戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1  contribution of acetylcholine to the airway constrictive and lumenal obstructive response after inha
2 sive and calcified coronary atherosclerosis, constrictive arterial remodeling, and greater disease pr
3 ted reaction intermediates and the effect of constrictive binding on the bound guest.
4  hosts and guests need to be balanced with a constrictive binding to allow thermally activated chemic
5  This reaction pathway, which is promoted by constrictive binding within the supramolecular cavity of
6                                   Rationale: Constrictive bronchiolitis (ConB) is a relatively rare a
7 stopathologic evidence of deployment-related constrictive bronchiolitis (DRCB) has been identified in
8        Primary bronchiolar disorders include constrictive bronchiolitis (obliterative bronchiolitis,
9  to the airway epithelium play a key role in constrictive bronchiolitis after lung transplantation, t
10                  These results, particularly constrictive bronchiolitis and polarizable crystals, sup
11                            All soldiers with constrictive bronchiolitis had normal results on chest r
12  remaining 11 soldiers, diagnoses other than constrictive bronchiolitis that could explain the presen
13 ion is associated with a progressive form of constrictive bronchiolitis that targets conducting airwa
14 ation and pulmonary edema in severe cases to constrictive bronchiolitis, being a more distant consequ
15 re similar, since all database biopsies have constrictive bronchiolitis, one has lung fibrosis with t
16 an analysis of biopsy samples showed diffuse constrictive bronchiolitis, which was possibly associate
17    Among the five soldiers biopsied, all had constrictive bronchiolitis.
18 f sustained targeted club cell injury causes constrictive bronchiolitis.
19 transforming growth factor-B and ameliorated constrictive bronchiolitis.
20 nsforming growth factor-beta and ameliorated constrictive bronchiolitis.
21 diers having changes that were diagnostic of constrictive bronchiolitis.
22 pathologic hypertrophy, and restrictive from constrictive cardiomyopathy.
23 uronal cells from the proximal stump and the constrictive, circumferential forces imposed by the cont
24          Penile injection therapy and vacuum constrictive devices are reasonable choices for men in w
25          Division can occur, however, when a constrictive force brings the midcell into a compressed
26 one can drive membrane constriction, or if a constrictive force from the tubulin homolog FtsZ is requ
27 st that Z-ring contraction serves as a major constrictive force generator to limit the progression of
28 eak mechanism drives division with a smaller constrictive force sufficient to bring the midcell into
29 Lmnb1 (-/-) neurons when exposed to external constrictive forces (migration into a field of tightly s
30 its the division machinery, and may generate constrictive forces necessary for cytokinesis.
31                        Histology confirmed a constrictive form of bronchiolitis caused by expansion o
32 -up echocardiograms showed resolution of the constrictive hemodynamics without pericardiectomy.
33  patients (57%), restrictive in 12 (34%) and constrictive in 3 (9%).
34 ents (40.5%), restrictive in 17 (40.5%), and constrictive in 8 (19%).
35 nical hypersensitivity after sciatic chronic constrictive injury (CCI) in MrgprX1 mice.
36  Here, we created infraorbital nerve chronic constrictive injury (ION-CCI) in rats, an animal model o
37 le rats following infraorbital nerve chronic constrictive injury (ION-CCI).
38  to dose of 10 mg/kg of 1 in the rat chronic constrictive injury model of neuropathic pain.
39 analgesic efficacy data from the rat chronic constrictive injury model of neuropathic pain.
40 l models of neuropathic pain, transection or constrictive injury to peripheral nerves produces ectopi
41 rization is responsible for the formation of constrictive membrane curvature.
42                  In addition, in the chronic constrictive model of neuropathic pain, compound 34 sign
43 ed in cold allodynia using rats with chronic constrictive nerve injury (CCI), a neuropathic pain mode
44                                 The cause of constrictive pericardial disease was indeterminate in 67
45    In acute pericarditis, the development of constrictive pericarditis (<0.5%) and pericardial tampon
46 ve pulmonary disease without an overlap with constrictive pericarditis (39.5+/-18.8 cm/s vs. 4.2+/-3.
