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1 re observed for a larger multimeric complex (Concanavalin A).
2 ssessed after perfusion with FITC-conjugated concanavalin A.
3  separate group of mice by administration of concanavalin A.
4  of the second through sixth generations and Concanavalin A.
5 pon aggregation of unilamellar vesicles with concanavalin A.
6 ssessed by labeling adherent leukocytes with concanavalin A.
7 d has a surface that is capped by the lectin concanavalin A.
8  which is only manifested in the presence of concanavalin A.
9 te and sodium azide, and one model biotoxin, concanavalin A.
10 th immobilized Sambucus nigra agglutinin and concanavalin A.
11 at were resistant to the cytotoxic action of concanavalin A.
12 - and bivalent ligands for the legume lectin concanavalin A.
13 10 is inducible by interferons (IFNs) and by concanavalin A.
14 after stimulation of cells with the mitogen, concanavalin A.
15 n of peripheral blood mononuclear cells with concanavalin A.
16 cation exchange column and was shown to bind concanavalin A.
17 ho-proliferative response to alloantigens or concanavalin A.
18 tic liver damage induced by the injection of concanavalin A.
19 ose-containing bath solution or treated with concanavalin A.
20 ction in response to either C. pneumoniae or concanavalin A.
21 d Th-2 cytokine responses to SME antigen and concanavalin A.
22 ion-induced programmed cell death induced by concanavalin A.
23 ting the enzyme's aggregation induced by the concanavalin A.
24  binding of the polymers to the plant lectin concanavalin A.
25 s not evident after 48-hour stimulation with concanavalin-A.
26 8.6 kDa), and the tetrameric protein complex Concanavalin A (103 kDa).
27          Pretreatment of HEK-hCaR cells with concanavalin A (250 microg/ml) to block CaR internalizat
28 GC-5 cells were differentiated with succinyl concanavalin A (50 microg/mL) and transferred to a press
29                                              Concanavalin A, a lectin selective for high mannose and
30                                              Concanavalin A, a lectin that binds glucose and mannose
31 ectin binding experiments using succinylated concanavalin A, a plant lectin with high affinity for ma
32 scriptional regulation of CSF-2 and CSF-3 in concanavalin A-activated MSCs requires MT1-MMP signaling
33       GPI-IL-12-induced the proliferation of concanavalin A-activated T cells and induced IFN-gamma s
34 ene (aSMase(-/-)), we found that thymocytes, concanavalin A-activated T cells, and lipopolysaccharide
35                    Mannosidase digestion and concanavalin A adsorption indicate that the terminal res
36 eted cryptococcal antigens were separated by concanavalin A affinity chromatography into adherent (ma
37 tionation, diethylaminoethyl chromatography, concanavalin A affinity chromatography, and chromatofocu
38                          APS was purified on concanavalin A affinity columns to minimize the loss of
39 valent affinity capture using NeutrAvidin or concanavalin A agarose beads or directly via covalent co
40  was also lost when they were incubated with concanavalin A-agarose beads, suggesting that the inhibi
41                         After binding to the concanavalin A-agarose beads, the inhibitor in BAL fluid
42 f oocytes with 20% sucrose and 250 microg/ml concanavalin A, agents that inhibit the endocytosis of R
43 ctional valency of a triantennary analog for concanavalin A and D. grandiflora lectin are observed.
44 rate analogs to the Man/Glc-specific lectins concanavalin A and Dioclea grandiflora lectin was invest
45 a of infected insect cells using immobilized concanavalin A and immobilized BACE inhibitor, P10-P4' S
46   B10.BR splenic lymphocytes stimulated with concanavalin A and interleukin 2 were infected with a re
47 from broccoli flower buds was purified using concanavalin A and ion-exchange chromatography.
48                               Here, by using Concanavalin A and LC-MS analysis, we identified a novel
49                                              Concanavalin A and lectins from pea and lentil, also man
50 nt experimental hepatitis models, induced by concanavalin A and Listeria monocytogenes.
