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1 c interneurons and the substantia nigra pars compacta.
2 RRK2 expression in the substantia nigra pars compacta.
3 fused locally into the substantia nigra pars compacta.
4 lase expression in the substantia nigra pars compacta.
5 the thalamus, and the substantia nigra pars compacta.
6 inergic neurons of the substantia nigra zona compacta.
7 ventral tegmental area/substantia nigra pars compacta.
8 arkably, little to the substantia nigra pars compacta.
9 and very few fibers within substantia nigra compacta.
10 inergic neurons of the substantia nigra pars compacta.
11 ne (DA) neurons in the substantia nigra pars compacta.
12 ne (DA) neurons in the substantia nigra pars compacta.
13 x, caudate-putamen and substantia nigra pars compacta.
14 raphe nucleus, and the substantia nigra pars compacta.
15 inergic neurons in the substantia nigra pars compacta.
16 egmental area (VTA) or substantia nigra pars compacta.
17 substantia nigra pars reticulata and/or pars compacta.
18 ventral tegmental area and substantia nigra compacta.
19 of degeneration of the substantia nigra pars compacta.
20 inergic neurons in the substantia nigra pars compacta.
21 nd inflammation in the substantia nigra pars compacta.
22 al tegmental area, and substantia nigra zona compacta.
23 inergic neurons in the substantia nigra pars compacta.
24 ession is noted in the substantia nigra pars compacta.
25 opaminergic neurons in substantia nigra pars compacta.
26 inergic neurons in the substantia nigra pars compacta.
27 inergic neurons in the substantia nigra pars compacta.
28 lesser extent, in the substantia nigra pars compacta.
29 opamine neurons in the substantia nigra pars compacta.
30 inergic neurons of the substantia nigra pars compacta.
31 ral tegmental area and substantia nigra pars compacta.
32 neuron activity in the substantia nigra pars compacta.
33 opamine neurons of the substantia nigra pars compacta.
34 inergic neurons in the substantia nigra pars compacta.
35 ine neurons from mouse substantia nigra pars compacta.
36 sal GL or in the right substantia nigra pars compacta.
37 s. 36+/-2.6 O.D. BMP-7-treated, p<0.05; pars compacta: 29.0+/-4.9 O.D. control vs. 64.4+/-6.9 O.D. BM
39 opamine neurons of the substantia nigra pars compacta, a deficit in dopamine neurotransmission and th
40 of serotonin, and the substantia nigra pars compacta, a major source of dopamine, while monkeys perf
42 gmental area (VTA) and substantia nigra pars compacta activates inhibitory postsynaptic D2-receptors
43 ons of 6-OHDA into the substantia nigra pars compacta and 1 week later were tested for startle after
44 nergic (DA) neurons in substantia nigra pars compacta and age-dependent L-DOPA-sensitive motor dysfun
45 oducing neurons in the substantia nigra pars compacta and decreased striatal dopamine levels are the
46 opaminergic neurons of substantia nigra pars compacta and in the CA1/2 pyramidal cells of hippocampus
47 inergic neurons in the substantia nigra pars compacta and intracellular inclusion called Lewy bodies.
48 inergic neurons in the substantia nigra pars compacta and locus ceruleus, without Lewy body pathology
49 gmented neurons of the substantia nigra pars compacta and locus coeruleus in all four DYT1 dystonia c
51 ing cell levels in the substantia nigra pars compacta and nigrostriatal terminal density in vivo, in
52 ergic neurons from the substantia nigra pars compacta and noradrenergic neurons from the locus coerul
54 sion, particularly the substantia nigra pars compacta and other dopamine-synthesizing cell groups.
