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1 ith the reversion event having occurred in a common lymphoid progenitor.
2 MPP-2, MPP-3, common myeloid progenitor, and common lymphoid progenitor.
3 s at a developmental stage that precedes the common lymphoid progenitor.
4 , developed into lymphoid lineage-restricted common lymphoid progenitors.
5 n(+) cells depleted osteolectin(+) cells and common lymphoid progenitors.
6  while promoting T cell differentiation from common lymphoid progenitors.
7  of peri-arteriolar osteolectin(+) cells and common lymphoid progenitors.
8 rm repopulating activity, HSC quiescence and common lymphoid progenitors.
9  suggesting a differentiation bottleneck for common lymphoid progenitors.
10 e progenitors, hematopoietic stem cells, and common lymphoid progenitors.
11  plays a critical role in the development of common lymphoid progenitors.
12               Here we show that a clonogenic common lymphoid progenitor, a bone marrow-resident cell
13                             ILCs derive from common lymphoid progenitors, although the transcriptiona
14                          We propose that the common lymphoid progenitor and common myeloid progenitor
15 elopmental intermediate between the upstream common lymphoid progenitor and the downstream NKP, previ
16  of lymphoid-primed multipotent progenitors, common lymphoid progenitors and B cell progenitors, norm
17                          In contrast to MPP, common lymphoid progenitors and B lineage cell numbers w
18                                   Clonogenic common lymphoid progenitors and clonogenic common myeloi
19 rred via developmental intermediates such as common lymphoid progenitors and common myeloid progenito
20 also of their downstream progenitors such as common lymphoid progenitors and common myeloid progenito
21 ymphopoiesis was unaffected, and bone marrow common lymphoid progenitors and hematopoietic stem cells
22 ng heterodimer that stimulates the growth of common lymphoid progenitors and immature B and T lymphoi
23             In bone marrow from mutant mice, common lymphoid progenitors and pre-pro-B cells appeared
24     Ras inhibition impaired proliferation of common lymphoid progenitors and pre-pro-B cells but not
25                                           In common lymphoid progenitors and T-cells Firre exerts a c
26 hanism resulting in reduced cell survival of common lymphoid progenitors and thymocytes at the double
27 mmitted progenitors in both the bone marrow (common lymphoid progenitor) and thymus (proT1) maintain
28 ipheral leukocyte counts, early depletion of common lymphoid progenitors, and a monocytic bias in com
29 ive haematopoietic stem cells, are absent on common lymphoid progenitors, and are upregulated after m
30 tic stem cells, early lymphocyte precursors, common lymphoid progenitors, and early T lineage progeni
31 mphopoiesis at the single cell level: HSPCs, common lymphoid progenitors, and mature B cells.
32      Strikingly, ectopic GATA-1 reprogrammed common lymphoid progenitors as well as granulocyte/monoc
33 s altered, we show that ILC2 generation from common lymphoid progenitors, as well as ILC2 homeostasis
34 f cell-fate-restriction events that occur as common lymphoid progenitors become committed to each of
35 LL1), FLT3L and IL-7 to hepatocytes expanded common lymphoid progenitors, boosted de novo thymopoiesi
36 emokine receptor CCR9 by a subset of MPP and common lymphoid progenitors but not hemopoietic stem cel
37 that the observed expression of the IL-7R on common lymphoid progenitors, but not ETPs, results in di
38  a reduction of hematopoietic stem cells and common lymphoid progenitors causing a severely depleted
39 bly by influencing lineage commitment from a common lymphoid progenitor cell.
40 f B cells, T cells, natural killer cells and common lymphoid progenitor cells and an enhanced myeloid
41 et (FMD) increases the levels of bone marrow common lymphoid progenitor cells and cytotoxic CD8(+) tu
42                  IL-7R is first expressed on common lymphoid progenitor cells and is not detected on
43                                              Common lymphoid progenitor cells and M2 macrophages were
44               The expression of Pax5 commits common lymphoid progenitor cells to B-lymphoid lineage d
45     Marrow-derived hematopoietic stem cells, common lymphoid progenitor cells, and developing B and m
46        ILC2s develop in the bone marrow from common lymphoid progenitor cells, but little is known ab
47 , we sort-purified mouse bone marrow-derived common lymphoid progenitor cells, early thymic progenito
48 ymphocyte development and can be detected in common lymphoid progenitor cells.
49 progenitor cells and the bone marrow-derived common lymphoid progenitor cells.
50 methylated at CpG residues among fetal liver common lymphoid progenitor cells.
51                        Our data suggest that common lymphoid progenitor (CLP) cells could be responsi
52 he phenotypic changes of reporter-expressing common lymphoid progenitor (CLP) cells in the bone marro
53  hematopoietic stem cell (HSC) expansion and common lymphoid progenitor (CLP) depletion in a model of
54 t in FOXO1-deficient mice is arrested in the common lymphoid progenitor (CLP) LY6D(+) cell stage.