47 h significant reductions in the incidence of constrictive pericarditis (4.4% vs. 7.8%; hazard ratio,
48 and differentiate between rare diseases like constrictive pericarditis (CP) and restrictive cardiomyo
49 ted to be useful for differentiating chronic constrictive pericarditis (CP) and restrictive cardiomyo
50 venous flow (PV) velocities in patients with constrictive pericarditis (CP) and to describe the influ
51                           Differentiation of constrictive pericarditis (CP) from restrictive cardiomy
52  peptide (BNP) measurements to differentiate constrictive pericarditis (CP) from restrictive cardiomy
53                     The clinical spectrum of constrictive pericarditis (CP) has been affected by a ch
54                                              Constrictive pericarditis (CP) is a disabling disease, a
55 between restrictive cardiomyopathy (RCM) and constrictive pericarditis (CP) is challenging and, despi
56 atheterization criteria for the diagnosis of constrictive pericarditis (CP) rely on equalization of i
57 study was to compare myocardial mechanics of constrictive pericarditis (CP) with restrictive cardiomy
58  and specific criterion for the diagnosis of constrictive pericarditis (CP), but simultaneous ventric
59                                           In constrictive pericarditis (CP), E' is not reduced, despi
60  to determine the association of etiology of constrictive pericarditis (CP), pericardial calcificatio
61 theterization criterion for the diagnosis of constrictive pericarditis (CP).
62  the causes and natural history of transient constrictive pericarditis (CP).
63  (n = 162), peritoneal disease (n = 10), and constrictive pericarditis (n = 2).
64           Patients with surgically confirmed constrictive pericarditis (n=130) at Mayo Clinic (2008-2
65 We present our institutional experience with constrictive pericarditis after lung transplant in an ef
66 stics and identify variables associated with constrictive pericarditis after lung transplantation.
67 mal in a substantial number of patients with constrictive pericarditis after pericardiectomy.
68 rmed in 5 patients with surgically confirmed constrictive pericarditis and 12 patients (control subje
69 d echocardiographic data of 50 patients with constrictive pericarditis and 44 with restrictive cardio
70                                              Constrictive pericarditis and chronic obstructive pulmon
71 elocity was 13% +/- 6% and -8% +/- 7% in the constrictive pericarditis and control groups, respective
72 ction after pericardiectomy in patients with constrictive pericarditis and correlated postoperative D
73 city duration are increased in patients with constrictive pericarditis and may be helpful in diagnosi
74 ides a clinically useful distinction between constrictive pericarditis and restrictive cardiomyopathy
75 y data sets derived from patients with known constrictive pericarditis and restrictive cardiomyopathy
76 elocity is the main diagnostic criterion for constrictive pericarditis by Doppler echocardiography, i
77 mited cutaneous scleroderma in whom calcific constrictive pericarditis developed.
78 icacy of pericardiectomy, some patients with constrictive pericarditis fail to improve postoperativel
79 measuring PVF have included: differentiating constrictive pericarditis from restriction, estimation o
80 rly diastolic velocity to help differentiate constrictive pericarditis from restrictive cardiomyopath
81 ular expansion velocities in differentiating constrictive pericarditis from restrictive cardiomyopath
82 classifier was evaluated for differentiating constrictive pericarditis from restrictive cardiomyopath
83   Echocardiography allows differentiation of constrictive pericarditis from restrictive myocardial di
84 e chronic obstructive pulmonary disease from constrictive pericarditis in patients with a respiratory
85 tion on a plain radiograph strongly suggests constrictive pericarditis in patients with heart failure
86                                              Constrictive pericarditis is a potentially reversible ca
87                                              Constrictive pericarditis is a rare, but increasingly re
88                            However, calcific constrictive pericarditis is considered rare in the Unit
89                    A physiologic hallmark of constrictive pericarditis is enhanced ventricular interd
90                                              Constrictive pericarditis is the result of a spectrum of
91  may occur after pericardiectomy surgery for constrictive pericarditis patients; however, its mechani
92                                              Constrictive pericarditis represents a serious hemodynam
93                                 Diagnosis of constrictive pericarditis should be considered in patien
94  inflammation and may identify patients with constrictive pericarditis that will improve with anti-in
95                             In patients with constrictive pericarditis treated with anti-inflammatory
96 om 1985 to 1995, a total of 58 patients with constrictive pericarditis underwent pericardiectomy and
97                                              Constrictive pericarditis was confirmed by the surgical
98 ease or severe tricuspid regurgitation after constrictive pericarditis was considered but ruled out.
99 m onset to peak inspiration in patients with constrictive pericarditis were significantly different f
100     In 12 patients with surgically confirmed constrictive pericarditis who had < 25% respiratory vari
101   We identified 41 consecutive patients with constrictive pericarditis who had a cardiovascular magne
102 measured at 26 locations in 11 patients with constrictive pericarditis who underwent intraoperative t
103 tive patients undergoing pericardiectomy for constrictive pericarditis without tricuspid valve surger
104 dinal axis expansion (Ea) in 8 patients with constrictive pericarditis, 7 patients with restriction a
105 ed in 18% of patients with surgically proven constrictive pericarditis, although the histopathologica
106 des an important opportunity to evaluate for constrictive pericarditis, and definite diagnostic crite
107 those with neoplastic infiltration, effusive-constrictive pericarditis, or loculated effusions, fluor
108 omy is indicated for chronic or irreversible constrictive pericarditis, refractory recurrent pericard
109 or absent during the evaluation of suspected constrictive pericarditis, repeat Doppler recording of m
110 because of cardiovascular complications (one constrictive pericarditis, two right heart failures with
111 is may be associated with increased risk for constrictive pericarditis.
