ライフサイエンスコーパス: conduction velocity

1 rnodes along axons altering action potential conduction velocity.

2 ectrophysiology, increasing action potential conduction velocity.

3 increased gadolinium uptake has lower local conduction velocity.

4 tive effects on maximal phase 0 upstroke and conduction velocity.

5 coordination deficits and exhibit decreased conduction velocity.

6 -9)), indicating that INPP4B regulates nerve conduction velocity.

7 longer than non-VT channels, and have slower conduction velocity.

8 evertheless, there is a 38% reduction in PNS conduction velocity.

9 mation processing in addition to speeding up conduction velocity.

10 h surrounding neural structures to determine conduction velocity.

11 s that intercellular coupling determines the conduction velocity.

12 a 2:1 atrioventricular block, and slowing of conduction velocity.

13 bit impaired motor function and slower nerve conduction velocity.

14 poral coordinate transformation via measured conduction velocity.

15 eralgesia, cold allodynia, and sciatic nerve conduction velocity.

16 nant of the actual value of action potential conduction velocity.

17 of the auditory nerve and results in altered conduction velocity.

18 the effects of levosimendan on muscle fiber conduction velocity.

19 n turn, has a significant influence on nerve conduction velocity.

20 decreased sodium current density and slower conduction velocity.

21 is due to the increase of the dispersion of conduction velocity.

22 , reduced peak sodium current and diminished conduction velocity.

23 ndpoint assessments of action potentials and conduction velocity.

24 increased P-wave duration and slowed atrial conduction velocity.

25 the gene desert upstream of TBX3 in cardiac conduction velocity.

26 s were analyzed for amplitude, duration, and conduction velocity.

27 daptations in axon myelination for increased conduction velocity.

28 lin outfoldings that impair the normal nerve conduction velocity.

29 omeres, smaller action potentials, and lower conduction velocities.

30 xons, delayed myelination, and reduced nerve conduction velocities.

31 d stable structures with highly reproducible conduction velocities.

32 ties and displayed a mild reduction of nerve conduction velocities.

33 set and the degree of slowing of motor nerve conduction velocities.

34 lation and the horses with the slowest nerve conduction velocities.

35 ceps muscle contractility, and sciatic nerve conduction velocities.

36 sarcomeric structure, action potentials and conduction velocities.

37 Schwann cells (~5%), and evaluation of nerve conduction velocity (0.13-0.28 m/sec), previously unreal

38 , bipolar EGM amplitude (0.08+/-0.11 mV) and conduction velocity (0.27+/-0.19 m/s) were lower than th

39 versus 64%, P=0.008), exhibited slower local conduction velocity (0.49+/-0.43 versus 0.93+/-0.57 m/s,

40 In 1D tissue with normal longitudinal conduction velocity (0.55 m/s), the numbers of contiguou

41 ime (103+/-14 versus 43+/-13 ms), and slower conduction velocity (0.87+/-0.23 versus 1.39+/-0.21 m/s)

42 mm of the leading wavefront (30%, P<0.0001); conduction velocity (1.0+/-0.7 m/s, P=0.002) and voltage

43 discontinuity (qualitative) and decrease in conduction velocity (13%) and electrogram amplitude (21%

44 nduction velocity=3.1+/-0.1 cm/s) or strong (conduction velocity=22.1+/-0.4 cm/s) electrical coupling

45 icular myocytes (AP duration=153.2+/-2.3 ms, conduction velocity=22.3+/-0.3 cm/s) seamlessly interfac

46 es, (2) greater slowing of motor and sensory conduction velocities, (3) less prolonged motor distal l

47 sodium (Na(v)1.5) channels with either weak (conduction velocity=3.1+/-0.1 cm/s) or strong (conductio

48 MEHP exposure also slowed epicardial conduction velocity (35 versus 60 cm/s), which may be pa

49 /- 1 ms; p < 0.001), and slower longitudinal conduction velocity (62 +/- 2 cm/s vs. 70 +/- 1 cm/s; p

50 is reached, beyond which no further gains in conduction velocity accrue.

51 Conduction velocity across BB is around 90 cm/s.

52 axonal degeneration and may lead to impaired conduction velocity across surviving axons after stroke.

53 Simultaneous optical Vm mapping showed that conduction velocity, action potential duration, and dVm/

54 ulthood, resulting in a 30% reduction in the conduction velocity along the adult sciatic nerves.