55 nsistent with multilineage progenitor (MLP), common lymphoid progenitor (CLP), and B lineage-restrict
56                    NFIL3 was required in the common lymphoid progenitor (CLP), and was essential for
57 ive DDT decreased the numbers of MPP-derived common lymphoid progenitor (CLP), common myeloid progeni
58 ne of these developmental intermediates, the common lymphoid progenitor (CLP), which can give rise to
59                                The number of common lymphoid progenitors (CLP) and their pre-pro-B an
60 est, there was heterogeneity among canonical common lymphoid progenitors (CLP) in bone marrow.
61 ow multipotent progenitors (MPP) switch into common lymphoid progenitors (CLP) or common myeloid prog
62 id-primed multipotential progenitors (LMPP), common lymphoid progenitors (CLP), and B/T cell precurso
63 n protein renders functionally defined HSCs, common lymphoid progenitors (CLP), and precursor B-lymph
64 D44(Hi) and similar in some respects to the "common lymphoid progenitors" (CLP) identified by others.
65 eviously reported that two subpopulations of common lymphoid progenitors, CLP-1 and CLP-2, coexist in
66                   Strikingly, >50% hGM-CSFR+ common lymphoid progenitors (CLPs) and >20% hGM-CSFR+ pr
67  which generate fewer than normal numbers of common lymphoid progenitors (CLPs) and common myeloid pr
68 ed lymphopenia and substantial reductions of common lymphoid progenitors (CLPs) and lymphoid precurso
69 rkably, CD11a was critical for generation of common lymphoid progenitors (CLPs) and lymphoid-primed m
70 studies using highly enriched populations of common lymphoid progenitors (CLPs) and MPs from the bone
71 al of 2 putative thymus seeding populations, common lymphoid progenitors (CLPs) and multipotent proge
72 )CD27(+) subset is predominantly composed of common lymphoid progenitors (CLPs) and multipotent proge
73 tment point when NK progenitors develop from common lymphoid progenitors (CLPs) and that E4bp4 must b
74                                              Common lymphoid progenitors (CLPs) are descended from HS
75                                              Common lymphoid progenitors (CLPs) are lymphoid-lineage-
76                                              Common lymphoid progenitors (CLPs) are the first bone ma
77  the V(D)J recombinase is active as early as common lymphoid progenitors (CLPs) but not in the upstre
78                                              Common lymphoid progenitors (CLPs) clonally produce both
79 hroid but not lymphoid genes, whereas single common lymphoid progenitors (CLPs) coexpress T and B lym
80                             We now show that common lymphoid progenitors (CLPs) from mice with active
81 th which hematopoietic stem cells (HSCs) and common lymphoid progenitors (CLPs) from neonates and adu
82 f both Rac1 and Rac2 inhibited production of common lymphoid progenitors (CLPs) in bone marrow and su
83 ) haematopoietic progenitor cells (LSKs) and common lymphoid progenitors (CLPs) in bone marrow.
84 s) in the thymus are thought to develop from common lymphoid progenitors (CLPs) in the bone marrow (B
85 l embryonic stem (ES) cells fail to generate common lymphoid progenitors (CLPs) resulting in a comple
86              In the fetal liver, a subset of common lymphoid progenitors (CLPs) that expresses the in
87  showed that the extrathymic precursors were common lymphoid progenitors (CLPs) that included CD19(-)
88 endritic cell (pDC) lineage derived from the common lymphoid progenitors (CLPs) that is dependent on
89                                The number of common lymphoid progenitors (CLPs) were not affected; ho
90 ulated the proliferation of pre-pro-B cells, common lymphoid progenitors (CLPs), and colony-forming u
91 rified HSCs, multipotent progenitors (MPPs), common lymphoid progenitors (CLPs), and common myeloid p
92 p to 1000-fold toward cells with features of common lymphoid progenitors (CLPs), and lymphoid differe
93 egakaryocyte-erythrocyte progenitors (MEPs), common lymphoid progenitors (CLPs), and pro-T and pro-B
94 on CpG sites throughout the genome for MPPs, common lymphoid progenitors (CLPs), common myeloid proge
95  depleted common myeloid progenitors (CMPs), common lymphoid progenitors (CLPs), granulocyte-macropha
96 tion with common myeloid progenitors (CMPs), common lymphoid progenitors (CLPs), granulocyte-macropha
97 phoid lineage-committed progenitors, such as common lymphoid progenitors (CLPs), maintain a latent my
98  common dendritic cell progenitors (CDPs) or common lymphoid progenitors (CLPs), respectively.