51 , T cell proliferation upon stimulation with concanavalin A and phytohemagglutinin A was only 40-50%
52 ion lymphocytes were examined in response to concanavalin A and purified protein derivative (PPD) in
53 or the ability to proliferate in response to concanavalin A and purified protein derivative (PPD) in
54 tely 100 kDa, contained sugars that bound to concanavalin A and ricin.
55  protein receptors such as the plant lectins concanavalin A and the highly toxic mistletoe Viscum alb
56 her mannose/glucose binding lectins, such as concanavalin A and the pea, lentil and Calystegia sepium
57  in C57BL/6 mice by intravenous injection of concanavalin A and then analyzed liver concentrations of
58 unction of DR signaling in T cell-dependent (concanavalin A) and independent (lipopolysaccharide/gala
59 tures of the protein complexes streptavidin, concanavalin A, and C-reactive protein under charge redu
60 superoxide dismutase, dimeric and tetrameric concanavalin A, and heptameric GroES and Gp31; ranging i
61 asurements were performed with cytochrome c, concanavalin A, and poly-L-lysine, biomolecules that exh
62 y from Escherichia coli lipopolysaccharides, concanavalin A, and Pseudomonas aeruginosa exotoxin A.
63 ulin and enolase), tetrameric (streptavidin, concanavalin A, and pyruvate kinase), and pentameric (C-
64 an inducible pattern on T cells activated by concanavalin A, anti-CD3 mAb in combination with anti-CD
65 r induction of selective EC injury using the concanavalin A/anti-concanavalin A model and after ische
66 nti-bovine serum albumin (BSA) antibody, and concanavalin A are embedded in distinct regions of a 7.5
67 that of the wild type and did not react with concanavalin A as did wild-type LAM.
68 ives supported the integration of the lectin concanavalin A as well as the enzymes alpha1-2,3 mannosi
69                       MIPP co-localized with concanavalin A at the endoplasmic reticulum, suggesting
70 suggesting that the polymers bind the lectin concanavalin A at the same site as natural carbohydrates
71 investigated by bromelain IgE inhibition and concanavalin A binding assays using sera of cypress poll
72 th a putative sequence tract RYRY that mimic concanavalin A-binding glycans.
73 n chromium-release assays against a panel of Concanavalin A blast targets.
74 ng BiP/Grp78 messenger RNA (mRNA) in splenic concanavalin A blasts.
75 l activation, as stimulation of T cells with concanavalin A, but not phorbol 12-myristate 13-acetate
76        A model of reactive lymph nodes after concanavalin A challenge served as an additional control
77 ver, CD160(-/-) mice are more susceptible to Concanavalin A challenge, and display elevated serum AST
78 ZM-INVINH1 with the glycoprotein fraction by concanavalin A chromatogaphy suggests that ZM-INVINH1 in
79                                              Concanavalin A chromatography effectively enriched and p
80                                     Finally, concanavalin A-coated colloids were allowed to adsorb on
81         The two gammaGTases were resolved by concanavalin A (Con A) affinity chromatography, indicati
82 s been created using Alexa Fluor 647-labeled concanavalin A (Con A) and a fourth-generation PAMAM Ale
83 e material was derivatized with two lectins, Concanavalin A (Con A) and Aleuria aurantia lectin (AAL)
84  monitoring the specific interaction between Concanavalin A (Con A) and D-(+)-mannose.
85 ion of the electrical synapse was blocked by concanavalin A (Con A) and dynamin inhibitory peptide (D
86 e data available on the binding constants of Concanavalin A (Con A) and glycogen and Con A-mannan usi
87 bohydrate platform for the immobilization of Concanavalin A (Con A) and is capable of LPS binding mea
88 ew as the sensing probes for lectins such as concanavalin A (Con A) based on the molecular recognitio
89 eral crystal structures of the legume lectin concanavalin A (Con A) bound to closely related carbohyd
90 set of serial lectin columns consisting of a concanavalin A (Con A) column coupled to an SNA column f
91 sed to measure the adhesive strength between concanavalin A (Con A) coupled to an AFM tip and Con A r
92  of alpha-Man to the mannose-specific lectin concanavalin A (Con A) even though homogeneous beta-Lact
93 el on their surfaces was probed using lectin concanavalin A (Con A) from Canavalia ensiformis.
