ライフサイエンスコーパス: colony formation

1 h P50-siRNA caused a significant decrease in colony formation.

2 with a dramatic reduction of cell growth and colony formation.

3 ling potential pathogens without waiting for colony formation.

4 resulting in enhanced apoptosis and reduced colony formation.

5 GFB signaling and increase proliferation and colony formation.

6 he minimal cell number needed for successful colony formation.

7 itro elevated the low ROS level and promoted colony formation.

8 ges in morphology, which occur prior to iPSC colony formation.

9 overexpression that results in enhanced AML colony formation.

10 ll lines and reduced their proliferation and colony formation.

11 nhibited cytokine-independent megakaryocytic colony formation.

12 rregular trilineage anemia, with deficits in colony formation.

13 increased tumor cell apoptosis and inhibited colony formation.

14 for proliferation, apoptosis, migration, and colony formation.

15 n naive hESCs reduces cell proliferation and colony formation.

16 f DNA methyltransferases (DMNT) each reduced colony formation.

17 with the increased frequency of aberrant 3D colony formation.

18 in, decreases sphere formation, and inhibits colony formation.

19 that none of them have the ability to induce colony formation.

20 e the dynamics in fitness over the course of colony formation.

21 ion, and decrease mammosphere and progenitor colony formation.

22 s with high endogenous GRHL1 levels promoted colony formation.

23 , camptothecin, or etoposide and assayed for colony formation.

24 indicated that PML is involved in erythroid colony formation.

25 arget of ROS, and enhanced proliferation and colony formation.

26 rmally bonded to control embryonic stem cell colony formation.

27 ation, survival, invasion, dissemination and colony formation.

28 ion of pluripotency and epithelial genes and colony formation.

29 increased activity severely delayed wrinkled colony formation.

30 leading to autocrine Wnt signalling and CSC colony formation.

31 buting cells on surfaces prior to and during colony formation.

32 paired proliferation and anchorage-dependent colony formation.

33 ryptophan repeat can support LIF-independent colony formation.

34 ults in an elevated IFN response and reduces colony formation.

35 fect on increasing the frequency of aberrant colony formations.

36 rrier of Ink4a/Arf locus, thus enhancing iPS colonies formation.

37 lity, proliferation, cell cycle progression, colony formation, 3D spheroid growth, and tumor xenograf

38 To automatically analyse and determine iPSC colony formation, a machine learning-based classificatio

39 oundly reduced cell migration, invasion, and colony-formation abilities of cells overexpressing EMSY

40 YAP1 mutation was shown to increase the colony formation ability and invasion potential of lung

41 tently, TIPE1 inhibited both cell growth and colony formation ability of cultured HCC cell lines, whi

42 eased apoptosis and decrease in invasion and colony formation ability of HNSCC cell lines.

43 dition, the rate of proliferation as well as colony formation ability were significantly decreased in

44 enzalutamide increased cell death, decreased colony formation ability, and resensitized cells to enza

45 CMLD-2 treatment hindered both migration and colony formation ability.

46 cells, such as increased cell proliferation, colony formation, anchorage-independent growth in soft a

47 allow accurate and unbiased determination of colony formation and anchorage independent growth over t

48 ficantly inhibited tumor cell proliferation, colony formation and anchorage-independent cell growth.

49 s of alpelisib on cancer cell proliferation, colony formation and apoptosis and additionally blocked

50 A549R cells resulted in increased levels of colony formation and cell migration as well as reduced a

51 This subsequently reduced the level of colony formation and cell migration facilitating ATO-ind

52 ge-dependent and -independent proliferation, colony formation and cell migration, alterations of 3D-s

53 OPK-032 (TOPK specific inhibitor) attenuated colony formation and cell proliferation of skin cancer c

54 6 and IL-8 expression dramatically inhibited colony formation and cell survival in vitro and stanched

55 3 binding, contributing to the inhibition of colony formation and cell-cycle arrest.

56 , and anti-PTN antibody suppressed human CML colony formation and CML repopulation in vivo.

57 CRC cells inhibited their proliferation and colony formation and decreased their glucose consumption

58 short telomeres to support alveolar organoid colony formation and found that type 2 alveolar epitheli

59 also can be used to monitor the dynamics of colony formation and growth.

