ライフサイエンスコーパス: common carotid

1 ia produced by transient 20 min occlusion of common carotid and vertebral arteries in rats caused a d

2 tion of the combined thickness of the distal common carotid arterial far wall intima-media complex (c

3 end-tidal CO2 values were measured (mm Hg); common carotid arterial flow also was measured (mL/min).

4 ced by hypovolemic hypotension and bilateral common carotid arterial occlusion, which was followed by

5 Common carotid arteries (CCA) excised from 3-month, 8-mo

6 ned with a 30-min occlusion of the bilateral common carotid arteries (CCA) in male rats.

7 in gerbils by a 5-min occlusion of bilateral common carotid arteries (CCA).

8 nced magnetic resonance (MR) imaging in both common carotid arteries (CCAs) and in one internal carot

9 manent coil filter directly placed into both common carotid arteries (CCAs) was designed to capture e

10 right middle cerebral artery (MCA) and both common carotid arteries (CCAs).

11 ) compared with sham and contralateral right common carotid arteries (RCs).

12 ernal iliac arteries but not the brachial or common carotid arteries and not correlated significantly

13 sed to assess the thickness of the bilateral common carotid arteries and the mean maximal IMT of 12 s

14 -Evans rats by a bilateral occlusion of both common carotid arteries and the right middle cerebral ar

15 focal ischemia was induced by occluding both common carotid arteries and the right middle cerebral ar

16 ements of the intima-media thickness of both common carotid arteries blinded from the randomization a

17 Common carotid arteries decreased in size after ipsilate

18 was induced in gerbils by occlusion of both common carotid arteries for 5 min.

19 and 15 days after bilateral occlusion of the common carotid arteries for 5 min.

20 idative stress was induced by occluding both common carotid arteries for 90 min, followed by reperfus

21 Transient occlusions of common carotid arteries for periods between 20 and 40 mi

22 t B-mode ultrasonography of the internal and common carotid arteries in 1994-1996 and again in 1998-2

23 s definition and between-group comparison of common carotid arteries intima-media thickness change.

24 The change in common carotid arteries intima-media thickness was -2.69

25 Intima-media thickness of the common carotid arteries is a marker of atherosclerosis a

26 cerebral ischemia induced by middle cerebral/common carotid arteries occlusion (MCA/CCAo) induced up-

27 The common carotid arteries of 17 wild-type (WT) mice and 8

28 ric chloride injury of the midportion of the common carotid arteries of apoE(-/-) mice (n=22) induced

29 perimental BVGs were then implanted into the common carotid arteries of nude rats.

30 e in vivo efficacy of the 12-LO Rz, the left common carotid arteries of rats were injured with a ball

31 m donor WT mice that had been anastamosed to common carotid arteries of recipient WT mice (WT:WT) exh

32 ssion of intima-media thickness (IMT) of the common carotid arteries over 18 months was determined ul

33 The intima-media thickness (IMT) of the common carotid arteries was measured ultrasonographicall

34 Images of the common carotid arteries were acquired from three indepen

35 The levels of NFATc1 in injured right common carotid arteries were increased at 72 h, 1 week,

36 ve rats, the middle cerebral artery and both common carotid arteries were occluded for 4.9+/-0.13 h (

37 The common carotid arteries were surgically exposed, and ult

38 l thickness (IMT) and plaque height from the common carotid arteries were used as noninvasive markers

39 nd 14 days after burn and endothelium-intact common carotid arteries were used for ex vivo force/rela

40 ene (eNOS) did not remodel their ipsilateral common carotid arteries whereas wild-type mice did.

41 p115 RhoGEF tyrosine phosphorylation in rat common carotid arteries, and siRNA-mediated down-regulat

42 ivascular application to balloon-injured rat common carotid arteries, halofuginone versus vehicle (n=

43 ty liver and intimal medial thickness of the common carotid arteries, subcutaneous adipose tissue, an

44 eased in the left and increased in the right common carotid arteries.

45 an posterior-wall CIMT of the left and right common carotid arteries.

46 y compared with its levels in uninjured left common carotid arteries.

47 ared by injection of epoxy resin through the common carotid arteries.

48 of the right middle cerebral artery and both common carotid arteries.

49 0.001) but not intima-media thickness of the common carotid artery (0.0%, P=0.99).