94                         Hepatitis induced by concanavalin A (Con A) in mice is well known to be a T-l
95                                 Injection of concanavalin A (Con A) into mice recapitulates the histo
96                                              Concanavalin A (Con A) kills procyclic (insect) forms of
97 vation and expansion were studied by using a concanavalin A (Con A) liver injury model followed by pa
98 g the direct pili-mannose binding as well as Concanavalin A (Con A) mediated lipopolysaccharides (LPS
99 ial, we have developed a nanoscale chelating Concanavalin A (Con A) monolithic capillary prepared usi
100                        These sensors utilize Concanavalin A (Con A) protein hydrogels with a 2D PC em
101 ntaining 2-D photonic crystal sensor detects Concanavalin A (Con A) through shifts in the 2-D diffrac
102 ible binding of a mobile fluorophore-labeled Concanavalin A (Con A) to immobile pendant glucose moite
103 -SNPs with fluorescein isothiocyanate (FITC)-concanavalin A (Con A) was determined using a fluorescen
104 sembled monolayer in combination with lectin concanavalin A (Con A) was used as molecular recognition
105          Conformational structure changes in concanavalin A (Con A), a legume lectin protein which is
106 achite green (MG-dextran), was conjugated to concanavalin A (Con A), an enzyme with specific affinity
107 ns including Maackia amurensis lectin (MAL), concanavalin A (Con A), and wheat germ agglutinin (WGA).
108  were cultured in the absence or presence of concanavalin A (Con A), phorbol myristate acetate (PMA)/
109          Construction of biosensors based on Concanavalin A (Con A), Sambucus nigra agglutinin type I
110  excellent selectivity to its target lectin, concanavalin A (Con A), surpassing the formerly used lin
111 lines, we identify a plant-derived compound, Concanavalin A (Con A), which differentially kills p53-n
112 ran-coated nanoparticles are aggregated with concanavalin A (Con A), which results in a significant s
113                                              Concanavalin A (Con A), which selectively blocks KAR des
114 mmune or viral hepatitis, and of mice during concanavalin A (Con A)-induced hepatitis (CIH).
115                                              Concanavalin A (Con A)-induced injury is an established
116                                      We used concanavalin A (Con A)-induced liver injury to study the
117 dhesion of the alpha-mannose-specific lectin concanavalin A (Con A).
118 ramide (alphaGalCer), anti-CD3 antibody, and concanavalin A (Con A).
119 nsity on the clustering of a model receptor, concanavalin A (Con A).
120 nd clustering of a model protein, the lectin concanavalin A (Con A).
121 ell lymphoma cells (TIB 155) stimulated with concanavalin A (Con A).
122 tive protein (CRP), transthyretin (TTR), and concanavalin A (Con A).
123 nsing through polydopamine (PDA)-immobilized concanavalin A (Con A).
124 ondrial O2 consumption) in mice treated with concanavalin A (Con A).
125  UV-vis spectroscopy during its binding with concanavalin A (Con A).
126 ) kainate receptors, two unrelated proteins, concanavalin-A (Con-A) and postsynaptic density protein
127 riefly, the well-known lectin macromolecule (concanavalin A, Con A) monolayer was functionalized on 3
128 -HIV antibody 2G12 (shortest distance 31 A), concanavalin A (ConA) (shortest distance 72 A), RCA120 (
129                                   The lectin concanavalin A (ConA) activates immune cells, resulting
130  rate, compared to wild-type (wt) mice, upon concanavalin A (ConA) administration.
131 y, including T/NKT cell hepatitis induced by concanavalin A (ConA) and alpha-galactosylceramide (alph
132 CM electrodes present a higher adsorption of Concanavalin A (ConA) and Bovine Serum Albumin (BSA) pro
133 arbohydrates to the Man/Glc-specific lectins concanavalin A (ConA) and Dioclea grandiflora lectin (DG
134 arbohydrates to the Man/Glc-specific lectins concanavalin A (ConA) and Dioclea grandiflora lectin (DG
135 binding of the carbohydrate-binding proteins concanavalin A (ConA) and jacalin to arrays composed of
136 cterized chemical inhibitors of endocytosis: concanavalin A (conA) and phenylarsine oxide (PAO).
