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1 houghts (obsessions) and repetitive actions (compulsions).
2 es (obsessions) and by repetitive behaviors (compulsions).
3 ives rise to inflexibilities like habits and compulsion.
4 SertKI mice showed an enhanced transition to compulsion.
5 rtant for developing interventions to reduce compulsion.
6  experiencing problems with self-control and compulsion.
7 iable and the complementary idea of exposure compulsion.
8 r the stochastic nature of the transition to compulsion.
9 mma oscillations were highly correlated with compulsion.
10  the percentage change of these bands during compulsion.
11 ism may underlie diverse disorders involving compulsion.
12 d not control malaria; its use required mass compulsion.
13 -predicting stimuli, which may contribute to compulsion.
14  under negative affect, and less by habit or compulsion.
15  signaling had no effect on the emergence of compulsions.
16 sumably effective in reducing obsessions and compulsions.
17 ases with prominent distress when preventing compulsions.
18 itating illness consisting of obsessions and compulsions.
19 t, and durable improvement in obsessions and compulsions.
20 gers, and the intent of ERP is to extinguish compulsions.
21 triggers but prevented from expressing their compulsions.
22 on, and 4) hoarding: hoarding obsessions and compulsions.
23 te disinhibition and apathy, and may exhibit compulsions.
24  are more specific to the phenotype than are compulsions.
25  disorder characterized by obsessions and/or compulsions.
26 the mechanisms regulating the development of compulsions.
27 g personalized provocation of obsessions and compulsions.
28 ssions and repeating, ordering, and counting compulsions; 2) forbidden thoughts: aggression, sexual,
29 ng disturbances (80%), hypersexuality (43%), compulsions (29%), and depressed mood (48%).
30 ligious, and somatic obsessions and checking compulsions, 3) cleaning: cleaning and contamination, an
31 der had a higher rate of both obsessions and compulsions, an earlier age at onset, and more comorbid
32 is impaired in psychiatric disorders such as compulsion and addiction, via overreliance on MF control
33  characteristics on the associations between compulsion and disengagement.
34 rly model by integrating circuits underlying compulsion and negative reinforcement.
35 l real-time neurophysiological biomarkers of compulsion and warrant further assessment of the use of
36  underlying neurophysiological mechanisms of compulsion and warrants further assessment of the use of
37 ions may be a potential mechanism leading to compulsions and an important addition to the goal/habit
38  people in the community have obsessions and compulsions and associated levels of interference.
39                                 Because both compulsions and cocaine-induced behaviors may represent
40 eview, we examine the psychological basis of compulsions and compulsivity and their underlying neural
41 s associated with severity of obsessions and compulsions and degree of poor insight in BDD, suggestin
42 m-avoiding) actions-can trigger and maintain compulsions and is directly linked to compulsion severit
43               The presence of obsessions and compulsions and mental disorders was assessed using the
44               Yet, how these beliefs lead to compulsions and obsessions remains unclear.
45                                         When compulsions and obsessive thoughts took over her world,
46               However, it is unknown whether compulsions and reversal learning deficits share a commo
47 ecific semantic impairment (23%) and complex compulsions and rigid thought process (18%).
48 tween eating disorder-related obsessions and compulsions and thickness of the anterior midbody, rostr
49  reduced binge-eating-related obsessions and compulsions, and SGAs reduced symptoms of depression (MD
50 such as hallucinations, depression, anxiety, compulsions, and the suffering stemming from them, canno
51 atients experience in dismissing obsessions, compulsions, and worry despite recognition that these sy
52  (LFP) signals in this area during obsession/compulsion are lacking.
53                                         Many compulsions are an attempt to avoid perceived dangers, a
54                               Obsessions and compulsions are common in the adult population, have the
55 that patients know that their obsessions and compulsions are excessive, but their symptoms neverthele
56 caine, suggests that cortical-limbic induced compulsions are significantly different in their origin
57 king rituals, and the need to perform mental compulsions before discarding any item.
58 rcuits control the expression of established compulsions, but little is known about the mechanisms re
59 evaluated for the presence of obsessions and compulsions by means of the Structured Clinical Intervie
60                      These data suggest that compulsions can be triggered not by impaired cortical-li
61 asses: unaffected (class 1), ritual/symmetry compulsions (class 2), germ/contamination obsessions (cl
62 ted behaviors suggest that the expression of compulsions could in part involve loss of control of suc
63 iatal circuits leading to the development of compulsions (defined here as continued reward seeking in
64           We identify two general markers of compulsion: delta and alpha LFP power was significantly
65 verarching learning theories, to outline how compulsion develops in addiction.
66  strong stimulus-response associations, or a compulsion driven by insensitivity to costs imposed on d
67 ed that binge eating reflects a pathological compulsion driven by the "addictive" properties of foods
68 dividual variability in the speed with which compulsions emerged was predicted by DMS dopamine axon a
69 (DMS) and the dorsolateral striatum (DLS) as compulsions emerged.
70 re recruited in heavy-drinking humans during compulsion for alcohol, highlighting the importance of a
71 ives had higher rates of both obsessions and compulsions; however, this finding is more robust for ob
72 Levels of discontinuity were associated with compulsion (IDR 0.973 [95% CI 0.96-0.99, p<0.0001].
73  reform agenda which could mark a new era of compulsion in international trade law.
74 estigates the goal/habit imbalance theory of compulsion in obsessive-compulsive disorder (OCD), which
75 nt behaviour, and the ensuing development of compulsion in vulnerable individuals.