50 markers in adults: intima-media thickness of common carotid artery ([Formula: see text]), carotid pla

51 nd increased intimal-medial thickness of the common carotid artery (CCA IMT), a purported index of at

52 individuals aged 30 to 70 years, we measured common carotid artery (CCA) and carotid bulb IMT, and al

53 ndent association of whole-grain intake with common carotid artery (CCA) and internal carotid artery

54 maximal intima-media thickness (IMT) of the common carotid artery (CCA) and the internal carotid art

55 Common carotid artery (CCA) balloon angioplasty injury w

56 We assessed internal (ICA) and common carotid artery (CCA) haemodynamics (indicative of

57 No LOD scores >2.0 were observed for common carotid artery (CCA) IMT.

58 Obesity has been associated with increased common carotid artery (CCA) intima-media thickness (IMT)

59 Bilateral common carotid artery (CCA) ligation (n = 24) or sham-op

60 Mice underwent unilateral common carotid artery (CCA) ligation.

61 e application of a microvascular clip to the common carotid artery (CCA) might decrease variability o

62 rebral artery (MCA) with bilateral temporary common carotid artery (CCA) occlusion for 90 min.

63 botic MCA occlusion coupled with ipsilateral common carotid artery (CCA) occlusion.

64 1.25 m/sec, internal carotid artery (ICA) to common carotid artery (CCA) peak-systolic velocity ratio

65 sized that variant geometric patterns of the common carotid artery (CCA) predict the incidence of car

66 The right common carotid artery (CCA) was balloon-injured in 21 Ne

67 sed Doppler ultrasound waveforms of the ECA, common carotid artery (CCA), and internal carotid artery

68 Intima-media thickness (IMT) measures of the common carotid artery (CCA), internal carotid artery (IC

69 ltrasonography and wave form analysis of the common carotid artery (CCA), with its conversion to the

70 temporary (1 h) occlusion of the ipsilateral common carotid artery (CCA).

71 hyperplasia caused by balloon injury to the common carotid artery (CCA).

72 media thickness of the right far wall of the common carotid artery (CCA-IMT) and carotid artery bifur

73 Intima-media thickness of the common carotid artery (CCA-IMT), pulse wave velocity (PW

74 oupled with a temporary 1-h occlusion of the common carotid artery (CCAo).

75 particles, and intima-media thickness of the common carotid artery (CIMT) in elderly men residing in

76 arotid artery reduced blood flow in the left common carotid artery (LC) compared with sham and contra

77 ht common carotid artery (RCCA) and the left common carotid artery (LCCA), and fasting lipid profile

78 on, sex, and systolic blood pressure for the common carotid artery (r2=0.17); age, race, and systolic

79 ), intima-media thickness (IMT) of the right common carotid artery (RCCA) and the left common carotid

80 he PSV in the ICA to that in the ipsilateral common carotid artery (VICA/VCCA) were compared with the

81 ks and underwent wire denudation of the left common carotid artery after 1 week of feeding.

82 e report that inward remodeling of the mouse common carotid artery after ligation of the ipsilateral

83 s compared with its levels in uninjured left common carotid artery and both CsA and GFPVIVIT negated

84 than that in lean and obese subjects for the common carotid artery and bulb.

85 balloon withdrawal injury (BWI) of the left common carotid artery and continued on this drug regimen

86 beginning before balloon injury of the right common carotid artery and continuing for 14 days afterwa

87 hind limb CTA were anastomosed to recipient common carotid artery and external jugular vein without

88 Intima-media thickness of the walls of the common carotid artery and internal carotid artery may ad

89 ischemia by permanent ligation of the right common carotid artery and temporary occlusion of the rig

90 ng, and the distal descending aorta, and the common carotid artery and the brachial artery were asses

91 sured the mean intima-media thickness of the common carotid artery and the maximum intima-media thick

92 from the proximal and distal portion of the common carotid artery and the mean value was taken.

93 We bilaterally and transiently occluded the common carotid artery and then examined the molecular an

94 us patients (nine men, two women) undergoing common carotid artery angiography.