137 UVL patients exhibited normal ATP release to Concanavalin A (ConA) and phytohemagglutinin (PHA; 190+/
138 K using the receptor internalization blocker concanavalin A (ConA) and the carboxyl terminus-truncate
139                         The jack bean lectin concanavalin A (ConA) and the Dioclea grandiflora lectin
140 d from a detailed analysis of the binding of concanavalin A (ConA) and wheat germ agglutinin (WGA) to
141 on, cells treated with ICZ increased surface concanavalin A (ConA) binding, corroborating an increase
142                                              Concanavalin A (ConA) bound to PTP1 and to the polar tub
143 ated in T cell-mediated hepatitis induced by concanavalin A (ConA) but are less extensively elevated
144                                              Concanavalin A (ConA) causes immune cell-mediated liver
145 anisms in optical glucose sensors based upon Concanavalin A (ConA) has tended to prevent the sensors'
146 mpetitive binding assays based on the lectin Concanavalin A (ConA) have displayed significant potenti
147         Competitive binding assays utilizing concanavalin A (ConA) have the potential to be the basis
148                                              Concanavalin A (ConA) inhibited phi C31 infection of S.
149 peptide reacted strongly with both lotus and concanavalin A (ConA) lectins, it bound to lotus stronge
150 robe was designed by noncovalent assembly of concanavalin A (ConA) on gold nanoparticles (AuNPs).
151 ects of T cell-mediated hepatitis induced by concanavalin A (ConA) on the regenerative response in vi
152 ls of hepatitis, including administration of concanavalin A (ConA) or alpha-galactosyl-ceramide, whic
153 in unstimulated or lipopolysaccharide (LPS), concanavalin A (ConA) or phytohemagglutinin (PHA) stimul
154          The treatment of splenic cells with concanavalin A (ConA) plus CT enhanced the production of
155 pha-D-mannopyranoside residues to the lectin concanavalin A (ConA) show increasing negative cooperati
156  cells showed enhanced responses to in vitro concanavalin A (ConA) stimulation when compared with WT
157 ide (LPS) plus D-galactosamine (D-Galn), and concanavalin A (ConA) to identify the function of extrac
158    Here, we show that treatment of mice with concanavalin A (ConA) to induce liver injury triggered a
159                               The binding of Concanavalin A (ConA) to mannose-functionalized self-ass
160 l apoptosis induced by the administration of concanavalin A (ConA) to pregnant mothers.
161                                              Concanavalin A (ConA) was used as an activator of lympho
162 opaeus (gorse, furze), Triticum vulgaris and Concanavalin A (ConA) was used for probes to evaluate bi
163  coefficients that showed mannose to bind to concanavalin A (conA) with 3.7 times greater affinity th
164 e binding affinity of mannose and glucose to concanavalin A (ConA), a 106 KDa homotetramer protein, i
165 ension of macroporous hydrogel particles and concanavalin A (ConA), a glucose-specific lectin, that a
166                                Starting from concanavalin A (ConA), a mannose (Man)-binding protein,
167 tein, PZR displays a strong association with concanavalin A (ConA), a member of the plant lectin fami
168 tal model of autoimmune hepatitis induced by concanavalin A (ConA), a process involving T cell activa
169                     MSCs were activated with concanavalin A (ConA), a Toll-like receptor (TLR)-2 and
170 feration and IL-2 production induced by PHA, concanavalin A (conA), and anti-TCR MAb.
