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1 r carcinoma in situ (LCIS) were followed for contralateral breast cancer.
2 is associated with risk of breast cancer and contralateral breast cancer.
3 and cumulative risks of breast, ovarian, and contralateral breast cancer.
4 rrence of invasive locoregional, distant, or contralateral breast cancer.
5 ugh few had a clinically significant risk of contralateral breast cancer.
6  lower risks of breast cancer recurrence and contralateral breast cancer.
7 free survival, overall survival, and time to contralateral breast cancer.
8 -fold (95% CI, 1.9 to 26.5) elevated risk of contralateral breast cancer.
9 ld (95% CI, 1.03-19.0) increased risk of ER- contralateral breast cancer.
10 y higher risk of developing a second primary contralateral breast cancer.
11 d the absolute occurrence of ipsilateral and contralateral breast cancer.
12 e a substantially reduced risk of developing contralateral breast cancer.
13 ifen might play in the reduction of risk for contralateral breast cancer.
14 ed as potential risk factors for synchronous contralateral breast cancer.
15                      Eight women developed a contralateral breast cancer.
16 .22-0.63; p<0.0001), but having no effect on contralateral breast cancer (0.84, 0.45-1.58; p=0.6).
17  for disease recurrence or the occurrence of contralateral breast cancer, 0.66; P=0.01 by a two-sided
18 cancer specific mortality (0.72, 0.45-1.16), contralateral breast cancer (1.18, 0.64-2.16), or depres
19              Among the 704 participants with contralateral breast cancer, 108 (15.3%) were identified
20 ed 5 years reduces the risk of recurrence or contralateral breast cancer 15 years after starting trea
21 ion, and 5.6% (5.5% to 5.6%) had developed a contralateral breast cancer, 3.1% (3.0% to 3.2%) more th
22 es (324 vs 375, 0.86, 0.74-0.99, p=0.04) and contralateral breast cancers (35 vs 59, 42% reduction, 1
23 tases (89% v 92%, respectively; P = .16), or contralateral breast cancer (6% v 6%, respectively; P =
24 ving disease recurrence or the occurrence of contralateral breast cancer (67 with letrozole and 98 wi
25 ated that desires to decrease their risk for contralateral breast cancer (98%) and improve survival (
26                                              Contralateral breast cancer accounts for around 60% of t
27 vivors had an increased risk of metachronous contralateral breast cancer (adjusted hazard ratio [HR],
28        Information is limited on the risk of contralateral breast cancer after a diagnosis of breast
29  the hazard ratio (HR) for the occurrence of contralateral breast cancer after CPM was 0.03 (95% CI,
30 d smoking on risk of second primary invasive contralateral breast cancer among breast cancer survivor
31 47 years; IQR, 40-55 years) eligible for the contralateral breast cancer analysis, 426 were diagnosed
32 ve invasive breast cancer and second primary contralateral breast cancer and 728 matched control wome
33 ated breast cancer are at increased risk for contralateral breast cancer and ovarian cancer and there
34                 Probabilities for developing contralateral breast cancer and ovarian cancer, dying fr
35            Aromatase inhibitors prevent more contralateral breast cancers and cause fewer side effect
36 y with increased uterine cancer (but reduced contralateral breast cancer), and chemotherapy with incr
37  seems to protect against the development of contralateral breast cancer, and although women who unde
38 mic heart disease, cerebrovascular diseases, contralateral breast cancer, and depression.
39  WBI and 10 PBI; P = .28), 20 patients had a contralateral breast cancer, and eight patients had dist
40 ated PRS have an elevated risk of developing contralateral breast cancer, and that the PRS can consid
41                      The risks of subsequent contralateral breast cancer are substantial for women wh
42   This study assessed the risk of subsequent contralateral breast cancer associated with carrying a B
43 ns were significantly more likely to develop contralateral breast cancer at 5 years (31% v 4%, P=.000
44 rolled in the trial who did not have a known contralateral breast cancer at the time of surgical plan
45          Given these patients' high risk for contralateral breast cancer, bilateral mastectomy is inc
46 ctic mastectomy [CPM]), although the risk of contralateral breast cancer (CBC) has decreased in recen
47 reast cancer survivors at risk of developing contralateral breast cancer (CBC) is increasing.
48                                              Contralateral breast cancer (CBC) is the most frequent n
49 enes other than BRCA1, BRCA2, and CHEK2 with contralateral breast cancer (CBC) risk and breast cancer
50 ersus routine surveillance as an alternative contralateral breast cancer (CBC) risk management strate
51  PRS of 313 germline variants (PRS(313)) and contralateral breast cancer (CBC) risk.
52       Rates of IBTR, distant recurrence, and contralateral breast cancer (CBC) were among the end poi
53 idences of locoregional recurrence (LRR) and contralateral breast cancer (CBC) were assessed with com
54 er diagnosis, roughly 5% of patients develop contralateral breast cancer (CBC).
55 tify factors predictive of HRL and/or occult contralateral breast cancer (CBC).
56 fy genetic and environmental determinants of contralateral breast cancer (CBC).
57 er diagnosis, roughly 5% of patients develop contralateral breast cancer (CBC).
58 netic resonance imaging (MRI) detects occult contralateral breast cancers (CBCs) in women with breast
59 tes of in-breast tumor recurrence (IBTR) and contralateral breast cancers (CBCs).
60  lower risks of breast cancer recurrence and contralateral breast cancer compared with placebo.