60 sis identified multiple factors that recruit colony formation and highlights novel facets of LVCP fun

61 out (PKRKO) mice has increased potential for colony formation and HSPCs are more actively proliferati

62 ssels were assayed at postoperative day 7 by colony formation and immunofluorescence.

63 -expression analyses, together with in vitro colony formation and in vivo adoptive humanized mouse tr

64 expression of Y102F mutant of WASp decreases colony formation and in vivo tumor growth.

65 rols, ARC-deficient RCCs exhibited decreased colony formation and increased apoptosis in vitro.

66 of PDE10 by short hairpin RNA also inhibits colony formation and increases doubling time of colon tu

67 pression of TET1 catalytic domain suppressed colony formation and induced apoptosis of tumor cells of

68 I-CBP112 resulted in substantially impaired colony formation and induced cellular differentiation wi

69 ir proliferation and tumor growth, impairing colony formation and inducing cellular senescence.

70 R1 in prostate cancer cells markedly reduced colony formation and inhibited tumor growth in animals.

71 lase inhibition (GSK-J1, 2,4-PDCA) decreased colony formation and invasion in three-dimensional model

72 tion of ESRRA impaired anchorage-independent colony formation and invasion of OSCC cells.

73 ted with epithelial surface adherence, micro-colony formation and invasion, without overt disease.

74 ted phenotypes including cell proliferation, colony formation and invasiveness, which were restored b

75 nt mouse HSPCs and suppresses human leukemic colony formation and leukemia progression of primary hum

76 MP production, induces apoptosis, and blocks colony formation and mammary tumor growth.

77 ignificantly inhibited anchorage-independent colony formation and mammosphere formation, indicating c

78 The combination treatment decreased colony formation and migration, and induced cell cycle a

79 feration and repressed anchorage-independent colony formation and migration, but increased cell invas

80 rgize with inhibitors of Aurora B to inhibit colony formation and oncogenic transformation.

81 ity attenuates cell proliferation, soft-agar colony formation and orthotopic GBM growth in NOD/SCID m

82 3A, supports cell survival, drug resistance, colony formation and proliferation in vitro, and promote

83 ort functions, secretes factors that promote colony formation and proliferation of purified quiescent

84 G12D/+)-induced splenomegaly and spontaneous colony formation and prolongs the survival of CMML-beari

85 ibited growth factor-independent KRASG12D BM colony formation and sensitized cells to a low dose of t

86 CaP cells expressing Lin28 was determined by colony formation and soft agar assays.

87 tion that results in ER-stress-mediated cell-colony formation and survival, growth, and invasion, whi

88 zygous Aalpha deletion resulted in decreased colony formation and tumor growth across multiple cell l

89 d inhibited cancer cell migration, invasion, colony formation and tumor sphere formation in vitro, bu

90 ignificantly reduced the cell proliferation, colony formation and tumorigenesis abilities of glioblas

91 ckdown of AIB1 decreased cell proliferation, colony formation and tumorigenesis of these CRC cells.

92 SECs, induced cell proliferation, migration, colony formation and tumorigenesis.

93 In vitro colony formation and wound healing experiments were perf

94 ne(R) and PKC412(R) had higher capability of colony formation and wound healing than parental cells i

95 The results of colony formation and xenograft assays showed a mutation

96 ted ICN1 to facilitate anchorage-independent colony formation and xenograft tumor growth with increas

97 found that E6, but not E7, recovers FA iPSC colony formation and, furthermore, that p53 inhibition i

98 profibrotic gene expression, cell migration, colony formation, and activation of the transcription fa

99 sed cell proliferation, migration, invasion, colony formation, and anchorage-dependent growth in HCC

100 e expression suppression of CSR1, suppressed colony formation, and blocked cell cycle entry to the S

101 vity against melanoma cells in cytotoxicity, colony formation, and cell invasion studies.

102 in (i) a decrease in cellular proliferation, colony formation, and cellular migration; (ii) induction

103 Alterations of cell proliferation, colony formation, and cellular senescence were evaluated

104 demonstrated superior hematopoietic growth, colony formation, and diversification compared to ALD an

105 that promote cell proliferation, anchor-free colony formation, and epithelial-mesenchymal transition

106 CC cells abrogates cell branching, invasion, colony formation, and growth in a murine xenograft model

107 signaling, transwell invasion and soft agar colony formation, and in vivo promoted lung metastasis i

108 ted in decreased cell proliferation, reduced colony formation, and increased apoptosis.