171 oteins, namely, transthyretin (TTR), avidin, concanavalin A (conA), and human serum amyloid P compone
172                     In situ competition with concanavalin A (ConA), another high-mannose specific lec
173                                     By using concanavalin A (ConA), as a recognition template, peptid
174                 This was followed by binding concanavalin A (ConA), glucose oxidase (GOx), and Au nan
175  which consists of three mixtures of lectins concanavalin A (ConA), jacalin (JAC), and wheat germ agg
176 ure induced by Fas-agonistic antibody (Jo2), concanavalin A (ConA), or D-galactosamine/lipopolysaccha
177  and lipopolysaccharide (LPS), or the lectin concanavalin A (ConA), suggesting that physiologic level
178 tested with several plant lectins, including concanavalin A (conA), Vicia villosa isolectin B4 (VVL-B
179     On the basis of the model system glucose-Concanavalin A (ConA), we explore the application of Tra
180 tosamine (GaIN)/lipopolysaccharide (LPS) and concanavalin A (ConA)-induced acute liver failure (ALF),
181 lated by natural killer T (NKT) cells during concanavalin A (ConA)-induced acute liver injury.
182 ore D-Gal/LPS-induced FH and before or after concanavalin A (ConA)-induced FH.
183 ated the effects of THC in a murine model of concanavalin A (ConA)-induced hepatitis.
184 o chronic choline-deficient diet exacerbates concanavalin A (ConA)-induced liver hepatitis, which is
185  of the molecular and cellular mechanisms of concanavalin A (ConA)-induced liver injury have provided
186 ) mice, germ-free (GF) mice are resistant to Concanavalin A (ConA)-induced liver injury.
187 y susceptible to liver destruction following concanavalin A (ConA)-induced T cell activation.
188 DTA mice) exhibited enhanced lymph node (LN) concanavalin A (ConA)-induced Th1 responses after tick i
189 ingly, NK cells mediated hypersensitivity to concanavalin A (ConA)-mediated hepatitis in GNMT(-/-) mi
190 to Fas-specific antibody or co-cultured with concanavalin A (ConA)-stimulated hepatic mononuclear cel
191                                              Concanavalin A (ConA)-stimulated peripheral blood mononu
192  increased production of interferon-gamma by concanavalin A (ConA)-stimulated spleen T cells and expr
193 d interferon-gamma (IFN-gamma) production in concanavalin A (conA)-stimulated spleen T cells, and dim
194  sensitive to apoptosis upon activation with concanavalin A (ConA).
195 from autoimmune hepatitis induced by mitogen concanavalin A (ConA).
196 P) and then with the Fas agonist Jo2 or with concanavalin A (ConA).
197 ere identified by perfusion with fluorescent concanavalin A (ConA).
198 cose- and mannose-specific binding protein - Concanavalin A (ConA).
199 chemical displacement sensor for the protein concanavalin A (ConA).
200 , nanogold particles (NGPs), antibodies, and Concanavalin A (ConA).
201 er injury in the lipopolysaccharide/GalN and concanavalin A (ConA)/GalN models, but not in a ConA-onl
202      Activation of CD8(+) T lymphocytes with concanavalin A (ConA)/interleukin-2 (IL-2), and activati
203                                   The lectin concanavalin A conjugated to colloidal gold particles wa
204 that treatment of OAT1-expressing cells with concanavalin A, depletion of K(+) from the cells, or tra
205       Circular dichroism, mass spectrometry, concanavalin A detection, immunoblotting, enzyme-linked
206                    Blocking endocytosis with concanavalin A eliminated the accumulation of fluorescen
207 protease domain of MDC9, Western analysis of concanavalin A-enriched glomerular microsomal extracts d
208 uired for TNFalpha-induced injury induced by concanavalin A/GalN but not by ConA alone.
209 ulin G, avidin:biotin, antibody:antigen, and concanavalin A:glycoprotein interactions are used to dem
210 or LcL were compared with those obtained for concanavalin A i.e. lectin, which interacts with the car
211              Following enzymatic hydrolysis, concanavalin A, immobilized or soluble, was added to the
212 ttenuated the suppression of the response to concanavalin A in immunized mice, providing further evid
213 s attenuated by blockade of endocytosis with concanavalin A, indicating a critical role for internali
214 ruited iNKT cells were anergic and prevented concanavalin A-induced (ConA-induced) hepatitis by speci
215 imicrobial susceptibility assay based on the concanavalin A-induced clustering of dextran-coated gold
216  that myosin phosphorylation is critical for concanavalin A-induced gathering of surface receptors.