95 rnal carotid arteries were ligated, reducing common carotid artery blood flow from 8.0+/-0.6 to 0.5+/

96 lic strain rate were regressed on IMT of the common carotid artery defined by ultrasound, with adjust

97 Sprague-Dawley rats were subjected to left common carotid artery denudation, after which arteries w

98 ll measures of C-IMT and the interadventitia common carotid artery diameter (ICCAD) were associated w

99 The left common carotid artery divided into the internal carotid,

100 Gy (range 0.1-24.0 Gy) and average bilateral common carotid artery dose was 21.5 Gy (range 0.6-38.1 G

101 ross-sectional association of education with common carotid artery elasticity.

102 n intima-media thickness of the right distal common carotid artery far wall in computer image process

103 mode ultrasound quantification of the distal common carotid artery far wall intima-media thickness (I

104 e, the annual progression rate of the distal common carotid artery far wall intima-media thickness de

105 rial end point was the rate of change in the common carotid artery far-wall intima-media thickness (I

106 schemia was induced by ligation of the right common carotid artery followed by 2 hrs of hypoxia (8% o

107 to formation of an occlusive thrombus in the common carotid artery following experimentally induced e

108 nt mice a Western diet and occluded the left common carotid artery for 2 days.

109 which was induced by occluding the bilateral common carotid artery for 5 min.

110 cles by Sir Astley Cooper on ligature of the common carotid artery for aneurysm, John Abernethy on mi

111 th with PHIV, virally suppressed by ART, the common carotid artery IMT did not progress over 2 years.

112 d I/M ratio seem preferable to estimation of common carotid artery IMT in imaging cardiovascular risk

113 morphisms was undertaken with 6 internal and common carotid artery IMT phenotypes using an additive m

114 trasound (22 MHz) to estimate the individual common carotid artery IMTs in 55 women at PE diagnosis a

115 2 expression was also increased in the right common carotid artery in a time-dependent manner after b

116 for the internal carotid artery than for the common carotid artery in all ethnic groups.

117 earing (n = 4) coils were implanted into the common carotid artery in nude rats.

118 Compliance was significantly lower in the common carotid artery in symptomatic and asymptomatic wo

119 Velocity in the common carotid artery increased with distance (toward th

120 Ligation of the murine common carotid artery induces a reproducible remodeling

121 Left common carotid artery injury was induced with a guidewir

122 After common carotid artery injury, knockdown of Nox4 by adeno

123 CA, as well as neointima formation, after ZO common carotid artery injury.

124 +1 serving; 200-250 g/d) intakes slows 12-mo common carotid artery intima media thickness (CCA IMT) p

125 integrity, and T-cell activation, as well as common carotid artery intima media thickness and pulse w

126 associated with a significantly thicker mean common carotid artery intima, thinner media, and higher

127 Outcomes included changes in right common carotid artery intima-media thickness (CCA-IMT) a

128 Data on common carotid artery intima-media thickness (CCA-IMT) w

129 We investigated the association of common carotid artery intima-media thickness (CCA-IMT) w

130 (LAMP-LD) and evaluated the association with common carotid artery intima-media thickness (cCIMT) usi

131 Coronary artery calcification (CAC) and common carotid artery intima-media thickness (CIMT) are

132 herosclerosis, as measured by progression of common carotid artery intima-media thickness (cIMT), in

133 Mean common carotid artery intima-media thickness (IMT) and p

134 Mean common carotid artery intima-media thickness (IMT) and p

135 Ultrasound assessment of the common carotid artery intima-media thickness (IMT) durin

136 tween years since bilateral oophorectomy and common carotid artery intima-media thickness (IMT).

137 progression of atherosclerosis, measured as common carotid artery intima-media thickness (IMT).

138 Coprimary outcomes included common carotid artery intima-media thickness and coronar

139 Common carotid artery intima-media thickness has been as

140 cation is strongly inversely associated with common carotid artery intima-media thickness in the Athe

141 Europeans (fully-adjusted P=0.004) and with common carotid artery intima-media thickness in the Fram

142 difference in the rates of change in either common carotid artery intima-media thickness or coronary

143 Common carotid artery intima-media thickness was measure

144 d progression of coronary artery calcium and common carotid artery intima-media thickness.

145 ography, and B-mode ultrasonography measured common carotid artery intima-medial thickness (IMT) in 5

146 naive (inversely) cells were associated with common carotid artery intimal media thickness (CC IMT) i

147 s progression of atherosclerosis measured by common carotid artery intimal medial thickness (IMT).

