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1 re observed for a larger multimeric complex (Concanavalin A).
2 ssessed after perfusion with FITC-conjugated concanavalin A.
3 separate group of mice by administration of concanavalin A.
4 of the second through sixth generations and Concanavalin A.
5 pon aggregation of unilamellar vesicles with concanavalin A.
6 ssessed by labeling adherent leukocytes with concanavalin A.
7 d has a surface that is capped by the lectin concanavalin A.
8 which is only manifested in the presence of concanavalin A.
9 te and sodium azide, and one model biotoxin, concanavalin A.
10 th immobilized Sambucus nigra agglutinin and concanavalin A.
11 at were resistant to the cytotoxic action of concanavalin A.
12 - and bivalent ligands for the legume lectin concanavalin A.
13 10 is inducible by interferons (IFNs) and by concanavalin A.
14 after stimulation of cells with the mitogen, concanavalin A.
15 n of peripheral blood mononuclear cells with concanavalin A.
16 cation exchange column and was shown to bind concanavalin A.
17 ho-proliferative response to alloantigens or concanavalin A.
18 tic liver damage induced by the injection of concanavalin A.
19 ose-containing bath solution or treated with concanavalin A.
20 ction in response to either C. pneumoniae or concanavalin A.
21 d Th-2 cytokine responses to SME antigen and concanavalin A.
22 ion-induced programmed cell death induced by concanavalin A.
23 ting the enzyme's aggregation induced by the concanavalin A.
24 binding of the polymers to the plant lectin concanavalin A.
25 s not evident after 48-hour stimulation with concanavalin-A.
28 GC-5 cells were differentiated with succinyl concanavalin A (50 microg/mL) and transferred to a press
31 ectin binding experiments using succinylated concanavalin A, a plant lectin with high affinity for ma
32 scriptional regulation of CSF-2 and CSF-3 in concanavalin A-activated MSCs requires MT1-MMP signaling
34 ene (aSMase(-/-)), we found that thymocytes, concanavalin A-activated T cells, and lipopolysaccharide
36 eted cryptococcal antigens were separated by concanavalin A affinity chromatography into adherent (ma
37 tionation, diethylaminoethyl chromatography, concanavalin A affinity chromatography, and chromatofocu
39 valent affinity capture using NeutrAvidin or concanavalin A agarose beads or directly via covalent co
40 was also lost when they were incubated with concanavalin A-agarose beads, suggesting that the inhibi
42 f oocytes with 20% sucrose and 250 microg/ml concanavalin A, agents that inhibit the endocytosis of R
43 ctional valency of a triantennary analog for concanavalin A and D. grandiflora lectin are observed.
44 rate analogs to the Man/Glc-specific lectins concanavalin A and Dioclea grandiflora lectin was invest
45 a of infected insect cells using immobilized concanavalin A and immobilized BACE inhibitor, P10-P4' S
46 B10.BR splenic lymphocytes stimulated with concanavalin A and interleukin 2 were infected with a re
51 , T cell proliferation upon stimulation with concanavalin A and phytohemagglutinin A was only 40-50%
52 ion lymphocytes were examined in response to concanavalin A and purified protein derivative (PPD) in
53 or the ability to proliferate in response to concanavalin A and purified protein derivative (PPD) in
55 protein receptors such as the plant lectins concanavalin A and the highly toxic mistletoe Viscum alb
56 her mannose/glucose binding lectins, such as concanavalin A and the pea, lentil and Calystegia sepium
57 in C57BL/6 mice by intravenous injection of concanavalin A and then analyzed liver concentrations of
58 unction of DR signaling in T cell-dependent (concanavalin A) and independent (lipopolysaccharide/gala
59 tures of the protein complexes streptavidin, concanavalin A, and C-reactive protein under charge redu
60 superoxide dismutase, dimeric and tetrameric concanavalin A, and heptameric GroES and Gp31; ranging i
61 asurements were performed with cytochrome c, concanavalin A, and poly-L-lysine, biomolecules that exh
62 y from Escherichia coli lipopolysaccharides, concanavalin A, and Pseudomonas aeruginosa exotoxin A.
