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1 romatin structure of breakpoint regions in a common fragile site.
2 essor gene, like FHIT and WWOX, located at a common fragile site.
3 cular demonstration of a germline break in a common fragile site.
4  and promotes replication of DNA lesions and common fragile sites.
5 arge proportion of these breaks occurring at common fragile sites.
6 n secondary structures that are hallmarks of common fragile sites.
7 kpoints of lost regions located in genes and common fragile sites.
8 demonstrate that OPT domains are enriched at common fragile sites.
9 s that resemble those of aphidicolin-induced common fragile sites.
10 ny characteristics similar to those in other common fragile sites.
11 lated with the presence of previously mapped common fragile sites.
12 nt known to result in chromosome breakage at common fragile sites.
13 inhibit DNA replication induce expression of common fragile sites.
14 ility regions are the most characteristic of common fragile sites.
15 it gene and asked whether it also contains a common fragile site and if it is unstable in mouse tumor
16       The tumor suppressor gene FHIT spans a common fragile site and is highly susceptible to environ
17 s evidence indicates that telomeres resemble common fragile sites and present a challenge for DNA rep
18 ty of ORC sites are strongly associated with common fragile sites and recurrent deletions in cancers.
19 to replication stress, certain loci, such as common fragile sites and telomeres, remain under-replica
20 toward understanding the genomic features of common fragile sites and the cellular processes that mon
21 14.2, is the most frequently expressed human common fragile site, and allelic losses at FRA3B have be
22 recessive cancer genes, 17 were of sequenced common fragile sites, and 178 were in genomic regions th
23                                              Common fragile sites appear to arise through incomplete
24                                              Common fragile sites are chromosomal loci prone to break
25                                              Common fragile sites are highly unstable regions of the
26                                              Common fragile sites are loci that form chromosome gaps
27                                 In contrast, common fragile sites are present in all individuals and
28                                              Common fragile sites are regions of human chromosomes pr
29                                              Common fragile sites are regions that show elevated susc
30         Our results support a model in which common fragile sites are sequences that initiate replica
31                                              Common fragile sites are specific regions in the human g
32                                              Common fragile sites are specific regions of the genome
33                                              Common fragile sites, as their name implies, are present
34                                          The common fragile site at 3p14.2 (FRA3B) is the most sensit
35                                          The common fragile site at chromosomal band 3p14.2 (FRA3B) i
36            FRA7G is an aphidicolin-inducible common fragile site at human chromosomal band 7q31.2.
37 highly destabilized chromosomes and specific common fragile site breakage.
38 uced replication stress induces breakages at common fragile sites, but the exact causes of fragility
39                           Thus, the study of common fragile sites can provide insight not only into t
40 uently at KRTAP-1 than at the representative common fragile site (CFS) FRA16D.
41      The cloning and characterization of the common fragile site (CFS) FRA6E (6q26) identified Parkin
42                                              Common fragile sites (CFS) are chromosomal regions that
43 ites (ERFS), while ERFS and late-replicating common fragile sites (CFS) are equally fragile in respon
44 t the completion of replication at so-called common fragile sites (CFS) occurs very late in the cell
45      Although distinct from late-replicating common fragile sites (CFS), the stability of ERFSs and C
46 of hard-to-replicate genomic regions, namely common fragile sites (CFS).
47 e chromosomes, which preferentially occur at common fragile sites (CFS).
48  loci with DNA secondary structures, such as common fragile sites (CFSs) and palindromic repeats.
49                                              Common fragile sites (CFSs) are chromosomal loci with in
50                                              Common fragile sites (CFSs) are difficult-to-replicate g
51                                  Chromosomal common fragile sites (CFSs) are genetically unstable reg
52                                              Common fragile sites (CFSs) are genomic regions prone to
53                                              Common fragile sites (CFSs) are genomic regions that are
54                                              Common fragile sites (CFSs) are hot spots of chromosomal
55                                              Common fragile sites (CFSs) are large genomic regions pr
56                                              Common fragile sites (CFSs) are loci that preferentially
57                                              Common fragile sites (CFSs) are regions of profound geno
58                                              Common fragile sites (CFSs) are regions prone to chromos
59                                              Common fragile sites (CFSs) are regions susceptible to r
60                                              Common fragile sites (CFSs) are specific breakage-prone
61                                  Chromosomal common fragile sites (CFSs) are unstable genomic regions
62                                              Common fragile sites (CFSs) are unstable loci that provi
63 se cleaves late replication intermediates at common fragile sites (CFSs) during early mitosis to trig
64 n in cervical cancer and the position of the common fragile sites (CFSs) has been observed at both th
65 n in cervical cancer and the position of the common fragile sites (CFSs) has been observed at the cyt
66                                              Common fragile sites (CFSs) represent large, highly unst
67  from genomic deletions and duplications and common fragile sites (CFSs) seen as breaks on metaphase
68 ragile metaphase telomeres that resemble the common fragile sites (CFSs), and the association of sist
69             AT repeats are abundant in human common fragile sites (CFSs), genomic regions that underg
70 ol eta is also required for the stability of common fragile sites (CFSs), whose rearrangements are co
71 m fragile telomeres-structures that resemble common fragile sites (CFSs)-but how they are formed is n
72  novo copy number variant (CNV) formation at common fragile sites (CFSs).
73 cult-to-replicate genomic regions, including common fragile sites (CFSs).
74 entially integrates at loci containing human common fragile sites (CFSs).
