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1 inated in SMKO mice (approximately 5%-10% of control levels).
2 ase-specific heparan sulfate, was reduced to control level.
3 beta-amyloid, P-tau] were reduced almost to control level.
4 deficits but fails to enhance growth beyond control level.
5 re to hyperoxia but then decreased below the control level.
6 D) activity of only 6 (+/-5) % of the normal control level.
7 -treated cells rescued KGDHC activity to the control level.
8 ssed PRC protein at approximately 15% of the control level.
9 n followed by a recovery significantly below control level.
10 n of I(NaL) decreased both parameters to the control level.
11 d physiological L-type Ca(2+) current to the control level.
12 control and brings the depression down to a control level.
13 ing to 80% and 107%, respectively, above the control level.
14 ding to 39% and 38%, respectively, above the control level.
15 -Q138 to the cytoplasm restores viability to control levels.
16 n of ISG15 attenuates viral resistance to WT control levels.
17 dietary potassium, which also returned BP to control levels.
18 ued bacterial burden in the ethanol group to control levels.
19 ethylated and methylated INS DNA returned to control levels.
20 d total cholesterol (TC) with respect to the control levels.
21 acellular pH and restored enzyme activity to control levels.
22 the cell-cycle distribution and apoptosis to control levels.
23 of respiration with glutamate/malate back to control levels.
24 freezing response in MK-801-treated rats to control levels.
25 administered methamphetamine (0.05 mg/kg) at control levels.
26 glycemic ischemia that gradually returned to control levels.
27 C-1alpha target gene expression, compared to control levels.
28 acids, trimethylamine and cadaverine towards control levels.
29 nAg, it was nonetheless significantly above control levels.
30 reducing CP-AMPAR transmission in the NAc to control levels.
31 levels in all tissues examined compared with control levels.
32 fate and beta-hexosaminidase accumulation to control levels.
33 testinal IL-1beta and TNF-alpha cytokines to control levels.
34 not sufficient to restore differentiation to control levels.
35 sed mitochondrial aconitase activity back to control levels.
36 ection with a low-dose challenge compared to control levels.
37 and peat, but remained elevated relative to control levels.
38 14 days post-treatment increased again above control levels.
39 monocytes, where DC numbers are about double control levels.
40 icated by decreased relative impedance below control levels.
41 %), and activity ( approximately 108%) above control levels.
42 e (+15%, p<.01), but force did not return to control levels.
43 nockout mice and restored tumor incidence to control levels.
44 ession and cellular location to sinus rhythm control levels.
45 of citrate synthase to levels 36% to 44% of control levels.
46 nal hyperexcitability in epileptic tissue to control levels.
47 rate of apoptosis in active neurons back to control levels.
48 restored PKCepsilon mRNA and protein to the control levels.
49 -LTP by 125% (P<0.001), thus restoring it to control levels.
50 I cell epithelial permeability to protein to control levels.
51 spiking activity and spike amplitude toward control levels.
52 g" myofilament Ca(2+) responsiveness back to control levels.
53 d paired-pulse facilitation deficits back to control levels.
54 rations, and prolonged plasma coagulation to control levels.
55 signaling pathway or proteasome function to control levels.
56 1 and HeyL rescued TrkC(+) neuron numbers to control levels.
57 genesis by surviving interneurons overshoots control levels.
58 rm and rebounded in epileptic animals beyond control levels.
59 ression whereas mGlu3 mRNA was comparable to control levels.
60 ium and cellular ATP content are returned to control levels.
61 maintained in the contralateral hind paw at control levels.
62 ioral and neurochemical measures returned to control levels.
63 [(14)C] sucrose permeability, back to saline control levels.
64 ta-stimulated nuclear and cytoplasmic p65 to control levels.
65 e immune response attenuates neurogenesis to control levels.
66 ately 2.5-fold [P <0.03], respectively) over control levels.
67 ored mitochondrial and cytoplasmic Ca(2+) to control levels.
68 d biotinylated EAAC1 to approximately 50% of control levels.
69 verapamil to maintain AP duration (APD) near control levels.
70 rate of reepithelialization, which exceeded control levels.
71 vels restored autophagy and lysosome size to control levels.
72 growth, aging, asthma prevalence, and asthma control levels.