55 The enhancement of conduction velocity along with the evolution of jaws lik

56 ts further demonstrated spatially discordant conduction velocity alternans which resulted in nonunifo

57 travelling wave and from which instantaneous conduction velocity amplitude and direction can be compu

58 action potential amplitudes, improved nerve conduction velocities and ameliorated muscle strength.

59 rving spatially uniform properties with high conduction velocities and contractile stresses.

60 th hypo- and demyelination, slowing of nerve conduction velocities and disturbed nodal architecture.

61 We sought to validate omnipole-derived conduction velocities and explore potential application

62 tensity in 78 dye-injected DRG neurons whose conduction velocities and hindlimb sensory receptive fie

63 fat diet to a MUFA-rich high-fat diet; nerve conduction velocities and intraepidermal nerve fiber den

64 /R98C) mice develop weakness, abnormal nerve conduction velocities and morphologically abnormal myeli

65 cular myocyte monolayers demonstrated slowed conduction velocity and a reduced maximum capture rate a

66 tural changes were associated with decreased conduction velocity and conduction block.

67 oretical optimal cell length with respect to conduction velocity and consider the possibility of epha

68 rdiac adipose tissue-secreted factors slowed conduction velocity and contained proteins with capacity

69 rm sodium channel distribution increases the conduction velocity and decreases the time delays over g

70 density, A Pupstroke velocity, AP duration, conduction velocity and EAD incidence, as well as reflec

71 rthermore, inner-zone GCs have faster axonal conduction velocity and elicit faster synaptic potential

72 ding critical clinical metrics such as nerve conduction velocity and histomorphometry are necessary t

73 The local conduction velocity and image intensity ratio in the lef

74 rophysiological substrate revealed decreased conduction velocity and increased AP duration (APD) hete

75 positively correlated with decreasing nerve conduction velocity and increasing pain severity.

76 y ameliorated DPN by improving sciatic nerve conduction velocity and increasing thermal and mechanica

77 examine the association between left atrial conduction velocity and LGE in patients with atrial fibr

78 c-Src inhibition improved Cx43 levels and conduction velocity and lowered arrhythmia inducibility

79 is best known for its role in increasing the conduction velocity and metabolic efficiency of long-ran

80 critical for the re-establishment of axonal conduction velocity and metabolic support to the axons.

81 Such dysregulations led to defects in conduction velocity and motor and sensory functions that

82 Axon myelination increases the conduction velocity and precision of action potential pr

83 Nerve conduction velocity and psychophysical data were acquire

84 mpanied by decreased motor and sensory nerve conduction velocity and reduced compound muscle action p

85 argeted manipulation of Cx43 levels improved conduction velocity and reduced ventricular tachycardia

86 as nociceptors or non-nociceptors based upon conduction velocity and response to transient receptor p

87 ficiency or proportionate increases in their conduction velocity and support the view that the intern

88 ion potential timing is shaped by the axonal conduction velocity and the duration of synaptic transmi

89 a(V)1.5, plays a pivotal role in setting the conduction velocity and the initial depolarization of th

90 e interpeak latency, indicative of increased conduction velocity and thereby enhanced functional repa

91 Conduction velocity and threshold for alternans monotoni

92 icted that this geometry helps to adjust the conduction velocity and timing of action potentials with

93 caliber plays a crucial role in determining conduction velocity and, consequently, in the timing and

94 atrial and left atrial voltage distribution, conduction velocities, and electrogram characteristics w

95 c nerve conduction delays depend on RGC axon conduction velocities, and velocity is primarily determi

96 velocity, as assessed by the SDs of APD and conduction velocity, and atrial fibrillation inducibilit

97 well as magnetic field correlates of the AP conduction velocity, and directly determines the AP prop

98 longitudinal conduction velocity, transverse conduction velocity, and dV/dt(max) in fibrotic monolaye

99 width helps to control transmitter release, conduction velocity, and firing patterns and understandi

100 Longitudinal conduction velocity, transverse conduction velocity, and normalized action potential ups

101 eir connections, as well as by the diameter, conduction velocity, and synaptic efficacy of their axon

102 een connexin43 (Cx43) expression, myocardial conduction velocity, and ventricular tachycardia in a mo

103 the 62 capsaicin-sensitive C-fibres studied (conduction velocity approximately 0.5 m s(-1)), 71% were

