ライフサイエンスコーパス: congestion

1 revealed cardiomegaly and pulmonary vascular congestion.

2 left atrial hypertension, exacerbating lung congestion.

3 ents with HFpEF without worsening left heart congestion.

4 -occupancy vehicle (HOV) policies can combat congestion.

5 ccuracy and better cope with single molecule congestion.

6 ces, for overall mortality, or with downwind congestion.

7 the circulation and thereby relieving tissue congestion.

8 on between regional function and presence of congestion.

9 oteins are largely precluded due to spectral congestion.

10 scular mortality were suggested with greater congestion.

11 single-occupancy cars often leads to traffic congestion.

12 flaps failed in the DIEP group due to venous congestion.

13 asal itching, sneezing, watery discharge and congestion.

14 mand explains the percentage of time lost in congestion.

15 t cavity, but it still suffers from spectral congestion.

16 city on their routes determines the level of congestion.

17 distance accompanied by repulsive electronic congestion.

18 nts and lung function and increases in nasal congestion.

19 ute decompensated heart failure and clinical congestion.

20 in and it may lead to prostatic and urethral congestion.

21 mbined impact of traffic control and traffic congestion.

22 kely to fully address the problem of orbital congestion.

23 on fraction, left ventricular mass, and lung congestion.

24 m relief by addressing signs and symptoms of congestion.

25 from sample ions and thereby reduce spectral congestion.

26 ivity and LV end-diastolic pressure and lung congestion.

27 ge, microvascular thrombosis, and neutrophil congestion.

28 diomyocyte apoptosis and minimized pulmonary congestion.

29 nts are not very effective in treating nasal congestion.

30 3% in subjects without both asthma and nasal congestion.

31 ical techniques for identifying each type of congestion.

32 d afterload, which in turn lead to pulmonary congestion.

33 and symptoms of heart failure referred to as congestion.

34 association with SCAI stages and hemodynamic congestion.

35 and signs of HF, mostly related to systemic congestion.

36 five patients with symptoms of chronic nasal congestion.

37 ood volume and thereby reduces intravascular congestion.

38 treatment effects of sacubitril/valsartan on congestion.

39 sover design, we examined the association of congestion 0-150 m, 151-300 m, and 301-1,000 m upwind of

40 The most common adverse events were nasal congestion (16% of patients) and myalgia (14%).

41 y (30% vs. 8%) lymphoma, episcleral vascular congestion (40% vs. 16%), anterior chamber reaction (30%

42 the subgroup reporting both asthma and nasal congestion, 55.8% had insomnia symptoms compared with 35

43 damage (70/70) and capillary dilatation and congestion (70/70), often accompanied by microthrombi (2

44 those patients with moderate-to-severe lung congestion, 71% were asymptomatic or presented slight sy

45 82%), chronic cough (99%), and chronic nasal congestion (97%).

46 c link between systemic microvasculature and congestion, a central feature of the syndrome, has yet b

47 ractive (or repulsive) forces resulting from congestion, accessibility, and travel demand.

48 This suggests that the steric congestion afforded by these bulky substituents imposes

49 ency of abdominal pain, diarrhoea, and nasal congestion after all three vaccinations was significantl

50 al examination of the affected organs showed congestions, aggregations and multinucleated hepatocytes

51 d and capacity exist, the possible limits of congestion alleviation by only modifying route choices h

52 However, we find many individual sites of congestion along the mRNAs where the probability of ribo

53 n, and a considerable contributor to traffic congestion and air pollution causing substantial adverse

54 ctorily explain the detrimental link between congestion and cardiorenal function.

55 V) afterload in VA-ECLS can worsen pulmonary congestion and compromise myocardial recovery.

56 However, owing to their distinct steric congestion and conformational restriction, the formation

57 arch is urgently needed, given the burden of congestion and data suggesting sustained benefits of ear

58 onstrate that LukED and HlgAB cause vascular congestion and derangements in vascular fluid distributi

59 he pathogenesis of PCV may include choroidal congestion and dilatation.