73 years (QALYs), and productivity loss across control levels.
74 cells in PD patients were reduced to healthy control levels.
75 either prevented or restored mtDNA damage to control levels.
76 rofiles of Trim63 (MuRF1) and Chrna1 back to control levels.
77 E activities were decreased to 32% to 85% of control levels.
78 use exogenous PKA restored all parameters to control levels.
79 unction and intensity of fibrosis to healthy control levels.
80 ile at 800 MPa, all combinations reduced the control levels.
81 eturned tumor-infiltrating CD4(+) T cells to control levels.
82 extent, the midbrain-rebounded or surpassed control levels.
83 gned to receive conventional dietary advice [control; level 0 (L0)] or PN advice on the basis of curr
84 ion was decreased by 25-30%, restored to the control levels 1 day after extubation, and secondarily d
86 the RPP to the left kidney was maintained at control levels (125 +/- 2 mmHg) by continuous servocontr
87 of nitrogen addition, and did not recover to control levels 20 years after nitrogen addition ceased.
91 ws that plant diversity decreased well below control levels after 10 years of chronic high rates (95-
95 the OVX groups, responding was enhanced from control levels after ExA self-administration in estradio
98 onfirmed by restoration of cell migration to control levels after overexpression of beta1 integrin in
99 lly higher in young Tg1 mice but falls below control levels after postnatal week 12, approximately co
104 s disease were largely comparable to healthy control levels, albeit with altered microstate topograph
106 H]thymidine incorporation that was twice the control level, along with complete retention of GSIS.
107 creased short-term but recovered only toward control levels, although in epileptic rats excitatory sy
108 creased concentration of RCIIs to 10% of the control level and, interestingly, an increased antenna c
109 l hippocampus restores DA neuron activity to control levels and also blocks the expression of locomot
110 neuroinflammation, return Arc expression to control levels and attenuate cognitive deficits induced
111 y respond to local TGF-beta-induced cues and control levels and distribution of Sox2 to form airways.
112 hose in striatum and cerebral cortex fell to control levels and hippocampal corticosterone remained e
113 l myofibres increases transient amplitude to control levels and protects them from loss after OSI.
114 tformin reduces mitochondrial respiration to control levels and significantly improves both motor fun
116 decreased the MKP-1 protein levels to 15% of control levels and was associated with sustained phospho
117 nd UMUC3 in a dose-dependent manner to 6% of control levels and was significant at 1 nmol/L in J82, T
118 on being one of the important processes that controls levels and activities of signaling molecules.
119 reatly elevated ( approximately 50-fold from control levels), and were highly correlated to liver dam
120 cantly increased, NCC expression returned to control levels, and body weight, plasma and urinary elec
121 on, autoantibodies progressively returned to control levels, and both soluble Abeta and axonal degene
122 ributed an anti-inflammatory role at various control levels, and recently we could show that annexin
124 mately 2-fold (P<0.05) over DHF, approaching control levels, and this effect was recapitulated with a
125 s of PRKN/PINK1 mutations were comparable to control levels; and (iv) that circulating cell-free mtDN
127 ntrol system propose that rules at different control levels are organized along an anterior-to-poster
128 e fundamental frequency partially recover to control levels as concussion symptoms abate, suggesting
131 ntly perturbed at 3 or 7 dpi and returned to control levels at 14 or 28 dpi, indicating that the meta
132 heart tissue 10-14 d after MI, returning to control levels at 21 d, whereas NE levels were stable.
134 oportion of cells in S and M phase exceeding control levels at 5-6 and 6 dpi, respectively, suggestin
137 bolism for both cell lines are kept close to control levels at room temperature and at 34 degrees C a
139 e mitofusins by shRNA to ~45% or ~52% of the control levels attenuated the htau-enhanced mitochondria
142 thylphenidate restored inhibitory ability to control levels but only partially corrected stability de
143 detraining (D7), C/F levels were similar to control levels, but both basal VEGF and TSP-1 were eleva
144 sion of beta1 integrin was reduced to 35% of control levels, but function remained normal at baseline
146 as very high at week 1 and decreased to near control level by 6 weeks without any RMC autophagy.
150 e reduced at 30 minutes and then returned to control levels by 2 hours following induction of LTP.
