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1 horesis and single cell gel electrophoresis (Comet Assay).
2 lar zinc increased DNA single-strand breaks (comet assay).
3 ity to repair DNA lesions as measured by the comet assay.
4 unhooking capacity measured using a modified comet assay.
5 of damage could be evaluated in real time by comet assay.
6 eased after IGF-IR activation as measured by comet assay.
7 ibutable to DNA damage, as determined by the comet assay.
8 om TPZ-induced DNA damage measured using the comet assay.
9 ed from measurements of DNA damage using the comet assay.
10 ononuclear cells from 13 pSS patients, using comet assay.
11 ucible values obtained from SDFR and neutral comet assay.
12  doses of 0.1 to 1.5 kGy and analyzed by DNA Comet Assay.
13 -/-)p53(-/-) MEFs as demonstrated by neutral Comet assay.
14  assessed DNA damage by means of the neutral comet assay.
15 T3-L1 normal cells was detected by using the comet assay.
16 ential induced DNA damage in vitro using the Comet Assay.
17 dants and the DNA damage was measured by the Comet assay.
18 xtracts induced DNA damage, evaluated with a comet assay.
19 llowing gamma-irradiation as assessed by the comet assay.
20 nternalization, as evaluated by the alkaline comet assay.
21 sed DNA double-strand breaks as shown by the comet assay.
22 ated with accelerated DNA repair measured by comet assay.
23  gammaH2A.X expression/foci formation and by comet assay.
24 amage measured by H2AX phosphorylation and a comet assay.
25 mmunolocalization of gamma-H2AX foci and the COMET assay.
26 X) and increased DNA breaks, as evidenced by comet assays.
27 trand breaks, as reflected in gammaH2A.X and comet assays.
28 estern blot analysis, immunofluorescence and comet assays.
29 e repairs, as shown by radio-sensitivity and Comet assays.
30 rm of histone H2AX (gamma-H2AX) and alkaline Comet assays.
31 he alkaline single cell gel electrophoresis (comet) assay.
32  by using a single-cell gel electrophoresis (comet) assay.
33 beling, and single-cell gel electrophoresis (COMET) assays.
34 ubsequently measured as DNA breaks using the comet assay (7 groups of volunteers, in six countries).
35               Moreover, through the Alkaline Comet Assay, a technique that quantifies DNA damage at t
36 ncluding DNA damage in lymphocytes (with the comet assay), activity of detoxifying enzymes (glutathio
37 tion of the single cell gel electrophoresis (comet) assay after administration of a therapeutic dose.
38                  Compared with halo assay or comet assay alone, this method allows automated analysis
39               Clonogenic survival assays and comet assays also show that Spy1 expression enhances cel
40               Using both a modified alkaline comet assay and a DNA cleavage assay, we demonstrate tha
41 breaks (DSBs) that are detectible by neutral COMET assay and as gamma-H2AX foci that colocalize with
42 firming this phenomenon, we have revealed by comet assay and currently the most sensitive method of D
43 yed ICL processing as revealed by a modified Comet assay and gamma-H2AX foci persistence.
44 nhanced DNA damage, as measured by using the comet assay and gammaH2AX staining.
45 he offspring, stability was assessed via the Comet assay and highly sensitive, semiautomated confocal
46  DNA damage following BPA exposure using the comet assay and immunofluorescence staining, and used ce
47 898E-reconstituted cells, as revealed by the comet assay and IR-induced Rad51 foci formation assay.
48 induced DSBs as revealed by both the neutral comet assay and measurements of the specific DNA damage
49    This study demonstrates, by using neutral comet assay and pulsed field gel electrophoresis, that h
50                 Here, we confirm by specific comet assay and pulsed-field electrophoresis that cells
51 ine-dependent DNA damage, as measured by the comet assay and the expression of the phosphorylated for
52                 Moreover, as measured by the Comet assay and the induction of gamma-H2A.X, quercetin,
53 h was associated with DNA damage analyzed by comet assay and up-regulation of p53.
54             An Alu retrotransposition assay, COMET assays and 53BP1 foci staining show that the SpORF
55 ged the induction of DNA damage as judged by Comet assays and gammahistone 2AX (gammaH2AX) and checkp
56 emcitabine-induced DNA damage as measured by comet assays and quantification of gammaH2AX foci.
