ライフサイエンスコーパス: comatose

1 noncomatose patients who subsequently became comatose.

2 core and cumulative doses of sedatives while comatose.

3 roups based on their best examination as (1) comatose, (2) arousable (eye opening or attending toward

4 ale scores grouped into four levels denoted "comatose" (-5), "deeply sedated" (-4 to -3), "lightly se

5 Before LT, the majority of patients were comatose (76%), on hemodialysis (16%), and ICU-bound.

6 mpiric use of naloxone and flumazenil in the comatose adult patient who presents to the emergency dep

7 domized 2-by-2 factorial trial, in which 789 comatose adult patients who had out-of-hospital cardiac

8 We examined a prospective cohort of 103 comatose adult patients who were unconscious 48 hours af

9 Comatose, adult out-of-hospital cardiac arrest patients

10 atients were assessed for eligibility and 98 comatose adults (Glasgow Coma Scale [GCS] score of </=8

11 Consecutive comatose adults admitted after cardiac arrest, identifie

12 Therapeutic hypothermia is recommended for comatose adults after witnessed out-of-hospital cardiac

13 ed temperature management is recommended for comatose adults and children after out-of-hospital cardi

14 get of 63 mm Hg as compared with 77 mm Hg in comatose adults who had been resuscitated after an out-o

15 -by-2 factorial design, we randomly assigned comatose adults with out-of-hospital cardiac arrest in a

16 Patients still comatose after a witnessed OHCA of presumed cardiac orig

17 multicenter cohort were 46 patients who were comatose after CA.

18 Patients who are comatose after cardiac arrest continue to be a challenge

19 ng out-of-hospital cardiac arrest who remain comatose after initial resuscitation are at high risk of

20 degrees C, for pediatric patients who remain comatose after resuscitation from out-of-hospital cardia

21 been standard of care for patients remaining comatose after resuscitation from out-of-hospital cardia

22 dicate ICP monitoring in patients who remain comatose after resuscitation if the admission computed t

23 n-traumatic cardiac arrest patients who were comatose after return of spontaneous circulation.

24 as follows: age >/=18, nontrauma arrest, and comatose after return of spontaneous circulation.

25 than 17 years, nontrauma cardiac arrest, and comatose after return of spontaneous circulation.

26 Brains of comatose ALF mice were processed for histological and bi

27 rature-sensitive paralytic mutations in NSF (comatose) also block synaptic transmission, but over a m

28 lar synapses of Drosophila double mutant for comatose (an NSF mutant) and Kum (a SERCA mutant), and p

29 chieved spontaneous circulation but remained comatose and (except one) died within 58 hrs with multip

30 severe malarial anemia and can also benefit comatose and acidotic malaria patients.

31 uillain-Barre syndrome, or when a patient is comatose and cardinal signs may be lacking.

32 All patients were initially comatose and had invasive brain monitoring placed.

33 between 0.4 hours (for patients who are not comatose and had no prior seizure) and 16.4 hours (comat

34 thalamus, brain stem, and cerebellum between comatose and noncomatose patients acutely after TBI.

35 However, compared between comatose and noncomatose patients, CMRglc values in the

36 se and had no prior seizure) and 16.4 hours (comatose and prior seizure).

37 ositive for delirium, 13% were classified as comatose, and 62% were delirium-free and coma-free.

38 are units who were possibly brain stem dead (comatose, apparently apnoeic with unresponsive pupils) d

39 Eighty-one percent of patients who were comatose at admission survived.

40 itated by prehospital personnel yet remained comatose at arrival to the hospital.

41 r than 18 years) patients with OHCA who were comatose at hospital admission.

42 H were restricted to those patients who were comatose at hospital admission.

43 was performed in 241 of 941 (25.6%) persons comatose at hospital admission.

44 Over half of patients were comatose at presentation (53%).

45 the primary admission diagnosis and 48% were comatose at the time of cEEG.

46 ain networks had typically become nonhubs of comatose brain networks and vice versa.