55 pamine (DA) neurons in substantia nigra pars compacta and restored DA levels in striatum using two di
56 in neurons within the substantia nigra pars compacta and striatum, two regions critically involved i
57 inergic neurons in the Substantia Nigra pars compacta and terminal dopamine fiber density in the stri
58 inergic neurons in the substantia nigra pars compacta and the accumulation of the protein alpha-synuc
59 ergic neurons from the substantia nigra pars compacta and the presence, in affected brain regions, of
60 icking, in vivo in the substantia nigra pars compacta and the serum of 1-methyl-4-phenyl-1,2,3,6-tetr
61 c projections from the substantia nigra pars compacta and the ventral tegmental area to the basal gan
62 neurons located in the substantia nigra pars compacta and the ventral tegmental area, which form the
63 mine by neurons in the substantia nigra pars compacta and ventral tegmental also influences basal gan
65 nucleus, raphe nuclei, substantia nigra pars compacta and ventral tegmental area homologs, superficia
66 2 were more numerous in the substantia nigra compacta and ventral tegmental area in the Ts65Dn compar
67 opamine neurons in the substantia nigra pars compacta and ventral tegmental area regulate behaviours
68 ic (DA) neurons in the substantia nigra pars compacta and ventral tegmental area regulate extrapyrami
71 ogous to the mammalian substantia nigra pars compacta and was renamed accordingly; a group of gamma-a
72 (PT; homologous to the substantia nigra pars compacta and/or ventral tegmental area of mammals) to th
73 aminergic neurons, A9 (substantia nigra pars compacta) and A10 (ventral tegmental area), have distinc
74 within neurons of the substantia nigra pars compacta, and dual labeling and confocal imaging confirm
76 he ventral tier of the substantia nigra pars compacta, and the lowest levels in the ventral tegmental
77 Mn accumulation in the substantia nigra pars compacta, and thus, can represent a link between Mn expo
78 was upregulated in the substantia nigra pars compacta, and trkB was elevated in the striatum in MDMA-
80 inergic neurons of the substantia nigra pars compacta are defective in Parkinson's disease, but the s
81 ne neurons in the substantia nigra (SN) pars compacta are selectively lost during the progression of
82 Because the VTA and substantia nigra pars compacta are the sole sources of striatal and limbic for
83 mic nucleus (STN), and substantia nigra pars compacta, are conserved throughout vertebrate phylogeny
84 and A10 region of the substantia nigra pars compacta as well as the technical and surgical steps ass
87 eactive neurons in the substantia nigra pars compacta (both in total and in subregions) were performe
88 in neurons not only in substantia nigra pars compacta but also in other extrastriatal areas of the br
90 opamine neurons in the substantia nigra pars compacta, but not in the adjacent ventral tegmental area
92 eactive neurons in the substantia nigra pars compacta by 2-fold (p < 0.05) in animals lesioned with 6
97 gic neurons within the substantia nigra pars compacta coupled with depletion of striatal dopamine.
98 inergic neurons in the substantia nigra pars compacta coupled with intracytoplasmic inclusions known
99 opamine neurons in the substantia nigra pars compacta, culminating in severe motor symptoms, includin
100 ral tegmental area and substantia nigra pars compacta DA neurons in the post-reward period had a sign
101 neurons of the brain's substantia nigra pars compacta die in Parkinson's disease has been an enduring
102 cordings of identified substantia nigra pars compacta dopamine neurons in R1441C LRRK2 transgenic rat
103 reby properly maintain substantia nigra pars compacta dopaminergic neurons and their innervation in t
105 We show that, in rat substantia nigra pars compacta dopaminergic neurons, the voltage dependences o
107 is expressed in human substantia nigra pars compacta glia as well as tyrosine hydroxylase-positive n
108 forming an ultra-short substantia nigra pars compacta --> SNr dopamine pathway that regulates the fir
109 alpha-synuclein in the substantia nigra pars compacta impacts visual processing in a well-established
111 grostriatal neurons of substantia nigra pars compacta in Parkinson's disease represents the best-stud
112 e vulnerability of the substantia nigra pars compacta in PD, why the disorder is age related, and the
113 ty (TH-ir) in the substantia nigra (SN) pars compacta, in the lesioned hemisphere [31.7+/-5.2 (optica
114 ral tegmental area and substantia nigra pars compacta innervate the majority of the cortex, including
115 inergic neurons of the substantia nigra pars compacta is a cardinal feature of Parkinson's disease (P
116 ation of DA neurons in substantia nigra pars compacta is a key neuropathological feature in Parkinson
117 inergic neurons in the substantia nigra pars compacta is the primary cause for motor symptoms observe
118 onial pterobranch hemichordate, Rhabdopleura compacta, is expressed in a dramatically different patte
119 inergic neurons of the substantia nigra pars compacta leading to abnormal activity within the basal g
121 n subtypes outside the substantia nigra pars compacta may be compensating at a molecular level for di
122 ucleus and dorsomedial substantia nigra pars compacta) mesodiencephalic dopaminergic neuron subset, u
124 kB) receptor occurs in substantia nigra pars compacta neurons and is required for neuroprotection.
