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1 within the cortical collecting duct and the connecting tubule.
2 y homologous nephron progenitor cell-derived connecting tubule.
3 f split-opened cortical collecting ducts and connecting tubules.
6 rin-dependent HCO(3)(-) secretion within the connecting tubule and cortical collecting duct, or gave
8 egments from the transition zone of the late connecting tubule and early cortical collecting duct dem
9 cell types of the nephron progenitor-derived connecting tubule and ureteric progenitor-derived collec
10 nit was more apically distributed within the connecting tubules and cortical collecting ducts of Pon3
11 he thick ascending limb of Henle's loop, the connecting tubule, and in some, but not all, cells of co
12 part of the distal convoluted tubule and the connecting tubule, and regulation results from the inter
13 ron, i.e., the distal convoluted tubule, the connecting tubule, and the cortical collecting duct.
15 from scRNA-seq pointed to proximal tubules, connecting tubules, and principal cells as likely cellul
16 ed syntaxin-4 mRNA in glomeruli, vasa recta, connecting tubules, and thin descending limbs of Henle's
17 cted rat cortical collecting ducts (CCD) and connecting tubules, but not in proximal tubules, medulla
19 mmunofluorescence ANKRA is also expressed in connecting tubule cells and principal cells of collectin
20 gene expression, primarily in principal and connecting tubule cells and, to a lesser extent, in segm
21 B and non-A-non-B intercalated cells of the connecting tubule (CNT) and the cortical collecting duct
22 split-open distal convoluted tubule 2 (DCT2)/connecting tubule (CNT) from mice on NL or LS/LM diets.
23 beta1-subunit is also expressed in the renal connecting tubule (CNT), a segment of the aldosterone-se
24 I constituted 35% to 40% of all cells in the connecting tubule (CNT), cortical collecting duct (CCD),
25 dn1 double-knockout mice (DE(AC) ), and Edn1 connecting tubule/collecting duct-specific conditional k
26 studied four groups of mice: wild-type mice, connecting tubule/collecting duct-specific Dot1l conditi
30 lower blood [K(+) ], more functional ENaC in connecting tubules/cortical collecting ducts, higher ami
31 absorption in the late distal convoluted and connecting tubules (DCT2/CNT) is initiated by Ca(2+) inf
32 AQP2 trafficking in cortical collecting and connecting tubules in parallel with an increase in urine
33 ed ASDN remodeling, involving a reduction in connecting tubule mass and attenuation of epithelial sod
35 rm the late distal convoluted tubule (DCT2), connecting tubule segments 1 and 2 (CNT1 and CNT2, respe
36 /NOTCH signaling, which expands the cortical connecting tubule with PCs and replaces acid-secreting a