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1 llular calcium, allowing the generation of a concentration response curve.
2 his resulted in a leftward shift of the Ca2+ concentration-response curve.
3 homeostasis and pathology on a steep agonist concentration-response curve.
4 r leftward shifts of the acetylcholine (ACh) concentration-response curve.
5 eptor can alter the EC(50) value of the 5-HT concentration-response curve.
6 ing characteristics of the antibodies beyond concentration-response curves.
7  and 3) allow the measurement of single-cell concentration-response curves.
8 on broader health impacts or on the shape of concentration-response curves.
9 able from the experimental single compounds' concentration-response curves.
10 escence intensity was quantified to generate concentration-response curves.
11 higher concentrations, producing bell-shaped concentration-response curves.
12 hibition, as determined from single chemical concentration-response curves.
13 agonists caused rightward shifts in the PGE2 concentration-response curves.
14 ply through the analysis of their respective concentration-response curves.
15 ffect models were used to pool city-specific concentration-response curves.
16 old to the right the 8-iso-PGE and 8-iso-PGE concentration-response curves.
17 lent cations did not significantly alter ATP concentration-response curves.
18 sthetized male C57BL/6J mice (3 months old), concentration-response curves (10(-9) m to 10(-5) m, 0.5
19                  Efficiencies estimated from concentration-response curves (23 agonists, 53 mutations
20 c agonist oxotremorine, we revealed a unique concentration-response curve and a sensitivity to repeat
21 not result in a lateral shift in the agonist concentration-response curve and are unlikely to involve
22 of the high ACh-sensitivity component of the concentration-response curve and contribute directly to
23                          Analysis of the CHA concentration-response curve and inhibition of the CHA-i
24        The parameter is derived from agonist concentration-response curves and comprises the maximal
25                                Acetylcholine concentration-response curves and maximum function were
26 uman GPCRs resulted in 1500 pathway-specific concentration-response curves and revealed a great diver
27 eceptor tyrosine phosphorylation had similar concentration-response curves and were inhibited by the
28 t 10 to 100 microM, it shifts mGluR5 agonist concentration-response curves approximately 2-fold to th
29 ate immunoassay reagents through analyses of concentration-response curves as well as antibody-antige
30         The slope of the underlying in-vitro concentration-response curves contributes as well.
31                               The shape of a concentration-response curve (CRC) is determined by unde
32 etested on the SyncroPatch 384PE to generate concentration-response curves (CRCs).
33                 Operational model fitting of concentration-response curve data from cells subjected t
34 ntra- and interinhibitor comparisons of dose/concentration response curves demonstrated the absence o
35                          Analysis of the CHA concentration-response curves demonstrated that this inc
36                              Conversely, the concentration-response curve describing the enhancement
37 Incubation with 3.5 mM Ca2+ shifted the 5-HT concentration-response curve downward and to the right,
38                                   Whole-cell concentration-response curves enabled the agonists to be
39 ed method for quantifying biomarkers wherein concentration-response curves estimated using samples of
40 and efficacious constrictor, with a biphasic concentration-response curve, followed by vasopressin, s
41  two- to threefold shift to the right in the concentration response curves for arachidonic acid relea
42 n a rightward shift in both the PS- and Ca2+-concentration response curves for PKCalpha membrane asso
43  caused a shift to the right in the collagen concentration response curves for protein tyrosine phosp
44 42A/C296A exhibited no additive shift in the concentration-response curve for 2-MeSADP.
45 ontrast to the wild type A(2A) receptor, the concentration-response curve for agonist-induced cAMP ac
46                                          The concentration-response curve for alphabeta-MeATP had an
47                                          The concentration-response curve for ATP on human P2X4 in th
48      RA-2 at 100 nM right-shifted the KCa3.1 concentration-response curve for Ca(2+) activation.
49  trials, has been shown to induce a biphasic concentration-response curve for down-regulating protein
50 -/-)) platelets display an inhibition in the concentration-response curve for GPVI-specific agonist-i
51                      Compared with AuIB, the concentration-response curve for inhibition of alpha3bet
52 er than that of ethanol and the slope of the concentration-response curve for isoflurane less steep t
53                  The serotonin (2-30 microM) concentration-response curve for LTP was bell shaped as
54                                          The concentration-response curve for myocyte shortening by t
55  several days with VDH, exhibited a U-shaped concentration-response curve for neuroprotection against
56 ors, Con G produced a rightward shift in the concentration-response curve for NMDA, providing support
57                         The flexible ambient concentration-response curve for O3 showed evidence of n
58                                          The concentration-response curve for oxo-M was shifted to th
59     In preparations from untreated rats, the concentration-response curve for PAD in response to 0.1-
60 uced an apparent noncompetitive shift in the concentration-response curve for spermine potentiation o
61 e pore was supported by a right shift in the concentration-response curve for tetraethylammonium; sim
62 with the enzyme pyruvate kinase, to generate concentration-response curves for >60,000 compounds in a
