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1 r carcinoma in situ (LCIS) were followed for contralateral breast cancer.
2 is associated with risk of breast cancer and contralateral breast cancer.
3 and cumulative risks of breast, ovarian, and contralateral breast cancer.
4 rrence of invasive locoregional, distant, or contralateral breast cancer.
5 ugh few had a clinically significant risk of contralateral breast cancer.
6 lower risks of breast cancer recurrence and contralateral breast cancer.
7 free survival, overall survival, and time to contralateral breast cancer.
8 -fold (95% CI, 1.9 to 26.5) elevated risk of contralateral breast cancer.
9 ld (95% CI, 1.03-19.0) increased risk of ER- contralateral breast cancer.
10 y higher risk of developing a second primary contralateral breast cancer.
11 d the absolute occurrence of ipsilateral and contralateral breast cancer.
12 e a substantially reduced risk of developing contralateral breast cancer.
13 ifen might play in the reduction of risk for contralateral breast cancer.
14 ed as potential risk factors for synchronous contralateral breast cancer.
15 Eight women developed a contralateral breast cancer.
16 .22-0.63; p<0.0001), but having no effect on contralateral breast cancer (0.84, 0.45-1.58; p=0.6).
17 for disease recurrence or the occurrence of contralateral breast cancer, 0.66; P=0.01 by a two-sided
18 cancer specific mortality (0.72, 0.45-1.16), contralateral breast cancer (1.18, 0.64-2.16), or depres
20 ed 5 years reduces the risk of recurrence or contralateral breast cancer 15 years after starting trea
21 ion, and 5.6% (5.5% to 5.6%) had developed a contralateral breast cancer, 3.1% (3.0% to 3.2%) more th
22 es (324 vs 375, 0.86, 0.74-0.99, p=0.04) and contralateral breast cancers (35 vs 59, 42% reduction, 1
23 tases (89% v 92%, respectively; P = .16), or contralateral breast cancer (6% v 6%, respectively; P =
24 ving disease recurrence or the occurrence of contralateral breast cancer (67 with letrozole and 98 wi
25 ated that desires to decrease their risk for contralateral breast cancer (98%) and improve survival (
27 vivors had an increased risk of metachronous contralateral breast cancer (adjusted hazard ratio [HR],
29 the hazard ratio (HR) for the occurrence of contralateral breast cancer after CPM was 0.03 (95% CI,
30 d smoking on risk of second primary invasive contralateral breast cancer among breast cancer survivor
31 47 years; IQR, 40-55 years) eligible for the contralateral breast cancer analysis, 426 were diagnosed
32 ve invasive breast cancer and second primary contralateral breast cancer and 728 matched control wome
33 ated breast cancer are at increased risk for contralateral breast cancer and ovarian cancer and there
36 y with increased uterine cancer (but reduced contralateral breast cancer), and chemotherapy with incr
37 seems to protect against the development of contralateral breast cancer, and although women who unde
39 WBI and 10 PBI; P = .28), 20 patients had a contralateral breast cancer, and eight patients had dist
40 ated PRS have an elevated risk of developing contralateral breast cancer, and that the PRS can consid
42 This study assessed the risk of subsequent contralateral breast cancer associated with carrying a B
43 ns were significantly more likely to develop contralateral breast cancer at 5 years (31% v 4%, P=.000
44 rolled in the trial who did not have a known contralateral breast cancer at the time of surgical plan
46 ctic mastectomy [CPM]), although the risk of contralateral breast cancer (CBC) has decreased in recen
49 enes other than BRCA1, BRCA2, and CHEK2 with contralateral breast cancer (CBC) risk and breast cancer
50 ersus routine surveillance as an alternative contralateral breast cancer (CBC) risk management strate
53 idences of locoregional recurrence (LRR) and contralateral breast cancer (CBC) were assessed with com
58 netic resonance imaging (MRI) detects occult contralateral breast cancers (CBCs) in women with breast
61 ation Epidemiology [WECARE]) of asynchronous contralateral breast cancer conducted during the period
