ライフサイエンスコーパス: control diet

1 er whipping cream (MFGM diet) or butter oil (control diet).

2 tic insulin sensitivity in comparison to the control diet.

3 the DASH and HF-DASH diets compared with the control diet.

4 ne-supplemented diets when compared with the control diet.

5 ation, then weaned onto either obesogenic or control diet.

6 ls, compared to offspring from mothers fed a control diet.

7 les, and LDL peak diameter compared with the control diet.

8 lective cathepsin S inhibitor RO5444101 or a control diet.

9 on lipoprotein risk factors compared with a control diet.

10 om donors on either a high-fat diet (HFD) or control diet.

11 nd Cox7a1 knockout mice fed with high fat or control diet.

12 and serum levels 16-fold, compared with the control diet.

13 ed with the Se-enriched diet compared to the control diet.

14 spring; post weaning, offspring were fed the control diet.

15 iet supplemented with nuts compared with the control diet.

16 r content of major volatile compounds to the control diet.

17 ice fed the fish oil diet as compared to the control diet.

18 ostly sucrose) or a calorie-matched-per-gram control diet.

19 nduced inflammatory response faster than the control diet.

20 ls in comparison to F1 mice from dams on the control diet.

21 r concentrations compared with the low-dairy control diet.

22 Ex, in comparison to sedentary animals fed a control diet.

23 t mice were sham- or whey-sensitized and fed control diet.

24 d either a diet enhanced with spirulina or a control diet.

25 those found in individuals who were fed the control diet.

26 riglyceride concentrations compared with the control diet.

27 and Bifidobacterium breve M-16V (GF/Bb) or a control diet.

28 ion after the consumption of a fish oil or a control diet.

29 ubly deficient diet compared to mice fed the control diet.

30 rs, relative to the offspring of males fed a control diet.

31 erance compared with offspring of dams fed a control diet.

32 weeks of age, and all pups were weaned onto control diet.

33 olesterol levels compared with animals fed a control diet.

34 similar to that noted in Abcc6(-/-) mice on control diet.

35 ith 32.4% of calories as ethanol or pair-fed control diet.

36 ining Lieber-DeCarli diet or were pair-fed a control diet.

37 rsible when the rats were switched back to a control diet.

38 gnificantly different from the abrasive-free control diet.

39 78- fold greater total As than birds fed the control diet.

40 + extra-virgin olive oil, MedDiet + nuts, or control diet.

41 aecalibacterium prausnitzii than patients on control diet.

42 g a 12% alcohol liquid diet or an isocaloric control diet.

43 ) before and after 12 wk of a 20% ER diet or control diet.

44 5% calorie restriction diet or an ad libitum control diet.

45 esophageal tissues compared with mice fed a control diet.

46 iterranean diet supplemented with nuts, or a control diet.

47 ethanol for 8 weeks, as a model of ALD, or a control diet.

48 cations, were randomized to either DASH or a control diet.

49 hich is high in fiber and whole grains, or a control diet.

50 nd cholesterol levels in mice fed Western or control diets.

51 syndrome components than did guideline-based control diets.

52 compared with consumption of non-whole-grain control diets.

53 = 28%) of diets including nuts compared with control diets.

54 lso had shorter survival times than mice fed control diets.

55 L/6 mice with colitis, compared with mice on control diets.

56 ithelial progenitors) compared with mice fed control diets.

57 flammatory agent sulindac vs mice on a chow (control) diet.

58 respectively), or restricted calorie (50% of control) diets.

59 ndividuals kept in the laboratory and fed on controlled diets.

60 ener to diabetics and others on carbohydrate-controlled diets.

61 r the modified diet, yet decreased after the control diet (0.02 +/- 0.01 mumol/L and -0.03 +/- 0.02 m

62 in fasting LDL cholesterol observed with the control diet (0.03 +/- 0.06 mmol/L and 0.19 +/- 0.05 mmo

63 Rats were fed for 5weeks with control diet (0.15mug Se/g feed) or Se-enriched diet for

64 ol/L, respectively) (P < 0.05) than with the control diet (0.89 +/- 0.12 and 0.84 +/- 0.11 mmol/L, re

65 ), whereas a decrease was observed after the control diet (-0.51% +/- 0.15%; P< 0.0001).

66 vated in Kcnmb1(-/-) under basal conditions (control diet, 0.6% K) and increased significantly more t

67 embryonic days 12-17 and then returned to a control diet (1.1 g choline chloride/kg).

