ライフサイエンスコーパス: compulsive

1 scalation of alcohol drinking from casual to compulsive?

2 f obsessive-compulsive disorder (OCD), where compulsive actions are recognized as disproportionate, w

3 entify patterns of individual differences in compulsive "addiction-like" aggressive behavior.

4 avior, suggesting an evolutionary origin for compulsive aggression.

5 Compulsive alcohol consumption is a core, treatment-resi

6 for individual variance in vulnerability to compulsive alcohol drinking.

7 essive drinking, for which treatments exist, compulsive alcohol seeking is therefore another key feat

8 ually predicted the vulnerability to develop compulsive alcohol seeking, but only in subjects depende

9 Compulsive alcohol seeking, maintained in the face of th

10 ng predicts and underlies the development of compulsive alcohol seeking.

11 mine predicts and underlies the emergence of compulsive alcohol seeking.

12 this predicted the subsequent development of compulsive alcohol-seeking behavior.

13 c relapsing disorder that is associated with compulsive alcohol-seeking behavior.

14 d both alcohol seeking and alcohol intake in compulsive and non-compulsive rats, indicating its thera

15 disrupted NAc D2R signaling are involved in compulsive and perseverative feeding behaviors.

16 OCD is the key example of the 'obsessive-compulsive and related disorders', a group of conditions

17 We examined twenty low-compulsive and twenty high-compulsive participants, recr

18 tive behavior therapy for anxiety, obsessive-compulsive, and posttraumatic stress disorders and to ev

19 tive behavior therapy for anxiety, obsessive-compulsive, and posttraumatic stress disorders is unclea

20 yses revealed a positive correlation between compulsive behavior (measured with the Repetitive Behavi

21 isordered eating, and a factor consisting of compulsive behavior and intrusive thoughts.

22 t understanding highlights three elements of compulsive behavior as it applies to pathological overea

23 ation of targeted stimulation treatments for compulsive behavior disorders.

24 al regions in the pathology and treatment of compulsive behavior disorders.SIGNIFICANCE STATEMENT The

25 The presence of perseverative or compulsive behavior was more common in the TDP-B and TDP

26 ay be attributed to KOR-induced increases in compulsive behavior.

27 seems to be associated with the severity of compulsive behavior.

28 such as drug seeking, social withdrawal, and compulsive behavior.

29 n the putamen, which may contribute to their compulsive behavior.

30 hypocaloric feeding schedules (HFS) exhibit compulsive behavioral responses involving food anticipat

31 nate hypothalamic response and expression of compulsive behavioral responses involving meal anticipat

32 Compulsive behaviors (e.g., addiction) can be viewed as

33 l as a transdiagnostic impairment underlying compulsive behaviors and representing a promising therap

34 in significantly lower scores for obsessive-compulsive behaviors at 16 weeks.

35 Hyperactivity in striatum is associated with compulsive behaviors in obsessive-compulsive disorder (O

36 l may underlie deficits in stopping unwanted compulsive behaviors in the disorder.

37 asure of distress associated with preventing compulsive behaviors significantly correlated with TSPO

38 (+/-) mice manifested tic-like behaviors and compulsive behaviors that could be rescued by the tic-re

39 ined phenotypes included social interaction, compulsive behaviors, aggression, hyperactivity, anxiety

40 gest synaptic adhesion as a key component in compulsive behaviors, and show that targeted sequencing

41 e to both in vivo striatal hyperactivity and compulsive behaviors, and support a potential role for p

42 control over stereotyped and repetitive and compulsive behaviors, respectively.

43 Sapap3 knock-out (Sapap3-KO) mouse model of compulsive behaviors, which also exhibits hyperactivity

44 are often comorbid with the overlap based on compulsive behaviors.

45 glutamate transporter EAAC1 to the onset of compulsive behaviors.

46 be beneficial for decreasing stress-induced compulsive behaviors.

47 (most notably nicotine addiction) and other compulsive behaviors.

