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1 trasonographically in the distal 1 cm of the common carotid artery.
2 ents a distant athero-protected UF site, the common carotid artery.
3 animals underwent balloon angioplasty of the common carotid artery.
4 were subjected to balloon angioplasty of the common carotid artery.
5 monitored with a cannula placed in the left common carotid artery.
6 cipation in response to ligation of the left common carotid artery.
7 he external jugular vein is connected to the common carotid artery.
8 al assessments of the far wall of the distal common carotid artery.
9 otein E knockout mice subjected to injury of common carotid artery.
10 nt dye, after balloon angioplasty of the rat common carotid artery.
11 rmed after endothelial denudation of the rat common carotid artery.
12 gonads underwent balloon injury to the right common carotid artery.
13 ere subjected to balloon injury of the right common carotid artery.
14 rrected velocity was measured with US in the common carotid artery.
15 0 after carotid artery stenting for the left common carotid artery.
16 separate pseudoaneurysms involving the left common carotid artery.
17 n injection followed by ligation of the left common carotid artery.
18 order via a diagnostic catheter located in a common carotid artery.
19 bility of plasma lipids and stiffness of the common carotid artery.
20 as elicited with a hydraulic occluder on the common carotid artery.
21 Carotid artery IMT was measured in the left common carotid artery.
22 nonocclusive silastic collar around the left common carotid artery.
23 tal grey matter and B-mode ultrasound of the common carotid artery.
24 low-disturbed left and the undisturbed right common carotid artery.
25 eased in the left and increased in the right common carotid arteries.
26 an posterior-wall CIMT of the left and right common carotid arteries.
27 y compared with its levels in uninjured left common carotid arteries.
28 ared by injection of epoxy resin through the common carotid arteries.
29 of the right middle cerebral artery and both common carotid arteries.
31 ression of the intima-media thickness of the common carotid artery, 0.032 vs. 0.046 mm; P=0.01; and p
33 e report that inward remodeling of the mouse common carotid artery after ligation of the ipsilateral
34 ernal iliac arteries but not the brachial or common carotid arteries and not correlated significantly
35 sed to assess the thickness of the bilateral common carotid arteries and the mean maximal IMT of 12 s
36 -Evans rats by a bilateral occlusion of both common carotid arteries and the right middle cerebral ar
37 focal ischemia was induced by occluding both common carotid arteries and the right middle cerebral ar
38 s compared with its levels in uninjured left common carotid artery and both CsA and GFPVIVIT negated
40 balloon withdrawal injury (BWI) of the left common carotid artery and continued on this drug regimen
41 beginning before balloon injury of the right common carotid artery and continuing for 14 days afterwa
42 hind limb CTA were anastomosed to recipient common carotid artery and external jugular vein without
43 Intima-media thickness of the walls of the common carotid artery and internal carotid artery may ad
44 ischemia by permanent ligation of the right common carotid artery and temporary occlusion of the rig
45 ng, and the distal descending aorta, and the common carotid artery and the brachial artery were asses
46 sured the mean intima-media thickness of the common carotid artery and the maximum intima-media thick
48 We bilaterally and transiently occluded the common carotid artery and then examined the molecular an
49 p115 RhoGEF tyrosine phosphorylation in rat common carotid arteries, and siRNA-mediated down-regulat
50 French catheter sheath was inserted into the common carotid artery, and a 0.035-cm (0.014-in) guidewi
52 AChT-immunoreactive cells in segments of the common carotid artery, aorta, and pulmonary artery appea
53 ial chemoreceptors have been isolated to the common carotid artery, aorta, and pulmonary artery of tu
54 knowledge, the first case of isolated right common carotid artery arising from the right pulmonary a
55 of the intima-medial thickness (IMT) of the common carotid artery, as an indicator of atherosclerosi
56 ements of the intima-media thickness of both common carotid arteries blinded from the randomization a
57 rnal carotid arteries were ligated, reducing common carotid artery blood flow from 8.0+/-0.6 to 0.5+/
58 t cases, paraganglioma is located around the common carotid artery, but may also be located within th
59 arotid sites; changes in maximum CIMT of the common carotid artery, carotid bulb, and internal caroti
60 Carotid IMT was measured at 3 locations (common carotid artery, carotid bulb, internal carotid ar
64 nd increased intimal-medial thickness of the common carotid artery (CCA IMT), a purported index of at
65 individuals aged 30 to 70 years, we measured common carotid artery (CCA) and carotid bulb IMT, and al
66 ndent association of whole-grain intake with common carotid artery (CCA) and internal carotid artery
67 maximal intima-media thickness (IMT) of the common carotid artery (CCA) and the internal carotid art
71 Obesity has been associated with increased common carotid artery (CCA) intima-media thickness (IMT)