57 DNA repair as revealed by gammaH2AX foci and comet assays and to the up-regulation of Ku70 and DNA-de
58  in single-strand breaks (as measured by the comet assay) and 7,8-dihydro-8-oxo-2'-deoxyguanosine (8-
59  DNA single-strand breaks (SSBs) (assayed by comet assays) and ATM autophosphorylation (at Ser-1981).
60 vity to etoposide-induced DNA damage (yH2AX, Comet assays) and growth inhibition was restored to leve
61 ense and damage, biomarkers of genotoxicity (comet assay), and behavioral biomarkers (feeding and bur
62 orescent microscopy, imaging flow cytometry, comet assay, and RT-qPCR.
63 -GFP reporter assays, qRT-PCR, Western blot, comet assays, and immunofluorescence.
64 ll viability experiments and the single cell Comet assay are consistent with NCP reactivity.
65 ngths are highlighted by the adoption of the comet assay as an in vivo test for genotoxicity in anima
66 duced DNA strand breaks in the hOGG1-coupled comet assay as well as 8-oxo-dGuo (as measured by LC-ESI
67 , VP-16-induced DSBs as monitored by neutral comet assay, as well as DNA damage signals (e.g. gamma-H
68 NA damage responses, as evidenced by neutral comet assay, as well as H2AX, Chk2 and p53 phosphorylati
69 but induced DNA damage, as determined by the comet assay, at all concentrations tested.
70 tivation, spontaneous DNA damage by alkaline comet assay, basal micronuclei levels, the number of lar
71 mical analysis of gammaH2AX focus formation, comet assay-based DNA strand break analysis, and measuri
72 essed on peripheral blood lymphocytes by the comet assay before and after exposing cells to a tobacco
73                                          The Comet assay (CA) is a sensitive/simple measure of genoto
74 he alkaline single cell gel electrophoresis (comet) assay can be combined with fluorescent in situ hy
75 CA) providing recommendations for describing comet assay conditions and results.
76 ted an intercalative mode of binding and the comet assay confirmed the DNA damaging effects.
77         Single cell gel electrophoresis (the comet assay), continues to gain popularity as a means of
78                                This modified comet assay could represent a stand-alone test or an adj
79    Induction of minority MOMP was tested via comet assay, CyQuant, annexin V, JC-1, cytochrome C subc
80                                              Comet assays demonstrated that DNA repair was delayed in
81         Additionally, a neutral and alkaline comet assay displayed a persistent level of intrinsic DN
82 eotides in the DNA, irresolvable DNA damage (comet assay), disruption of cellular bioenergetics (Seah
83 lated using published potencies based on the comet assay for Chinese hamster ovary cells (assesses th
84 0) and lethal dose (LD(50)), Ames tests, and Comet assays for in vitro DNA damage.
85 utagen-sensitivity assay) and DNA damage (by comet assay) for individuals in higher-risk subgroups am
86           Our high-throughput variant of the comet assay greatly increases the number of samples anal
87                                      Neutral comet assay has been available for two decades to evalua
88                  Various applications of the comet assay have been validated by research groups in ac
89  all three groups were used for the alkaline comet assay in the presence and absence of catalase.
90 onitored by formation of gamma-H2AX foci and comet assay, in an ATM (ataxia-telangiectasia mutated)-d
91                Sources of variability in the comet assay include variations in the protocol used to p
92                                          The comet assay indicated that SPD reduced 4NQO-induced DNA
93        gammaH2AX IR-induced foci (IRIFs) and comet assays indicated ineffective NHEJ DNA repair in p1
94                                              Comet assays indicated that strand breaks accumulated, a
95 ct of Usp and Imu1-3 was assessed by MTT and Comet assays, infection assays, caspase 3/7 activity, fl
96                                          The comet assay is a versatile method to detect nuclear DNA
97                                          The comet assay is a widely used test for the detection of D
98                                 The alkaline comet assay is an established method for detecting DNA s
99                DNA damage as measured by the comet assay is associated with the development of multip
100                                    Thus, DNA Comet Assay may be a practical quarantine control method
101 nt for the Minimum Information for Reporting Comet Assay (MIRCA) providing recommendations for descri
102 ts with pediatric SLE, including the neutral comet assay (NCA), colony survival assay (CSA), irradiat
103                                              Comet assay of epithelial cells obtained from scraping t
104 aser scanning cytometer and then perform the comet assay on the same cells.