68 0.05 mmol/L, respectively) compared with the control diet (1.12 +/- 0.11 and 1.19 +/- 0.05 mmol/L, re

69 etal offspring from obese dams reversed to a control diet (1.3-fold, P>0.05).

70 C57BL/6 female mice were fed either a control diet (10% energy from fat) or a high-fat diet (4

71 y rats were fed a HFD (45% fat) or a matched control diet (10% fat) from weaning for 14 weeks.

72 ima Otsuka (L) and OLETF (O) rats consumed a control diet (10% kcal fat, 3.5% sucrose) or a WD (45% k

73 re assigned to a Control diet and BEOs diet (Control diet + 120 mg/kg BEOs), were challenged with C.

74 Male C57BL/6J mice were fed either a control diet (13.5% kcal from fat) or a very high-fat di

75 nergetic diets, each comprising 1) no dairy (control diet), 2) 3 daily servings of 1% fat milk, and 3

76 ere randomized into three dietary groups: 1) control diet, 2) zinc-deficient diet for 3 weeks, and 3)

77 ,t11-CLA, and iTFA, in the context of highly controlled diets (24 d each), on lipoprotein risk factor

78 cheese (5.7 +/- 0.4 g/d) diets than with the control diet (3.9 +/- 0.3 g/d) (P < 0.001).

79 ng the low FODMAP diet (14/27, 52%) than the control diet (4/25, 16%, P=.007).

80 fter the milk diet were lower than after the control diet (-4.1%; P = 0.009).

81 y) were lower than in those who followed the control diet (5.14 +/- 0.18 and 3.06 +/- 0.15 mmol/L, re

82 pe and nSIRT1OE Tg mice were fed with either control diet (6.2% fat) or a HFD (36% fat) for 2 months.

83 hemoglobin increased tumor load in Min mice (control diet: 67 +/- 39 mm(2); 2.5% hemoglobin diet: 114

84 e scores (81.9 +/- 1.2) than patients on the control diet (78.3 +/- 1.2, P = .042).

85 ts increased triglycerides compared with the control diet (+9.9%, P = 0.01 and +10.5%, P = 0.007, res

86 C57BL6/J mice were fed either a control diet, a diet containing choline (1.2%) or a diet

87 ring which all participants received a low-K control diet, a significant racial difference remained (

88 y individuals who consumed in random order a control diet, a standard DASH diet, and a higher-fat, lo

89 neered diets including a standard laboratory control diet, a vitamin D null diet, and a vitamin D enr

90 with walnuts at ~15% energy (30-60 g/d) or a control diet (abstention from walnuts).

91 L1B mice fed the HFD to L2-IL1B mice fed the control diet accelerated tumor development.

92 iterranean diet supplemented with nuts, or a control diet (advice on a low-fat diet).

93 nean diet supplemented with mixed nuts, or a control diet (advice to reduce dietary fat).

94 aFMT in the weight-loss phase compared with control diet aFMT, significantly prevented weight regain

95 is is only partially corrected by a low-fat, control diet after weaning.

96 Seven-weeks old Wistar rats received control diet (AIN-93G), Western diet with and without a

97 Healthy, lean men ingested a eucaloric control diet and a 3-day hypercaloric high-fat diet (inc

98 Chicks were assigned to a Control diet and BEOs diet (Control diet + 120 mg/kg BEO

99 hypercaloric diet for 6 weeks or a eucaloric control diet and measured intrahepatic triglyceride cont

100 e randomized into five groups (n = 8-14): C (control diet and sedentary), F (fed the fructose-rich di

101 WT) mice were fed a high-fat diet (HFD) or a control diet and studied at 6 months of age.

102 re established in two groups (n = 13/group): control diet and Western diet fed for 41 weeks.

103 om mice on wheat-containing, ATI-containing, control diets and transplanted to intestines of mice wit

104 e C57BL6/J mice were fed ethanol or pair-fed control diets and treated or not with HA35.

105 at- or ATI-containing diets or a wheat-free (control) diet and then given dextran sodium sulfate to i

106 were first lowered by a combination of a 3-d controlled diet and prolonged exercise.