74 e application of a microvascular clip to the common carotid artery (CCA) might decrease variability o
77 1.25 m/sec, internal carotid artery (ICA) to common carotid artery (CCA) peak-systolic velocity ratio
78 sized that variant geometric patterns of the common carotid artery (CCA) predict the incidence of car
80 sed Doppler ultrasound waveforms of the ECA, common carotid artery (CCA), and internal carotid artery
81 Intima-media thickness (IMT) measures of the common carotid artery (CCA), internal carotid artery (IC
82 ltrasonography and wave form analysis of the common carotid artery (CCA), with its conversion to the
85 media thickness of the right far wall of the common carotid artery (CCA-IMT) and carotid artery bifur
88 nced magnetic resonance (MR) imaging in both common carotid arteries (CCAs) and in one internal carot
89 manent coil filter directly placed into both common carotid arteries (CCAs) was designed to capture e
91 particles, and intima-media thickness of the common carotid artery (CIMT) in elderly men residing in
93 lic strain rate were regressed on IMT of the common carotid artery defined by ultrasound, with adjust
94 Sprague-Dawley rats were subjected to left common carotid artery denudation, after which arteries w
95 ll measures of C-IMT and the interadventitia common carotid artery diameter (ICCAD) were associated w
97 Gy (range 0.1-24.0 Gy) and average bilateral common carotid artery dose was 21.5 Gy (range 0.6-38.1 G
99 n intima-media thickness of the right distal common carotid artery far wall in computer image process
100 mode ultrasound quantification of the distal common carotid artery far wall intima-media thickness (I
101 e, the annual progression rate of the distal common carotid artery far wall intima-media thickness de
102 rial end point was the rate of change in the common carotid artery far-wall intima-media thickness (I
103 schemia was induced by ligation of the right common carotid artery followed by 2 hrs of hypoxia (8% o
104 to formation of an occlusive thrombus in the common carotid artery following experimentally induced e
107 idative stress was induced by occluding both common carotid arteries for 90 min, followed by reperfus
111 cles by Sir Astley Cooper on ligature of the common carotid artery for aneurysm, John Abernethy on mi
112 markers in adults: intima-media thickness of common carotid artery ([Formula: see text]), carotid pla
113 ivascular application to balloon-injured rat common carotid arteries, halofuginone versus vehicle (n=
114 th with PHIV, virally suppressed by ART, the common carotid artery IMT did not progress over 2 years.
115 d I/M ratio seem preferable to estimation of common carotid artery IMT in imaging cardiovascular risk
116 morphisms was undertaken with 6 internal and common carotid artery IMT phenotypes using an additive m
118 trasound (22 MHz) to estimate the individual common carotid artery IMTs in 55 women at PE diagnosis a
119 t B-mode ultrasonography of the internal and common carotid arteries in 1994-1996 and again in 1998-2
120 2 expression was also increased in the right common carotid artery in a time-dependent manner after b
123 Compliance was significantly lower in the common carotid artery in symptomatic and asymptomatic wo
129 s definition and between-group comparison of common carotid arteries intima-media thickness change.
131 +1 serving; 200-250 g/d) intakes slows 12-mo common carotid artery intima media thickness (CCA IMT) p
132 integrity, and T-cell activation, as well as common carotid artery intima media thickness and pulse w
133 associated with a significantly thicker mean common carotid artery intima, thinner media, and higher
137 (LAMP-LD) and evaluated the association with common carotid artery intima-media thickness (cCIMT) usi
139 herosclerosis, as measured by progression of common carotid artery intima-media thickness (cIMT), in
144 tween years since bilateral oophorectomy and common carotid artery intima-media thickness (IMT).
148 cation is strongly inversely associated with common carotid artery intima-media thickness in the Athe
149 Europeans (fully-adjusted P=0.004) and with common carotid artery intima-media thickness in the Fram
150 difference in the rates of change in either common carotid artery intima-media thickness or coronary
153 ography, and B-mode ultrasonography measured common carotid artery intima-medial thickness (IMT) in 5
155 naive (inversely) cells were associated with common carotid artery intimal media thickness (CC IMT) i
156 s progression of atherosclerosis measured by common carotid artery intimal medial thickness (IMT).
158 sion of intima-media thickness in the distal common carotid artery is a useful surrogate end point fo
160 of arterial remodeling in which flow in the common carotid artery is interrupted by ligation of the
161 arotid artery reduced blood flow in the left common carotid artery (LC) compared with sham and contra
162 ht common carotid artery (RCCA) and the left common carotid artery (LCCA), and fasting lipid profile
164 y 4 (P4) SD rats were subjected to bilateral common carotid artery ligation (BCAL), followed by 8% ox
165 -ischemia (HI) damage in adult mice by right common carotid artery ligation followed by hypoxia.