148 sion of intima-media thickness in the distal common carotid artery is a useful surrogate end point fo

149 Trifurcation of the common carotid artery is an unusual variation.

150 of arterial remodeling in which flow in the common carotid artery is interrupted by ligation of the

151 the common gamma chain (FcR gamma) had their common carotid artery ligated.

152 y 4 (P4) SD rats were subjected to bilateral common carotid artery ligation (BCAL), followed by 8% ox

153 -ischemia (HI) damage in adult mice by right common carotid artery ligation followed by hypoxia.

154 gments the reduction in blood flow caused by common carotid artery ligation in wild-type mice.

155 preconditioning ischemic insult (unilateral common carotid artery ligation) to 12- to 14-week-old mi

156 natal day 7 rat pups were subjected to right common carotid artery ligation, followed by 8% oxygen in

157 (P4) SD rat pups were subjected to bilateral common carotid artery ligation, followed by exposure to

158 Regions of interest were drawn within the common carotid artery lumen and immediately adjacent to

159 MRA) was performed to longitudinally monitor common carotid artery luminal areas in living mice pre-

160 bal cerebral ischaemia in mouse is bilateral common carotid artery occlusion (BCCAo) and the neuropat

161 Bilateral common carotid artery occlusion (BCCAO) in a patient wit

162 poperfusion and VaD was induced by bilateral common carotid artery occlusion (BCCAO) in adult male Sp

163 Chronic bilateral common carotid artery occlusion (BCCAO) in adult rats ha

164 Permanent bilateral common carotid artery occlusion (BCCAO) was used as a mo

165 n long-term experimental permanent bilateral common carotid artery occlusion (BCCAO).

166 ischemia was induced by 10 min of bilateral common carotid artery occlusion and hypotension in rats.

167 re in adult mice that consists of unilateral common carotid artery occlusion and hypoxia with tightly

168 y-six fasted Wistar rats underwent bilateral common carotid artery occlusion and severe hypotension (

169 tized gerbils by a 5-min period of bilateral common carotid artery occlusion followed by reperfusion

170 terial pressure of 30-35 mm Hg and bilateral common carotid artery occlusion for 8 min).

171 2Y12R-mediated cerebrovascular adaptation to common carotid artery occlusion resulting in hypoperfusi

172 re subjected to global ischemia by bilateral common carotid artery occlusion under controlled ventila

173 bal cerebral ischemia (tGCI) using bilateral common carotid artery occlusion with hypotension.

174 and Fpr2/3(-/-)) were subjected to bilateral common carotid artery occlusion, followed by reperfusion

175 bal ischemic injury was induced by bilateral common carotid artery occlusion, whereas severe focal st

176 at (HF) diet and modeled VCID via unilateral common carotid artery occlusion.

177 ser-Doppler probe, combined with ipsilateral common carotid artery occlusion.

178 ral global ischemia was induced by bilateral common carotid artery occlusion.

179 ined in anaesthetized rats with a unilateral common carotid artery occlusion.

180 3 h of middle cerebral artery and bilateral common carotid artery occlusion.

181 pe (Wt) littermates after 3 min of bilateral common carotid artery occlusion.

182 anular zone 1-2 weeks after 10 min bilateral common carotid artery occlusions.

183 rterial remodeling in which flow in the left common carotid artery of FVB mice was interrupted by lig

184 The stents were deployed into the common carotid artery of rats and their impact on vascul

185 The right common carotid artery of wild-type and Cav-1 -/- mice wa

186 We show here, using an intact porcine common carotid artery perfusion culture model, that nico

187 verlapping associations between internal and common carotid artery phenotypes at P<5.0e(-6).

188 Surgical cutdown for access to the common carotid artery provides a more direct route for c

189 Endovascular therapy in patients with common carotid artery pseudoaneurysms offers a reliable

190 stenosis; two additional parameters, ICA-to-common carotid artery PSV ratio and ICA end-diastolic ve

191 rotid for 14 days to lower blood flow in the common carotid artery reduced the lumen diameter of caro

192 UDP end labeling (TUNEL) in the injured rat common carotid artery show that TUNEL-positive cells in

193 38 (95% CI, -0.0064 to -0.0013) mm/y for the common carotid artery sites (P<.001), -0.0040 (95% CI, -

194 mean CIMT for the rosuvastatin group for the common carotid artery sites was 0.0004 (95% CI, -0.0011

195 carotid artery sites and in mean CIMT of the common carotid artery sites.