47 All FHF rats became comatose by 24 hours postoperatively.

48 wing a high performance in identifying early comatose CA survivors who will reach functional independ

49 Prognostication studies on comatose cardiac arrest (CA) patients are limited by lac

50 ppropriate time frame for prognostication in comatose cardiac arrest survivors treated with therapeut

51 In initially comatose cardiac arrest survivors, improvements in funct

52 therapeutic hypothermia improves outcomes in comatose cardiac arrest survivors.

53 en, 281 [79.2%] men) with STEMI-CS excluding comatose cardiac arrest were enrolled; 179 were randomiz

54 hin 6 hours after the return of circulation, comatose children older than 48 hours and younger than 1

55 Among comatose children who survived in-hospital cardiac arres

56 In comatose children who survived out-of-hospital cardiac a

57 In comatose children, with Glasgow Motor Scale score less t

58 The Drosophila comatose (comt) gene encodes an NSF homolog, dNSF1.

59 temperature-sensitive paralytic phenotype in comatose (comt) mutants.

60 temperature-sensitive Drosophila NSF mutant comatose (comt) to study the function of NSF in neurotra

61 Among those candidates, we identified Comatose (Comt), the homologue of the N-ethylmaleimide s

62 ve cerebral malaria and 264 age-matched, non-comatose controls were followed up for a median of 495 d

63 Whilst the morphology of comatose (cts) embryos is not altered, physiological ana

64 COMATOSE (CTS) encodes a peroxisomal ATP-binding cassett

65 Here we show that the Arabidopsis COMATOSE (CTS) locus is required for this transition, an

66 The peroxisomal ABC transporter, Comatose (CTS), a full length transporter from Arabidops

67 Using COMATOSE (CTS), a plant representative of the ABCD famil

68 phenotype comparable with that reported for comatose (cts-2), a mutant in a peroxisomal ABC transpor

69 CI, 1.34-3.44; p = 0.002), the percentage of comatose days (66.0% 31.3% vs 36.0% 36.9%, adjusted diff

70 alyses were used to assess the percentage of comatose days, sedative medications used, and the associ

71 ns, we have undertaken a genetic analysis of comatose (dNSF-1) in Drosophila.

72 Ventilator-dependent, comatose, do not resuscitate, and female patients were e

73 tire vesicle pool can be depleted in shibire comatose double mutants, demonstrating that NSF activity

74 logram and electrocardiogram signals in four comatose dying patients before and after the withdrawal

75 ors had favorable outcomes despite remaining comatose for 1 to 2 weeks postarrest.

76 Fifteen comatose (Glasgow Coma Scale score of < or = 7) adult pa

77 The incidence of survival of comatose hospital patients decreased by 29% from 1975 to

78 Comatose humans have upper airway soft tissue obstructio

79 In 1960, measurements on comatose humans with or without cardiac arrest, with or

80 umented bacterial aspiration pneumonia among comatose ICU patients with symptoms suggesting either ba

81 atus, resuscitation from cardiac arrest, and comatose immediately after resuscitation.

82 large number of patients who were initially comatose, in whom the ultimate outcome is unclear.

83 peroxisomal ATP-binding cassette transporter COMATOSE is required for the accumulation of benzoylated

84 Mutation of the COMATOSE locus in Arabidopsis results in a marked reduct

85 behavioral paralysis that normally occurs in comatose mutants, indicating NSF activity is not require

86 of mammalian NSF (G274E) and Drosophila NSF (comatose) mutants revealed an evolutionarily conserved N

87 ac mutant was identified as an enhancer of a comatose mutation's effects on general locomotion.

88 Characterization of 16 comatose mutations demonstrates that NSF mediates disass

89 Comatose neurocritically ill adults with an absolute dif

90 Performance Category 3-5, severe disability, comatose, or death).

91 able neurologic outcome in a large cohort of comatose out-of-hospital cardiac arrest patients treated

92 The classification of the comatose patient has been greatly improved with the use

93 A comatose patient has neither wakefulness nor awareness;

94 approach for the clinical management of the comatose patient.

95 evels of the inflammatory mediator IL-6 than comatose patients (129.3 vs. 35.0 pg/mL, p=0.05).

