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1 s can trigger ventricular fibrillation (VF) (commotio cordis).
2 lows in recreational and competitive sports (commotio cordis).
3 act has been described in young individuals (commotio cordis).
4 e that there is individual susceptibility to commotio cordis.
5 rts-that of the Brugada syndrome and that of commotio cordis.
6 in our experimental model and in victims of commotio cordis.
7 ith regulation balls, may reduce the risk of commotio cordis.
8 e provides a great degree of protection from commotio cordis.
9 by comparison with similar phenomena seen in commotio cordis.
10 nt in humans and contribute to the rarity of commotio cordis.
11 n athletes (9% vs 1%; P < 0.001), as well as commotio cordis (5% compared with 1% in nonathletes; P =
12 a that caused structural damage (416 [22%]), commotio cordis (65 [3%]), and heat stroke (46 [2%]).
14 rdiomyopathy, coronary artery anomalies, and commotio cordis are more common in young athletes than i
15 ng chest blows causing sudden cardiac death (commotio cordis) are often associated with competitive s
21 due to low-energy trauma to the chest wall (commotio cordis) has been described in young sports part
24 rrow, subject-specific vulnerable window for commotio cordis-induced VF that exists both in time and
28 cities, suggesting that the predilection for commotio cordis is related in a complex manner to the pr
31 nagement of blunt thoracic aortic trauma and commotio cordis, or sudden death following blunt precord
32 ogical data from a well-established model of commotio cordis predicts the reduction in SCD with chest
35 In our previously reported swine model of commotio cordis, ventricular fibrillation (with T-wave s
36 urtailed VF occurrence in a porcine model of commotio cordis, VF has been suggested to arise from abn
37 odel of sudden death from chest wall impact (commotio cordis), we sought to define the chest wall are