196 Here we used a bilateral common carotid artery stenosis (BCAS) mouse model of VaD

197 Here, we create bilateral common carotid artery stenosis in mice, which effectivel

198 le C57Bl/6J mice were subjected to bilateral common carotid artery stenosis or a sham operation and f

199 to receive vehicle or drug immediately after common carotid artery thrombosis (CCAT).

200 We report a case of left common carotid artery trifurcation in a 74-year-old man.

201 aphy and CT angiography findings of the left common carotid artery trifurcation were described with i

202 emia as follows: permanent ligation of right common carotid artery under halothane anesthesia, 2-h re

203 s embolic stroke propensity through the left common carotid artery using an idealized aortic arch mod

204 The distance from the bifurcation at which common carotid artery velocity is measured should be sta

205 se in the mean intima-media thickness of the common carotid artery was 1.13 (95% CI, 1.02 to 1.24), w

206 Intima-media thickness of the left common carotid artery was associated with cognitive impa

207 (4) vehicle, and balloon injury of the right common carotid artery was carried out.

208 ntricle for I.C.V. injection of drugs, and a common carotid artery was catheterized to measure mean a

209 Preintrusive atherosclerosis in the common carotid artery was evaluated every 6 months with

210 The left common carotid artery was ligated just proximal to the c

211 Bypass grafting of the jugular vein to the common carotid artery was performed 2 weeks after starti

212 The far wall of the common carotid artery was significantly more echogenic t

213 its were anesthetized, and the right or left common carotid artery was surgically exposed.

214 y stenosis and intima-media thickness of the common carotid artery were assessed by using duplex ultr

215 trasonic flow probe and meter applied to the common carotid artery while cerebral blood flow was meas

216 Left common carotid artery wire injury in apolipoprotein E-/-

217 ayed in cultured endothelial cells and mouse common carotid artery with or without OGD treatment.

218 ypoxia, induced by permanent ligation of the common carotid artery with transient hypoxia, resulted i

219 nd and arterial applanation tonometry on the common carotid artery) was lower (P:<0.05) in middle-age

220 ression of the intima-media thickness of the common carotid artery, 0.032 vs. 0.046 mm; P=0.01; and p

221 French catheter sheath was inserted into the common carotid artery, and a 0.035-cm (0.014-in) guidewi

222 AChT-immunoreactive cells in segments of the common carotid artery, aorta, and pulmonary artery appea

223 ial chemoreceptors have been isolated to the common carotid artery, aorta, and pulmonary artery of tu

224 of the intima-medial thickness (IMT) of the common carotid artery, as an indicator of atherosclerosi

225 t cases, paraganglioma is located around the common carotid artery, but may also be located within th

226 arotid sites; changes in maximum CIMT of the common carotid artery, carotid bulb, and internal caroti

227 Carotid IMT was measured at 3 locations (common carotid artery, carotid bulb, internal carotid ar

228 nternal carotid artery is anastomosed to the common carotid artery, this obviates the need for patch

229 f endovascular balloon occlusion of the left common carotid artery, which created an aneurysm at the

230 tal grey matter and B-mode ultrasound of the common carotid artery.

231 low-disturbed left and the undisturbed right common carotid artery.

232 trasonographically in the distal 1 cm of the common carotid artery.

233 ents a distant athero-protected UF site, the common carotid artery.

234 animals underwent balloon angioplasty of the common carotid artery.

235 were subjected to balloon angioplasty of the common carotid artery.

236 monitored with a cannula placed in the left common carotid artery.

237 cipation in response to ligation of the left common carotid artery.

238 he external jugular vein is connected to the common carotid artery.

239 al assessments of the far wall of the distal common carotid artery.

240 otein E knockout mice subjected to injury of common carotid artery.

241 nt dye, after balloon angioplasty of the rat common carotid artery.

242 rmed after endothelial denudation of the rat common carotid artery.

243 gonads underwent balloon injury to the right common carotid artery.

244 ere subjected to balloon injury of the right common carotid artery.

245 n injection followed by ligation of the left common carotid artery.

246 rrected velocity was measured with US in the common carotid artery.

247 order via a diagnostic catheter located in a common carotid artery.

248 bility of plasma lipids and stiffness of the common carotid artery.

249 as elicited with a hydraulic occluder on the common carotid artery.