107 f C57BL/6J mice, as compared with mice fed a control diet, and that these chromatin changes are assoc

108 libitum HFr developed HS in contrast to the control diet, and the extent of ectopic fat was related

109 ing cells in liver, compared with mice given control diets, as well as higher levels of serum IgA and

110 D-supplemented mothers following reversal to control diet at weaning was interrogated by methylation-

111 participants were instructed to adhere to a controlled diet based on ITF-rich vegetables (providing

112 t (FASD) (5-fold higher than recommended) or control diets before mating and during pregnancy.

113 k, and up to 13.5+/- 4.1g/100mL FW in weight control diet beverage.

114 red with mineralocorticoid-excess mice fed a control diet, both high-fiber diet and acetate supplemen

115 ulate the immune response, compared with the control diet, but had no significant effect on markers o

116 een wild-type (WT) and Ucp1(-/-) mice on the control diet, but MR increased EE by 31% and reduced adi

117 HF-tau in the hippocampus of animals fed the control diet, but not in the irradiated animals fed the

118 n a high-potassium diet than from those on a control diet, but this was not a result of altered expre

119 0) or 20 (T20) added taurine (g/kg), while a control diet (C+) included two-fold higher amount of fis

120 increased feeding in lean rats fed a low-fat control diet (CD) [192 +/- 5 g (ghrelin+CD) vs. 152 +/-

121 Healthy males consumed a control diet (CD) and on two further occasions an isocal

122 A control diet (CD) and Western diet (WD), which contains

123 , canola oil, berries, and fish, whereas the control diet (CD) group consumed low-fiber cereal produc

124 fructose-rich diet mouse (FRD) myocytes vs. control diet (CD) mice, in the absence of significant ch

125 ansgenic counterparts (NT) were fed either a control diet (CD) or a high-fat diet (HFD) for up to 10

126 Female mice were placed on control diet (CD) or folic acid-supplemented diet (FASD)

127 -trained females were randomly assigned to a control diet (CD), TRF, or TRF plus 3 g/d HMB (TRFHMB).

128 type (WT) and miR-155 knockout (KO) mice fed control diet (CD); however, miR-155 KO mice fed high-fat

129 cluding 57 g pistachios/d) and an isocaloric control diet (CD, 55 E% carbohydrates and 30 E% fat) for

130 awley rats were weaned onto HFD (45% fat) or Control Diet (CD; 10% fat).

131 F0 females were fed control diet (CD; 10%kcal from fat) or HFD (60%kcal from

132 he absence of SR Ca(2+) load alterations vs. control-diet (CD) myocytes.

133 Offspring (F(1) ) of mothers on control diet (CF(1) ) and MO diet (MOF(1) ) delivered sp

134 nding order are the powder of inulin, weight control diet, coffee mixed, instant beverage, supplement

135 Female C57BL/6 J mice were fed a control diet (CON) or a high-fat diet (HFD) with or with

136 her a lower-protein (1.2 g . kg(-1) . d(-1)) control diet (CON) or a higher-protein (2.4 g . kg(-1) .

137 is, adult offspring of rat dams either fed a control diet (CON) or one deficient in choline (DEF) dur

138 % carbohydrate) (CHO) as well as a eucaloric control diet (CON).

139 In animal studies, compared with the control diet, consumption of high-Pi diet for 12 weeks d

140 One growth trial used a control diet containing fish meal (FM) as the main prote

141 Another growth trial compared a control diet containing fish oil (FO) as the main lipid

142 whereas their eight male siblings were fed a control diet containing pig fat as the main fat source.

143 Neonatal mice were subjected to a control diet (CTR) or experimental diet with altered rel

144 Mice fed the control diet designed to simulate Western human diet dis

145 showed that SNAP-25b-deficient mice fed with control diet developed hyperglycemia, liver steatosis, a

146 Compared with control diets, diets enriched with nuts did not increase

147 When compared with control diets, diets supplemented with walnuts resulted

148 was to evaluate the contribution of tightly controlled diets differing in carbohydrate and fat conte

149 sensitivity compared with Gipr(-/-) mice on control diet during IU/L.

150 dams were fed either a corn oil-based HF or control diet during pregnancy.

151 to 7.1-fold, P<0.001) compared with that in control diet-exposed animals and is reversible in fetal

152 aternal high-fat diet exposure compared with control diet-exposed animals.

153 -derived suppressor cells; MDSC) compared to control diet fed mice (p < 0.05).

154 strongly upregulated or downregulated versus control-diet fed groups but actually reversed in directi

155 ce fed an HFD (GSH- deficient) compared with control diet-fed group.