61 ation Epidemiology [WECARE]) of asynchronous contralateral breast cancer conducted during the period
62 ancer and are judged to be at high risk of a contralateral breast cancer, contralateral risk-reducing
63                                              Contralateral breast cancer developed in 0.5% of women w
64                                              Contralateral breast cancer developed in 2.7% of women w
65 his nested case-control study, patients with contralateral breast cancer diagnosed 1 year or more aft
66 ancer, 109 with ovarian cancer, and 245 with contralateral breast cancer during follow-up.
67 rature review of the risk of ipsilateral and contralateral breast cancer events among breast cancer s
68 w established a low risk for ipsilateral and contralateral breast cancer events in most older breast
69 y had above-average risks of ipsilateral and contralateral breast cancer events, most did not have an
70  and 10-year cumulative) risks of developing contralateral breast cancer following a first invasive b
71                         The relative risk of contralateral breast cancer for BRCA1 mutation carriers
72 e age-specific risks of breast, ovarian, and contralateral breast cancer for mutation carriers and to
73                            Letrozole reduced contralateral breast cancer frequency in the first 10 ye
74 essed the long-term risks of ipsilateral and contralateral breast cancer in a cohort of young women w
75 tion is available on the subsequent risks of contralateral breast cancer in mutation carriers.
76                 The annual incidence rate of contralateral breast cancer in the letrozole group was 0
77 stic accuracy for additional ipsilateral and contralateral breast cancer in women with nondense breas
78 mary endpoints were rates of ipsilateral and contralateral breast cancer, in relation to germline BRC
79 al prophylactic mastectomy (CPM) in reducing contralateral breast cancer incidence and breast cancer
80 silateral breast tumour recurrence rates and contralateral breast cancer incidence are scarce, but to
81                                    Regarding contralateral breast cancer incidence, there were six ev
82  (local, regional, or distant recurrence, or contralateral breast cancer, invasive disease, or ductal
83                             The true risk of contralateral breast cancer is not associated with the d
84                 In particular, the risk of a contralateral breast cancer is reviewed to help guide th
85 sk of the relatively uncommon outcome of ER- contralateral breast cancer may now need to be tallied a
86 isk of hormone receptor-specific subtypes of contralateral breast cancer (n = 303 ER+ and n = 52 ER-
87 ntinued until the first event of recurrence, contralateral breast cancer, new primary malignant neopl
88 oking were all positively related to risk of contralateral breast cancer (odds ratio [OR], 1.4; 95% C
89 en for >or=5 years had a reduced risk of ER+ contralateral breast cancer [odds ratio, 0.4; 95% confid
90 oophorectomy was not associated with risk of contralateral breast cancer or IBTR.
91 ory of HRT did not have an increased risk of contralateral breast cancer or second non-breast cancer
92       Participants previously diagnosed with contralateral breast cancer or unilateral breast cancer
93  in regional recurrence, distant metastases, contralateral breast cancers, or new breast cancers were
94  ipsilateral breast tumor recurrence (IBTR), contralateral breast cancer, ovarian cancer, and ovarian
95 time to distant recurrence, incidence of new contralateral breast cancer, overall survival, and death
96  regimens, a significant excess incidence of contralateral breast cancer (rate ratio 1.18, SE 0.06, 2
97                                              Contralateral breast cancer rates are declining as well
98 95% CI, 2.0- to 5.8-fold) increased risks of contralateral breast cancer, respectively.
99                                              Contralateral breast cancer risk decreased significantly
100 sed on promising data involving reduction of contralateral breast cancer risk in adjuvant studies, se
101 women with low-penetrance mutations (assumed contralateral breast cancer risk of 24% and ovarian canc
102 hose with high-penetrance mutations (assumed contralateral breast cancer risk of 65% and ovarian canc
103 , smoking, and alcohol consumption influence contralateral breast cancer risk, affording breast cance
104 een these three exposures and second primary contralateral breast cancer risk.
105 use for <5 years was not associated with ER- contralateral breast cancer risk.
106 ts, the current evidence for ipsilateral and contralateral breast cancer risks in older survivors of
107  Secondary end points were overall survival, contralateral breast cancer, second primary cancer, and
108                             The incidence of contralateral breast cancer seems to be reduced signific
109 silateral recurrences, nine (29.0%) were new contralateral breast cancers, six (19.4%) were distant r
110 ast examination and mammography in detecting contralateral breast cancer soon after the initial diagn
111 sease-free survival and a lower incidence of contralateral breast cancer than those with placebo, but
112                                          For contralateral breast cancer, the cumulative risk 20 year
113   Efficacy of letrozole versus tamoxifen for contralateral breast cancer varied significantly over ti
114  a trend toward higher risk for relatives of contralateral breast cancer vs unilateral breast cancer
115 the 20-year radiation-related excess risk of contralateral breast cancer was estimated to increase fr
116                  The absolute excess risk of contralateral breast cancer was greater in younger than
117 r who underwent breast MR imaging at which a contralateral breast cancer was not identified, patient
118 arly dependent on TN status, but the risk of contralateral breast cancer was not.
119                                  The risk of contralateral breast cancer was significantly reduced (h
120 e corresponding risks of any recurrence or a contralateral breast cancer were 17%, 22%, and 26%, resp
121       For the 357 postmenopausal women, 50.3 contralateral breast cancers were predicted, whereas onl
122 ers more accurately perceived their risk for contralateral breast cancer, whereas women without a kno
123                              Life-tables for contralateral breast cancers, which consider age at firs
124 even patients were diagnosed with subsequent contralateral breast cancer, yielding 5- and 10-year act

 
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