product, ES-62, is protective against collagen-induced arthritis (CIA), a model of rheumatoid
164 lator of inflammation was examined in murine collagen-induced arthritis (CIA), a model of rheumatoid
165 inflammation, contribute to the progress of collagen-induced arthritis (CIA), a mouse model for RA.
166 toring and quantifying joint inflammation in collagen-induced arthritis (CIA), a mouse model for RA.
167 ene knockout (KO) mice to assess its role in collagen-induced arthritis (CIA), a mouse model of human
168 arthritis shows any similarities with RA or collagen-induced arthritis (CIA), a mouse model of RA.
169 mmunoregulatory protein for the treatment of collagen-induced arthritis (CIA), a mouse model of rheum
170 ets, Vgamma1(+) and Vgamma4(+) cells, during collagen-induced arthritis (CIA), a mouse model that sha
171 eptibility of CD73-deficient C57BL/6 mice to collagen-induced arthritis (CIA), a well-established mou
172 table for use in DBA/1LacJ mice that develop collagen-induced arthritis (CIA), an animal model of hum
173 and evaluated their therapeutic potential in collagen-induced arthritis (CIA), an animal model of RA.
174 elayed the onset and reduced the severity of collagen-induced arthritis (CIA), and reduced paw tissue
175 immune responses, led to early resolution of collagen-induced arthritis (CIA), and reduced spontaneou
179 We assessed pain behavior and mechanisms in collagen-induced arthritis (CIA), the model of preclinic
181 cient in IL-1Ra (IL-1Ra(-/-)) were bred with collagen-induced arthritis (CIA)-susceptible DBA/1 mice
207 response gene with CII elicits an arthritis (collagen-induced arthritis [CIA]) that resembles rheumat
208 nificantly reduced incidence and severity of collagen-induced arthritis compared with wild-type (WT)
210 more effective than monotherapy in improving collagen-induced arthritis disease even when administere
212 nic mice developed more severe and sustained collagen-induced arthritis due to the enhanced Th1 respo
214 3, ASC, and caspase-1 in the pathogenesis of collagen-induced arthritis have not been characterized.
215 arthritis, IFN-gamma is required, whereas in collagen-induced arthritis, IL-17 is necessary for devel
216 , HNK stabilized the severity of symptomatic collagen-induced arthritis in both CD40-LMP1 transgenic
217 elayed the onset and reduced the severity of collagen-induced arthritis in DBA/1 mice by induction of
218 n DBA/1-TCR-beta transgenic mice, as well as collagen-induced arthritis in DBA/1 mice, both animal mo
220 e autoimmune inflammatory process of chronic collagen-induced arthritis in DBA/1-TCR-beta transgenic
221 We showed that this peptide ameliorates collagen-induced arthritis in DBA/1J mice and protects a
223 protein LTbetaR-Ig blocked the induction of collagen-induced arthritis in mice and adjuvant arthriti
224 nistration of ERG240 reduces the severity of collagen-induced arthritis in mice and crescentic glomer
225 eptide significantly reduced the severity of collagen-induced arthritis in mice by reducing levels of
226 25 in vivo down-regulates the progression of collagen-induced arthritis in mice via targeting of the
227 8 kinase activities and clinical symptoms of collagen-induced arthritis in mice were all diminished.
229 itis in the models of adjuvant arthritis and collagen-induced arthritis in rats and mice, respectivel
232 We studied serotonin's involvement during collagen-induced arthritis in wild-type and Tph1(-/-) mi
233 ne models of chronic inflammation, including collagen-induced arthritis, inflammatory bowel disease,
237 4) enhanced Th17 expansion, and in mice with collagen-induced arthritis it facilitated disease onset,
238 ecule on T lymphocytes, CD40(+) T cells from collagen-induced arthritis mice were examined in paralle
239 so potent in treating established disease in collagen-induced arthritis mice with beneficial effects
240 eutic action of tolerogenic DCs in a type II collagen-induced arthritis model and to investigate thei
242 ing of GLPG0634 in a therapeutic set-up in a collagen-induced arthritis model in rodents resulted in
243 n vivo, because inhibition of C5orf30 in the collagen-induced arthritis model markedly accentuated jo
244 odel of systemic lupus erythematosus and the collagen-induced arthritis model of rheumatoid arthritis
246 cells to the inflammatory site, we used the collagen-induced arthritis model to determine the freque
248 fficacy in adjuvant-induced arthritis model, collagen-induced arthritis model, and allergic asthma mo
252 xin-1 increased skewing in Th1 cells; in the collagen-induced arthritis model, treatment of mice with
260 Here, we examine the therapeutic effects in collagen-induced arthritis of the second generation PAD
262 e, IL-35 inhibited clinical manifestation of collagen-induced arthritis or could cease further diseas
263 ntigens emulsified in adjuvant oils, such as collagen-induced arthritis or experimental autoimmune en
266 kine analysis shows that mice protected from collagen-induced arthritis produce lower amounts of Th1
267 n up to 1 month before the clinical onset of collagen-induced arthritis protected mice from severe jo
270 s biomarkers in RA, enhance tissue damage in collagen-induced arthritis (see the related article begi
271 Crucially, ES-62 was also found to suppress collagen-induced arthritis severity and progression when
272 tained anti-inflammatory activity in vivo in collagen induced arthritis studies in mouse but had redu
276 This motif is similar to one recognized by collagen-induced arthritis-susceptible HLA-DR1- and HLA-
279 hma-like allergic lung inflammation and of a collagen-induced arthritis, the CpdX antiinflammatory ac
281 es are a prerequisite for the development of collagen-induced arthritis, their presence is insufficie
282 ivo studies were conducted in a rat model of collagen-induced arthritis to evaluate the therapeutic p
289 termine the role CD28 costimulation plays in collagen-induced arthritis, we have generated DQ8 transg
291 ation, late phase cutaneous anaphylaxis, and collagen-induced arthritis were aggravated, in contrast
297 er inflammation of the knee joints following collagen-induced arthritis, when compared with control m
298 *0401 gene contributes to the development of collagen-induced arthritis, whereas DRB1*0402 prevents t
299 ays a dominant role in the susceptibility to collagen-induced arthritis, whereas FcgammaRIIb on B cel
300 ent development of inflammation in mice with collagen-induced arthritis, whereas sorted CD25+CD39+CD4