156 Saline-injected and control diet-fed leptin receptor-deficient mice were use

157 mutant APP in mice or Abeta42 infusion into control diet-fed mice to mimic obese levels impaired NO

158 In control diet-fed mice, a 9-day CAP exposure was sufficie

159 In control diet-fed mice, exposure to CAP for 30 days decre

160 Splenocytes of control-diet-fed whey-sensitized donors transferred immu

161 Compared with controls, Diet-fed mice had a significant increase in Hc

162 Compared with dams on a control diet, female C57BL/6J mice fed an obesogenic die

163 y feeding adult zebrafish a Se-elevated or a control diet followed by collection of larvae from both

164 er either continued to be fed an SPI diet or control diet for 1 or 3 wk.

165 ts of an EtOH-containing (Lieber-DeCarli) or control diet for 11 weeks and exposed to cigarette smoke

166 high-fat and high-sucrose (HF/HS) diet or a control diet for 12 weeks to investigate interindividual

167 w-fat dairy, and fruits and vegetables) or a control diet for 12 wk.

168 F1 rats were maintained on the control diet for 14 weeks after weaning and then underwe

169 2 mice were placed on a 4% alcohol diet or a control diet for 14 weeks.

170 h Microcystis to contrast with those given a control diet for 16 days.

171 ne-deficient (MCD) diet or the corresponding control diet for 2 weeks and characterized for histologi

172 with deuterium, and then pair-fed alcohol or control diet for 2 weeks.

173 n ethanol-containing liquid diet or pair-fed control diet for 4 (11% total kcal;early response) or 25

174 eeks of cholesterol-rich diet, a switch to a control diet for 4 weeks reduced serum cholesterol and s

175 Db/db and db/m mice were fed the MCD or control diet for 4 weeks to characterize differences in

176 e, and cholesterol (HTF-C diet) or a low fat control diet for 4 weeks.

177 received 5% ethanol-containing or isocaloric control diet for 5 weeks.

178 L/6 mice were fed an ethanol-containing or a control diet for 5 weeks.

179 HED pigs (n = 7) was transitioned back to a control diet for an additional six weeks.

180 rranean diet (MedDiet) intervention versus a control diet for cardiovascular prevention, with a media

181 ted with extra virgin olive oil or nuts vs a control diet for primary cardiovascular prevention.

182 tal day 21, Gen-1 mice were then kept on the control diet for the remainder of their life.

183 /6NTac mice fed a 60% high-fat diet (HFD) or control diet for up to 16 weeks.

184 (vol/vol) ethanol (11% calories) or pair-fed control diets for 2 days, 2 weeks or 5 weeks and superim

185 in-2 (Plin2-null) were fed either Western or control diets for 30 weeks.

186 cess to ethanol-containing diets or pair-fed control diets for 4 or 25 days.

187 e- and choline-deficient (MCD), high-fat, or control diets for 8-16 weeks.

188 diet containing 5% alcohol or an isocaloric (control) diet for 4 weeks.

189 clear cells, healthy humans were placed on a controlled diet for 1 week, then given fish oil and bora

190 ral Clinical Research Center and placed on a controlled diet for 7 days.

191 t reduction in arachidonic acid intake) or a control diet from the 15th wk of pregnancy to 4 mo of la

192 l levels in serum compared to group fed on a control diet (groundnut oil).

193 p and -0.086 (95% CI: -0.115, -0.057) in the control diet group (P = 0.491).

194 ter for Gjb2-CKO mice fed ACEMg than for the control diet group.

195 anean diet supplemented with mixed nuts, and control diet groups, respectively, corresponding to rate

196 fetuses derived from Pemt(-/-) dams fed the control diet had 25-50% less phospholipid-DHA as compare

197 ntly affected by walnut diets more than with control diets (HDL cholesterol: WMD = -0.2, P = 0.8; tri

198 mineralocorticoid excess-treated mice with a control diet, high-fiber diet, or acetate supplementatio

199 (80% restriction), LR (80% restriction) and control diet in 10-month-old C57BL/6J male mice.

