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1     The survival of mice was not affected by complement deficiency.
2 deficiency considered clinically relevant (3 complement deficiencies, 1 autoimmune neutropenia, 10 hu
3                    Corroborating human data, complement deficiencies and blockade reduced tumor growt
4 nd the nature of the disease process in rare complement deficiencies and complement-driven diseases.
5  disease (IMD) is increased in patients with complement deficiency and human immunodeficiency virus (
6                           The association of complement deficiency and lupus has been known for over
7  the recognized autoimmune manifestations of complement deficiency and then describes new data derive
8  B and T lymphocytes, with the proposal that complement deficiency causes incomplete maintenance of p
9 the early pathogenesis of prion diseases and complement deficiency could result in decreased PrP(Sc)
10 rgeted to the transplanted heart and, unlike complement deficiency, did not affect immunity to infect
11 tissue damage, whereas inherited or acquired complement deficiencies highly favor the development of
12 omplement activation in Cfh(+/-) mice versus complement deficiency in Cfh(-/-) animals.
13 ection or creation of hybrid cell lines with complementing deficiencies in expression of HLA class II
14 h as macrophage activation, interferon-1 and complement deficiency may contribute to atherogenesis.
15  second hypothesis, generated primarily from complement deficiencies, relates to an impairment in the
16                           On the other hand, complement deficiency strikingly reduced the incidence a
17                               In contrast to complement deficiency, targeted complement inhibition wi
18  By using various serum sources with defined complement deficiencies, we demonstrate that, under phys
19                      Using mice with various complement deficiencies, we demonstrated that the intrap
20 properdin mutated individuals, and 4 digenic complement deficiencies were identified.
21 vide insight into the genetic association of complement deficiency with susceptibility to systemic lu