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1 versus 16%; P < 0.001) than in patients with community-acquired infection.
2 us is a leading cause of both nosocomial and community-acquired infection.
3 devices might be responsible for the rise of community acquired infections.
4 timicrobial resistance, especially regarding community-acquired infections.
5 the etiological agent of many hospital- and community-acquired infections.
6 ial resistance in both hospital-acquired and community-acquired infections.
7 hypervirulent strains associated with severe community-acquired infections.
8 us is now one of the key causative agents of community-acquired infections.
9 g our ability to treat routine hospital- and community-acquired infections.
10 thogen and a leading cause of nosocomial and community-acquired infections.
11 ria, and its presence in both nosocomial and community-acquired infections.
12 us, which is a major cause of nosocomial and community-acquired infections.
13 vant pathogens responsible for hospital- and community-acquired infections.
14 d to distinguish health-care-associated from community-acquired infections.
15 he number of methicillin-resistant S. aureus community-acquired infections.
16 or causative agent of numerous hospital- and community-acquired infections.
17 us is a major cause of severe nosocomial and community-acquired infections.
18 lity, have traditionally been categorized as community-acquired infections.
19 human pathogen causing hospital-acquired and community-acquired infections.
20 ureus is responsible for many nosocomial and community-acquired infections.
21 increasing number of serious nosocomial and community-acquired infections.
22 weeks to months, considerably faster than in community-acquired infections.
23 hours after LTCC admission so as to exclude community-acquired infections.
24 nce of nosocomial transmission compared with community-acquired infection?
25 reas in the smaller group of inpatients with community-acquired infections, 12 capsid and 9 polymeras
27 with shorter times to antimicrobial therapy: community-acquired infections (-53 min; p < 0.001) and h
28 17 of 31; P = 0.05); rates were similar for community-acquired infections (80% [124 of 156] versus 8
29 nclude a predisposition to opportunistic and community-acquired infections, an altered response to va
30 ains have spread throughout hospitals, while community acquired infections and other sources ensure a
31 rology, we estimated both the probability of community-acquired infection and within-household transm
32 charges) for nosocomial-acquired infections, community-acquired infections, and sepsis, respectively.
33 ra days) for nosocomial-acquired infections, community-acquired infections, and sepsis, respectively.
34 deaths) for nosocomial-acquired infections, community-acquired infections, and sepsis, respectively.
35 ions are nosocomial, but a growing number of community-acquired infections are caused by hypervirulen
36 es (surgical prophylaxis, broad-spectrum for community-acquired infections, broad-spectrum for hospit
42 Of the bacteria typically responsible for community-acquired infections, clarithromycin-resistant
43 us aureus is a major cause of nosocomial and community-acquired infections for which a vaccine is gre
44 cephalosporins cannot be recommended even in community-acquired infection in our cirrhotic population
46 ic characteristics have been responsible for community-acquired infections in France and Switzerland.
47 antibiotics but are highly virulent, causing community-acquired infections in healthy individuals.
48 n humans, are now emerging as agents of both community-acquired infections in humans as well as hospi
49 he magnitude and term of morbidity of acute, community-acquired infections in immune competent patien
50 influenza virus (PIVi) infections are common community-acquired infections in lung transplant recipie
51 changed markedly due to strong increases in community-acquired infections in successive periods of v
52 Staphylococcus aureus, is a major source of community-acquired infections in the USA, Canada, and Eu
53 has an appropriate spectrum of activity for community-acquired infections, including those caused by
54 auses toxin-mediated nosocomial diarrhea and community-acquired infections; no preventive vaccine is
56 anges to antibiotic treatment guidelines for community-acquired infections of the upper and lower res
57 infection (OR, 2.752 [95% CI, 1.100-6.886]), community-acquired infection (OR, 10.432 [95% CI, 3.623-
58 rmpA (OR, 17.398 [95% CI, 4.224-71.668]) and community-acquired infection (OR, 6.844 [95% CI, 1.905-2
60 ntibiotic-resistant bacteria responsible for community-acquired infections such as Salmonella spp, Ca
61 e its being a leading cause of nosocomal and community-acquired infections, surprisingly little is kn
62 ll death rate was 13%, ranging from 5% after community-acquired infections to 25% after infections ac
63 th Pneumocystis by cohabitation, to resemble community-acquired infection, underwent lung assessments
68 in an inpatient stay are considered to have community-acquired infection, while those developing sym
69 ticipants and their risk for colonisation or community-acquired infection with priority bacterial pat
70 virulent Klebsiella pneumoniae causes severe community-acquired infections, with its mucoid phenotype