200 tochondrial acetyl-proteome during CR versus control diet in mice that were wild-type or lacked the p

201 In a trial of the low FODMAP diet vs a control diet in patients with quiescent IBD, we found no

202 itiative (n = 153) were provided with a 2-wk controlled diet in which each individual's menu approxim

203 f 15 female subjects who were placed on well-controlled diets in which choline levels were manipulate

204 , Mediterranean, and high-protein diets with control diets including low-fat, high-GI, American Diabe

205 As expected, the control diet increased plasma lipids, whereas the MFGM d

206 TI-containing diets to intestines of mice on control diets increased the severity of colitis in these

207 atory gene expression in SAT compared with a control diet independently of body weight change in indi

208 epresents an important model for genetically controlled, diet-induced HCC.

209 Our studies identify a regulatory mechanism controlling diet-induced insulin resistance by highlight

210 s (0.5%, w/w) in supplementation with AIN76A control diet inhibited the growth of SCC1 tumor xenograf

211 med a systematic review and meta-analysis of controlled diet-intervention studies in nondiabetic subj

212 tions caused by Type 2 diabetes mellitus and controlled diet is one of them.

213 (43%) in the seven patients assigned to the control diet (log-rank p=0.03).

214 We fed male Sprague-Dawley rats a control diet, MCD diet, or betaine-supplemented MCD (MCD

215 eduction of 67; standard error, 78) than the control diet (mean reduction of 34; standard error, 50),

216 ereotypical behavior compared with mice with control diet microbiota in the absence of significant di

217 Compared with the control diet, milk- and cheese-based diets attenuated sa

218 followed a diet low in FODMAPs (n = 27) or a control diet (n = 25), with dietary advice, for 4 weeks.

219 striction diet (n=143, 66%) or an ad libitum control diet (n=75, 34%).

220 (TMD-Nuts; n=100), with respect to a low-fat control diet (n=96).

221 fat contents and compositions were compared: control diet [nondairy diet (~500 mg Ca/d)], milk diet [

222 serious adverse events than did those on the control diet (none vs nine; p=0.0005).

223 low sodium-DASH diet versus the high sodium-control diet on SBP were -5.3, -7.5, -9.7, and -20.8 mm

224 l, and both (low sodium-DASH vs. high sodium-control diets) on systolic blood pressure (SBP) by basel

225 supplementation, at 5-fold the level in the control diet, on the NTD and vertebral phenotypes in Apo

226 achidonic acid-balanced diet compared with a control diet] on the body weights and compositions of th

227 omized into two groups and received either a control diet or a DHA-supplemented diet for 7-8 weeks.

228 ice (Fbgalpha(+/-) mice) were fed either the control diet or a diet containing 0.025% alpha-naphthyli

229 f females feeding for two and five days on a control diet or a diet containing either a low or a high

230 as induced in C57BL/6 mice, which were fed a control diet or a diet containing resveratrol during eit

231 SJL/J mice were infected with TMEV and fed a control diet or a diet containing resveratrol during the

232 sition after 2, 4 and 8 weeks of consuming a control diet or a diet supplemented with fish oil.

233 Mice were fed a control diet or a diet supplemented with the FXR agonist

234 h-old wild-type and APP/PS1 mice on either a control diet or a diet that induces hyperhomocysteinemia

235 e [WT]) and diabetic ob/ob mice fed either a control diet or a methionine- and choline-deficient (MCD

236 receptor-deficient LDLr(-/-) mice were fed a control diet or a Western diet for 19 weeks.

237 ine decarboxylase (Hdc(-/-)) mice were fed a control diet or an HFD coupled with a high fructose corn

238 Rats were fed a control diet or an isocaloric diet enriched in cocoa for

239 s ligand (B6.gld) were given either high-fat control diet or ethanol diet by intragastric cannulation

240 ices from rats that were exposed to either a control diet or HFD from pregnancy day 13.

241 these biologic indicators in hPXR mice fed a control diet or HFD revealed further differences between

242 After 16 weeks on either a control diet or HFD, WT mice showed greater weight gain,

243 ith an intervention by feeding mice either a control diet or one containing the brain permeable beta-

244 7 tissues/fractions of young and old mice on control diet or one of 2 diet regimens (caloric restrict

245 agouti, were fed either a phytoestrogen-free control diet or one of six experimental diets: diets 1-3

246 wild type (WT) and Pemt(-/-) mice were fed a control diet, or a diet supplemented with 3 g/kg of DHA,

247 isolated from adult mice fed standard chow, control diets, or DHA supplemented diets.

248 and vegetables but otherwise similar to the control diet; or the DASH diet, which is rich in fruits,

249 ats were fed either an ethanol-containing or control diet over 14 weeks and euthanized 3 or 24 hours

250 g in the 3 consecutive 4-h samples after the control diet (P </= 0.005).