60 osis and extensive vascular damage: vascular congestion and dilation, endothelial necrosis, alteratio

61 Venous congestion and edema within the optic nerve related to h

62 , defined as worsening signs and symptoms of congestion and elevated natriuretic peptide level requir

63 aging showed transient decreases in vascular congestion and exudation after intravitreal anti-VEGF in

64 charged ions, can be used to alleviate this congestion and facilitate the identification of addition

65 ic lethality at mid-gestation, with systemic congestion and hypoxia.

66 ly reduced peritubular capillary erythrocyte congestion and improved histologic scores of tubular inj

67 2 (sST2) is released in response to vascular congestion and inflammatory and pro-fibrotic stimuli, an

68 ding with resultant gravity-dependent venous congestion and inflammatory vasculitis.

69 It was designed to overcome the spectral congestion and line broadening that occur due to sample

70 ) 18 years and older with moderate-to-severe congestion and NPs were randomized to twice-daily EDS-FL

71 flects LV preload independent of right heart congestion and pericardial restraint, was similar in TR

72 -term changes in markers of organ damage and congestion and relate them to 180-day mortality in patie

73 olume Overload to Assess Treatment Effect on Congestion and Renal Function (PROTECT) trial and valida

74 olume Overload to Assess Treatment Effect on Congestion and Renal Function study.

75 olume Overload to Assess Treatment Effect on Congestion and Renal Function) study in 1,969 patients w

76 olume Overload to Assess Treatment Effect on Congestion and Renal Function), a trial comparing the ef

77 As expected, nasal congestion and rhinorrhea were most common, but interest

78 Some also present with nasal congestion and subjective olfactory impairment.

79 okine levels, nasal secretion weights, nasal congestion and symptom scores were determined.

80 ive, unidentified target(4); however, steric congestion and the instability of bilobalide(1,2,6) have

81 ifferent forms of congestion - intravascular congestion and tissue congestion - and hypothesize that

82 atory distress in >80% of cases, daily nasal congestion and wet cough starting soon after birth, and

83 s (vs 50-64 years), symptoms of cough, nasal congestion and wheezing, and longer interval from illnes

84 Women had more symptoms and signs of congestion and worse quality of life.

85 estion - intravascular congestion and tissue congestion - and hypothesize that differentiating betwee

86 agnosis of patients presenting with dyspnea, congestion, and a normal ejection fraction.

87 ead to isobaric interferences, mass spectral congestion, and ambiguous ion identification.

88 p included peripheral edema, headache, nasal congestion, and anemia.

89 uctural alterations, the absence of vascular congestion, and attenuation of glomerulosclerosis in tre

90 ConA induced inflammation, sinusoidal congestion, and extensive midzonal hepatocyte death in c

91 from systolic dysfunction, hypertrophy, lung congestion, and fibrosis induced by chronic isoprotereno

92 Despite worse symptoms, more congestion, and lower quality of life, women had similar

93 se alveolar damage, capillary dilatation and congestion, and microthrombosis.

94 lar hypertrophy, diastolic dysfunction, lung congestion, and myocardial oxidative stress without affe

95 Sensors can measure air quality, traffic congestion, and other aspects of urban environments.

96 tand the relationships among symptom relief, congestion, and outcomes in patients with acute decompen

97 recapitulate the pulsatile forces induced by congestion, and performed microarray and RNA-sequencing

98 nd extent of hepatic inflammation, fibrosis, congestion, and portal hypertension.

99 terioration, attenuated hypertrophy and lung congestion, and prevented apoptosis and fibrotic remodel

100 ure to seasonal pollen causes itching, nasal congestion, and repeated sneezing, with profound effects

101 is information is often obscured by spectral congestion, and site-selective information is not availa

102 y, striped interstitial fibrosis, glomerular congestion, and tubular microcalcification were all grea

103 distribution, trauma center access, traffic congestion, and urbanicity (P < 0.05).