167 preconditioning ischemic insult (unilateral common carotid artery ligation) to 12- to 14-week-old mi
168 natal day 7 rat pups were subjected to right common carotid artery ligation, followed by 8% oxygen in
169 (P4) SD rat pups were subjected to bilateral common carotid artery ligation, followed by exposure to
170 Regions of interest were drawn within the common carotid artery lumen and immediately adjacent to
171 MRA) was performed to longitudinally monitor common carotid artery luminal areas in living mice pre-
172 cerebral ischemia induced by middle cerebral/common carotid arteries occlusion (MCA/CCAo) induced up-
173 bal cerebral ischaemia in mouse is bilateral common carotid artery occlusion (BCCAo) and the neuropat
175 poperfusion and VaD was induced by bilateral common carotid artery occlusion (BCCAO) in adult male Sp
179 ischemia was induced by 10 min of bilateral common carotid artery occlusion and hypotension in rats.
180 re in adult mice that consists of unilateral common carotid artery occlusion and hypoxia with tightly
181 y-six fasted Wistar rats underwent bilateral common carotid artery occlusion and severe hypotension (
182 tized gerbils by a 5-min period of bilateral common carotid artery occlusion followed by reperfusion
184 2Y12R-mediated cerebrovascular adaptation to common carotid artery occlusion resulting in hypoperfusi
185 re subjected to global ischemia by bilateral common carotid artery occlusion under controlled ventila
187 and Fpr2/3(-/-)) were subjected to bilateral common carotid artery occlusion, followed by reperfusion
188 bal ischemic injury was induced by bilateral common carotid artery occlusion, whereas severe focal st
197 ric chloride injury of the midportion of the common carotid arteries of apoE(-/-) mice (n=22) induced
199 e in vivo efficacy of the 12-LO Rz, the left common carotid arteries of rats were injured with a ball
200 m donor WT mice that had been anastamosed to common carotid arteries of recipient WT mice (WT:WT) exh
201 rterial remodeling in which flow in the left common carotid artery of FVB mice was interrupted by lig
204 ssion of intima-media thickness (IMT) of the common carotid arteries over 18 months was determined ul
209 stenosis; two additional parameters, ICA-to-common carotid artery PSV ratio and ICA end-diastolic ve
210 l complication rate in the IJV group was 3% (common carotid artery puncture-4 cases, perivascular hem
211 on, sex, and systolic blood pressure for the common carotid artery (r2=0.17); age, race, and systolic
212 ), intima-media thickness (IMT) of the right common carotid artery (RCCA) and the left common carotid
214 rotid for 14 days to lower blood flow in the common carotid artery reduced the lumen diameter of caro
216 UDP end labeling (TUNEL) in the injured rat common carotid artery show that TUNEL-positive cells in
217 38 (95% CI, -0.0064 to -0.0013) mm/y for the common carotid artery sites (P<.001), -0.0040 (95% CI, -
218 mean CIMT for the rosuvastatin group for the common carotid artery sites was 0.0004 (95% CI, -0.0011
221 in 2 C57BL/6J stroke mouse models (bilateral common carotid artery stenosis [BCCAS] and middle cerebr
223 le C57Bl/6J mice were subjected to bilateral common carotid artery stenosis or a sham operation and f
224 ty liver and intimal medial thickness of the common carotid arteries, subcutaneous adipose tissue, an
225 tandard cerebrovascular surgery cases in rat common carotid arteries, such as proximal occlusion, tra
226 nternal carotid artery is anastomosed to the common carotid artery, this obviates the need for patch
229 aphy and CT angiography findings of the left common carotid artery trifurcation were described with i
230 emia as follows: permanent ligation of right common carotid artery under halothane anesthesia, 2-h re
231 s embolic stroke propensity through the left common carotid artery using an idealized aortic arch mod
232 The distance from the bifurcation at which common carotid artery velocity is measured should be sta
233 he PSV in the ICA to that in the ipsilateral common carotid artery (VICA/VCCA) were compared with the
234 The intima-media thickness (IMT) of the common carotid arteries was measured ultrasonographicall
235 se in the mean intima-media thickness of the common carotid artery was 1.13 (95% CI, 1.02 to 1.24), w
238 ntricle for I.C.V. injection of drugs, and a common carotid artery was catheterized to measure mean a
241 Bypass grafting of the jugular vein to the common carotid artery was performed 2 weeks after starti
244 nd and arterial applanation tonometry on the common carotid artery) was lower (P:<0.05) in middle-age
247 ve rats, the middle cerebral artery and both common carotid arteries were occluded for 4.9+/-0.13 h (
249 l thickness (IMT) and plaque height from the common carotid arteries were used as noninvasive markers
250 nd 14 days after burn and endothelium-intact common carotid arteries were used for ex vivo force/rela
251 y stenosis and intima-media thickness of the common carotid artery were assessed by using duplex ultr
252 ene (eNOS) did not remodel their ipsilateral common carotid arteries whereas wild-type mice did.
253 f endovascular balloon occlusion of the left common carotid artery, which created an aneurysm at the
254 trasonic flow probe and meter applied to the common carotid artery while cerebral blood flow was meas
256 ayed in cultured endothelial cells and mouse common carotid artery with or without OGD treatment.
257 ypoxia, induced by permanent ligation of the common carotid artery with transient hypoxia, resulted i