125 Unilateral lesions of substantia nigra pars compacta neurons created rats with hyperexcitable trigem
126 spontaneous firing of substantia nigra pars compacta neurons, isolated from transgenic mice in which
127 dopaminergic (DAergic) substantia nigra pars compacta neurons, only select downregulation of the PHD2
129 characterised by the presence of severe pars-compacta nigral-cell loss, and accumulation of aggregate
131 ell body counts in the substantia nigra pars compacta of 6-OHDA- and free-3NT-treated mice (injected/
132 ta from the cortex and substantia nigra pars compacta of aged LRRK2 transgenic animals revealed alter
133 le DA neurons from the substantia nigra pars compacta of controls and subjects with idiopathic Parkin
134 opamine neurons of the substantia nigra pars compacta of mice following systemic administration of a
135 opamine neurons in the substantia nigra pars compacta of monkeys (Macaca mulatta) during a reaching t
141 so up-regulated in the substantia nigra pars compacta of post-mortem PD brains as compared with age-m
146 opamine neurons of the substantia nigra pars compacta, other regions of the midbrain, and also the hi
147 in injured IL-6 (+/+) and IL-6 (-/-) SN pars compacta (pc), microgliosis was severely compromised in
148 amygdalopallii pars basalis (PoAb) and pars compacta (PoAc), the nucleus taeniae amgygdalae (TnA) an
150 inergic neurons of the substantia nigra pars compacta preferentially terminate in patch-like regions
151 leasing neurons of the substantia nigra pars compacta produce an extraordinarily dense and expansive
152 (DA) neurons in their substantia nigra pars compacta, progressing to bilateral degeneration of the n
153 inergic neurons in the substantia nigra pars compacta, reproducing an important pathological feature
154 Neuronal counts in the substantia nigra pars compacta revealed that neuronal loss in the parkin cases
155 ne hydroxylase mRNA in substantia nigra pars compacta (SC) (93%; P<0.05) compared with control rats.
156 l proliferation in the substantia nigra pars compacta (SN(C)) with a time-dependent adoption of a neu
157 the laterally-located substantia nigra pars compacta (SN) and medially-located ventral tegmental are
158 inergic neurons in the substantia nigra pars compacta (SN) leads to debilitating motor dysfunction.
160 inergic neurons in the substantia nigra pars compacta (SNc) and consequent depletion of striatal dopa
161 ks the majority of the substantia nigra pars compacta (SNc) and experiences striatal denervation.
162 ine hydroxylase (TH) in the substantia nigra compacta (SNc) and in two subdivisions of the ventral te
163 nergic neurones in the substantia nigra pars compacta (SNc) and may contribute to excitotoxic cell de
166 opamine neurons of the substantia nigra pars compacta (SNc) and the importance of protein aggregation
167 inergic neurons in the substantia nigra pars compacta (SNc) and the presence of intracytoplasmatic in
168 amine neurons from the substantia nigra pars compacta (SNc) and ventral tegmental area (VTA) are invo
169 dopamine centers, the substantia nigra pars compacta (SNc) and ventral tegmental area (VTA), densely
170 f RET in the maintenance of substantia nigra compacta (SNC) and ventral tegmental area (VTA), we bred
172 ns that project to the substantia nigra pars compacta (SNc) are activated by a visual CS for food.