104 athing, fast breathing, runny nose, or nasal congestion; and current runny nose, nasal congestion, fe

105 beneficial effect on airway symptoms (nasal congestion, anterior rhinorrhea, loss of sense of smell,

106 Rhinitis medicamentosa with nasal congestion appears readily reversible with suitable trea

107 Uncontrolled asthma and nasal congestion are important, treatable risk factors for ins

108 rizing alkoxycarbonylation reveal the steric congestion around the diphosphine coordinated Pd-center

109 r vaccinating infants, who may develop nasal congestion as a result of intranasal vaccination.IMPORTA

110 equences such as reduced organ perfusion and congestion as well as maladaptive neurohormonal activati

111 peremia) and during venous occlusion (venous congestion), as assessed with skin capillaroscopy, and a

112 We evaluated whether spironolactone improved congestion at 4 months and whether improvement in conges

113 ry outcome irrespective of clinical signs of congestion at baseline (P=0.16 for interaction), and tre

114 ne the association between physical signs of congestion at baseline and during study follow-up with q

115 Steric congestion at the indole-C3 position and improved pai-pa

116 Furthermore, residual congestion before discharge from hospital is associated

117 rt failure-associated splanchnic circulation congestion, bowel wall edema, and impaired intestinal ba

118 versus 320 pg/mL; all P<0.01), more signs of congestion, but no significant difference in left ventri

119 lactone reduced the total number of signs of congestion by -0.10 ( P=0.005) signs, jugular venous dis

120 Increased severity of congestion by a simple orthodema assessment is associate

121 reducing preload on the heart, and relieves congestion by clearing interstitial fluid without excess

122 oval sometimes worsens economic damages from congestion by increasing launch incentives.

123 Detection of exercise-induced pulmonary congestion by lung ultrasound is an independent predicto

124 pray (alpha adrenergic agonist) causes nasal congestion by rhinitis medicamentosa.

125 JNJ-39220675 relieved allergen-induced nasal congestion by using standard nasal symptom scoring; howe

126 entilatory control and efficiency, pulmonary congestion, capillary stress failure, and eventually pul

127 however, whether exercise-induced pulmonary congestion carries prognostic implications is unknown.

128 In London, congestion-charging and a citywide low-emission zone fai

129 osis is important, as it can lead to hepatic congestion, cirrhosis and Budd-Chiari syndrome (BCS) and

130 ly the result of its relative lack of steric congestion compared to the trityl radical.

131 Spironolactone improved congestion compared with placebo.

132 Under congestion conditions, the UFP vehicle EFs increased fro

133 Chronic passive hepatic congestion (congestive hepatopathy) leads to hepatic fib

134 that this relation is reversed on all other congestion control algorithms for a region of the parame

135 (max-min fairness and proportional fairness) congestion control schemes, and analyse how the algorith

136 tween the two fields of complex networks and congestion control.

137 tear film breakup time (TBUT), conjunctival congestion, corneal haze, vascularization, conjunctivali

138 ically treating these two different forms of congestion could improve the outcomes of patients with a

139 t to take the opposite action, as in traffic congestion, crowd dispersion, and division of labor.

140 Despite intent to relieve congestion, current therapy often fails to relieve ortho

141 We leveraged finely resolved congestion data to investigate short-term associations w

142 Absence of pulmonary congestion detected by LUS implied a negative predictive

143 namely congestion propagation rate beta and congestion dissipation rate mu.

144 s infection is followed by intense pulmonary congestion due to an extensive influx of macrophages and

145 aluated whether sacubitril/valsartan reduced congestion during follow-up and whether improvement in c

146 panied by RWMA, abnormal LVCR, and pulmonary congestion during stress, and shows independent value ov

147 gns of organ damage and more rapid relief of congestion during the first days after admission.

148 t has been found that 0.5% PVI can attenuate congestion, edema and pain induced by pressure sores.

149 it is unclear how the clinical components of congestion (eg, peripheral edema, orthopnea) contribute

150 With dependence on the steric congestion encompassing the N-C-C-N fragment of the Ar-B

151 nd respiratory mortality with greater upwind congestion, especially congestion near the decedent's ho

152 th respect to pollution, energy consumption, congestion, etc.

153 ention and redistribution result in systemic congestion, eventually causing organ dysfunction due to

154 al congestion; and current runny nose, nasal congestion, fever, chest indrawing, or cyanosis.