63                                          The concentration-response curves for 5-HT and Ang II were s
64           EC(50) values were determined from concentration-response curves for a range of agonists.
65                                   The pooled concentration-response curves for all three causes were
66          In the presence of betaAR blockade, concentration-response curves for AR agonists suggest th
67 he Ca(2+), phorbol ester, and diacylglycerol concentration-response curves for Cdc42-induced activati
68 isoforms showed a rightward shift in agonist concentration-response curves for eliciting calcium rele
69                                          The concentration-response curves for enhancement of steady-
70 ity between these compounds, we examined the concentration-response curves for ethanol and isoflurane
71                   Both compounds shifted the concentration-response curves for extracellular Ca2+ to
72               For alpha1beta1delta subunits, concentration-response curves for GABA were displaced la
73          In the contraction experiments, the concentration-response curves for histamine-induced cont
74 hyl acetate leads to rightward shifts in the concentration-response curves for inhibition of [(125)I]
75 ole-cell recording was used to determine the concentration-response curves for lanthanum for the thre
76                                      Glycine concentration-response curves for NMDARs containing NR2A
77                                Comparison of concentration-response curves for presynaptic and postsy
78                                              Concentration-response curves for reduction in Cmpk by s
79                                  Smoke PM2.5 concentration-response curves for respiratory hospitaliz
80                            Comparison of the concentration-response curves for the effects of isoflur
81           IC(50) values were determined from concentration-response curves for three commonly used pu
82 dicated by a parallel rightward shift of the concentration response curve from an EC(50) of 2.7 +/- 0
83                                          The concentration response curves generated for these end po
84                                          The concentration-response curves generated by application o
85 TP currents at 100 microM was < 0.1, the ATP concentration-response curve had an EC50 of 56 microM an
86                                   The Ang II concentration response curves in abdominal aorta and fem
87                                              Concentration-response curves, in which the effect of va
88 atory subunit of the channel, shifts the ATP concentration-response curve into a range in which the c
89 actor for the slope of a quantal, population concentration-response curve is individual variability.
90 SUR2B currents by MgATP explains how the ATP concentration-response curve is shifted to the right in
91           Propofol (3 microM) increased GABA concentration-response curve maximal currents similarly
92 tive-gating modulator and shifts the calcium-concentration response curve of KCa3.1 to the left.
93                                            A concentration response curve of this blocking agent reve
94         ADP receptor antagonists shifted the concentration-response curve of collagen- or CRP-induced
95 ium chelator 5,5'-dimethyl-BAPTA shifted the concentration-response curve of convulxin-induced platel
96  nonequilibrium conditions we found that the concentration-response curve of pyramidal GABAA receptor
97 124183 caused a marked leftward shift of the concentration-response curve of the A3 receptor agonists
98 g the time it took to produce an eight-point concentration-response curve of the effect of propofol o
99 s surmountable antagonist rightward-shifting concentration-response curves of all three agonists in a
100                   BPTU rightward shifted the concentration-response curves of both 2-methylthioadenos
101                                              Concentration-response curves of endothelial-dependent a
102 pH (to 8.5) or lowering (to 6.5) shifted the concentration-response curves of GABA to the left or rig
103                                              Concentration-response curves of intracellular cyclic GM
104                                              Concentration-response curves of isoproterenol-stimulate
105 s, all of which are based on the analysis of concentration-response curves of ligands according to cl
106                                         Drug concentration-response curves of neural activity were id
107                           Data obtained from concentration-response curves of the volatile anesthetic
108 e rising phase of the glutamate steady state concentration-response curve overlapped with the wildtyp
109                              Whole-cell GABA concentration--response curves performed with and withou
110 entration-response curves were compared with concentration-response curves predicted by concentration
111 .2 mM) produced a downward shift of the 5-HT concentration-response curve, reducing the maximal respo
112 s, generating a characteristic 'bell-shaped' concentration-response curve, reminiscent of RAFi-driven
113                                  Equilibrium concentration-response curves revealed a lower potency f
114                                       Ca(2+) concentration-response curves revealed that differences
115  Furthermore, cyanopindolol shifted the 5-CT concentration-response curve rightward, increasing the E
116 nically for potency against BTK using IC(50) concentration-response curves; selectivity using a 270-k
117 ng culture media, was complex, with the GABA concentration-response curves shifting laterally with re
118                                              Concentration-response curves showed that pLV-S particle
119                                  Equilibrium concentration-response curves showed that recombinant hu
120 gers or ADP receptor antagonists shifted the concentration-response curve slightly to the right at lo
121            Initial estimates based on fitted concentration response curves suggested that maximal inh
122 orrelated with the EC(50) values of the 5-HT concentration-response curves, suggesting that these mut
123 alveolar concentration curve is a population concentration-response curve that describes the relation
124 channels exhibited a monophasic steady state concentration-response curve that simply plateaued at hi
125 lso produced a sinistral displacement of the concentration-response curves that described the augment
126 luK2/GluK4 and GluK2/GluK5 have steady state concentration-response curves that were bell-shaped in r
127 M PGE2 was without significant effect on the concentration response curve to exogenously added acetyl
128 sed GABAA receptors and in shifting the GABA concentration-response curve to lower concentrations.