104 pacing (n=11), flecainide reversibly reduced conduction velocity ( approximately 30% at cycle length

105 ength, no significant change was detected in conduction velocity ( approximately 43 m/s) measured eit

106 of nerves reach a plateau beyond which their conduction velocities are no longer sensitive to increas

107 Predictions that conduction velocities are sensitive to the distance betw

108 In postinfarction reentrant VT, conduction velocities are slowest at the proximal and di

109 host axons and restored nodes of Ranvier and conduction velocity as efficiently as CNS-derived precur

110 This study examined AP rise times and conduction velocity as the depolarizing wavefront approa

111 ock their action potentials and reduce their conduction velocities, as expected from previous knowled

112 P increased spatial heterogeneity of APD and conduction velocity, as assessed by the SDs of APD and c

113 The local conduction velocity at each point was calculated using p

114 matical modeling predicted a 25% decrease in conduction velocity at the lesion epicenter due to short

115 PP1-treated mice had restored conduction velocity at the scar border (PP3: 32 cm/s, PP

116 ain-Barre syndrome, we determined that nerve conduction velocities between the sciatic notch and the

117 amples are provided, showing a difference in conduction velocity between two different types of cardi

118 with respect to their sensory modalities and conduction velocities, but also in their relative roles

119 fibers, specifically sural or sciatic nerve conduction velocities, but significantly improved measur

120 ination is not only important for maximizing conduction velocity, but also for limiting hyperexcitabi

121 altered Na(v)1.5 function can reduce cardiac conduction velocity by 28% compared with control.

122 Myelin sheath length directly impacts axonal conduction velocity by influencing the spacing between n

123 Conduction velocities calculated from omnipolar electrog

124 Axonal conduction velocity can change substantially during ongo

125 roduced significant deficits in caudal nerve conduction velocity, caudal amplitude and digital nerve

126 esults in more rapid AP upstrokes and higher conduction velocities compared with the bulk myocardium.

127 ocytes with WT and mutant channels increased conduction velocity compared with noninfected cells.

128 e with severe PAD have poorer peroneal nerve conduction velocity compared with people with mild PAD o

129 termine atrial effective refractory periods, conduction velocity, conduction heterogeneity index, and

130 cantly higher myelinated fiber densities and conduction velocities consequent to acute sciatic nerve

131 stigate how these changes in myelination and conduction velocity contribute to signal integration in

132 , we find that the variation in diameter and conduction velocity correlates with the mean diameter, c

133 e function, we measured unmyelinated C-fiber conduction velocities (CV) in nerves of SIV-infected mac

134 t sites exhibiting rate-dependent slowing in conduction velocity (CV restitution) or local slowing ev

135 VT with comparison of circuit dimensions and conduction velocity (CV) across a wide range of both sta

136 othesis that alterations to action potential conduction velocity (CV) and conduction anisotropy in le

137 (Tyrode's solution), which acts to increase conduction velocity (CV) close to the tissue-fluid inter

138 showed slight (7%) reduction of ventricular conduction velocity (CV) compared to 16 wild-type hearts

139 Characterizing myocardial conduction velocity (CV) in patients with ischemic cardi

140 udied the effect of interstitial fibrosis on conduction velocity (CV) in the left atrial appendage of

141 Reduced conduction velocity (CV) in the myocardium is well known

142 Conduction velocity (CV) is an important property that c

143 approach produces anisotropic cultures with conduction velocity (CV) profiles that closer resemble n

144 ion potential duration (APD) restitution and conduction velocity (CV) restitution curves in these pat

145 (STM) (alpha, tau) and their interplay with conduction velocity (CV) restitution on alternans format

146 ive refractory period (ERP) restitution, and conduction velocity (CV) restitution.Temporal and spatia

147 Evaluation included atrial voltage, conduction velocity (CV), and refractoriness (7 sites, 2

148 pendency of I(Kr) enables it to control APD, conduction velocity (CV), and wavelength (WL) at the exc

149 Effects of mutations on AP duration (APD), conduction velocity (CV), effective refractory period (E

150 ), has been proposed as a key determinant of conduction velocity (CV).