109 profoundly suppressed cell proliferation and colony formation, and induced cell cycle arrest accompan

110 ion of CBX6 inhibited cell proliferation and colony formation, and induced cell cycle arrest along wi

111 ulted in decreased tumor cell proliferation, colony formation, and invasion in vitro Blocking endogen

112 Crispr/Cas9 greatly promotes cell viability, colony formation, and invasion of cancer cells in vitro

113 ssed MAN2A1-FER had increased proliferation, colony formation, and invasiveness and formed larger (>2

114 oma cells enhanced tumor cell proliferation, colony formation, and invasiveness, in vitro.

115 cogenic properties, including proliferation, colony formation, and motility.

116 ncident with increased cell death, decreased colony formation, and reduced tumor growth in an in vivo

117 IC50, suppresses PCa cell proliferation and colony formation, and reduces migration and invasion.

118 SSTs, increased proliferation, migration and colony formation, and SHH pathway activation.

119 /progenitor cells, inhibited cell growth and colony formation, and significantly prolonged survival i

120 ly increases apoptosis of CMLSCs, suppresses colony formation, and significantly prolongs survival in

121 and BCL2 markedly suppressed proliferation, colony formation, and survival of Ph(+) ALL cells ex viv

122 as the percentage of CD34(+) cells in cycle, colony formation, and survival, demonstrating its partic

123 an increased RBC mass, spontaneous erythroid colony formation, and the JAK2V617F mutation.

124 of EMT, the loss of TIC-mediated clonogenic colony formation, and the loss of cell motility and inva

125 TD1A inhibits colorectal cancer cell growth, colony formation, and tumor engraftment.

126 signaling and inhibited cell proliferation, colony formation, and tumor growth in subcutaneous and o

127 purine synthesis, RAS-dependent cancer cell-colony formation, and tumor growth.

128 h tenascin C exhibited enhanced adhesion and colony formation as mediated by integrin alpha9beta1.

129 ramatically altered cell invasion potential, colony formation, as well as tumorigenesis in orthotopic

130 Colony formation assay from explanted heart tubes and ge

131 micals were tested for their cytotoxicity by colony formation assay in cells of different BRCA2 statu

132 In cancer research, colony formation assay is a gold standard for the invest

133 Finally, colony formation assay revealed a dramatic decrease in t

134 ly, as assessed in vitro and in vivo using a colony formation assay, a spheroid formation assay and a

135 igher anchorage independence growth (AIG) in colony formation assay, and exhibited augmented motility

136 illing, the ability of cells to survive in a colony formation assay, and proliferation rates after ra

137 n vitro and in vivo, which were confirmed by colony formation assay, transwell invasion assay, and tu

138 ntification is a promising technique for the colony formation assay.

139 -irradiation at doses of 0, 2, 4, or 6 Gy by colony formation assay.

140 -dependent hypersensitivity by hematopoietic colony formation assays and phospho-STAT5 (pSTAT5) flow

141 AF WT, transformed NIH3T3 cells in soft-agar colony formation assays, increased kinase activity in vi

142 onferred resistance to ionizing radiation in colony formation assays, suggesting that DYRK1A expressi

143 yzed by immunoblotting and proliferation and colony formation assays.

144 o radiation-induced cell death determined in colony-formation assays.

145 esulted in shortened lag-phase and increased colony formation at sub-inhibitory concentrations of van

146 ed to not only increased tumor formation and colony formation but also increased tumor dispersal to s

147 significantly reduced cell proliferation and colony formation but induced tumor cell senescence.

148 JQ1 inhibited colony formation, but increased apoptosis in HD-MB03 and

149 al inhibition of Gln uptake blocks soft agar colony formation by Hace1(-/-) MEFs.

150 It strongly inhibits colony formation by MM cells while sparing surrounding B

151 enous IL-33 promoted cytokine production and colony formation by primary CD34+ MPN stem/progenitor ce

152 ng this pathway can partially reduce myeloid colony formation by Rcor1-deficient erythroid progenitor

153 lows Runx1 to increase Cebpa and granulocyte colony formation by Runx1-deleted murine marrow.