48 d structural imaging predictors of impulsive-compulsive behaviour (ICB) in de novo Parkinson's diseas

49 Tics and obsessive-compulsive behaviour (OCB) significantly improved over t

50 th improvement in tics or comorbid obsessive-compulsive behaviour and to predict clinical outcomes ac

51 For obsessive-compulsive behaviour, both targets showed that connectiv

52 improvements in tics and comorbid obsessive-compulsive behaviour, compare the networks across surgic

53 Impulse control disorders/other compulsive behaviours ('ICD behaviours') occur in Parkin

54 Thirty-one patients with impulsive compulsive behaviours and Parkinson's disease who donate

55 We emphasize the multidimensional nature of compulsive behaviours and the utility of computational m

56 The majority of patients with impulsive compulsive behaviours had dopamine dysregulation syndrom

57 ients with Parkinson's disease and impulsive compulsive behaviours had lower alpha-synuclein load and

58 Impulsive compulsive behaviours in Parkinson's disease have been l

59 Parkinson's disease cases without impulsive compulsive behaviours matched by age, sex, disease durat

60 duals with Parkinson's disease and impulsive compulsive behaviours was confirmed on western blotting.

61 ers and pathological groups characterized by compulsive behaviours, by posing it as an evidence accum

62 the ventral striatum resulting in impulsive compulsive behaviours.

63 t alcoholism and other addiction-related and compulsive behaviours.

64 e, caloric restriction (CR) induces bouts of compulsive binge feeding separated by prolonged fasting

65 Compulsive, binge eating of highly palatable food consti

66 ism as a novel pharmacological treatment for compulsive, binge eating.

67 e hypothesized that TAAR1 may have a role in compulsive, binge-like eating; we tested this hypothesis

68 by pathological uncertainty or doubt despite compulsive checking with impaired performance.

69 prefrontal cortex (PFC) it may contribute to compulsive cocaine intake.

70 hanol targets dorsolateral striatum to drive compulsive consumption is poorly understood.

71 , reduced ethanol consumption, and abrogated compulsive consumption of ethanol with the added bittera

72 in results in excessive barbering, obsessive compulsive digging and lack of cage exploration.

73 d phenotypic dimensions: a general impulsive-compulsive dimension; and two narrower phenotypes relate

74 Autism spectrum disorder (ASD), obsessive-compulsive disorder (OCD) and attention-deficit/hyperact

75 iated with compulsive behaviors in obsessive-compulsive disorder (OCD) and related illnesses, but it

76 behavioral alterations related to obsessive-compulsive disorder (OCD) and the role of TNFalpha and r

77 een linked with the development of obsessive-compulsive disorder (OCD) and tic disorders, a concept t

78 perseverative behaviors related to obsessive-compulsive disorder (OCD) and Tourette syndrome (TS).

79 The association between obsessive-compulsive disorder (OCD) and Tourette's/chronic tic dis

80 DSM provides distinct criteria for obsessive-compulsive disorder (OCD) and various types of anxiety d

81 icits in goal-directed behavior in obsessive-compulsive disorder (OCD) are caused by impaired frontos

82 utism spectrum disorder (ASD), and obsessive-compulsive disorder (OCD) are common neurodevelopmental

83 Anorexia nervosa (AN) and obsessive-compulsive disorder (OCD) are often comorbid and likely

84 tism spectrum disorders (ASDs) and obsessive compulsive disorder (OCD) are often comorbid with the ov

85 ion (DBS) for treatment-refractory obsessive-compulsive disorder (OCD) can be considered an establish

86 Patients with obsessive-compulsive disorder (OCD) can be described as cautious a

87 For a small percentage of obsessive-compulsive disorder (OCD) cases exhibiting additional ne

88 ion abnormalities in patients with obsessive-compulsive disorder (OCD) during both processes.

89 ion (DBS) for treatment refractory obsessive-compulsive disorder (OCD) has not been examined.

90 e and Compulsive Symptoms (OCS) or Obsessive Compulsive Disorder (OCD) in the context of schizophreni

91 Mode Network (DMN) deactivation in Obsessive Compulsive Disorder (OCD) in the transition between a re

92 anual of Mental Disorders (DSM-5), obsessive-compulsive disorder (OCD) included a new "tic-related" s

93 Obsessive-compulsive disorder (OCD) is a chronic and disabling con

94 Obsessive-compulsive disorder (OCD) is a debilitating neuropsychia

95 Obsessive-compulsive disorder (OCD) is a disabling condition, ofte

96 Obsessive-compulsive disorder (OCD) is a highly prevalent and chro

97 even when genomic data are limited.Obsessive-compulsive disorder (OCD) is a neuropsychiatric disorder

98 Obsessive-compulsive disorder (OCD) is a neuropsychiatric disorder

99 Obsessive-compulsive disorder (OCD) is a psychiatric disorder char

100 Obsessive compulsive disorder (OCD) is a severe illness that affec

101 Obsessive-compulsive disorder (OCD) is a severe, chronic neuropsyc

102 Obsessive-compulsive disorder (OCD) is associated with high risk o

103 Obsessive-compulsive disorder (OCD) is associated with increased m

104 Obsessive-compulsive disorder (OCD) is commonly associated with al

105 ath by suicide in individuals with obsessive-compulsive disorder (OCD) is largely unknown.