96 One hundred ten comatose patients (aged 24-76 years) who had experienced

97 e BOLD fMRI signals from two classes of post-comatose patients (minimally conscious state and unrespo

98 nter prospective cohort study included adult comatose patients admitted to the intensive care unit (I

99 o rapidly induce and maintain hypothermia in comatose patients after cardiac arrest and a small numbe

100 res provides unequalled prognostic value for comatose patients after cardiac arrest and enables bedsi

101 come prediction of approximately half of all comatose patients after cardiac arrest.

102 dside insight in the neurologic prognosis of comatose patients after cardiac arrest.

103 measures contribute to outcome prediction of comatose patients after cardiac arrest.

104 Two hundred eighty-three consecutive comatose patients after cardiac arrest.

105 domized clinical trial, Ghrelin Treatment of Comatose Patients After Cardiac Arrest: A Clinical Trial

106 In this predefined analysis of comatose patients after OHCA, hypothermia did not lead t

107 logy assessments, or functional survival for comatose patients after out-of-hospital cardiac arrest.

108 strictive or liberal oxygenation strategy in comatose patients after resuscitation for cardiac arrest

109 prognostic performance in postcardiac arrest comatose patients and whether quantitative pupillary lig

110 ble, the results were abnormal for all eight comatose patients as well as for all three noncomatose p

111 Excluding evaluations of comatose patients because of lack of characteristic deli

112 Parasitized comatose patients dying of other causes are less likely

113 the utility of the neurologic examination in comatose patients from nontraumatic causes in the modern

114 Acute administration of exenatide to comatose patients in the intensive care unit after OHCA

115 In febrile comatose patients living in malaria-endemic areas, overl

116 ncephalography and electrocardiography in 48 comatose patients on the first day after cardiac arrest

117 Among comatose patients receiving mechanical ventilation, thos

118 Eighty-two percent (n = 1674) of comatose patients recovered consciousness during inpatie

119 Comatose patients resuscitated after out-of-hospital car

120 We randomly assigned 120 consecutive comatose patients resuscitated from OHCA in a double-bli

121 stemic inflammation and myocardial injury in comatose patients resuscitated from out-of-hospital card

122 In-hospital mortality in comatose patients resuscitated from out-of-hospital card

123 Acutely comatose patients secondary to brain injury.

124 postresuscitation care for resuscitated but comatose patients that includes therapeutic hypothermia

125 We studied a cohort of consecutive comatose patients treated after cardiac arrest from Janu

126 A total 91 comatose patients were enrolled in the study.

127 We randomly assigned comatose patients who had been resuscitated after an out

128 In comatose patients who had been resuscitated after out-of

129 A total of 110 comatose patients who had experienced OHCA from a cardia

130 l outcomes after global ischaemia-hypoxia in comatose patients who have had cardiac arrest, and is on

131 Among 249 comatose patients who received prehospital care, 205 die

132 Among 101 comatose patients who were hospitalized, 63 died; the od

133 hin 6 hours after the return of circulation, comatose patients who were older than 2 days and younger

134 d, HIPAA-compliant retrospective study of 80 comatose patients with cardiac arrest who underwent diff

135 s safe and can provide metabolic support for comatose patients with fulminant hepatic failure for up

136 This study investigated whether comatose patients with greater duration and magnitude of

137 This prospective cohort study included 90 comatose patients with high-grade spontaneous subarachno

138 age should be considered in all stuporous or comatose patients with intraventricular haemorrhage and

139 study, we prospectively enrolled consecutive comatose patients with irreversible brain injury undergo

140 Eighty comatose patients with out-of-hospital cardiac arrest we

141 ermia at 32-34 degrees C for 12-24 hours for comatose patients with out-of-hospital cardiac arrest wi

142 during postresucitation intensive care among comatose patients with out-of-hospital cardiac arrest wi

143 thesized musical instrument tones in 22 post-comatose patients with severe brain injury causing varia

144 ylaxis upon the first day of the ICU stay in comatose patients with severe brain injury could enlarge

145 nd 1 intensive care unit (ICU) that included comatose patients with suspected acute poisoning and a G

146 Among comatose patients with suspected acute poisoning, a cons

147 Controls were comatose patients without malaria (n = 6) and age/sex-ma

148 For postresuscitation comatose patients, early quantitative pupillary light re

149 After inclusion of 98 comatose patients, the trial was stopped early at the re

150 lography responses to auditory stimuli in 94 comatose patients, under hypothermia and after re-warmin

151 poor long-term clinical outcomes in acutely comatose patients.