129           Transfection with bcl-2 shifts the concentration-response curve to NCS but does not change
130 he GM-treated group demonstrated a preserved concentration-response curve to the alpha(1) adrenergic
131  as His and Trp produced a shift of the GABA concentration-response curve to the left, whereas replac
132 slower closing rates, thus shifting the GABA concentration-response curve to the left.
133 T1B autoreceptor but shifted the sumatriptan concentration-response curve to the right (P < 0.05).
134 ncentration of 100 nM, shifted the muscarine concentration-response curve to the right by around 50-f
135 concentration of Waglerin-1 shifted the GABA concentration-response curve to the right in a parallel
136            Zn2+ (100 microM) shifted the ATP concentration-response curve to the right in a parallel
137           IL-2 (2 ng/ml) shifted the kainate concentration-response curve to the right in a parallel
138 dditionally, IL-2 (1 ng/ml) shifted the NMDA concentration-response curve to the right, significantly
139 e channel opening rate and shifting the GABA concentration-response curve to the right.
140                                              Concentration-response curves to 5-HT2C agonists were fi
141 al pressure on the diameter was assessed and concentration-response curves to different constrictor a
142 r in a video-monitored perfusion system, and concentration-response curves to phenylephrine and acety
143 was also observed to a greater extent on the concentration-response curves to selective hmGluR2/3 ago
144                 No obvious difference of the concentration-response curve was found among three group
145 ted group, a non-saturating 5-HT-induced PAD concentration-response curve was generated.
146                                     The GABA concentration-response curve was shifted to the left by
147 sin; the maximal response was lower, and the concentration-response curve was shifted to the right in
148                                              Concentration-response curves were classified to rapidly
149                                     Measured concentration-response curves were compared with concent
150                                              Concentration-response curves were constructed and compa
151                                   Cumulative concentration-response curves were constructed for Ang I
152                                         GABA concentration-response curves were depressed in a mixed/
153 r 30 min, and after a 60-min washout period, concentration-response curves were determined for the ad
154                                     The GABA concentration-response curves were enhanced (pH 5.4) or
155                       Using Optimul and LTA, concentration-response curves were generated for arachid
156 sidustat, vadadustat, and FG-2216, for which concentration-response curves were generated, allowing f
157 pioids reached equilibrium with the KOR, and concentration-response curves were generated.
158 egardless of the Galpha subunit present, the concentration-response curves were leftward shifted when
159                            The slopes of the concentration-response curves were more shallow than bef
160                                      Agonist concentration-response curves were similar for all 12 fu
161                                          ERC concentration-response curves were used across multiple
162 nduce parallel rightward shifts in the VU-29 concentration-response curve, whereas 5MPEP inhibits CPP
163 beta2 nAChRs, as evidenced by monophasic ACh concentration-response curves, whereas injections with 1
164 itive antagonist hybrids produce bell-shaped concentration-response curves, whereas the agonist-compe
165 tive high-throughput screen (qHTS), based on concentration-response curves, which was designed to ide
166  produced a rightward shift in the CP 93,129 concentration-response curve, while spiperone had no aff
167 sthetic concentrations but there was a sharp concentration-response curve with only minimal effects o
168 odel to obtain a unified fit of the multiple concentration-response curves with a single set of param
169                                              Concentration-response curves with Gsalpha suggested the
170 onist-competitive antagonist hybrids produce concentration-response curves with reduced but plateaued
171 roposed approach performed well for 14-point-concentration-response curves with typical levels of res
172        CMPI produced a left shift of the ACh concentration-response curve without altering ACh effica
173 produced a 4-fold rightward shift in the ACh concentration-response curve without altering maximum AC
174  (Emax) of the nACh alpha 7 receptor agonist concentration-response curve, without significantly affe

 
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