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1 jacent to the sarcomere in support of muscle contractile performance.
2 y relative to its demands can impair cardiac contractile performance.
3 gy metabolism, neurotransmitter release, and contractile performance.
4 ts in relaxation (diastole) while preserving contractile performance.
5  does not impair the normal left ventricular contractile performance.
6 s harbored a spontaneous capacity to improve contractile performance.
7 siological approach to study skeletal muscle contractile performance.
8 y thin filament length regulation and muscle contractile performance.
9 myocardial apoptosis and resultant decreased contractile performance.
10 hosphate content and failure to sustain high contractile performance.
11 tinct pathways of ventricular remodeling and contractile performance.
12 increase blood pressure and left ventricular contractile performance.
13 f pathologic hypertrophy and improvements in contractile performance.
14 als exhibited hypertrophy without diminished contractile performance.
15 odeling, and (3) enhance in vivo and ex vivo contractile performance.
16 eurohormonal system activity, and myocardial contractile performance.
17 es and produced a relative improvement in LV contractile performance.
18  vasodilation and improves LV relaxation and contractile performance.
19 ing as dominant negative alleles that impair contractile performance.
20 showed a 73% decrease in baseline isovolumic contractile performance.
21  electrical pacing rate increased isovolumic contractile performance.
22 hemically injured heart enhances ventricular contractile performance.
23 the effect of resveratrol on skeletal muscle contractile performance, a primary marker of skeletal mu
24 s (STG-MI), however, showed preserved global contractile performance, abrogated apoptosis, and preven
25                          In order to compare contractile performance across experimental platforms, w
26                        Left ventricular (LV) contractile performance after MI is an important predict
27 in missense mutations, like TTNtvs, diminish contractile performance and are pathogenic.
28                               In this study, contractile performance and calcium transients were meas
29 , endothelin enhanced both adult rat myocyte contractile performance and cardiac troponin-I phosphory
30 orbide dinitrate restores Ca(2+) cycling and contractile performance and controls superoxide producti
31 re CHF, allopurinol produced no change in LV contractile performance and did not alter the response t
32 ical hypertrophy by contributing to enhanced contractile performance and efficiency.
33 y examined left ventricular (LV) and myocyte contractile performance and electrophysiologic variables
34                                              Contractile performance and energetics were measured usi
35 e depletion of cardiomyocyte iron, impairing contractile performance and explaining why a transferrin
36  frank DCM, defined as LV dilation, impaired contractile performance and LVEDD > or = 112% plus FS <
37  LV pump function, enhanced isolated myocyte contractile performance and normalized myocyte action po
38 rat hearts exhibiting physiologic values for contractile performance and oxygen consumption.
39  may potentially account for the compromised contractile performance and reduced functional reserve i
40       This activity translates into improved contractile performance and survival in preclinical hear
41 reater benefits than after waking for muscle contractile performance and systemic glucose tolerance.
42 erations in cTnC-cTnT binding may compromise contractile performance and trigger pathological remodel
43  skeletal muscle weight, fiber size, in vivo contractile performance, and expression of mRNAs and pro
44  pMultistem improved BZ energetics, regional contractile performance, and global left ventricular eje
45 arction increased survival, enhanced cardiac contractile performance, and halted ventricular remodeli
46  reduced cardiomyocyte hypertrophy, improved contractile performance, and prevented early lethality.
47 e defects in both whole heart energetics and contractile performance, and that the magnitude of these
48  the ATPase would be consistent with reduced contractile performance, and the results suggest reduced
49 ndent (type 1) diabetes results in depressed contractile performance associated with diminished sarco
50  spectroscopy while simultaneously measuring contractile performance at four work states.
51 al adaptations to muscle disuse, we compared contractile performance at the molecular, cellular and w
52     This new method allows quantification of contractile performance at the single-cell level, which
53 o determine if heart failure further impairs contractile performance by altering myofibrillar protein
54 ies indicating that low levels of NO augment contractile performance by inhibition of a cGMP-dependen
55                                              Contractile performance, carbohydrate oxidation, and oxy
56  transgenic mice showed improved recovery of contractile performance during reperfusion after ischemi
57 nsic mechanical and neural factors in muscle contractile performance during running, which has been l
58 TNF-alpha induced a 7% to 40% increase in LV contractile performance during the 60-minute infusion.
59 ATP synthesis and ATP consumption, improving contractile performance following ischemia-reperfusion i
60 hanisms underlying the accompanying improved contractile performance have been largely ignored.