106 Obsessive-compulsive disorder (OCD) is prevalent and without adequ

107 structural covariance networks in obsessive-compulsive disorder (OCD) may provide clues to the patho

108 elatives of these SCZ patients, 13 obsessive-compulsive disorder (OCD) patients, 18 unaffected first-

109 urine and blood plasma samples of obsessive-compulsive disorder (OCD) patients.

110 ere, chronic, treatment-refractory obsessive-compulsive disorder (OCD) patients.

111 Autism spectrum disorder (ASD) and obsessive-compulsive disorder (OCD) share inhibitory control defic

112 Pediatric obsessive-compulsive disorder (OCD) sometimes appears rapidly, eve

113 Treatments for obsessive-compulsive disorder (OCD) tend to be of mixed efficacy b

114 h severe, debilitating symptoms of obsessive-compulsive disorder (OCD) that have proven refractory to

115 in the CSTC pathway is involved in obsessive compulsive disorder (OCD), a neuropsychiatric disorder c

116 tionships among Tourette syndrome, obsessive-compulsive disorder (OCD), and attention deficit hyperac

117 is a commonly occurring symptom in obsessive-compulsive disorder (OCD), and is associated with worse

118 rs (DDs), anxiety disorders (ADs), obsessive-compulsive disorder (OCD), and posttraumatic stress diso

119 y (CBT) is effective for pediatric obsessive-compulsive disorder (OCD), but non-response is common.

120 effective first-line treatment for obsessive-compulsive disorder (OCD), but only some patients achiev

121 ognitive behavioral treatments for obsessive-compulsive disorder (OCD), some patients continue to be

122 has a known impact on the risk for obsessive-compulsive disorder (OCD), there is also evidence that t

123 ated by the ego-dystonic nature of obsessive-compulsive disorder (OCD), where compulsive actions are

124 iatal dysfunction in the Sapap3-KO obsessive-compulsive disorder (OCD)-relevant mouse model, with M2

125 unction similar to humans with the obsessive-compulsive disorder (OCD)-spectrum disorder, trichotillo

126 ption for patients with refractory obsessive-compulsive disorder (OCD).

127 ent for severe treatment-resistant obsessive-compulsive disorder (OCD).

128 s an emerging treatment for severe obsessive-compulsive disorder (OCD).

129 in both major depression (MDD) and obsessive-compulsive disorder (OCD).

130 harmacological agents for treating obsessive-compulsive disorder (OCD).

131 le behavior influenced by anxiety, Obsessive Compulsive Disorder (OCD).

132 glutamate transporter EAAC1, with obsessive-compulsive disorder (OCD).

133 tational basis for OC symptoms and obsessive compulsive disorder (OCD).

134 s with severe treatment-refractory obsessive-compulsive disorder (OCD).

135 striatum in the neuropathology of obsessive compulsive disorder (OCD).

136 it did not predict the severity of obsessive-compulsive disorder (R(2) = .11%, p empirical = .11, Q =

137 opsychiatric conditions, including obsessive-compulsive disorder and addiction.

138 healthy individuals in depression, obsessive-compulsive disorder and attention-deficit hyperactivity

139 orders that include schizophrenia, obsessive-compulsive disorder and bipolar disorder (BD).

140 r limb of the internal capsule for obsessive-compulsive disorder and possibly other psychiatric disor

141 lity of major depressive disorder, obsessive-compulsive disorder and schizophrenia within the putamen

142 es, including anxiety, depression, obsessive-compulsive disorder and schizophrenia, are frequent in p

143 ake inhibitors for childhood-onset obsessive-compulsive disorder and the anxiety disorders, suggest a

144 europsychiatric conditions such as obsessive-compulsive disorder and Tourette syndrome remain uncerta

145 to 'walk away', whereas those with obsessive compulsive disorder become more deliberative and cautiou

146 In contrast, patients with obsessive-compulsive disorder had slower processing speeds during

147 608 genes potentially involved in obsessive-compulsive disorder in human, dog, and mouse.