154 MERTK-mediated downstream signaling, reduced colony formation by up to 59%, and diminished tumor volu

155 170 not only promoted cell proliferation and colony formation by up-regulating the expression of cycl

156 tools to quantify the frequency of aberrant colony formations by the combined stressors of stiffness

157 Functionally, knockout of SNORD42A reduced colony formation capability and inhibited proliferation.

158 Colony formation capability of single cells in G(2)/M, b

159 und reductions in cellular proliferation and colony-formation capacities.

160 racts the decrease in cell proliferation and colony formation caused by UV-induced DNA damage.

161 4a potently inhibited cell proliferation and colony formation, causing an increase of the G2/M phase

162 CDH10 promoted cell proliferation, soft-agar colony formation, cell migration and cell invasion, and

163 ated with increased levels of cell survival, colony formation, cell migration and decreased cellular

164 tumorsphere formation, anchorage-independent colony formation, cell migration in vitro, and lung meta

165 1) significantly reduced cell proliferation, colony formation, cell migration, and/or growth of xenog

166 ecrease in medulloblastoma cell growth, cell colony formation, cell migration, invasion, and tumor sp

167 etion of SATB2 inhibited cell proliferation, colony formation, cell motility and expression of beta-c

168 d with controls showed significantly reduced colony formation, cell proliferation, induced cell cycle

169 Contrasting the reduced colony formation, cells attached on narrow patterns were

170 r in vitro hematopoietic-differentiation and colony formation (CFU assay).

171 increased capacity for anchorage-independent colony formation compared to CD44(lo) IPF MPCs.

172 of cancer cell extravasation and metastatic colony formation compared to stimulated cancer cells.

173 ve for long-term growth, as shown by reduced colony formation compared with cells expressing either c

174 -GBM astrocytes exhibited greater growth and colony formation compared with female Mes-GBM astrocytes

175 epletion of MLL target Ikzf2 in LSCs reduced colony formation, decreased proliferation, and increased

176 secretion, increased invasiveness, increased colony formation, decreased PTEN expression, and formati

177 ed a significant increase in reappearance of colony formation depth (P < 0.05), possibly indicating t

178 nd reduced colony-forming unit-megakaryocyte colony formation driven by JAK2-V617F, but was not suffi

179 s had rapid proliferation rates and enhanced colony formation efficiencies.

180 transformed cells and significantly reduces colony formation efficiency, in a dose-dependent manner,

181 udy showed that sesamol treatment suppressed colony formation, elicited S phase arrest during cell cy

182 into clusters induced a >15-fold increase in colony formation ex vivo and a >100-fold increase in met

183 ogenitor cells exhibited an elevation in RBC colony formation ex vivo.

184 Reduction in cht7 colony formation following N refeeding is explained by i

185 ckdown of Skp2 suppresses tumor cell growth, colony formation, glycolysis, and in vivo tumor growth.

186 nd were demonstrated to yield a high rate of colony formation (>/=85%) after removal from the array.

187 ng in a strong differentiation phenotype and colony formation impairment, confirming the potential of

188 f RSK2, and another RSK2 inhibitor increased colony formation implicating a role for this kinase in e

189 2 suppresses leukemia cell proliferation and colony formation in a manner dependent on WT1.

190 From colony formation in bacteria to wound healing and embryo

191 We observed a strong inhibition of colony formation in cells overexpressing these tsRNAs co

192 eral hematological cell lines, inhibition of colony formation in HEL cells, and analysis of apoptosis

193 uld promote cell proliferation and soft agar colony formation in HNSCC cells with low FGF19 expressio

194 nucleation, decreased cell proliferation and colony formation in human NSCLC cells, and reduced tumor

195 cytokine-independent growth, and/or enhanced colony formation in Jurkat T cells.

196 ion defect, triggers senescence and inhibits colony formation in liver, but also breast and lung canc

197 knockdown of CBL enhanced cell motility and colony formation in NSCLC cells, and these activities we

198 rowth, induced differentiation, and impaired colony formation in primary AML blasts.

199 cell proliferation and anchorage-independent colony formation in primary human melanocytes.

200 TGM2 suppressed colony formation in soft agar and tumor formation in a x

201 Colony formation in soft agar is the gold-standard assay

202 inhibitors abolished cell proliferation and colony formation in soft agar of KSHV-transformed cells

203 s suppresses growth proliferation, abolishes colony formation in soft agar, and decreases NO generati

204 d to cell hyperproliferation, enhanced tumor colony formation in soft agar, and increased xenograft t

205 icantly inhibits both cell proliferation and colony formation in soft agar, and induces apoptosis in

206 ing in decreased cell proliferation, reduced colony formation in soft agar, and induction of apoptosi

207 aling, induced apoptosis in culture, reduced colony formation in soft agar, and inhibited invasion of

208 is required for KSHV-induced proliferation, colony formation in soft agar, and NO generation of KSHV

209 owth conditions, however, including impaired colony formation in soft agar, spheroid formation, and x

210 ion of NFAT3 at Ser259 led to a reduction of colony formation in soft agar.

