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1 is may be associated with increased risk for constrictive pericarditis.
2 c tamponade requiring pericardiocentesis, or constrictive pericarditis.
3 dependently associated with the diagnosis of constrictive pericarditis.
4 c tamponade requiring pericardiocentesis, or constrictive pericarditis.
5 e and effective treatment for posttransplant constrictive pericarditis.
6 iae that progressed to cardiac tamponade and constrictive pericarditis.
7 ent treatments for pericardial effusions and constrictive pericarditis.
8 amponade, but also to suggest a diagnosis of constrictive pericarditis.
9 imilar to that observed in clinical cases of constrictive pericarditis.
10 It represents a novel animal model of constrictive pericarditis.
11 onsidered an essential diagnostic feature of constrictive pericarditis.
12 estrictive cardiomyopathy and preserved with constrictive pericarditis.
13 ication is a common finding in patients with constrictive pericarditis.
14 = 4.6-28.6 mo), 10 patients (0.8%) developed constrictive pericarditis.
15 formed promptly in symptomatic patients with constrictive pericarditis.
16 ose of 20 patients who had surgically proved constrictive pericarditis.
17 velocity, which is not seen in patients with constrictive pericarditis.
18 are less restrictive compared with those in constrictive pericarditis.
19 nd should be a valuable adjunct in assessing constrictive pericarditis.
20 eristic Doppler echocardiographic feature in constrictive pericarditis.
21 respiration in relation to the diagnosis of constrictive pericarditis.
22 ion could assist in noninvasively diagnosing constrictive pericarditis.
23 onsidered to prevent recurrence and effusive-constrictive pericarditis.
24 th corticosteroids considered for associated constrictive pericarditis.
25 variables were independently associated with constrictive pericarditis: (1) ventricular septal shift,
26 ve pulmonary disease without an overlap with constrictive pericarditis (39.5+/-18.8 cm/s vs. 4.2+/-3.
27 h significant reductions in the incidence of constrictive pericarditis (4.4% vs. 7.8%; hazard ratio,
28 dinal axis expansion (Ea) in 8 patients with constrictive pericarditis, 7 patients with restriction a
29 We present our institutional experience with constrictive pericarditis after lung transplant in an ef
30 stics and identify variables associated with constrictive pericarditis after lung transplantation.
32 ed in 18% of patients with surgically proven constrictive pericarditis, although the histopathologica
33 rmed in 5 patients with surgically confirmed constrictive pericarditis and 12 patients (control subje
34 d echocardiographic data of 50 patients with constrictive pericarditis and 44 with restrictive cardio
36 elocity was 13% +/- 6% and -8% +/- 7% in the constrictive pericarditis and control groups, respective
37 ction after pericardiectomy in patients with constrictive pericarditis and correlated postoperative D
38 city duration are increased in patients with constrictive pericarditis and may be helpful in diagnosi
39 y data sets derived from patients with known constrictive pericarditis and restrictive cardiomyopathy
40 ides a clinically useful distinction between constrictive pericarditis and restrictive cardiomyopathy
41 des an important opportunity to evaluate for constrictive pericarditis, and definite diagnostic crite
42 elocity is the main diagnostic criterion for constrictive pericarditis by Doppler echocardiography, i
43 ted to be useful for differentiating chronic constrictive pericarditis (CP) and restrictive cardiomyo
44 and differentiate between rare diseases like constrictive pericarditis (CP) and restrictive cardiomyo
45 venous flow (PV) velocities in patients with constrictive pericarditis (CP) and to describe the influ
46 peptide (BNP) measurements to differentiate constrictive pericarditis (CP) from restrictive cardiomy
50 between restrictive cardiomyopathy (RCM) and constrictive pericarditis (CP) is challenging and, despi
51 atheterization criteria for the diagnosis of constrictive pericarditis (CP) rely on equalization of i
52 study was to compare myocardial mechanics of constrictive pericarditis (CP) with restrictive cardiomy
53 and specific criterion for the diagnosis of constrictive pericarditis (CP), but simultaneous ventric
55 to determine the association of etiology of constrictive pericarditis (CP), pericardial calcificatio
59 icacy of pericardiectomy, some patients with constrictive pericarditis fail to improve postoperativel
60 measuring PVF have included: differentiating constrictive pericarditis from restriction, estimation o
61 rly diastolic velocity to help differentiate constrictive pericarditis from restrictive cardiomyopath
62 classifier was evaluated for differentiating constrictive pericarditis from restrictive cardiomyopath
63 ular expansion velocities in differentiating constrictive pericarditis from restrictive cardiomyopath
64 Echocardiography allows differentiation of constrictive pericarditis from restrictive myocardial di
65 e chronic obstructive pulmonary disease from constrictive pericarditis in patients with a respiratory
66 tion on a plain radiograph strongly suggests constrictive pericarditis in patients with heart failure
72 In acute pericarditis, the development of constrictive pericarditis (<0.5%) and pericardial tampon
75 phenomenon that has been labeled "transient constrictive pericarditis." No large studies have examin
76 those with neoplastic infiltration, effusive-constrictive pericarditis, or loculated effusions, fluor
77 may occur after pericardiectomy surgery for constrictive pericarditis patients; however, its mechani
78 omy is indicated for chronic or irreversible constrictive pericarditis, refractory recurrent pericard
79 or absent during the evaluation of suspected constrictive pericarditis, repeat Doppler recording of m
82 inflammation and may identify patients with constrictive pericarditis that will improve with anti-in
84 because of cardiovascular complications (one constrictive pericarditis, two right heart failures with
85 om 1985 to 1995, a total of 58 patients with constrictive pericarditis underwent pericardiectomy and
87 ease or severe tricuspid regurgitation after constrictive pericarditis was considered but ruled out.
88 m onset to peak inspiration in patients with constrictive pericarditis were significantly different f
89 In 12 patients with surgically confirmed constrictive pericarditis who had < 25% respiratory vari
90 We identified 41 consecutive patients with constrictive pericarditis who had a cardiovascular magne
91 measured at 26 locations in 11 patients with constrictive pericarditis who underwent intraoperative t
92 tive patients undergoing pericardiectomy for constrictive pericarditis without tricuspid valve surger