63 ulin and enolase), tetrameric (streptavidin, concanavalin A, and pyruvate kinase), and pentameric (C-
64 an inducible pattern on T cells activated by concanavalin A, anti-CD3 mAb in combination with anti-CD
65 r induction of selective EC injury using the concanavalin A/anti-concanavalin A model and after ische
66 nti-bovine serum albumin (BSA) antibody, and concanavalin A are embedded in distinct regions of a 7.5
68 ives supported the integration of the lectin concanavalin A as well as the enzymes alpha1-2,3 mannosi
70 suggesting that the polymers bind the lectin concanavalin A at the same site as natural carbohydrates
71 investigated by bromelain IgE inhibition and concanavalin A binding assays using sera of cypress poll
75 l activation, as stimulation of T cells with concanavalin A, but not phorbol 12-myristate 13-acetate
77 ver, CD160(-/-) mice are more susceptible to Concanavalin A challenge, and display elevated serum AST
78 ZM-INVINH1 with the glycoprotein fraction by concanavalin A chromatogaphy suggests that ZM-INVINH1 in
82 s been created using Alexa Fluor 647-labeled concanavalin A (Con A) and a fourth-generation PAMAM Ale
83 e material was derivatized with two lectins, Concanavalin A (Con A) and Aleuria aurantia lectin (AAL)
85 ion of the electrical synapse was blocked by concanavalin A (Con A) and dynamin inhibitory peptide (D
86 e data available on the binding constants of Concanavalin A (Con A) and glycogen and Con A-mannan usi
87 bohydrate platform for the immobilization of Concanavalin A (Con A) and is capable of LPS binding mea
88 ew as the sensing probes for lectins such as concanavalin A (Con A) based on the molecular recognitio
89 eral crystal structures of the legume lectin concanavalin A (Con A) bound to closely related carbohyd
90 set of serial lectin columns consisting of a concanavalin A (Con A) column coupled to an SNA column f
91 sed to measure the adhesive strength between concanavalin A (Con A) coupled to an AFM tip and Con A r
92 of alpha-Man to the mannose-specific lectin concanavalin A (Con A) even though homogeneous beta-Lact
97 vation and expansion were studied by using a concanavalin A (Con A) liver injury model followed by pa
98 g the direct pili-mannose binding as well as Concanavalin A (Con A) mediated lipopolysaccharides (LPS
99 ial, we have developed a nanoscale chelating Concanavalin A (Con A) monolithic capillary prepared usi
101 ntaining 2-D photonic crystal sensor detects Concanavalin A (Con A) through shifts in the 2-D diffrac
102 ible binding of a mobile fluorophore-labeled Concanavalin A (Con A) to immobile pendant glucose moite
103 -SNPs with fluorescein isothiocyanate (FITC)-concanavalin A (Con A) was determined using a fluorescen
104 sembled monolayer in combination with lectin concanavalin A (Con A) was used as molecular recognition
106 achite green (MG-dextran), was conjugated to concanavalin A (Con A), an enzyme with specific affinity
107 ns including Maackia amurensis lectin (MAL), concanavalin A (Con A), and wheat germ agglutinin (WGA).
108 were cultured in the absence or presence of concanavalin A (Con A), phorbol myristate acetate (PMA)/
110 excellent selectivity to its target lectin, concanavalin A (Con A), surpassing the formerly used lin
111 lines, we identify a plant-derived compound, Concanavalin A (Con A), which differentially kills p53-n
112 ran-coated nanoparticles are aggregated with concanavalin A (Con A), which results in a significant s
126 ) kainate receptors, two unrelated proteins, concanavalin-A (Con-A) and postsynaptic density protein
127 riefly, the well-known lectin macromolecule (concanavalin A, Con A) monolayer was functionalized on 3
128 -HIV antibody 2G12 (shortest distance 31 A), concanavalin A (ConA) (shortest distance 72 A), RCA120 (
131 y, including T/NKT cell hepatitis induced by concanavalin A (ConA) and alpha-galactosylceramide (alph
132 CM electrodes present a higher adsorption of Concanavalin A (ConA) and Bovine Serum Albumin (BSA) pro
133 arbohydrates to the Man/Glc-specific lectins concanavalin A (ConA) and Dioclea grandiflora lectin (DG
134 arbohydrates to the Man/Glc-specific lectins concanavalin A (ConA) and Dioclea grandiflora lectin (DG
135 binding of the carbohydrate-binding proteins concanavalin A (ConA) and jacalin to arrays composed of
136 cterized chemical inhibitors of endocytosis: concanavalin A (conA) and phenylarsine oxide (PAO).
137 UVL patients exhibited normal ATP release to Concanavalin A (ConA) and phytohemagglutinin (PHA; 190+/
138 K using the receptor internalization blocker concanavalin A (ConA) and the carboxyl terminus-truncate
140 d from a detailed analysis of the binding of concanavalin A (ConA) and wheat germ agglutinin (WGA) to
141 on, cells treated with ICZ increased surface concanavalin A (ConA) binding, corroborating an increase
143 ated in T cell-mediated hepatitis induced by concanavalin A (ConA) but are less extensively elevated
145 anisms in optical glucose sensors based upon Concanavalin A (ConA) has tended to prevent the sensors'
146 mpetitive binding assays based on the lectin Concanavalin A (ConA) have displayed significant potenti
149 peptide reacted strongly with both lotus and concanavalin A (ConA) lectins, it bound to lotus stronge
150 robe was designed by noncovalent assembly of concanavalin A (ConA) on gold nanoparticles (AuNPs).