148 Deep brain stimulation for obsessive-compulsive disorder is a rapidly developing treatment st

149 Obsessive-compulsive disorder is a severe and disabling psychiatri

150 Obsessive-compulsive disorder is a severe psychiatric disorder lin

151 A striking observation in obsessive-compulsive disorder is that patients know that their obs

152 Thus, subjects with obsessive compulsive disorder on subthalamic stimulation may be le

153 rge with the effective contacts in obsessive compulsive disorder patients localized within the anteri

154 f posttraumatic stress disorder or obsessive-compulsive disorder to date, although there is evidence

155 ence accumulation in subjects with obsessive-compulsive disorder towards a functional less cautious s

156 female) with treatment-refractory obsessive-compulsive disorder undergoing deep brain stimulation ta

157 polar disorder, schizophrenia, and obsessive-compulsive disorder were highly correlated (r = .443 to

158 ional impulsivity in subjects with obsessive compulsive disorder who have undergone deep brain stimul

159 iple surgical targets for treating obsessive-compulsive disorder with deep brain stimulation (DBS) ha

160 patients with treatment-resistant obsessive-compulsive disorder, a condition linked to abnormalities

161 ost-traumatic stress disorder, and obsessive-compulsive disorder, and determine whether any such alte

162 iated with Tourette's syndrome and obsessive-compulsive disorder, and dysfunction of corticostriatal

163 ) anorexia nervosa, schizophrenia, obsessive-compulsive disorder, and education years are negatively

164 sease, essential tremor, dystonia, obsessive-compulsive disorder, and epilepsy, chronic electrophysio

165 tress disorder, anxiety disorders, obsessive-compulsive disorder, and substance use disorders, and me

166 circuit in the pathophysiology of obsessive-compulsive disorder, and suggest a neural systems model

167 ers, major depressive disorder and obsessive-compulsive disorder, arguably characterized by behaviour

168 urodevelopmental disorders such as obsessive-compulsive disorder, attention deficit hyperactivity dis

169 y of tics, and symptom severity of obsessive-compulsive disorder, attention-deficit/hyperactivity dis

170 , bipolar disorder, schizophrenia, obsessive-compulsive disorder, autism spectrum disorders and even

171 dition to schizophrenia, including obsessive-compulsive disorder, autism, and alcoholism, occur more

172 l psychiatric disorders, including obsessive-compulsive disorder, major depressive disorder, posttrau

173 traumatic stress disorder, n = 91; obsessive-compulsive disorder, n = 92) alongside n = 201 healthy c

174 order with or without agoraphobia, obsessive-compulsive disorder, or posttraumatic stress disorder.

175 bia, generalized anxiety disorder, obsessive-compulsive disorder, or social anxiety disorder were ran

176 with higher levels associated with obsessive-compulsive disorder, schizophrenia, and anxiety disorder

177 nxiety, major depressive disorder, obsessive-compulsive disorder, schizophrenia, and Parkinson's dise

178 vasive neuromodulation targets for obsessive-compulsive disorder.

179 tia, alcohol-induced delusions and obsessive-compulsive disorder.

180 iated with Tourette's syndrome and obsessive-compulsive disorder.

181 er, major depressive disorder, and obsessive-compulsive disorder.

182 patients with treatment-refractory obsessive-compulsive disorder.

183 come of deep brain stimulation for obsessive-compulsive disorder.

184 ll as a new clinical indication in obsessive-compulsive disorder.

185 epressive disorders; 9% (7-10) for obsessive-compulsive disorder; 5% (3-6) for bipolar disorders; and

186 stant to extinction as observed in obsessive compulsive disorders (OCD).

187 according to the Questionnaire for Impulsive-Compulsive Disorders in Parkinson's Disease (QUIP).

188 e sleepiness, anxiety, depression, impulsive-compulsive disorders, blood pressure, urate, and rapid e

189 ilt proneness might be relevant to obsessive compulsive disorders.

190 We longitudinally tracked the development of compulsive drinking across a binge-drinking experience i

191 ing latent traits in alcohol consumption and compulsive drinking despite equal prior exposure to alco

192 ations for understanding the neural basis of compulsive drinking in alcohol use disorder.

193 ms that gate the transition from moderate to compulsive drinking remain poorly understood.

194 haracterize the addiction phenotype, such as compulsive drug consumption, impaired self-control, and

195 n is a long-lasting disease characterized by compulsive drug intake mediated in part by neuronal and

196 allmark of addictive behaviors that underlie compulsive drug seeking and taking in humans.

197 ought to allow drug-associated cues to drive compulsive drug seeking and taking.