151 SHR hearts had slower conduction velocity (CV; P<0.01 versus WKY), steeper CV

152 ed on the softer substrates displayed higher conduction velocities (CVs) and improved electrophysiolo

153 Ms) and as having C-, Adelta- or Aalpha/beta-conduction velocities (CVs).

154 observed that, in addition to reduced nerve conduction velocity, db/db mice also develop PNH.

155 than uninfected animals and the magnitude of conduction velocity decline correlated strongly with ext

156 Conduction velocity decreased from 0.46 +/- 0.02 m/sec t

157 propagation, action potential duration, and conduction velocity), disrupted by IR injury, were resto

158 reased gap junction lateralization and lower conduction velocity due to downregulation of Connexin 43

159 nal current, it does not significantly alter conduction velocity during cardiac fibrosis relative to

160 restoration of action potential duration and conduction velocity early after reperfusion.

161 t of standalone module for quantification of conduction velocity, employing multiple methodologies, c

162 ocities individually to achieve the specific conduction velocities essential for precise temporal int

163 Although myelination and conduction velocities eventually recovered, polyaxonal m

164 activity levels, and axonal myelination and conduction velocity exhibited no adaptations.

165 KEY POINTS: Changes in nerve conduction velocity following an impulse (i.e. velocity

166 tic agents and a moderate reduction of nerve conduction velocity for cisplatin and paclitaxel.

167 ome, but not healthy controls, sensory nerve conduction velocity for Digits 2 and 3 was slower than D

168 complement synaptic plasticity by adjusting conduction velocity for optimal spike-time arrival.

169 t was less negative, and slower upstroke and conduction velocity for rotors in the PV region than in

170 1) in all hearts, and reduced the transmural conduction velocity from 36 cm/s (95% CI, 30-42) to 32 c

171 ervention and training-induced changes in MU conduction velocity had an effect size of 2.1 (tracked M

172 treated mice showed increased sciatic nerve conduction velocities, improvement of myelination and re

173 Here we measured nerve conduction velocities in a rabbit model of limb lengthen

174 lso reveal that the reduction of motor nerve conduction velocities in affected patients is proportion

175 rdings in vitro and in vivo confirmed higher conduction velocities in low-frequency axons.

176 ner axonal diameters and internodal lengths, conduction velocities in mutant quadriceps nerves were a

177 VIP shortened APD and slowed conduction velocity in a dose-dependent manner.

178 In contrast, beta2-stimulation increased conduction velocity in both donor and failing hearts but

179 Here, we show that the action potential conduction velocity in both myelinated and unmyelinated

180 aluate NINM effects on pain rating and nerve conduction velocity in DPN patients.

181 x of peripheral neuropathy is the slowing of conduction velocity in large myelinated neurons and a la

182 Conduction velocity in RMCCs was slower when compared wi

183 ted macaques had significantly lower C-fiber conduction velocity in sural nerves than uninfected anim

184 coupling led to slow electrical propagation; conduction velocity in TBX18 NRCMs slowed by more than 5

185 d a marked reduction in the left ventricular conduction velocity in the absence of myocardial Cav1, w

186 Nevertheless, conduction velocity in the adult RVOT was similar to tha

187 ad less electrogram fractionation and faster conduction velocity in the anterior-septal border zone.

188 ciated with a significant reduction in nerve conduction velocity in the optic nerve.

189 Conduction velocity in the visual pathways correlated cl

190 ansfected cells and sensory neurons, slowing conduction velocity in unmyelinated peripheral nerve fib

191 es encoding molecular determinants of atrial conduction velocity, including Scn5a (Nav1.5) and Gja5 (

192 50 mum from the epicardial surface, whereas conduction velocity increased and AP duration was only s

193 two collaterals of a single axon adjust the conduction velocities individually to achieve the specif

194 ty may be because of the oscillations of the conduction velocity inside the tachycardia circuits.

195 Heterogeneity in regional conduction velocities is a critical substrate for functi

196 athy, but high activity-dependent slowing of conduction velocity is more common in fibromyalgia patie

197 in addition to decreased heart rate, atrial conduction velocity is persistently slower than control.

198 se and that alterations in myelin can modify conduction velocity, leading to changes in neural circui

199 ents with E90K and N98S had upper limb motor conduction velocities <38 m/s.

200 chniques, such as electromyography and nerve conduction velocity measurement, do not reliably predict

201 termined by abnormal motor and sensory nerve conduction velocity, mechanical allodynia, and loss of i

202 onal loss, which underlie slowed motor nerve conduction velocity (MNCV) and reduced compound muscle a

203 tary locomotor activity, reduced motor nerve conduction velocities (MNCVs) and muscle atrophy.