173 gmental area (VTA) and substantia nigra pars compacta (SNc) are not significantly modulated by anesth
174 gmental area (VTA) and substantia nigra pars compacta (SNC) contain dopamine neurons intermixed with
177 ns (DAs) of the rodent substantia nigra pars compacta (SNc) display varied electrophysiological prope
178 sease characterized by substantia nigra pars compacta (SNc) dopamine (DA) neuron loss and subsequent
180 have established that substantia nigra pars compacta (SNc) dopamine neurons are a key node in the ci
181 underlying the loss of substantia nigra pars compacta (SNc) dopaminergic neurons in Parkinson's disea
183 increased activity of substantia nigra pars compacta (SNc) dopaminergic neurons, elevated baseline e
184 epend on the firing of substantia nigra pars compacta (SNc) dopaminergic neurons, we identified and c
185 ogous to the mammalian substantia nigra pars compacta (SNc) evokes increasing activation of MLR cells
186 inergic neurons of the substantia nigra pars compacta (SNc) exhibit functional heterogeneity that lik
187 ic (DA) neurons of the substantia nigra pars compacta (SNc) govern movements requires a detailed know
190 receptor in regulating substantia nigra pars compacta (SNc) neuron physiology in both mice and rats.
191 ordings were made from substantia nigra pars compacta (SNC) neurons in horizontal brain slice prepara
192 s differs from that in substantia nigra pars compacta (SNc) neurons, where subthreshold calcium curre
193 much stronger in monkey than in rat SN pars compacta (SNc) neurons, whereas a moderate level of mGlu
196 rAAV2/5 vectors in the substantia nigra pars compacta (SNc) on one side of the brain; the other side
197 as seen in neighboring substantia nigra pars compacta (SNC) or substantia nigra pars reticulata.
201 ne (DA) release in the substantia nigra pars compacta (SNc) shows a limited dependence on extracellul
202 neurons, including the substantia nigra pars compacta (SNc) subpopulation that preferentially degener
203 e neuron in the monkey substantia nigra pars compacta (SNc) that retains past learned reward values s
204 gmental area (VTA) and substantia nigra pars compacta (SNc) to that of axonal dopamine release in the
205 atal pathway, from the substantia nigra pars compacta (SNc) to the dorsal striatum, and on related fu
206 low or Fluorogold into substantia nigra pars compacta (SNc) were combined with larger injections of T
207 neuron numbers in the substantia nigra pars compacta (SNC) were estimated using stereological method
208 ne (DA) neurons of the substantia nigra pars compacta (SNc) were found to exhibit sustained responses
209 olliculus (SC), to the substantia nigra pars compacta (SNc) where direct synaptic contacts are made w
210 inergic neurons in the substantia nigra pars compacta (SNc), but not in ventral tegmental area or sub
212 rst established in the substantia nigra pars compacta (SNc), but their validity in the VTA is uncerta
213 inergic neurons of the substantia nigra pars compacta (SNc), in addition to many other regions, inclu
214 gmental area (VTA) and substantia nigra pars compacta (SNc), no clear evidence for separate structure
215 neurons (DaNs) of the substantia nigra pars compacta (SNc), resulting in the characteristic hypokine
217 suggested that, in the substantia nigra pars compacta (SNc), the pacemaking relies more on Ca(2+) cha
218 he retrorubral field (RRF), substantia nigra compacta (SNc), ventral tegmental area (VTA), and ventro
219 to dopamine neurons in substantia nigra pars compacta (SNc), whereas matrix neurons receive sensorimo
237 or, a homologue of the substantia nigra pars compacta (SNc)/ventral tegmental area (VTA) is present i
239 nergic neurons of the substantia nigra, pars compacta (SNpc) akin to what is observed in Parkinson di
240 ic (DA) neurons in the substantia nigra pars compacta (SNpc) and of noradrenergic neurons in the locu
241 ntly diminished in the substantia nigra pars compacta (SNpc) and striatum, regions most affected in h
242 ese neurons in the rat substantia nigra pars compacta (SNpc) and ventral tegmental area (VTA), and co
243 opamine neurons in the substantia nigra pars compacta (SNpc) and widespread aggregates of the protein
245 opamine neurons in the substantia nigra pars compacta (SNpc) are greatly needed to effectively change
247 opamine neurons of the substantia nigra pars compacta (SNpc) by regulating the magnitude of the first
249 e hydroxylase-immunoreactive (TH-ir) SN pars compacta (SNpc) DA neurons are immunoreactive for estrog
250 DA) neurons within the substantia nigra pars compacta (SNpc) display a differential vulnerability to
251 generation of midbrain substantia nigra pars compacta (SNpc) dopaminergic (DA) neurons contributes to
252 acterized by a loss of substantia nigra pars compacta (SNpc) dopaminergic (DA) neurons, and can be mo
253 Neuronal loss in the substantia nigra pars compacta (SNpc) in Parkinson disease (PD) is not uniform
254 ogenetic activation of substantia nigra pars compacta (SNpc) neurons alleviates parkinsonism in acute
256 has been shown in the substantia nigra pars compacta (SNpc) of PD models when there has been a decre
257 sfer of RGS10 into the substantia nigra pars compacta (SNpc) of rats reduced microgliosis and protect
259 neurons at the ventral substantia nigra pars compacta (SNpc) preferentially degenerate in Parkinson's
261 inergic neurons in the substantia nigra pars compacta (SNpc) undergo natural cell death during develo
262 s containing CR in the substantia nigra pars compacta (SNpc) were relatively spared compared to those
263 Here, we show that the substantia nigra pars compacta (SNpc), a brain region where dopamine (DA) cell
264 opaminergic neurons in substantia nigra pars compacta (SNpc), and SNCA mice were more vulnerable.