151 ects of T cell-mediated hepatitis induced by concanavalin A (ConA) on the regenerative response in vi
152 ls of hepatitis, including administration of concanavalin A (ConA) or alpha-galactosyl-ceramide, whic
153 in unstimulated or lipopolysaccharide (LPS), concanavalin A (ConA) or phytohemagglutinin (PHA) stimul
155 pha-D-mannopyranoside residues to the lectin concanavalin A (ConA) show increasing negative cooperati
156 cells showed enhanced responses to in vitro concanavalin A (ConA) stimulation when compared with WT
157 ide (LPS) plus D-galactosamine (D-Galn), and concanavalin A (ConA) to identify the function of extrac
158 Here, we show that treatment of mice with concanavalin A (ConA) to induce liver injury triggered a
162 opaeus (gorse, furze), Triticum vulgaris and Concanavalin A (ConA) was used for probes to evaluate bi
163 coefficients that showed mannose to bind to concanavalin A (conA) with 3.7 times greater affinity th
164 e binding affinity of mannose and glucose to concanavalin A (ConA), a 106 KDa homotetramer protein, i
165 ension of macroporous hydrogel particles and concanavalin A (ConA), a glucose-specific lectin, that a
167 tein, PZR displays a strong association with concanavalin A (ConA), a member of the plant lectin fami
168 tal model of autoimmune hepatitis induced by concanavalin A (ConA), a process involving T cell activa
171 oteins, namely, transthyretin (TTR), avidin, concanavalin A (conA), and human serum amyloid P compone
175 which consists of three mixtures of lectins concanavalin A (ConA), jacalin (JAC), and wheat germ agg
176 ure induced by Fas-agonistic antibody (Jo2), concanavalin A (ConA), or D-galactosamine/lipopolysaccha
177 and lipopolysaccharide (LPS), or the lectin concanavalin A (ConA), suggesting that physiologic level
178 tested with several plant lectins, including concanavalin A (conA), Vicia villosa isolectin B4 (VVL-B
179 On the basis of the model system glucose-Concanavalin A (ConA), we explore the application of Tra
180 tosamine (GaIN)/lipopolysaccharide (LPS) and concanavalin A (ConA)-induced acute liver failure (ALF),
184 o chronic choline-deficient diet exacerbates concanavalin A (ConA)-induced liver hepatitis, which is
185 of the molecular and cellular mechanisms of concanavalin A (ConA)-induced liver injury have provided
188 DTA mice) exhibited enhanced lymph node (LN) concanavalin A (ConA)-induced Th1 responses after tick i
189 ingly, NK cells mediated hypersensitivity to concanavalin A (ConA)-mediated hepatitis in GNMT(-/-) mi
190 to Fas-specific antibody or co-cultured with concanavalin A (ConA)-stimulated hepatic mononuclear cel
192 increased production of interferon-gamma by concanavalin A (ConA)-stimulated spleen T cells and expr
193 d interferon-gamma (IFN-gamma) production in concanavalin A (conA)-stimulated spleen T cells, and dim
201 er injury in the lipopolysaccharide/GalN and concanavalin A (ConA)/GalN models, but not in a ConA-onl
202 Activation of CD8(+) T lymphocytes with concanavalin A (ConA)/interleukin-2 (IL-2), and activati
204 that treatment of OAT1-expressing cells with concanavalin A, depletion of K(+) from the cells, or tra
207 protease domain of MDC9, Western analysis of concanavalin A-enriched glomerular microsomal extracts d
209 ulin G, avidin:biotin, antibody:antigen, and concanavalin A:glycoprotein interactions are used to dem
210 or LcL were compared with those obtained for concanavalin A i.e. lectin, which interacts with the car
212 ttenuated the suppression of the response to concanavalin A in immunized mice, providing further evid
213 s attenuated by blockade of endocytosis with concanavalin A, indicating a critical role for internali
214 ruited iNKT cells were anergic and prevented concanavalin A-induced (ConA-induced) hepatitis by speci
215 imicrobial susceptibility assay based on the concanavalin A-induced clustering of dextran-coated gold
216 that myosin phosphorylation is critical for concanavalin A-induced gathering of surface receptors.
218 endogenous Gal-1 protected the liver against concanavalin A-induced hepatitis with the B6 genetic bac
219 henotype of these cells, reduced severity of concanavalin A-induced hepatitis, and alterations in the
225 K12-Fc inhibited in a dose-dependent manner concanavalin A-induced proliferation, but not anti-TcRal
228 y, IL-22TG mice were completely resistant to concanavalin A-induced T cell hepatitis with minimal eff
232 more, a non-selective endocytosis inhibitor, concanavalin A, inhibited the internalization of wild ty
234 nnosylated conjugated polymer (sugar-PPE) by Concanavalin A is positively dependent upon sugar-PPE co
236 were simultaneously quantified by combining concanavalin A lectin (ConA) perfusion labeling with a f
237 asured by fluorescein isothiocyanate-coupled concanavalin A lectin and acridine orange labeling.
