1 n athletes (9% vs 1%; P < 0.001), as well as
commotio cordis (5% compared with 1% in nonathletes; P =
2 a that caused structural damage (416 [22%]),
commotio cordis (65 [3%]), and heat stroke (46 [2%]).
3 rdiomyopathy, coronary artery anomalies, and
commotio cordis are more common in young athletes than i
4 This experimental model of
commotio cordis closely resembles the clinical profile o
5 The risk of
commotio cordis during sports activities has become more
6 Commotio cordis events occurred most commonly during org
7 The expanded spectrum of
commotio cordis illustrates the potential dangers implic
8 We developed a swine model of
commotio cordis in which a low-energy impact to the ches
9 Commotio cordis is being increasingly recognized as a ca
10 Whereas chest wall blows are common,
commotio cordis is rare.
11 cities, suggesting that the predilection for
commotio cordis is related in a complex manner to the pr
12 A physiological
commotio cordis model was utilized to assess variables t
13 lt in a significant reduction in the risk of
commotio cordis on the playing field.
14 ogical data from a well-established model of
commotio cordis predicts the reduction in SCD with chest
15 ng chest blows causing sudden cardiac death (
commotio cordis) are often associated with competitive s
16 External chest impacts (
commotio cordis) can cause mechanically induced prematur
17 due to low-energy trauma to the chest wall (
commotio cordis) has been described in young sports part
18 odel of sudden death from chest wall impact (
commotio cordis), we sought to define the chest wall are
19 s can trigger ventricular fibrillation (VF) (
commotio cordis).
20 lows in recreational and competitive sports (
commotio cordis).
21 act has been described in young individuals (
commotio cordis).
22 entricular cardiomyopathy, long QT syndrome,
commotio cordis, and Kawasaki disease.
23 In this experimental model of
commotio cordis, blockade of the K(+)(ATP) channel reduc
24 nagement of blunt thoracic aortic trauma and
commotio cordis, or sudden death following blunt precord
25 Commotio cordis, sudden cardiac death (SCD) caused by re
26 In an animal model of
commotio cordis, sudden death with chest-wall impact, we
27 In our previously reported swine model of
commotio cordis, ventricular fibrillation (with T-wave s
28 urtailed VF occurrence in a porcine model of
commotio cordis, VF has been suggested to arise from abn
29 Using our model of
commotio cordis, we evaluated individual animal suscepti
30 Commotio cordis, which is a recently described syndrome
31 rrow, subject-specific vulnerable window for
commotio cordis-induced VF that exists both in time and
32 of PVEM induction and its potential role in
commotio cordis-induced VF.
33 nt in humans and contribute to the rarity of
commotio cordis.
34 e that there is individual susceptibility to
commotio cordis.
35 rts-that of the Brugada syndrome and that of
commotio cordis.
36 in our experimental model and in victims of
commotio cordis.
37 ith regulation balls, may reduce the risk of
commotio cordis.
38 e provides a great degree of protection from
commotio cordis.
39 by comparison with similar phenomena seen in
commotio cordis.
40 Extramacular
commotio occurs mostly in an inferotemporal to temporal
41 well as intravitreal hemorrhages (14.7%) and
commotio retinae (21.1%).
42 d corneal abrasion (n = 7), hyphema (n = 9),
commotio retinae (n = 5), intraretinal hemorrhage (n = 3
43 Reduced VA after extramacular
commotio retinae may represent occult macular injury or
44 a retinae, retinal necrosis, and surrounding
commotio retinae with specific photoreceptor cell death
45 For extramacular
commotio retinae, 117 patients were identified, of whom
46 For macular
commotio retinae, 53 patients were identified, of whom 3
47 ost common retinal locations of extramacular
commotio retinae, in order of frequency, were inferotemp
48 acute traumatic maculopathy and extramacular
commotio retinae.
49 consistent with human and animal studies of
commotio retinae.