112 ication is a common finding in patients with constrictive pericarditis.
113 formed promptly in symptomatic patients with constrictive pericarditis.
114 ose of 20 patients who had surgically proved constrictive pericarditis.
115 velocity, which is not seen in patients with constrictive pericarditis.
116  are less restrictive compared with those in constrictive pericarditis.
117 nd should be a valuable adjunct in assessing constrictive pericarditis.
118 eristic Doppler echocardiographic feature in constrictive pericarditis.
119  respiration in relation to the diagnosis of constrictive pericarditis.
120 ion could assist in noninvasively diagnosing constrictive pericarditis.
121 onsidered to prevent recurrence and effusive-constrictive pericarditis.
122 th corticosteroids considered for associated constrictive pericarditis.
123 e and effective treatment for posttransplant constrictive pericarditis.
124 c tamponade requiring pericardiocentesis, or constrictive pericarditis.
125 dependently associated with the diagnosis of constrictive pericarditis.
126 c tamponade requiring pericardiocentesis, or constrictive pericarditis.
127 iae that progressed to cardiac tamponade and constrictive pericarditis.
128 ent treatments for pericardial effusions and constrictive pericarditis.
129 = 4.6-28.6 mo), 10 patients (0.8%) developed constrictive pericarditis.
130 amponade, but also to suggest a diagnosis of constrictive pericarditis.
131 imilar to that observed in clinical cases of constrictive pericarditis.
132        It represents a novel animal model of constrictive pericarditis.
133 onsidered an essential diagnostic feature of constrictive pericarditis.
134 estrictive cardiomyopathy and preserved with constrictive pericarditis.
135  phenomenon that has been labeled "transient constrictive pericarditis." No large studies have examin
136 variables were independently associated with constrictive pericarditis: (1) ventricular septal shift,
137 as composed of a combination of dilatory and constrictive phases.
138 ardial inflammation and fibrosis, leading to constrictive phenotype during the acute phase of disease
139  The KO mice exhibited a classic restrictive/constrictive phenotype with decreased cardiac output, in
140 e course of their illness, resolution of the constrictive physiologic features occurred at an average
141 ome patients with acute CP, the symptoms and constrictive physiologic features resolve with medical t
142 had become normal, and 1 patient who had had constrictive physiology had restrictive findings.
143        Right heart catheterization confirmed constrictive physiology in all cases.
144 ients with restrictive physiology and 5 with constrictive physiology the results had become normal, a
145 eg irons, or stocks were used to put tightly constrictive pressure around the extremities of POWs as
146 eparation in a 135-min cell cycle so the two constrictive processes are separated in both time and sp
147 ages of the cell division site show separate constrictive processes closing first the inner membrane
148 structure allows pore water to penetrate the constrictive region and to form a continuous water wire
149 ion by the cell types most likely to mediate constrictive remodeling after angioplasty.
150             Expression of TGF-beta3 inhibits constrictive remodeling after PTCA and reduces luminal l
151  a potential target for therapies to prevent constrictive remodeling and restenosis.
152 dense collagenous adventitia, which prevents constrictive remodeling by acting as an external scaffol
153 hese changes resulted in a trend toward more constrictive remodeling in low- compared with high-WSS s
154 c arteries contributes to intimal growth and constrictive remodeling leading to lumen loss.
155                                              Constrictive remodeling of the arterial wall was observe
156 ter plaque and necrotic core progression and constrictive remodeling, and high-WSS segments develop g
157 ement is vessel expansion and attenuation of constrictive remodeling.
158 t delay re-endothelialization and also cause constrictive remodeling.
159 ul in limiting intimal growth and preventing constrictive remodeling.
160 educes luminal loss after PTCA by inhibiting constrictive remodeling.
161 wall after angioplasty, where it may inhibit constrictive remodeling.
162 eceptor antagonist, will decrease the airway constrictive response and acute bronchial obstruction to
163 n littermates were used to examine the acute constrictive response of the developing retinal vessels
164    In contrast, beta2AR-OE mice had enhanced constrictive responses.
165        Thus, betaAR antithetically regulates constrictive signals, affecting bronchomotor tone/reacti
166                       Three major processes, constrictive vessel remodeling, intimal hyperplasia (IH)

 
Page Top