76 power was significantly increased during all compulsions in the external globus pallidus (GPe), nucle
77 gories used to group types of obsessions and compulsions in the Yale-Brown Obsessive Compulsive Scale
78 genic mouse model of cortical-limbic induced compulsions in which dopamine D1 receptor-expressing (D1
79                                              Compulsion is a cardinal symptom of drug addiction (seve
80                                     However, compulsion is observed in only a small proportion of ind
81 ughts (obsessions) and repetitive behaviors (compulsions), is associated with dysfunction in fronto-s
82     We identified no evidence that increased compulsion leads to improved readmission outcomes or to
83 m and NASPM-sensitive) specifically regulate compulsion-like alcohol consumption, while aINS more gen
84                                              Compulsion-like alcohol drinking (CLAD), where consumpti
85   Much remains unknown about the etiology of compulsion-like alcohol drinking, where consumption pers
86 ens core (NAcore) are critical for promoting compulsion-like alcohol drinking, where rats consume alc
87  lower dose systemic Ox1R inhibition reduced compulsion-like alcohol intake in both sexes, indicating
88 on, whereas miRNA-665-3p inhibition promoted compulsion-like behavior and also enhanced food addictio
89 ination, or responsiveness facilitates their compulsion-like behavior.
90 ing (rather than potentiation) of D1CT mouse compulsion-like behaviors by cocaine, suggests that cort
91 ted, the incidence of D1CT transgene-induced compulsion-like behaviors such as repeated leaping and p
92 at drinking onset had greater activity under compulsion-like conditions.
93 IC from 15 male rats during alcohol-only and compulsion-like consumption.
94 rmined for all licks in a session) predicted compulsion-like drinking, while a separate lick-associat
95 tegrated model for the role of AIC firing in compulsion-like drinking, with important relevance for h
96 nstem connections and alpha-1 NERs regulated compulsion-like intake and alcohol-only drinking (AOD).
97 subcortical projections from the AIC promote compulsion-like intake in rats and are recruited in heav
98 der stress, which is conceptually similar to compulsion-like responding (adaptive responding despite
99 of the transition from social consumption to compulsion-like seeking during relapse.
100 y may govern feeding more related to reward, compulsion, or generalized consumption than to energy ba
101 not found in other parts of the brain during compulsion, or in the BNST during noncompulsive behavior
102 ed by the phase of theta oscillations during compulsions, possibly reflecting pathological coupling o
103 ysthymia, anxiety, panic, phobia, obsession, compulsion, posttraumatic, care management, case managem
104 ic dopamine signaling is sufficient to drive compulsion, psychostimulants such as cocaine also boost
105 esponsible for improvement of obsessions and compulsions, rather than local impact at the stimulation
106                           It is thought that compulsions reflect a failure to override habitual behav
107 ems and the presence of other obsessions and compulsions related to their hoarding, such as fear of c
108 ring, arranging, counting, writing-rewriting compulsions, repetitive writing tics) and disinhibition
109 uggest that the motor circuits stimulated in compulsions represent only a subset of the parallel circ
110 and decreased during the initiation phase of compulsion, respectively, whereas beta increased after c
111 and decreased during the initiation phase of compulsion, respectively, whereas beta increased after c
112 7-positive patients had significantly higher compulsion scores than D8/17-negative patients.
113 ts with autism and may serve as a marker for compulsion severity within autism.
114 intain compulsions and is directly linked to compulsion severity.
115                   Subclinical obsessions and compulsions should be taken into account in research, in
116 , respectively, whereas beta increased after compulsion stopped.
117 , respectively, whereas beta increased after compulsion stopped.
118  behaviors, according to both the Yale-Brown compulsion subscale and CGI rating of obsessive-compulsi
119  Repetitive behaviors were measured with the compulsion subscale of the Yale-Brown Obsessive Compulsi
120                                    In mental compulsion subtypes, similar low-frequency increases wer
121 oral abnormalities uniquely resembling human compulsions, such as non-aggressive biting of cagemates
122 lsive disorder (OCD) involves obsessions and compulsions that cause impairment and distress, and whic
123 is characterized by recurrent obsessions and compulsions that create distress and interfere with dail
124 cluding generalized anxiety, depression, and compulsions that preceded the birth of their children.
125                                              Compulsion theory of addiction also has weak support.
126  cognitive distortions: 1) dichotomania, the compulsion to perceive quantities as dichotomous even wh
127             Alcoholism is characterized by a compulsion to seek and ingest alcohol, loss of control o
128 onically relapsing disorder characterized by compulsion to seek and take drugs and has been linked to
129 ically relapsing disorder characterized by a compulsion to seek and take drugs and the development of
130 ically relapsing disorder characterized by a compulsion to seek and take drugs, the development of de
131 ng 5-HT reversed the inherently high rate of compulsion transition with optogenetic dopamine self-sti
132                           Longer duration of compulsion was associated with later disengagement (HR 0
133 ical stimulation, in terms of suppression of compulsion, was significantly correlated with the percen
134 y aggressive and shameful thoughts), but not compulsions, was associated with help-seeking.
135                                              Compulsions were assessed with the Yale-Brown Obsessive
136                               Obsessions and compulsions were frequent in individuals with mental dis
137 e signal accelerated animals' transitions to compulsion, whereas inhibition delayed it.
138  understand the mechanisms of addictions and compulsions, which involve a failure of aversive outcome
139 l mechanisms that underlie the transition to compulsion with overarching learning theories, to outlin
140                                          The compulsion zone theory interprets BP simply as the maxim
141 statement paradigm within the context of the compulsion zone theory is a useful pharmacological bioas
142                                          The compulsion zone theory of cocaine self-administration wa
143               The thresholds delineating the compulsion zone were very stable and independent of the
144 nd upper limits, respectively, of a cocaine "compulsion zone".
145 ction while cocaine levels remain within the compulsion zone.
146 ing that SCH23390 also increased the cocaine compulsion zone.

 
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