75 late-sensitive RFSs share many features with common fragile sites (CFSs; which are found in all indiv
76 enomic regions, we assessed the stability of common fragile sites, chromosomal loci that are prone to
77                    Thus, this is the largest common fragile site cloned to date.
78                                   With three common fragile sites cloned, their mechanism of expressi
79      We found that aphidicolin (APH)-induced common fragile sites contain more sequence segments with
80                                   FRA7G is a common fragile site containing the candidate tumor suppr
81 cal to prevent mitotic catastrophe following common fragile site expression.
82                                              Common fragile sites form gaps at characteristic chromos
83 es of RET/PTC rearrangements, are located in common fragile sites FRA10C and FRA10G, and undergo DNA
84 ative tumor suppressor gene that maps to the common fragile site FRA16D on chromosome 16q23.3-24.1, i
85 the cloning of WWOX, a gene that maps to the common fragile site FRA16D region in chromosome 16q23.3-
86 e that WWOX may span the yet uncharacterized common fragile site FRA16D region.
87 3.3-24.1, a chromosome region that spans the common fragile site FRA16D.
88 ontig of >1 Mb across the second most active common fragile site, FRA16D (16q23.2).
89 omain containing oxidoreductase) gene at the common fragile site, FRA16D, is altered in many types of
90         The 16q23.2 breakpoint transects the common fragile site, FRA16D, providing a molecular demon
91               The FHIT gene, which spans the common fragile site FRA3B, has been shown to produce abe
92 scripts in a variety of tumors and spans the common fragile site FRA3B.
93 e gaps and breaks and breaks at the specific common fragile sites FRA3B and FRA16D were significantly
94 such independent integrations into 3p14.2, a common fragile site (FRA3B).
95    One putative target, 3p14.2, contains the common fragile site, FRA3B, a hereditary renal carcinoma
96 tidine triad gene (FHIT) encompasses a human common fragile site, FRA3B, that is susceptible to envir
97  within the FHIT gene, which encompasses the common fragile site, FRA3B.
98 involved in the fragility of the most active common fragile site, FRA3B.
99 large genes, including the two most unstable common fragile sites, FRA3B and FRA3D, thus linking tran
100 ated that the DNA probe for D7S522 spans the common fragile site FRA7G at 7q31.
101                Thus for the first two cloned common fragile sites, FRA7G and FRA3B, there is an assoc
102 31.1-31.2, a region that contains one of the common fragile sites, FRA7G.
103                                              Common fragile sites frequently coincide with the locati
104                                 The study of common fragile sites has its roots in the early cytogene
105 romosome structure, no biological effects of common fragile sites have been convincingly shown, altho
106 f cell cycle checkpoints and DNA repair, and common fragile sites have provided insight into understa
107 ntriguing component of chromosome structure, common fragile sites have taken on novel significance as
108 teresting component of chromosome structure, common fragile sites have taken on novel significance as
109 n chromosomal band 3p14.2 is the most active common fragile site in the human genome.
110 somal band 3p14.2, FRA3B, is the most active common fragile site in the human genome.
111 FRA3B, has been described as the most active common fragile site in the human genome.
112  chromosomal band 3p14.2, is the most active common fragile site in the human genome.
113  to a mechanistic basis for the structure of common fragile sites in mitotic chromosomes.
114  different from MiDAS at aphidicolin-induced common fragile sites in that they map to genomic regions
115    FRA3B at 3p14.2 is the most active of the common fragile sites in the human genome and is expresse
116 Parkin maps to FRA6E, one of the most active common fragile sites in the human genome, it represents
117 r led to dysregulation of DNA replication at common fragile sites in vitro.
118 orts the role of DNA secondary structures in common fragile site instability, provides a systematic m
119 Thus, the physical relationship of Fhit to a common fragile site is similar to that observed with the
120 f CCG repeats, the mechanism responsible for common fragile sites is unknown.
121       FRA3B is the most frequently expressed common fragile site localized within human chromosomal b
122      This work further demonstrates that the common fragile sites may play an important role in cance
123                 Recent findings suggest that common fragile sites may serve as markers of chromosome
124                    Correspondingly, genes at common fragile sites may sustain elevated levels of DNA
125 ues were associated with cancer breakpoints, common fragile sites, microRNA, and cancer-related genes
126 t 3p14.2 is the most highly expressed of the common fragile sites observed when DNA replication is pe
127 gene and is the most highly expressed of the common fragile sites observed when DNA replication is pe
128  3p14-p12 interval known to contain the most common fragile site of the human genome (FRA3B), the FHI
129      The 3p14.2 region spans the most active common fragile site of the human genome, encompassing a
130  oxidoreductase (WWOX) spans the second most common fragile site of the human genome, FRA16D, located
131                          The distribution of common fragile sites parallels the positions of neoplasi
132                                              Common fragile sites predispose to specific chromosomal
133 rspersed nuclear element 1s, suggesting that common fragile sites serve a function.
134 l cycle checkpoint kinases, CHK1 and CHK2 on common fragile site stability in human cells.
135 ing chromosome stability, and in particular, common fragile site stability.
136 wn genomic loci/regions include centromeres, common fragile sites, subtelomeres, and telomeres.
137 gions that are difficult to replicate (e.g., common fragile sites, telomeres, and repeated sequences)
138 dentification of a sequence element within a common fragile site that increases chromosome fragility.
139 ristics are possibly intrinsic properties of common fragile sites that may affect their replication a
140 ed with intrinsically unstable loci known as common fragile sites that occurs after cells experience
141 mal abnormalities occurred preferentially at common fragile sites upon conditional Hus1 inactivation.
142 o learn about the general characteristics of common fragile sites, we investigated the chromatin stru
143 ngements in tumors are often associated with common fragile sites, which are specific genomic loci pr

 
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