211 t tumor cells dependent on IRS1 activity for colony formation in soft agar.

212 romoted cancer cell growth and migration and colony formation in soft agar.

213 htened transformation potential, assessed by colony formation in soft agar.

214 ve mutant, suppressed cell proliferation and colony formation in soft-agar assays.

215 bit HRAS expression, metabolic activity, and colony formation in T24 cancer cells.

216 inhibition of this pathway prevents both CSC colony formation in the bone environment, and bone metas

217 ivity versus MAO A and remarkably inhibiting colony formation in THP-1 human leukemia cells, were ass

218 n addition, overexpression of MGL suppressed colony formation in tumor cell lines and knockdown of MG

219 y reduced cell viability, proliferation, and colony formation in vitro and delayed tumor growth in vi

220 ene knockdown inhibited TNBC cell growth and colony formation in vitro and growth of MDA-MB-468 xenog

221 Finally, FBXO32 inhibits colony formation in vitro and primary tumor initiation a

222 cell proliferation, migration, invasion, and colony formation in vitro and to inhibit HCC tumor growt

223 lthough all SLE-derived HSPCs exhibited poor colony formation in vitro compared with controls, SLE HS

224 Tumor growth was determined with colony formation in vitro or in vivo.

225 tin inhibits prostate cancer cell growth and colony formation in vitro, and growth of prostate cancer

226 ble transfectants suppressed cell growth and colony formation in vitro, and PTTG cell knockout also c

227 e properties including cellular invasion and colony formation in vitro, as well as tumor growth and m

228 iminate granulocyte/macrophage and erythroid colony formation in vitro.

229 ppression of either ZNF148 or CTNNB1 reduced colony formation in WNT-dependent, but not WNT-independe

230 c traits, including migration, invasion, and colony formation, in soft agar with CD66(high) cells.

231 n acute myeloid leukemia (AML) cells reduced colony formation, increased differentiation and apoptosi

232 Analysis of 3D colony formations indicate that high stiffness, within t

233 n the control of HSPC cycling, survival, and colony formation induced by CXCL12.

234 of U251MG cells with Cmpd1 reduced in vitro colony formation, induced cell cycle arrest in the G2/M

235 GRHL1 levels abrogated anchorage-independent colony formation, inhibited proliferation, and retarded

236 esulted in increased cellular proliferation, colony formation, invasion, and development of a multinu

237 80A mutant Nanog reduced cell proliferation, colony formation, invasion, migration and the CIC popula

238 KDM3A in S2-007 cells significantly reduced colony formation, invasion, migration, and spheroid form

239 Phenotypic profiling reveals that 3D colony formation is heterogeneous and increased stiffnes

240 levels and leading to reduced proliferation, colony formation, migration, and invasion.

241 e, as well as promoting their proliferation, colony formation, migration, and osteogenic potential.

242 We measured effects on proliferation, colony formation, migration, and/or growth of xenograft

243 and loss of NO66 function on cell viability, colony formation, migration, invasion, and tumor-induced

244 ce knockdown of MAPK7 reduces proliferation, colony formation, migration, tumour growth, macrophage r

245 adhesion and reduces cellular proliferation, colony formation, migration, wound healing, tumor growth

246 A first case study describes the colony formation of a rod-shaped species on a planar sub

247 2HG levels in vitro, and efficiently blocked colony formation of AML cells from IDH1-mutated patients

248 TX blocked an FGFR3-mediated increase in the colony formation of bladder cells.

249 suppressed the proliferation, invasion, and colony formation of breast cancer cells in vitro and tum

250 l cycle arrest, and attenuated migration and colony formation of breast cancer cells.