238 res show surprisingly high affinities toward Concanavalin A lectin receptor in comparison to their ho
240 n in guinea pig spleen cells stimulated with concanavalin A, lipopolysaccharide (LPS), phorbol myrist
242 tive EC injury using the concanavalin A/anti-concanavalin A model and after ischemia/reperfusion (I/R
243 nding site separation of the sugar sites for concanavalin A of 3.6-4.3 nm was determined and a critic
245 urement of the toxic effect of 100 microg/mL concanavalin A on macrophages and hepatocytes, but not o
247 nalized graphene indicate that adsorption of Concanavalin A onto graphene is accompanied by near-comp
248 no significant difference in the binding of concanavalin A or Aleuria aurantia lectin was detected.
249 n) and this was not blocked by agents (i.e., concanavalin A or hypertonic sucrose) that inhibit D1 re
250 , and the generation of activated T cells by Concanavalin A or L-PHA was also reduced in Fng tKO mice
251 Splenic T cells were then stimulated with concanavalin A or ovalbumin in vivo or in vitro, and CD2
253 d apoptosis following activation either with concanavalin A or with antibodies to CD3 and CD28 and le
254 ion in response to the homologous immunogen, concanavalin A, or lipopolysaccharide was similar for al
255 ncluded: (a) reduced IFN-gamma production by concanavalin A- or antigen-stimulated T cells; and (b) h
258 911) and cyclic peptide (D002) reactive with concanavalin A presented in a multiple antigen peptide (
259 interaction force between a polymer-tethered concanavalin-A protein (ConA) and a similarly tethered m
260 itogen-induced lymphoproliferative activity (concanavalin A, range: 74,218 dropping to 55,880 counts
261 arger than that of the wild type, had gained concanavalin A reactivity, and that the arabinan termini
262 ration in response to phytohemagglutinin and concanavalin A remained stable or increased for the Inte
265 of bacterial growth inhibition, addition of concanavalin A results in the formation of extensive dex
266 microbalance (QCM) to show that tripod-bound Concanavalin A retains its affinity for polysaccharides
267 ctra of the amide I region of poly-l-lysine, concanavalin A, ribonuclease A, and lysozyme show cross-
270 e, phenyl-Sepharose hydrophobic interaction, concanavalin A-Sepharose affinity and Superose 12 gel fi
271 Moreover, in response to aoHGE extracts or concanavalin A, splenocytes from ehrlichia-infected mice
274 , trough) and for lymphocyte functions using concanavalin A-stimulated whole blood assays to measure
275 ve loss of T cells, compromised responses to concanavalin A stimulation, and absence of inflammatory
277 nocytes from infected animals in response to concanavalin A, suggesting a role for NO in mediating th
278 -alpha(5)beta(1) interaction is inhibited by concanavalin A, suggesting that GT1b binds to mannose st
279 roteases and organic extraction but bound to concanavalin A, suggesting that IRI is a sulfated glycan
282 oclonal antibody (M1) and for the binding of concanavalin A to a glycoprotein have been determined.
283 Flipping was assayed by using the lectin Concanavalin A to capture M5-DLOs that had been transloc
284 endocytosis of transferrin and transport of concanavalin A to the lysosome are both impaired, confir
287 with BSA, and the affinity of photoattached concanavalin A toward ovalbumin was compared with that o
289 ies agrin-G3 as a functional analogue of the concanavalin A-type lectins, highlights functional simil
291 1.5-microm diameter microsphere coated with concanavalin A was inserted though an ablated hole in th
292 n with fluorescein isothiocyanate-conjugated concanavalin A, was increased in the absence of Dp71.
293 By using a Sepharose-immobilized lectin, concanavalin A, we isolated a fraction enriched with LRV
294 . coli 2443 with a fluorescent derivative of concanavalin A, we observed a similar helical organizati
295 oglobulin, ribonuclease A, E-cadherin 5, and concanavalin A were co-lyophilized with carbohydrates (t
296 ol) cows stimulated with the general mitogen concanavalin A were highly similar (overall r = 0.84), s
297 or all seven lectins, and similar to that of concanavalin A which is also a member of the Diocleinae
298 genes after exposure to interferon-gamma or concanavalin A, which resulted in minimal HLA-B27 up-reg
299 ned a high amount of mannose, as detected by concanavalin A, while the UT-A1 in lipid rafts was the m
300 pha-Man-(1--> 6)]-Man to bind to the lectin, Concanavalin A, with almost the same affinity as the tri