155 whether body elongation is linear or whether congestion forms within the flow and the body axis becom

156 or no congestion, patients with very severe congestion had a 4.2-fold risk of death (HR=4.20, 95% CI

157 Each of these two forms of congestion has a different pathophysiology and requires

158 Abdominal congestion (i.e., splanchnic venous and interstitial con

159 The steric congestion imparted by the 3,3'-disubstituted binaphthyl

160 of applied goals such as reducing automotive congestion, improving disaster response, and even predic

161 evere lung congestion in 45% and very severe congestion in 14% of the patients.

162 We detected moderate-to-severe lung congestion in 45% and very severe congestion in 14% of t

163 days significantly reduced indices of organ congestion in 6 rabbits with HF relative to indices of 6

164 as seen in all patients, and chronic hepatic congestion in 8 patients.

165 ble to prevent endothelial damage and tissue congestion in a model of heme overload.

166 ddress the acute use of tolvaptan to improve congestion in AHF.

167 ted to be responsible for prominent systemic congestion in Chagas disease.

168 ing a reassessment of the pathophysiology of congestion in congestive heart failure and the methods b

169 the liver and spleen contribute to systemic congestion in heart failure.

170 ultrasound can detect asymptomatic pulmonary congestion in hemodialysis patients, and the resulting B

171 e Na(+)-K(+) pump activity and reverse organ congestion in severe heart failure (HF).

172 Including the degree of pulmonary congestion in the model significantly improved the risk

173 Elevated venous pressure produces congestion in the orbit with resultant transudation of f

174 Reducing congestion in the outpatient setting is independently as

175 To eliminate spectral congestion in the T2DIR spectra, we use a subtraction me

176 limited to short-term use to prevent rebound congestion, in limited circumstances, patients receiving

177 ntagonist, JNJ-39220675, in preventing nasal congestion induced by exposing participants with ragweed

178 urther lung damage due to elevated pulmonary congestion, inflammatory cell infiltration, iron overloa

179 spectrally isolated absorptions to avoid the congestion inherent to protein spectra.

180 rvations, including capillary dilatation and congestion, interstitial edema, diffuse alveolar damage,

181 view, we describe the two different forms of congestion - intravascular congestion and tissue congest

182 ents with acute heart failure, and relief of congestion is a critical goal of therapy.

183 Congestion is a primary driver of symptoms in patients w

184 Congestion is a primary reason for hospitalization in pa

185 High ICU congestion is associated with a lower likelihood of ICU

186 ny AHF patients at discharge and more severe congestion is associated with increased morbidity and mo

187 Persistent congestion is associated with worse outcomes in acute he

188 We further identify that venous congestion is common and identifies patients with CS at

189 Collision risk from this orbital congestion is costly to satellite operators.

190 derivatives of C60 and C70 with high steric congestion is described using 1,3-dibenzoylpropane bis-p

191 Pulmonary congestion is highly prevalent and often asymptomatic am

192 oved understanding of the pathophysiology of congestion is of great importance in finding better and

193 odilators targeting decongestion, persistent congestion is present in many AHF patients at discharge

194 in heart failure, especially when abdominal congestion is present.

195 during follow-up and whether improvement in congestion is related to changes in clinical outcomes an

196 Congestion is the main reason for hospitalization in pat

197 Congestion is the most frequent cause for hospitalizatio

198 failure hospitalizations occur annually, and congestion is the predominant cause.

199 pressure, as an extreme marker of abdominal congestion, is correlated with renal dysfunction in adva

200 ded as a first-line test to assess pulmonary congestion, it has never been tested in this setting.

201 ed the association of the number of signs of congestion (jugular venous distention, edema, rales, and

202 teral vein as well as prostatic and urethral congestion leading to intermittent urinary hesitancy, wh

203 re is associated with splanchnic circulation congestion, leading to bowel wall oedema and impaired in

204 Chronic hepatic congestion leads to sinusoidal thrombosis and strain, wh

205 zed by attenuated heart enlargement and lung congestion, lower expression of cardiac stress genes (B-

206 on (i.e., splanchnic venous and interstitial congestion) manifests in a substantial number of patient

207 rtex veins ampullas, suggesting that outflow congestion may be a contributing factor to the pathogene

208 h its capacity is prepared for daily demand, congestion may rise when huge crowds gather in demonstra

209 red intestinal barrier function secondary to congestion, might further depress cardiac as well as ren

210 To improve traffic operations, develop new congestion mitigation strategies, and reduce negative tr

211 medical service were admitted under high ICU congestion (more than 90% of beds occupied).