251 ition, IFN-gamma increased proliferation and colony formation of CD34(+) stem/progenitor cells from C

252 IPO11 knockout decreased colony formation of CRC cell lines and decreased prolife

253 Colony formation of E-cadherin-negative cells was rescue

254 tudies showed decreased survival and reduced colony formation of Fan1(-/-) mouse embryonic fibroblast

255 Loss of TCF7L1 impaired growth and colony formation of HCT116 CRC cells and reduced tumor g

256 Depletion of JMJD1C impairs expansion and colony formation of human leukemic cell lines, with the

257 Depletion of PPP2R1A inhibited colony formation of Mad2-overexpressing HeLa cells but n

258 K578R mutant inhibited anchorage-independent colony formation of MCF7 breast cancer cell line.

259 roliferation, neoplastic transformation, and colony formation of mouse epidermal cells JB6 Cl41, huma

260 Depletion of Jmjd1c impairs growth and colony formation of mouse MLL-AF9 cells in vitro as well

261 ession of H1.3 decreases the growth rate and colony formation of OVCAR-3 cells.

262 ibition of FOXO3a restored proliferation and colony formation of Pdk1(-/-) melanoma cells.

263 (AVID200 and SD208) increased hematopoietic colony formation of SDS patient BM.

264 th robust proliferative potential, secondary colony formation on replating, and de novo blood vessel

265 istant cells, whereas it was unable to block colony formation or cell survival, suggesting that the r

266 dopting various lifestyles such as swimming, colony formation or dormancy.

267 f FDPS promotes oncogenic phenotypes such as colony formation (P < 0.01) and proliferation (P < 0.01)

268 ionally, diabetic ECFCs demonstrated delayed colony formation (p < 0.02), differential proliferative

269 r His-MDA-7/IL-24 inhibition of cell growth, colony formation, PARP cleavage, and apoptosis.

270 5-azacytidine reduced the proliferation and colony formation potential in ERBB2-positive breast canc

271 In addition, GSK2830371 suppressed the colony-formation potential of p53 wild-type NB cell line

272 itative and objective method to describe the colony formation process and the development of colony s

273 Hence, the colony formation process could be quantitatively represe

274 GRP1 overexpression bestows gain-of-function colony formation properties to bone marrow progenitors i

275 termining the reprogramming process and iPSC colony formation quantitatively, a mathematical model wa

276 cell cycle arrest at S phase, and decreased colony formation rate.

277 tivity to ADI-PEG 20, evidenced by decreased colony formation, reduced cell viability, and increased

278 The inhibition of colony formation, regulated by cisplatin, was more signi

279 Hormone-independent luminal progenitor colony formation required NF-kappaB, ataxia telangiectas

280 rotic targets, cell migration, and soft agar colony formation stimulated by TGF-beta required GLS1 ac

281 ellular assays and primary mouse bone marrow colony formation studies.

282 osis, decreased proliferation, and decreased colony formation, suggesting that SRSF2 has oncogenic fu

283 with loss of SOX7 promoted cell survival and colony formation, suppressed cellular apoptosis and prod

284 TMEM43 deficiency significantly affects colony formation, survival of anoikis-induced cell death

285 lation increased cell invasion and soft agar colony formation; this was dependent on NF-kappaB activa

286 Both diffusion inhibition zone tests and colony formation unit tests showed clear antimicrobial e

287 n for bacterial detection equal to 10(2)CFU (colony formation unit) for live bacteria detection with

288 In the absence of inhibitors, colony formation units (CFUs) per milliliter in blood fr

289 After 30 min irradiation, no colony formation was detected using TCP-C60 or TCP/TCP-C

290 r the periphery of the flasks, while spatial colony formation was statistically uniform at energies a

291 myeloma cells, whereas normal hematopoietic colony formation was unaffected.

292 nematics of single and pairs of hESCs impact colony formation, we study their mobility characteristic

293 ncer cell lines, both cell proliferation and colony formation were decreased.

294 in vitro Conversely, migratory potential and colony formation were enhanced in PHD3-deficient cells,

295 On day 1, in ARDSp, different patterns of colony formation were found, with nonstromal cells (main

296 f TIE2, and increase survival, invasion, and colony formation when expressed in human umbilical vein

297 auxiliary proteins had the ability to induce colony formation, while both Tax-3 and antisense protein

298 revealed an increased frequency of aberrant colony formation with the increased stiffness; however,

299 o decreased transwell invasion and soft agar colony formation, without affecting proliferation.

300 epithelial-to-mesenchymal transition (EMT), colony formation, xenograft-tumor growth in athymic mice