250 Carotid artery IMT was measured in the left common carotid artery.

251 nonocclusive silastic collar around the left common carotid artery.

252 The maximum internal and mean common carotid-artery intima-media thicknesses both pred

253 Segment-specific IMT measurements of common carotid, bifurcation, and internal carotid arteri

254 on pressure (16 +/- 3 to 18 +/- 3; p = .07); common carotid blood flow (48 +/- 5 to 82 +/- 5 mL/min;

255 caused a approximately 90% decrease in left common carotid blood flow and a approximately 50% increa

256 Left common carotid blood flow immediately decreased by appro

257 statistically significantly associated with common carotid, brachial, femoral arterial parameters (l

258 Common carotid (CC) and internal carotid (IC) regions-of

259 mately 50% increase in right (contralateral) common carotid flow.

260 A simple, defined common carotid focal stenosis yields reproducible murine

261 Common carotid IA diameter was greater in CAD cases than

262 on score = 1): P for trend = 0.023] and with common carotid IMT [>or=1.0 mm: OR (95% CI) for quartile

263 oronary artery calcification in men and with common carotid IMT and ABI in women (all P<0.05) after a

264 orted a protective effect of the A allele on common carotid IMT in women only (women: beta=-0.0047, P

265 This variant showed stronger effects on common carotid IMT in women, raising questions about the

266 Common carotid IMT progression from EDIC years 1 to 6 wa

267 ucleotide polymorphisms for association with common carotid IMT was undertaken in 5 independent Europ

268 Ultrasound measures of common carotid IMT were collected at this time and 7 yea

269 Internal and common carotid IMT were measured 8 and 14 years later by

270 There was also no significant association of common carotid IMT with premature or any parental CHD.

271 ared with quartile 1 = 1.33 (0.99, 1.79); ln(common carotid IMT): P for trend = 0.006].

272 Walking pace was associated favorably with common carotid IMT, ABI, and coronary artery calcificati

273 These associations were attenuated and, for common carotid IMT, no longer significant when lipids, h

274 y traditional and HIV-specific correlates of common carotid intima media thickness (CCIMT).

275 Common carotid intima media thickness (cIMT) and plaque

276 tified three genomic regions associated with common carotid intima media thickness and two different

277 imary end point was progression of mean-mean common carotid intima-media thickening (CIMT) measured b

278 osclerosis was determined by measurements of common carotid intima-media thickness (cCIMT, >80th perc

279 The primary end point was the change in common carotid intima-media thickness (CIMT) after 1 yea

280 The evidence that measurement of the common carotid intima-media thickness (CIMT) improves th

281 ssociation of coronary artery calcium (CAC), common carotid intima-media thickness (CIMT), aortic dis

282 onary artery calcification, and internal and common carotid intima-media thickness (IMT) were measure

283 ence in the change from baseline in the mean common carotid intima-media thickness after 14 months.

284 uted tomography and 52 869 participants with common carotid intima-media thickness measured by ultras

285 , many subclinical disease measures, such as common carotid intima-media thickness, ankle-arm index,

286 ith lower urinary albumin:creatinine ratios, common carotid intima-media thickness, measures of adipo

287 n the amount of subclinical atherosclerosis (common carotid intimal-media thickness) and 2) associate

288 es of subclinical disease (coronary calcium, common carotid intimal-medial thickness, and ankle-brach

289 Common carotid lumen diameter was marginally larger in i

290 sified events and non-events better than the common carotid mean IMT (net reclassification improvemen

291 protein were infused to the lumen of normal common carotids of CD-1 and C57BL/6 mice and atheroscler

292 Patients were excluded if the left common carotid or brachiocephalic artery had greater tha

293 nt surgical creation of stenoses at the left common carotid, right renal, and left external iliac art

294 The creation of an outflow common carotid stenosis generates clinically relevant (p

295 utflow carotid branches, creating an outflow common carotid stenosis, and constructing a midgraft ste

296 he flow waveform amplitudes were lowest with common carotid stenosis.

297 % via carotid branch ligation and by 80% via common carotid stenosis.

298 ted as the average of 3 segments: the distal common carotid, the carotid artery bifurcation, and the

299 ion tissue response, or by ligating the left common carotid to form an organized thrombus.

300 focal thickening of >1.3 mm at the level of common carotid, were evaluated using ultrasonography.