99                       We found that both the common lymphoid progenitors (CLPs), shown here and elsew
100  that of lin(-)Sca1(low)kit(low)IL7Ralpha(+) common lymphoid progenitors (CLPs), their cloning effici
101 r CXCR4 in MPPs reduced differentiation into common lymphoid progenitors (CLPs), which decreased lymp
102 plantation of low numbers of highly purified common lymphoid progenitors (CLPs)-a rare population of
103 promote early B lineage differentiation from common lymphoid progenitors (CLPs).
104 d compare multipotent progenitors (MPPs) and common lymphoid progenitors (CLPs).
105 inappropriate induction of Gata1 in HSCs and common lymphoid progenitors (CLPs).
106 and Flt3 ligand (FL) in pDC development from common lymphoid progenitors (CLPs).
107 the earliest lymphoid-committed progenitors (common lymphoid progenitors [CLPs]) and CMPs and their p
108                                              Common lymphoid progenitors could not be identified in t
109 trathymic T lineage-committed cells, whereas common lymphoid progenitors did not.
110 ously described lymphoid progenitors such as common lymphoid progenitors express TdT and relatively h
111 tors and Lin(-)Sca-1(+)cKit(Lo)IL-7Ralpha(+) common lymphoid progenitors from adult marrow efficientl
112 2L/L-selectin+ progenitors (LSP), as well as common lymphoid progenitors from C57BL6-Thy1.1-RAG-1/GFP
113 GATA-3 cannot generate early thymocytes from common lymphoid progenitors has thus far precluded inves
114                           PU.1 disruption in common lymphoid progenitors, however, did not prevent th
115  or most restricted progenitors but depleted common lymphoid progenitors, impairing lymphopoiesis, ba
116         Although we know they develop from a common lymphoid progenitor in the bone marrow (BM), the
117  regenerated the thymus and were superior to common lymphoid progenitors in magnitude and frequency o
118 provide direct evidence for the existence of common lymphoid progenitors in sites of early hematopoie
119                             ILC2s arise from common lymphoid progenitors in the bone marrow, are depe
120 ucer cell progenitors in the fetal liver and common lymphoid progenitors in the bone marrow.
121 ates that all newly made ILCs originate from common lymphoid progenitors in the bone marrow.
122 population of B/NK bipotent precursors among common lymphoid progenitors in the fetal liver and the b
123 stem, such as common myeloid progenitors and common lymphoid progenitors, increase the production of
124  required for the in vivo differentiation of common lymphoid progenitors into ILC lineage-restricted
125  progenitor (Lin-Sca1+Kit+ Flt3+) as well as common lymphoid progenitor (Lin-Sca1+CD117(lo)CD127+) po
126 ly, G-CSF activated STAT3 in early G-CSFR(+) common lymphoid progenitors of cDCs/pDCs but not in matu
127 itro proliferation and/or differentiation of common lymphoid progenitors, pre-pro-B cells, and hemato
128     Moreover, early lymphoid progenitors and common lymphoid progenitors produced significant numbers
129  from hematopoietic stem cells did not alter common lymphoid progenitor production but severely reduc
130 downstream multipotent progenitors (MPP) and common lymphoid progenitors rapidly generated T cells fo
131 scription signatures of E2A- and HEB-ablated common lymphoid progenitors significantly overlap.
132 hat extended to hematopoietic stem cells and common lymphoid progenitors, spared T cells and enhanced
133 -) mice is completely blocked at the LY6D(-) common lymphoid progenitor stage.
134              We have identified a clonogenic common lymphoid progenitor that can differentiate into T
135                           The existence of a common lymphoid progenitor that can only give rise to T
136                          Furthermore, unlike common lymphoid progenitors that readily generate B cell
137  contrast to the previously described murine common lymphoid progenitor, the alpha chain of the recep
138 hat mouse ILCs are derived downstream of the common lymphoid progenitor through lineage-restricted pr
139 ed factor that blocks B lymphopoiesis at the common lymphoid progenitor to preproB cell stage.
140 the adipocyte-derived factor occurred at the common lymphoid progenitor to preproB cell stage.
141 (ID) proteins block E-protein DNA binding in common lymphoid progenitors to allow ILC2 development.
142  lymphoid-primed multipotent progenitors and common lymphoid progenitors to the thymus decreases more
143 cell differentiation potential of HNF1A(-/-) common lymphoid progenitors was severely impaired in vit
144 competent myeloid but not lymphoid cells, as common lymphoid progenitors were decreased, and peripher
145 epression, operative in both fetal and adult common lymphoid progenitors, where T cell potential is s
146                      ILCs are generated from common lymphoid progenitors, which are subsequently comm
147 e in the bone marrow and differentiated from common lymphoid progenitors, which indicates they are di

 
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