198 characterized by impaired hedonic capacity, compulsive drug seeking, and high stress.

199 ction is a chronic disorder characterized by compulsive drug seeking, and involves repetitive cycles

200 s is causally involved in the development of compulsive drug seeking.

201 ial but the relationship of these changes to compulsive drug taking and abstinence is not clear.

202 nal states during drug abstinence that drive compulsive drug taking and seeking.

203 becomes sensitized during the development of compulsive drug taking with repeated withdrawal.

204 Under laboratory conditions linked to compulsive drug use and heightened relapse risk, drug om

205 ent of aDLS in the switch from controlled to compulsive drug use in vulnerable individuals remains to

206 g seeking, and involves repetitive cycles of compulsive drug use, abstinence, and relapse.

207 addiction is a chronic relapsing disorder of compulsive drug use.

208 glia, circuitry associated with habitual and compulsive drug use.

209 ke drugs; whereas "novelty-seeking" predicts compulsive drug-seeking behavior.

210 mphasize the conceptual distinctions between compulsive drug-seeking behaviour and compulsive drug-ta

211 Cocaine addiction is associated with compulsive drug-seeking, and exposure to the drug or to

212 etween compulsive drug-seeking behaviour and compulsive drug-taking behaviour (that is, use).

213 ne addiction is characterized by patterns of compulsive drug-taking, including preoccupation with obt

214 d altered appetitive motivation that induces compulsive eating and contributes to obesity.

215 omponents and the mechanisms contributing to compulsive eating are not yet clearly defined or underst

216 Here, we present evidence to relate compulsive eating behavior and addiction and to characte

217 Compulsive eating behavior is a transdiagnostic construc

218 Compulsive eating behavior is hypothesized to be driven

219 ay in turn contribute to the perpetuation of compulsive eating behavior.

220 mygdala, and the prefrontal cortex result in compulsive eating behaviors.

221 Compulsive eating characterizes many binge-related eatin

222 Measures of compulsive eating severity also directly correlated to l

223 A major need to improve understanding of compulsive eating through the integration of complex mot

224 The most prevalent subtype was compulsive eating, followed by punding, compulsive sexua

225 p pathological behaviours, such as gambling, compulsive eating, shopping, or disinhibited sexual beha

226 tandard chow with palatable diet, a model of compulsive eating.

227 s in the DA and DAT systems in this model of compulsive eating.

228 neurons are necessary for the development of compulsive ethanol consumption, we selectively ablated t

229 interneuron population as a key component of compulsive ethanol consumption.

230 t ethanol consumption paradigm followed by a compulsive ethanol drinking assay.

231 Hoxb8 mutant mice exhibit compulsive grooming and hair removal dysfunction similar

232 eversal impairments were not correlated with compulsive grooming severity.

233 d effortful responding for high-fat diet and compulsive grooming, whereas group-housed female mice di

234 xtended access to heroin, an animal model of compulsive heroin taking.

235 ereating underlying some forms of obesity is compulsive in nature and therefore contains elements of

236 ontribute to the transition from moderate to compulsive intake of cocaine.

237 to high levels of intoxication that leads to compulsive intake, the loss of control in limiting intak

238 ed cocaine intake, decreased motivation, and compulsive-like behavior to acquire cocaine, and it faci

239 to LHA (GABA) neurons, but without inducing compulsive-like behaviors, which we propose to require d

240 its that contribute to hypofrontality and to compulsive-like cocaine intake in addiction, and documen

241 self-administration to model moderate versus compulsive-like cocaine taking.

242 transmission within the CeA is implicated in compulsive-like cocaine-seeking.

243 The role of HCRT in the persistence of compulsive-like cocaine-taking has yet to be fully eluci

244 dult rodent brain) during abstinence blocked compulsive-like context-driven methamphetamine reinstate

245 ted with increased anxiety-like behavior and compulsive-like drinking in dependent rats.

246 e cell neurons (GCNs) in the DG and produced compulsive-like drug reinstatement.

247 e of adult neurogenesis during abstinence in compulsive-like drug reinstatement.

248 ditioned withdrawal to facilitate relapse to compulsive-like drug seeking.

249 We identified compulsive-like eating behavior in female rats through p

250 relin that differentiated rats with the most compulsive-like eating behavior.