212 y with greater upwind congestion, especially congestion near the decedent's home.

213 difficult to interpret due to large spectral congestion, necessitating modeling to elucidate key spec

214 wever, increasing histone-induced neutrophil congestion, neutrophil extracellular trap formation, and

215 of inflammation including epithelial damage, congestion, neutrophil infiltration, loss of mucin from

216 Coprimary end points were change in nasal congestion/obstruction at 4 weeks and summed bilateral p

217 ge density, the flow remains constant and no congestion occurs.

218 Furthermore, we found that CTX-induced congestion of capillaries in the terminal ileum correlat

219 Venous congestion of the epiescleral and retinal vessels were o

220 t under diverse conditions of excludability, congestion of the non-excludable public good, and arbitr

221 exist as a weak pi-dimers due to the steric congestion of the thioalkyl groups in the superimposed P

222 rization and explain the influence of steric congestion of the vinyl moiety on the isomerization path

223 All patients had dyspnoea, congestion on chest radiograph, increased brain natriure

224 cute heart failure and had dyspnea, vascular congestion on chest radiography, increased plasma concen

225 d further explored the impact of hemodynamic congestion on clinical outcomes.

226 Moreover, absence of pulmonary congestion on LUS provided an excellent negative predict

227 combination of both intravascular and tissue congestion, one phenotype can dominate.

228 tions, obesity, renal function, the state of congestion or decongestion, and whether patients are rec

229 ; p<0.0001) in SINUS-52; difference in nasal congestion or obstruction score was -0.89 (-1.07 to -0.7

230 to week 24 in nasal polyp score (NPS), nasal congestion or obstruction, and sinus Lund-Mackay CT scor

231 port of a daily cough for 3 months in a row, congestion or phlegm other than when accompanied by a co

232 Nasal congestion (OR 1.50), obesity (OR 1.54) and smoking (OR

233 CI: 0.851, 0.997; P = 0.043), and sinusoidal congestion (OR: 0.954; 95% CI: 0.912, 0.998; P = 0.039).

234 ns on the ISS were for sleep problems, pain, congestion, or allergy.

235 respect to cause of portal hypertension (ie, congestion- or cirrhosis-induced hypertension).

236 nists administered at high doses may relieve congestion, overcome diuretic resistance, and mitigate t

237 d outcomes even after adjusting for baseline congestion (P<0.001).

238 equent facility visits, have led to facility congestion, patient and healthcare provider dissatisfact

239 mpared with those patients having mild or no congestion, patients with very severe congestion had a 4

240 Conjunctival congestion persisted in 44% (11/25) in the MT group comp

241 Rehospitalizations for recurrent congestion portend poor outcomes independently of age an

242 multaneously, time-updated, but not baseline congestion, predicted outcomes.

243 sent a framework to describe the dynamics of congestion propagation and dissipation of traffic in cit

244 ristics for network traffic dynamics, namely congestion propagation rate beta and congestion dissipat

245 Finally, we examine congestion relief under a centralized routing scheme wit

246 Loop diuretics are used for congestion relief, and dose adaptations are usually a co

247 ction), many patients required diuretics for congestion relief.

248 Volume overload and fluid congestion remain primary issues for patients with chron

249 54%, 20%, and 4% had 0, 1, 2, and 3 signs of congestion, respectively, at baseline.

250 umonia, bronchospasm, atelectasis, pulmonary congestion, respiratory failure, pleural effusion, pneum

251 ry acidosis, impaired gas exchange, systemic congestion, respiratory support/replacement therapies, a

252 can include red itchy eyes, sneezing, nasal congestion, rhinorrhea, coughing, bronchoconstriction, a

253 eeway and arterial roads under free-flow and congestion scenarios.