204 MD, increased myelination and restored nerve conduction velocity, motor function and lifespan of the

205 s R98C animals, but it did not improve nerve conduction velocity, myelin thickness, G-ratios or myeli

206 plantation significantly improved tail nerve conduction velocity, Na(+)-K(+)-ATPase activity, and mor

207 n either behaviors or acute changes in nerve conduction velocity (NCV) and amplitude, but greater und

208 Nerve conduction velocity (NCV) significantly improved in only

209 ive correlations were found with sural nerve conduction velocities (NCVs) (r = -0.65, P < 0.001) and

210 A values were correlated to electrical nerve conduction velocities (NCVs) with Pearson correlation an

211 cts of each drug on caudal and digital nerve conduction velocity, nerve amplitude, and sciatic nerve

212 ment in the caudal SAN as shown by decreased conduction velocity, OAP amplitude, gradient and activat

213 uding Rin and lambda as well as waveform and conduction velocities of fully propagating action potent

214 ple carbon fibers per experiment, determined conduction velocities of some vagal signals in the affer

215 We measured conduction velocities of the ipsilateral and contralater

216 In addition, axon density was reduced and conduction velocities of the trigeminal and sciatic nerv

217 normalized the cisplatin-induced decrease in conduction velocity of Aalpha/Abeta-fibers and reduced t

218 hysiological measurements indicated that the conduction velocity of earthworm medial giant nerve fibe

219 Although the conduction velocity of sciatic nerves of mutant mice sho

220 We detected reduced nerve conduction velocity of the left RLn, compared to the rig

221 ulation significantly increased peak INa and conduction velocity of WT but not mutant channels.

222 ies have demonstrated a strong dependence of conduction velocity on internodal length.

223 t of adaptive, activity-dependent changes in conduction velocity on the large-scale phase synchroniza

224 No difference was seen at baseline for conduction velocity or dispersion of repolarization betw

225 or control groups: greater slowing in atrial conduction velocity (P<0.0001 and P<0.001); increased ab

226 group had modest but significant slowing in conduction velocity (P<0.01) but no change in conduction

227 image intensity ratio (0.20 m/s decrease in conduction velocity per increase in unit image intensity

228 Increased axonal diameter did not increase conduction velocity, possibly due to a failure to increa

229 odeling indicated that HCN2 channels control conduction velocity primarily by altering the resting me

230 on in compound muscle action potentials, low conduction velocities, prolonged distal latencies and pr

231 tion, fibrosis, increased vagal tone, slowed conduction velocity, prolonged cardiomyocyte action pote

232 correlations were found for the tibial nerve conduction velocity (r = -0.23; 95% CI: -0.44, -0.01; r

233 correlations were found for the tibial nerve conduction velocity (r = -0.24; 95% CI: -0.45, -0.01).

234 ed to a greater inverse correlation (r) with conduction velocity (r for adipose=0.39, r for discontin

235 their response properties, action potential conduction velocity, rate of adaptation to static indent

236 ternodes lengthened during postnatal growth, conduction velocities recovered to normal values and mut

237 excitability in the periinfarct zone; axonal conduction velocity recovered by 21 d post MCAO in KI mi

238 f R98C/+ mice but does not alter the reduced conduction velocities, reduced axonal diameters or clini

239 lation creates arrhythmogenic substrates via conduction velocity regulation and transmurally heteroge

240 Conduction velocity remained constant at chemically incr

241 entity, peripheral anatomy, central anatomy, conduction velocity, response properties in vitro and in

242 bility per se, but instead influences axonal conduction velocity, resting membrane potential, and noc

243 on potential duration restitution (APDR) and conduction velocity restitution (CVR) curves were genera

244 The mechanism of DEEPs relates to conduction velocity restitution magnified by zigzag cond

245 on of fibrillation by modulating APD and APD/conduction velocity restitution slopes in atrial tissue

246 entry by prolonging APD and changing APD and conduction velocity restitution slopes, thereby altering

247 By analyzing the effects of conduction velocity restitution, cellular dynamics, elec

248 All strategies prevented loss of nerve conduction velocity resulting from STZ-induced diabetes

249 al, P=0.0005; left atrial, P=0.0001), slower conduction velocities (right atrial, P=0.02; left atrial

250 ernodal lengths and associated reduced nerve conduction velocity seen in the absence of periaxin, sho

251 beta1-Stimulation significantly increased conduction velocity, shortened action potential duration

252 thermia attenuated both the ischemia-induced conduction velocity slowing (decreasing from 0.44 +/- 0.