217                                     Instead, concanavalin A-induced hepatitis was completely prevente
218 endogenous Gal-1 protected the liver against concanavalin A-induced hepatitis with the B6 genetic bac
219 henotype of these cells, reduced severity of concanavalin A-induced hepatitis, and alterations in the
220 ver regeneration, and in the murine model of concanavalin A-induced liver inflammation.
221                                   Similarly, concanavalin A-induced liver injury, where type 2 cytoki
222   These findings were confirmed in mice with concanavalin A-induced liver injury.
223 vents and treats both IL12-, IFN-gamma-, and concanavalin A-induced liver toxicity.
224 ion in A549 human lung epithelial cells, and concanavalin A-induced monocyte proliferation.
225  K12-Fc inhibited in a dose-dependent manner concanavalin A-induced proliferation, but not anti-TcRal
226 mal (BLV-negative) cows and had no effect on concanavalin A-induced proliferation.
227 ot wild type RLC almost completely abolished concanavalin A-induced receptor cap formation.
228 y, IL-22TG mice were completely resistant to concanavalin A-induced T cell hepatitis with minimal eff
229         Neutralization of CXCL10 ameliorated concanavalin A-induced tissue injury in vivo, which was
230                              The loss of the concanavalin A-induced, but not the lipopolysaccharide-i
231          Upon transplantation into mice with concanavalin-A-induced acute liver failure and fatal met
232 more, a non-selective endocytosis inhibitor, concanavalin A, inhibited the internalization of wild ty
233         Thymocyte proliferative responses to concanavalin A + interleukin-2 were also significantly d
234 nnosylated conjugated polymer (sugar-PPE) by Concanavalin A is positively dependent upon sugar-PPE co
235            Leukocyte adhesion was assayed by concanavalin A labeling.
236  were simultaneously quantified by combining concanavalin A lectin (ConA) perfusion labeling with a f
237 asured by fluorescein isothiocyanate-coupled concanavalin A lectin and acridine orange labeling.
238 res show surprisingly high affinities toward Concanavalin A lectin receptor in comparison to their ho
239 characterized member of its superfamily, the Concanavalin A-like lectins/glucanases.
240 n in guinea pig spleen cells stimulated with concanavalin A, lipopolysaccharide (LPS), phorbol myrist
241 lminant hepatitis, but was without effect on concanavalin A-mediated hepatitis.
242 tive EC injury using the concanavalin A/anti-concanavalin A model and after ischemia/reperfusion (I/R
243 nding site separation of the sugar sites for concanavalin A of 3.6-4.3 nm was determined and a critic
244 compared with that of commercially available concanavalin A on agarose beads.
245 urement of the toxic effect of 100 microg/mL concanavalin A on macrophages and hepatocytes, but not o
246              We have immobilized the protein Concanavalin A onto a self-assembled monolayer of multiv
247 nalized graphene indicate that adsorption of Concanavalin A onto graphene is accompanied by near-comp
248  no significant difference in the binding of concanavalin A or Aleuria aurantia lectin was detected.
249 n) and this was not blocked by agents (i.e., concanavalin A or hypertonic sucrose) that inhibit D1 re
250 , and the generation of activated T cells by Concanavalin A or L-PHA was also reduced in Fng tKO mice
251    Splenic T cells were then stimulated with concanavalin A or ovalbumin in vivo or in vitro, and CD2
252 hole spleen cells following stimulation with concanavalin A or PPD.
253 d apoptosis following activation either with concanavalin A or with antibodies to CD3 and CD28 and le
254 ion in response to the homologous immunogen, concanavalin A, or lipopolysaccharide was similar for al
255 ncluded: (a) reduced IFN-gamma production by concanavalin A- or antigen-stimulated T cells; and (b) h
256      Downregulation of chIL-17RA occurred in concanavalin A- or lipopolysaccharide-activated splenic
257 ower than that of maize and potato starches (concanavalin A precipitation method).