251 Tg mice fed the HM/LF diet compared with the control diet (P<0.001).

252 Compared with plasmas from mice fed a control diet, plasmas from mice fed a high-fat diet (HFD

253 Each subject received a phytosterol-controlled diet plus (1) ezetimibe placebo+phytosterol p

254 co1(-/-)Bco2(-/-) double knock-out mice to a controlled diet providing beta-carotene as the sole sour

255 compared with that of the weight-maintenance control diet) raised fasting plasma insulin concentratio

256 greater short-term improvements than did the control diets (random-effects model) for waist circumfer

257 ive access to either a high-fat or a matched control diet, rats received nonreinforced presentations

258 In the context of the control diet, reducing sodium (from high to low) was ass

259 Mice on a control diet served as age-matched controls.

260 Control diet (sham and MCT group) and isocaloric nutriti

261 o of suggested anti-inflammatory foods, or a control diet similar to the general dietary intake in Sw

262 Compared to a soy-free control diet, soy significantly shifted the cecal microb

263 Mice fed a control diet supplemented with homocysteine had a 3-fold

264 yogurt, or custard) with no red meat, and a control diet that contained neither red meat nor dairy.

265 ven amount of energy from free sugars with a control diet that provides the same amount of energy fro

266 h Initiative (WHI) were provided with a 2-wk controlled diet that mimicked each individual's habitual

267 Compared with the control diet, the DASH diet reduced hs-cTnI levels by 0.

268 Compared with the control diet, the fruit-and-vegetable diet reduced hs-cT

269 Compared with the control diet, the high-fructose diet significantly incre

270 Compared with the control diet, the HM/LF diet diminished endothelium-depe

271 sterol and triglyceride.In comparison with a control diet, the incorporation of cashews into typical

272 ia and tumors more rapidly than mice fed the control diet; the speed of tumor development was indepen

273 and PDACs than LSL-Kras/Ela-CreERT mice fed control diets; the mice fed the HFDs also had shorter su

274 All pups were weaned onto the control diet to specifically test the effects of early d

275 We fed alcohol or control diets to wild-type (WT) and IRF3 knock-out (KO)

276 igned to 8 weeks of monitored feeding with a control diet typical of what many Americans eat; a diet

277 After PN21 offspring were fed the same control diet until adulthood.

278 sodium, consuming the DASH compared with the control diet was associated with mean SBP differences of

279 Addition of the alginate to a control diet was investigated at both concentrations 2%

280 ence between the yogurt intervention and the control diet was only significant in one of these trials

281 ormal sex ratio observed in M. scalaris from control diets was affected by exposure to caffeine and p

282 eighted mean difference (WMD) between nut or control diets was estimated by using a random-effects me

283 Following a 3-day sodium-controlled diet, we infused Ang II (3 ng/kg per minute)

284 Slices from rats fed a control diet were bathed in artificial CSF replete with

285 of mice with CP (p = 0.055) and CP mice on a control diet were determined to spend more time at rest

286 Aged mice fed a control diet were exquisitely more susceptible to S. pne

287 (FASD, 10-fold higher than recommended) and control diet were fed to male Mthfr(+/+) and Mthfr(+/-)

288 Male F344 rats on either Test or Control diet were instrumented for electroencephalograph

289 were exposed to GOS/inulin mixture or fed a control diet were intraperitoneally sensitized to wheat

290 57BL/6 mice randomized to a high-fat diet or control diet were killed at 4-week intervals.

291 Wildtype mice (C57BLKS/J) fed the MCD or control diet were treated with SP600125; a c-Jun N-termi

292 holesterol from baseline between the OBG and control diets were analyzed by using random-effects meta

293 The 4 control diets were based on distinct national nutrition

294 eCarli diet containing alcohol or isocaloric control diets were fed to wild-type (WT) and MCP-1-defic

295 001) more on the New Nordic Diet than on the control diet, whereas normoglycemic individuals lost a m

296 (p = 0.032) compared to mice administered a control diet with restored serum lipase and amylase leve

297 We compared the effects of normal protein (control) diet with high protein diets containing whey, o

298 design, healthy volunteers (n=20) received a controlled diet with and without the serotonin precursor

299 respectively] compared with their respective control diets, with the largest effect size seen in the

300 less omega-3 PUFA than the conventional and control diets, yet contrary to expectations, together th