253 ardiac wavelength, due to the attenuation of conduction velocity slowing (p = 0.03), and the preserva

254 Unlike conduction velocity slowing and APD heterogeneity, the m

255 -1 and (R)-1 partially prevented motor nerve conduction velocity slowing in a mouse model of type 1 d

256 evented focal energy deprivation, transverse conduction velocity slowing, and the reentrant ventricul

257 phy of the dorsal cricoarytenoid muscles and conduction velocity testing of the innervating recurrent

258 er EGM amplitude with a significantly slower conduction velocity than the surrounding parts of the ci

259 ch a mechanism would allow for variations in conduction velocities that provide a degree of plasticit

260 Diabetes caused a decrease in nerve conduction velocity, thermal hypoalgesia, and a reductio

261 ed high-fat diets caused a decrease in nerve conduction velocity, thermal hypoalgesia, and intraepide

262 Conduction velocities (theta'), action potential wavelen

263 reased OPC maturation and promoted increased conduction velocities through lesions.

264 ectric uncoupling increased excitability and conduction velocity to 133% in 28% hMSC cocultures, but

265 The average conduction velocity to the Sp5I neurones was 0.94 +/- 0.

266 Longitudinal conduction velocity, transverse conduction velocity, and

267 ptomycin dramatically increased longitudinal conduction velocity, transverse conduction velocity, and

268 itigate the frequency-dependent decreases in conduction velocity typical of C-fiber axons.SIGNIFICANC

269 d initial activity-dependent acceleration of conduction velocity upon low frequencies of stimulation

270 e animal studies demonstrated improvement in conduction velocity using CNTf.

271 Mean effective conduction velocity was 89 cm/s.

272 nd adjusting for left atrial wall thickness, conduction velocity was associated with the local image

273 Median conduction velocity was higher (75 +/- 9 versus 65 +/- 1

274 Also, conduction velocity was lower in the RVOT than in the ri

275 Conduction velocity was measured within RMCCs and compar

276 the spatial profile of hERG-GFP expression; conduction velocity was not altered.

277 A marked decrease in conduction velocity was observed in strands composed of

278 of DeltaSIV hearts revealed that ventricular conduction velocity was preferentially decreased in the

279 mice with short mutant Schwann cells, nerve conduction velocity was reduced and motor function was i

280 ing revealed that selectively the transverse conduction velocity was slowed and anisotropy increased.

281 The conduction velocity was slower than that reported in nor

282 In heterocellular cultures, MI-Fb conduction velocities were different from Fb at all dens

283 Conduction velocities were higher and action potential d

284 Moreover, motor nerve conduction velocities were increased in treated Sh3tc2-/

285 n in the placebo controls (P=0.015), and the conduction velocities were significantly faster (P=0.005

286 Conduction velocities were slowest at the inward curvatu

287 Transmural dispersion of repolarization and conduction velocity were measured at baseline, during is

288 The action potential waveform and axonal conduction velocity were unaffected, suggesting that the

289 Abnormally high slowing of conduction velocity when first stimulated at 0.25Hz was

290 o reentry were harbored in regions with slow conduction velocity, where 3 isochrones were present wit

291 limited width critical isthmuses with slower conduction velocity, whereas sites with dPPI=0-30 ms are

292 Axonal conduction velocity, which ensures efficient function of

293 array platform and thereby the estimation of conduction velocity, which gradually increased during th

294 in a progressive slowing of action potential conduction velocity, which manifests as a progressive in

295 ial heterogeneity and decreases intra-atrial conduction velocity, which may play an important role in

296 ity mechanism for dynamically fine-tuning AP conduction velocity, which potentially has wide implicat

297 ) showed an age-dependent increase of axonal conduction velocity, which was positively correlated wit

298 nerability, shortened ERP and altered atrial conduction velocity, which, in combination, facilitate i

299 Conduction velocities within the shared isthmus were dep

300 nment pacing causes some perturbation of the conduction velocity within the tachycardia circuit, whic