152 g/min, P < 0.05) were significantly lower in comatose patients.

153 meeting readiness criteria, particularly for comatose patients.

154 cal care yet struggle to predict outcomes in comatose patients.

155 ement are associated with acute awakening in comatose patients.

156 regulation, and clinical outcomes in acutely comatose patients.

157 global network properties were conserved in comatose patients.

158 targeted temperature management) be used in comatose post-cardiac arrest patients?

159 Of our cohort of comatose postarrest patients, 59% (29 of 49) were male,

160 wn about when to assess neurologic status in comatose, postarrest patients receiving therapeutic hypo

161 Consecutive comatose postcardiac arrest patients were prospectively

162 Quantitative DWI in comatose postcardiac arrest survivors holds promise as a

163 Outcome prediction is challenging in comatose postcardiac arrest survivors.

164 or no analgesia/sedation, in a predominantly comatose sample.

165 ult patients 18 years or older who were in a comatose state after cardiac arrest were assessed for el

166 es, spend a considerable amount of time in a comatose state that can include time in burst suppressio

167 y in the descending aorta, and pre-operative comatose state were independent predictors, again with i

168 ltrates or cavities on chest radiograph; and comatose state, intubation, receipt of pressors, or deat

169 This brain injury, commonly manifested as a comatose state, is a marker of poor outcome and a major

170 Pre-operative cerebral malperfusion, comatose state, peripheral malperfusion, visceral malper

171 ents whose behavioral examination suggests a comatose state.

172 ventilated patients remaining unresponsive (comatose/stuporous or unable to follow commands) after s

173 multimodality physiological recordings in 48 comatose subarachnoid hemorrhage patients to better char

174 Twenty of 56 comatose survivors (32%) treated with hypothermia who aw

175 ic electroencephalographic (EEG) patterns in comatose survivors of cardiac arrest improves outcomes i

176 s not support early coronary angiography for comatose survivors of cardiac arrest without ST elevatio

177 ute ischemic stroke, induced hypothermia for comatose survivors of cardiac arrest, and endovascular c

178 the use of therapeutic hypothermia exist in comatose survivors of cardiac arrest, and recent trials

179 In comatose survivors of cardiac arrest, the incidence of a

180 Surface cooling improves outcome in selected comatose survivors of cardiac arrest.

181 rns detected on continuous EEG monitoring in comatose survivors of cardiac arrest.

182 hermia (TH) attenuates reperfusion injury in comatose survivors of cardiac arrest.

183 ctive cohort study, we included consecutive, comatose survivors of cardiac arrest.

184 t mild induced hypothermia be considered for comatose survivors of in-hospital cardiac arrest.

185 Among comatose survivors of OHCA, in comparison with hypotherm

186 rmia attenuates brain white matter injury in comatose survivors of out-of-hospital cardiac arrest (OH

187 Comatose survivors of out-of-hospital cardiac arrest exp

188 enation target for mechanical ventilation in comatose survivors of out-of-hospital cardiac arrest is

189 It is recommended that comatose survivors of out-of-hospital cardiac arrest sho

190 blood-pressure targets for the treatment of comatose survivors of out-of-hospital cardiac arrest who

191 In comatose survivors of out-of-hospital cardiac arrest, a

192 Among comatose survivors of out-of-hospital cardiac arrest, in

193 Targeted temperature management in comatose survivors of out-of-hospital cardiac arrest.

194 peutic hypothermia and neurologic outcome in comatose survivors of out-of-hospital cardiac arrest.

195 ia induced after successful resuscitation of comatose survivors of ventricular fibrillation cardiac a

196 The study cohort included 154 comatose survivors of witnessed out-of-hospital cardiopu

197 Adult (>18 years) comatose survivors without ST-segment elevation after re

198 control and neurological prognostication of comatose survivors, 2 topics for which there are more ro

199 (13.5%) were excluded because they remained comatose throughout the investigation.

200 m recorded (CAM-ICU-), and 27 (29%) remained comatose until extubation or death.

201 Patients who were comatose were less likely to receive these medications.