251 Collectively, this novel model of compulsive-like eating symptoms demonstrates adaptations

252 Furthermore, RO5256390 fully blocked compulsive-like eating when the palatable diet was offer

253 o the nucleus accumbens as modulating highly compulsive-like food self-administration behaviors that

254 of conditioned negative affect in sustaining compulsive-like heroin seeking and taking and providing

255 We speculate that the striking difference in compulsive-like locomotor behavior is also based on diff

256 Activation of LHA (GABA) neurons induced compulsive-like locomotor behavior; while LHA (Gal) neur

257 Obsessive compulsive (OC) symptoms involve excessive information g

258 ibutes to the development and persistence of compulsive opioid-seeking behavior.

259 al coincident neurological underpinnings for compulsive overeating and drug addiction.

260 te to the impaired emotional response and/or compulsive overeating characteristic of this disease.

261 motion detection paradigm revealed that high-compulsive participants had a reduced metacognitive abil

262 y, we investigate metacognitive abilities in compulsive participants using computational models, wher

263 amined twenty low-compulsive and twenty high-compulsive participants, recruited from a large populati

264 evidence was accumulated more slowly in high compulsive participants.

265 Addiction is characterized by a compulsive pattern of drug seeking and consumption and a

266 Compulsive patterns of drug use are thought to be the co

267 he general dimension, This general impulsive-compulsive phenotype may reflect a quantitative liabilit

268 from bi-factor modelling of 33 impulsive and compulsive problem behaviours.

269 l vulnerability to switch from controlled to compulsive, punishment-resistant alcohol seeking.

270 This effect was strongest in the most compulsive, punishment-resistant rats, and reinstatement

271 ing and alcohol intake in compulsive and non-compulsive rats, indicating its therapeutic potential to

272 quantified based on the Yale-Brown Obsessive Compulsive Scale (Y-BOCS) and the impact of complication

273 change in scores on the Yale-Brown Obsessive Compulsive Scale (Y-BOCS) from baseline until the 12-mon

274 with level I evidence (Yale-Brown Obsessive Compulsive Scale (Y-BOCS) score improved 37% during stim

275 the slope of change in Yale-Brown Obsessive-Compulsive Scale (Y-BOCS) scores across treatment.

276 as determined using the Yale-Brown Obsessive Compulsive Scale (YBOCS), and the primary efficacy endpo

277 lity, assessed with the Yale-Brown Obsessive Compulsive Scale (YBOCS).

278 modified version of the Yale-Brown Obsessive Compulsive Scale for hypochondriasis (H-YBOCS-M).

279 The Yale-Brown Obsessive Compulsive Scale measure of distress associated with pre

280 ased OCD symptoms (mean Yale-Brown Obsessive Compulsive Scale reduction 33%, 40% full responders) and

281 -refractory OCD (5 men; Yale-Brown Obsessive Compulsive Scale score >32) entered double-blind counter

282 ders (>=35% decrease of Yale-Brown Obsessive Compulsive Scale score).

283 s assessed by change in Yale-Brown Obsessive Compulsive Scale scores.

284 igher on the Children's Yale-Brown Obsessive Compulsive Scale, modified for pervasive developmental d

285 y efficacy measure, the Yale-Brown Obsessive Compulsive Scale, of >/=35% over the 3-year follow-up pe

286 oms, measured using the Yale-Brown Obsessive Compulsive Scale-Observer-Rated (Y-BOCS-OR) at 3, 6, and

287 were assessed with the Yale-Brown Obsessive Compulsive Scale.

288 inforcement until some individuals developed compulsive seeking behavior.

289 ehavior, including escalated use, tolerance, compulsive seeking, and dependence.

290 ng gambling disorder, binge eating disorder, compulsive sexual behaviour, and compulsive shopping occ

291 was compulsive eating, followed by punding, compulsive sexual behaviour, gambling and buying disorde

292 g disorder, compulsive sexual behaviour, and compulsive shopping occur in about 17% of patients with

293 as hypersexuality, pathological gambling or compulsive shopping.

294 A reduction in obsessive-compulsive symptom severity may result not only from dir

295 Obsessive and Compulsive Symptoms (OCS) or Obsessive Compulsive Disord

296 Impulsive and compulsive symptoms are common, tend to co-occur, and co

297 ive urgency, distress intolerance, obsessive-compulsive symptoms, disordered eating, and a factor con

298 deficits or depressive, manic, or obsessive-compulsive symptoms.

299 tional use, a subset of individuals develops compulsive use that does not stop even in light of major

300 rcing effects, transition from occasional to compulsive use, withdrawal-associated negative affective