254 A simple orthodema congestion score was generated based on symptoms of orth

255 1) and -0.90 versus -0.31 (P = .0140); Nasal Congestion Score, -0.89 versus -0.35 (P = .0004) and -0.

256 Secondary end points included the clinical congestion score, dyspnea assessment, net urine output,

257 week 24 in Nasal Polyp Score (NPS) and Nasal Congestion Score.

258 l airflow, nasal secretion weight, and nasal congestion scores.

259 uency, cough severity, cough bothersomeness, congestion severity, rhinorrhea severity, and cough effe

260 ions and possible effects on reducing clinic congestion should be investigated.

261 lds (no fever, and >/=2 of runny nose, nasal congestion, sneezing, sore throat, cough, swollen or ten

262 We describe the dynamics of congestion spread using these new parameters embedded wi

263 In vitro studies show that congestion stimulates HSC fibronectin (FN) fibril assemb

264 Change in congestion strongly predicted outcomes even after adjust

265 alternative treatment strategies to relieve congestion, such as ultrafiltration, may also result in

266 of Na(+)-K(+) pump inhibition and indices of congestion suggest pump inhibition is a useful target fo

267 amination with mild to moderate conjunctival congestion, suggestive of microsporidial superficial ker

268 loss without significant rhinorrhea or nasal congestion suggests that SARS-CoV-2 targets the chemical

269 patients hospitalized with AHF, dyspnea, and congestion, the addition of tolvaptan to a standardized

270 ction), and treatment with the drug improved congestion to a greater extent than did enalapril (P=0.0

271 Sacubitril/valsartan improved congestion to a greater extent than did enalapril.

272 Persistent congestion trumps serum creatinine increases in predicti

273 pEF) typically develop dyspnea and pulmonary congestion upon exercise.

274 of vehicle mode of operation (free flow and congestion) using (1) concurrent 5 min measurements of U

275 y, the method was shown to decrease spectral congestion via fragmentation of multiple (charge-reduced

276 ustment, each additional increase in sign of congestion was associated with a 30% to 60% increased ri

277 High ICU congestion was associated with a 9% lower likelihood of

278 t Association class, increasing time-updated congestion was associated with all outcomes (P<0.001).

279 Sputum production or chest congestion was associated with higher risk of antibiotic

280 Each reduction in sign of congestion was independently associated with a 4.0 (95%

281 Reducing congestion was independently associated with improved qu

282 n moderate/severe MR cases (n = 8), vascular congestion was more intense (rho = 0.841; P < .001), seq

283 stion at 4 months and whether improvement in congestion was related to quality of life as assessed by

284 The cause of congestion was traced back to significant variability in

285 esolution of emission sources (i.e., traffic congestion) was poor.

286 hose ICU admission decisions are affected by congestion, we found that, if congestion were not a barr

287 re affected by congestion, we found that, if congestion were not a barrier and more eligible patients

288 Patients with baseline congestion were older, more often female, had higher MAG

289 8380 participants, 0, 1, 2, and 3+ signs of congestion were present in 70%, 21%, 7%, and 2% of patie

290 All the assessments except nasal congestion were slightly better in Cedarcure((R)) group,

291 ately (1) hospitalized individuals with rest congestion, where diagnosis is more straightforward; and

292 adverse events: not transplanted- pulmonary congestion with epilepticus (likely not related) versus

293 The association of the number of signs of congestion with the primary outcome (cardiovascular deat

294 (Targeting Acute Congestion With Tolvaptan in Congestive Heart Failure [T

295 d describe the design of the Targeting Acute Congestion With Tolvaptan in Congestive Heart Failure St

296 The TACTICS-HF (Targeting Acute Congestion with Tolvaptan in Congestive Heart Failure) s

297 mes higher odds of death with greater upwind congestion within 150 m and 1.02 (95% CI: 0.95, 1.10) ti

298 For each 10-minute-km increase in upwind congestion within 150 m, the odds of cerebrovascular mor

299 ts with diabetes mellitus have more signs of congestion, worse quality of life, higher N-terminal pro

300 tiles of capillary recruitment during venous congestion yielded an odds ratio of 2.89 (95% confidence