62 ancer and are judged to be at high risk of a contralateral breast cancer, contralateral risk-reducing
65 his nested case-control study, patients with contralateral breast cancer diagnosed 1 year or more aft
67 rature review of the risk of ipsilateral and contralateral breast cancer events among breast cancer s
68 w established a low risk for ipsilateral and contralateral breast cancer events in most older breast
69 y had above-average risks of ipsilateral and contralateral breast cancer events, most did not have an
70 and 10-year cumulative) risks of developing contralateral breast cancer following a first invasive b
72 e age-specific risks of breast, ovarian, and contralateral breast cancer for mutation carriers and to
74 essed the long-term risks of ipsilateral and contralateral breast cancer in a cohort of young women w
77 stic accuracy for additional ipsilateral and contralateral breast cancer in women with nondense breas
78 mary endpoints were rates of ipsilateral and contralateral breast cancer, in relation to germline BRC
79 al prophylactic mastectomy (CPM) in reducing contralateral breast cancer incidence and breast cancer
80 silateral breast tumour recurrence rates and contralateral breast cancer incidence are scarce, but to
82 (local, regional, or distant recurrence, or contralateral breast cancer, invasive disease, or ductal
85 sk of the relatively uncommon outcome of ER- contralateral breast cancer may now need to be tallied a
86 isk of hormone receptor-specific subtypes of contralateral breast cancer (n = 303 ER+ and n = 52 ER-
87 ntinued until the first event of recurrence, contralateral breast cancer, new primary malignant neopl
88 oking were all positively related to risk of contralateral breast cancer (odds ratio [OR], 1.4; 95% C
89 en for >or=5 years had a reduced risk of ER+ contralateral breast cancer [odds ratio, 0.4; 95% confid
91 ory of HRT did not have an increased risk of contralateral breast cancer or second non-breast cancer
93 in regional recurrence, distant metastases, contralateral breast cancers, or new breast cancers were
94 ipsilateral breast tumor recurrence (IBTR), contralateral breast cancer, ovarian cancer, and ovarian
95 time to distant recurrence, incidence of new contralateral breast cancer, overall survival, and death
96 regimens, a significant excess incidence of contralateral breast cancer (rate ratio 1.18, SE 0.06, 2
100 sed on promising data involving reduction of contralateral breast cancer risk in adjuvant studies, se
101 women with low-penetrance mutations (assumed contralateral breast cancer risk of 24% and ovarian canc
102 hose with high-penetrance mutations (assumed contralateral breast cancer risk of 65% and ovarian canc
103 , smoking, and alcohol consumption influence contralateral breast cancer risk, affording breast cance
106 ts, the current evidence for ipsilateral and contralateral breast cancer risks in older survivors of
107 Secondary end points were overall survival, contralateral breast cancer, second primary cancer, and
109 silateral recurrences, nine (29.0%) were new contralateral breast cancers, six (19.4%) were distant r
110 ast examination and mammography in detecting contralateral breast cancer soon after the initial diagn
111 sease-free survival and a lower incidence of contralateral breast cancer than those with placebo, but
113 Efficacy of letrozole versus tamoxifen for contralateral breast cancer varied significantly over ti
114 a trend toward higher risk for relatives of contralateral breast cancer vs unilateral breast cancer
115 the 20-year radiation-related excess risk of contralateral breast cancer was estimated to increase fr
117 r who underwent breast MR imaging at which a contralateral breast cancer was not identified, patient
120 e corresponding risks of any recurrence or a contralateral breast cancer were 17%, 22%, and 26%, resp
122 ers more accurately perceived their risk for contralateral breast cancer, whereas women without a kno
124 even patients were diagnosed with subsequent contralateral breast cancer, yielding 5- and 10-year act