258 911) and cyclic peptide (D002) reactive with concanavalin A presented in a multiple antigen peptide (
259 interaction force between a polymer-tethered concanavalin-A protein (ConA) and a similarly tethered m
260 itogen-induced lymphoproliferative activity (concanavalin A, range: 74,218 dropping to 55,880 counts
261 arger than that of the wild type, had gained concanavalin A reactivity, and that the arabinan termini
262 ration in response to phytohemagglutinin and concanavalin A remained stable or increased for the Inte
263 -induced binding to rhodopsin immobilized on concanavalin-A resin.
264 A, avidin, monoclonal anti-BSA antibody, and concanavalin A, respectively.
265  of bacterial growth inhibition, addition of concanavalin A results in the formation of extensive dex
266 microbalance (QCM) to show that tripod-bound Concanavalin A retains its affinity for polysaccharides
267 ctra of the amide I region of poly-l-lysine, concanavalin A, ribonuclease A, and lysozyme show cross-
268                                   Given that Concanavalin A's tertiary structure is thought to be rel
269 , and Rac-1 coprecipitates with rhodopsin on Concanavalin A Sepharose.
270 e, phenyl-Sepharose hydrophobic interaction, concanavalin A-Sepharose affinity and Superose 12 gel fi
271   Moreover, in response to aoHGE extracts or concanavalin A, splenocytes from ehrlichia-infected mice
272                 PG27 significantly increased concanavalin A-stimulated in vitro IL-4 production by da
273               However, neither inhibition of concanavalin A-stimulated spleen cells nor keyhole limpe
274 , trough) and for lymphocyte functions using concanavalin A-stimulated whole blood assays to measure
275 ve loss of T cells, compromised responses to concanavalin A stimulation, and absence of inflammatory
276  and interleukin (IL)-4 (Th2 cytokine) after concanavalin A stimulation.
277 nocytes from infected animals in response to concanavalin A, suggesting a role for NO in mediating th
278 -alpha(5)beta(1) interaction is inhibited by concanavalin A, suggesting that GT1b binds to mannose st
279 roteases and organic extraction but bound to concanavalin A, suggesting that IRI is a sulfated glycan
280                    In DRG cells treated with concanavalin A the EC50 for ATPA was 341 nM.
281               Likewise, after treatment with concanavalin A, there was no change in either desensitiz
282 oclonal antibody (M1) and for the binding of concanavalin A to a glycoprotein have been determined.
283     Flipping was assayed by using the lectin Concanavalin A to capture M5-DLOs that had been transloc
284  endocytosis of transferrin and transport of concanavalin A to the lysosome are both impaired, confir
285 d targets (carbonic anhydrase, streptavidin, concanavalin A) to identify desired ligands.
286         This analysis implies that, although Concanavalin A tolerates the additional 6 arm GlcNAc pre
287  with BSA, and the affinity of photoattached concanavalin A toward ovalbumin was compared with that o
288 as there was no protection against CCl(4) or concanavalin A toxicity.
289 ies agrin-G3 as a functional analogue of the concanavalin A-type lectins, highlights functional simil
290 o O-linked N-acetylglucosamine or the lectin concanavalin A was detected.
291  1.5-microm diameter microsphere coated with concanavalin A was inserted though an ablated hole in th
292 n with fluorescein isothiocyanate-conjugated concanavalin A, was increased in the absence of Dp71.
293     By using a Sepharose-immobilized lectin, concanavalin A, we isolated a fraction enriched with LRV
294 . coli 2443 with a fluorescent derivative of concanavalin A, we observed a similar helical organizati
295 oglobulin, ribonuclease A, E-cadherin 5, and concanavalin A were co-lyophilized with carbohydrates (t
296 ol) cows stimulated with the general mitogen concanavalin A were highly similar (overall r = 0.84), s
297 or all seven lectins, and similar to that of concanavalin A which is also a member of the Diocleinae
298  genes after exposure to interferon-gamma or concanavalin A, which resulted in minimal HLA-B27 up-reg
299 ned a high amount of mannose, as detected by concanavalin A, while the UT-A1 in lipid rafts was the m
300 pha-Man-(1--> 6)]-Man to bind to the lectin, Concanavalin A, with almost the same affinity as the tri

 
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