105  absence of DNA damage, detectable by either comet assay or 53BP1 focus formation.
106 diol epoxide-induced Olive tail moments, the comet assay parameter for measuring DNA damage, were sig
107 oach is a significant advance on the current comet assay procedure.
108 ify the relevant conditions because detailed comet assay procedures are not always published.
109 pe, there are important modifications to the comet assay protocol to avoid the formation of additiona
110 her levels of DNA damage, as assessed by the comet assay, quantification of 8-oxoguanine, and poly(AD
111 determined with the use of an MTT test and a comet assay, respectively.
112  to MIRCA recommendations should ensure that comet assay results can be easily interpreted and indepe
113  correlated very well with rodent 1/TD50 and Comet assay results.
114                                          The comet assay revealed an increase in DNA damage by 95%.
115                                 In addition, COMET assays show that excess NEIL3 Zf-GRF repeat reduce
116                                          The comet assay showed that tiron's protective effect agains
117 lkaline single-cell gel electrophoresis (the comet assay), significantly decreased after consumption
118                    A modified version of the comet assay (single cell gel electrophoresis) was used t
119                           Using the alkaline comet assay (single-cell gel electrophoresis), we observ
120  including active RAS pulldown, reporter and Comet assays, small interfering RNA-mediated depletion o
121 trand DNA breaking has been observed through comet assay technique.
122          DNA damage was quantified using the Comet assay, telomere DNA length by Southern blotting an
123          Genotoxicity was measured using the Comet assay that can assess DNA damage in individual CHO
124 ion and genetic instability, as indicated by comet assays that showed increased numbers of DNA-strand
125 etermined the extent of DNA damage using the comet assay, the micronuclei assay, and the gamma-H2AX i
126      Compared with the microwell array based comet assay, this method can selectively capture and ana
127                              By coupling the comet assay to 8-oxoguanine glycosylase (hOGG1), which e
128 otocols that describe the application of the comet assay to a wide variety of cell types, species and
129                                    Using the comet assay to detect DNA damage, we find that reoxygena
130 n combination with DNA methylation sensitive comet assay to determine the effects of vitamin B12 or M
131 cancer and 153 healthy controls, we used the comet assay to investigate the roles of cell cycle check
132 mobilization and extracellular release using comet assays to measure DNA fragmentation, Qubit double-
133   Using the single cell gel electrophoresis (comet) assay to detect DNA strand breaks in murine hepat
134 g the single cell gel electrophoresis assay (comet assay) to assess DNA damage.
135 platform (a higher throughput more sensitive comet assay) to create the 'HepaCometChip', enabling the
136     We used single-cell gel electrophoresis (comet assay) to detect DNA-SSB in motor neurons.
137 et had lymphocyte DNA damage, as assessed by COMET assay, twice that found when they were fed the con
138   NO-dependent DNA damage as assessed by the comet assay was demonstrated during exposure of the thre
139                                          The comet assay was used to analyse the DNA damage.
140                                  The neutral comet assay was used to determine whether initial drug-i
141                       The specificity of the comet assay was validated by coupling it to human 8-oxo-
142 on of FPG-sensitive sites, measured with the comet assay, was 0.34 per 10(6) guanines.
143                                        Using comet assays, we demonstrate that LT induces overt DNA d
144 tion of the single-cell gel electrophoresis (Comet) assay, we have measured formation and repair of D
145  foci; DSB levels were determined by neutral comet assay; western blotting was used to evaluate prote
146 maged rapidly, within 12 h, as revealed by a comet assay, which detects broken DNA, and by staining f
147 idual cells by alkaline gel electrophoresis (comet assay), with and without the addition of the repai
148 -induced DNA strand breaks using an alkaline comet assay (+/-z-VAD-fmk cotreatment) and by levels of

 
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