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1  n = 4, 2 with ulcerative colitis and 2 with colonic cancer).
2 forms of gastrointestinal disease (primarily colonic cancers).
3       All patients had no disease other than colonic cancer.
4 l residues, an antigen associated with human colonic cancer.
5 red in 48 patients who underwent surgery for colonic cancer.
6 clooxygenase (COX)-2 is up-regulated in most colonic cancers and in inflammatory bowel disease in whi
7 a, and its expression is also upregulated in colonic cancers and most neoplasms, we investigated whet
8 te the importance of CME and CVL surgery for colonic cancer by comparison with a series of standard s
9 ing a panel of human pancreatic, breast, and colonic cancer cell lines, and in the GW-39 human coloni
10 ncreatic (PANC-1 HPAF, and MiaPaCa2) but not colonic cancer cells (Colo 320) significantly increased
11 which butyrate mediates growth inhibition of colonic cancer cells and thereby to elucidate the molecu
12 N-15 Fab' of mice implanted with GW-39 human colonic cancer cells increased their survival (P < 0.025
13 ffector of butyrate-induced growth arrest in colonic cancer cells, and may be an important molecular
14                                     In human colonic cancer cells, Sb prevented EGF-induced prolifera
15 oth mucin 2 (MUC2) and MUC5AC from HT29-18N2 colonic cancer cells.
16 kdown of 7343 human gene products in a human colonic cancer goblet cell line (HT29-18N2) revealed new
17                      The plane of surgery in colonic cancer has been linked to patient outcome althou
18                EC is the most frequent extra-colonic cancer in patients with hereditary non-polyposis
19  the amount of blood loss during surgery for colonic cancer influences long-term survival.
20                                        Human colonic cancer is associated with multiple genetic delet
21 ion in sporadic human and chemically induced colonic cancers may confer a relative growth advantage d
22  overexpressed in approximately 70% of human colonic cancers, previous studies have not detected freq
23 activation of PPARdelta induces expansion of colonic cancer stem cells (CSC) and promotes colorectal
24  countries and confirm the wide variation in colonic cancer surgery and the need for further standard
25 here is no solid data as to whether AL after colonic cancer surgery increases the risk of disease rec
26  are commonly seen in inherited and sporadic colonic cancers that exhibit microsatellite instability.
27 In the Apc(Pirc/+) (Pirc) rat model of early colonic cancer, this sex susceptibility was recapitulate
28 ies within a traditional stage III cohort of colonic cancer were identified.
29 y, we have demonstrated that growth of human colonic cancer xenografts is inhibited by treatment with