265 opaminergic neurons in substantia nigra pars compacta (SNpc), and there was no loss of dopaminergic n
266 ore TH-ir cells in the substantia nigra pars compacta (SNpc), but this difference was significant onl
267 neurons (DANs) in the substantia nigra pars compacta (SNpc), decrease of dopamine (DA) transmitter,
268 degeneration of DNs in substantia nigra pars compacta (SNpc), decreased striatal dopamine levels and
269 neurons located in the substantia nigra pars compacta (SNpc), expression of monoamines and indolamine
270 ensity loss within the substantia nigra pars compacta (SNpc), locus coeruleus, and ventral tegmental
271 g GDF5 into either the substantia nigra pars compacta (SNpc), the lateral ventricle (LV) or the stria
272 aminergic cells in the substantia nigra pars compacta (SNpc), ventral tegmental area (VTA), and retro
276 on was detected in the substantia nigra pars compacta, striatum, hippocampus, or selected regions of
277 ccumulation within the substantia nigra pars compacta, suggesting that nigrostriatal dopamine dysfunc
278 ccumulation within the substantia nigra pars compacta, suppression of microglial activation, and incr
280 inergic neurons in the substantia nigra pars compacta, the exact mechanism involved is still poorly u
282 rom frontal cortex and substantia nigra pars compacta tissue, isolated by several filtration and cent
283 allidus, thalamus, and substantia nigra pars compacta to various environmental or genetic insults.
284 and a 42% reduction in substantia nigra pars compacta tyrosine hydroxylase-positive neurons at 8 week
285 taining neurons in the substantia nigra pars compacta use pacemaking mechanisms common to neurons not
286 ps of neurons, such as substantia nigra pars compacta, ventral tegmental area and retrorubal field, t
287 pus and dentate gyrus, substantia nigra pars compacta, ventral tegmental area, geniculate nucleus and
288 the SN allowing readers to distinguish pars compacta ventralis and dorsalis from pars reticulata.
289 rminals in the ventral substantia nigra pars compacta (vSNc) or to the ventral tegmental area (VTA),
290 the nucleus tegmenti pedunculopontinus pars compacta was recognized as homologous to the mammalian s
291 TRPM2 and TRPM4mRNA in substantia nigra pars compacta.We propose that ICAN is selectively activated d
293 ommissural putamen and substantia nigra pars compacta were processed for tyrosine hydroxylase and dop
294 aminergic cells in the substantia nigra pars compacta were unaffected by METH or haloperidol alone or
295 dorsolaterally in the substantia nigra pars compacta, whereas neurons inhibited by the stimuli were
296 ic (DA) neurons of the substantia nigra pars compacta, which are preferentially lost in human Parkins
297 ify mDA neurons of the substantia nigra pars compacta while the late Shh and Gli1 lineages maintain t
298 inergic neurons in the substantia nigra pars compacta, with more than 40% of these neurons lost by ag
300 d neuron counts in the substantia nigra pars compacta, yet striatal medium spiny neuron dendritic spi