61 y, segmental ejection fraction (a measure of contractile performance) improved in injected scar segme
62 e favorably influences left ventricular (LV) contractile performance in heart failure remains uncerta
63 haracteristic, contributing to the decreased contractile performance in human and experimental failin
64 unction, ocular motility in intact mice, and contractile performance in isolated muscle preparations
65                       We measured isovolumic contractile performance in isolated perfused hearts from
66 entifies TnI as a potential target to modify contractile performance in normal and diseased myocardiu
67 ety of ways to adjust flight muscle size and contractile performance in order to meet demands imposed
68  normal basal cardiac function but depressed contractile performance in postinfarct and pressure over
69 gical functions including Ca(2+) cycling and contractile performance in skeletal and cardiac myocytes
70  (1) the thin filament is a key modulator of contractile performance in the failing human heart, (2)
71 (101) myofibrillar degeneration, to evaluate contractile performance in the mutant cardiomyocytes, an
72      The thin filament is a key modulator of contractile performance in the transition to failure in
73 s been shown to lead to increased myocardial contractile performance in vitro and in vivo.
74                                           LV contractile performance measured by the slopes of pressu
75  likely contributes to the improved cellular contractile performance observed after sustained LVAD su
76 hat RES attenuates HFD-induced reductions in contractile performance of EDL, but this response is not
77 lead to adverse effects on cell viability or contractile performance of EHMs (n=3, P=0.83, P=0.87).
78 n beta-MyHC dominant hiPSC-CMs would enhance contractile performance of hiPSC-CMs.
79  effects of high-fat diet consumption on the contractile performance of isolated fast twitch muscle a
80        Similar results were obtained for the contractile performance of myocytes isolated from a sepa
81 t that cell shape is critical in determining contractile performance of single cardiomyocytes by regu
82                                          The contractile performance of the heart is linked to the en
83 n early response of myofilaments to increase contractile performance of the heart.
84 n early response of myofilaments to increase contractile performance of the heart.
85 onstitution resulted in a marked recovery of contractile performance of the infarcted heart.
86 ac energetics using 31P NMR spectroscopy and contractile performance of the intact beating heart reve
87 s saline treatment completely diminished the contractile performance of the ischemic/reperfused myoca
88 ordinately maintain the hyperdynamic cardiac contractile performance of the PLN-KO mouse in the long
89 al oxidative capacity, apparently to support contractile performance of the TA.
90 sgenic hearts exhibited improved recovery of contractile performance over the reperfusion period.
91     TG hearts exhibited improved recovery of contractile performance over the whole reperfusion perio
92 e (CHF, n = 4) as determined by clinical and contractile performance parameters.
93 is often abrogated by progressively abnormal contractile performance per unit mass of myocardium, eve
94 BS2, a selective inhibitor of NOS2, improves contractile performance, preserves myocardial ATP, and r
95 itions, SERCA2a overexpressors show improved contractile performance relative to wild-type (WT) mice
96 ction of NO on myocardial O2 consumption and contractile performance remain unclear.
97 ated cardiac troponin-I in the adult myocyte contractile performance response to activated protein ki
98  myocytes expressing cTnI A164H maintained a contractile performance similar to that observed for the
99 At the level of isolated cardiac trabeculae, contractile performance, specifically of contractile kin
100 wed more myocardial damage and showed poorer contractile performance than hearts reperfused with high
101 ex plays a greater role in modulating muscle contractile performance than was recognized previously.
102 in both skeletal muscle strength and cardiac contractile performance that is not achievable with conv
103 tile work and a marked inability to increase contractile performance upon acute inotropic challenge i
104 oblasts, iPS treatment restored postischemic contractile performance, ventricular wall thickness, and
105                                        Local contractile performance was decreased in infarcted myoca
106 and time constant tau were much greater, and contractile performance was depressed.
107                                       Muscle contractile performance was determined in skinned muscle
108                        Left ventricular (LV) contractile performance was measured by the slopes of th
109 asal and isoproterenol-stimulated isovolumic contractile performance was preserved.
110 reperfusion, however, cardiac relaxation and contractile performance were greatly impaired in G6PD(de
111 responded promptly to the abrupt increase in contractile performance with epinephrine, during the lag
112 e in vivo translation of this improvement in contractile performance with PCO pretreatment was confou

 
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