152 days following vagotomy and were restored to control levels by 30 days and 60 days, respectively.
153 eOL degeneration, preOL density recovered to control levels by 7 days after ischemia and was ~2 fold
157 of these fibrogenic changes was restored to control levels by the blocking of CCN2 signaling in HRHF
158 ence of retinal waves, but quickly mature to control levels by the end of the second week, suggesting
162 cues, odour matching performance approached control levels, demonstrating an inability to retrieve b
164 Nighttime symptoms were the most common control-level determinant; there were few (<1%) instance
167 ed after 52 h of wakefulness was restored to control levels during a 14-h recovery sleep episode.
168 rate of spinal cord OPCs rose fivefold above control levels during EAE, and numbers of oligodendrogli
170 9 +/- 0.3 mm) responses could be restored to control levels during fetal treatment with NO synthase b
171 eased after hyperoxia at P8, but returned to control levels during recovery between P12 and P15.
172 nic plants, photosynthesis was maintained at control levels during stress and the flag leaf showed in
174 manifests itself in relative impedance above control levels followed by a slower phase of collapse an
175 genotype, but resembled baseline nonpregnant control levels following delivery in mothers with a CC g
177 ays after stroke revealed a normalization to control levels for miR-125b-5p and miR-143-3p starting a
179 pha, HSP70 and Caspase 11 all increased over control levels found in uninjured cells suggesting that
180 nged withdrawal, GABAA receptors returned to control levels in alcohol-dependent nonsmokers, but alco
181 er content, was persistently increased above control levels in AQP4(-/-) mice but not WT mice at 14 a
183 es, but remained below 24 hr vehicle-treated control levels in HCT116 Bax-ko cells (65%, p = 0.004) b
186 y from PI103 treatment, k(PL) remained below control levels in HT29 cells (74%, p = 0.02), and increa
189 ury synuclein immunofluorescence remained at control levels in neurons that were identified as "good
194 All these changes could be inhibited to the control levels in the presence of Nox2 inhibitor or supe
195 rleukin-1beta) and restored interleukin-6 to control levels in the renal cortex, indicating the prote
197 also restored PKCepsilon mRNA and protein to control levels in vivo in fetal hearts and in vitro in e
198 d that the density increases to reach almost control levels in volumes further than 50 microm from a
199 n reduced EADs and improved cell survival to control levels in WT beta(2a)-expressing ventricular myo
200 ), whereas CD8 T-cell counts were similar to controls' levels in HIV-positive/CMV-negative subjects.
201 were largely preserved (approximately 80% of control levels) in SMKO mice because of exaggerated acti
202 tistically elevated, when compared with mean control levels, in all treatment and dose groups, while
203 rate-dependent NADPH production above vector control levels, in contrast to the increased production
204 motor skills of Cln3(-/-) mice normalized to control levels, indicating a disease-modifying effect of
205 ation and transendothelial migration back to control levels, indicating that stathmin may influence t
206 ned depressed, but lactate levels fell below control levels, likely as a result of rapid renal lactat
207 in Ncx1(-/-) (retaining approximately 20% of control level NCX current) and control mice were similar
208 s increased significantly (P < 0.001) from a control level of 0.61+/-0.17% ID/g (percentage of inject
209 e of (64)Cu-NOTA-TRC105-Fab increased from a control level of 3.4 +/- 0.1 to 9.5 +/- 0.4 percentage i
210 ast 10 days of the recovery period, reaching control level of cumulative egg and nauplii production w
211 ntibiotics is mediated by two divergons that control levels of a beta-lactamase, PC1, and a penicilli
212 function primarily in feedback regulation to control levels of beta-catenin and thus prevent precocio
214 cal inputs were designed to spatiotemporally control levels of second messengers, IP3, phosphatidylin
215 his paradigm display resilience, maintaining control levels of social exploration and sucrose prefere
218 are absent on human T cells, were thought to control levels of T-cell activation in chimpanzees and w
220 function associated with impaired ability to control levels of the CTLA-4 ligands, CD80 and CD86.
221 ts showed that a key function of Magoh is to control levels of the microcephaly-associated protein Li
223 ined fluorinated nano-building blocks with a controlled level of reactive functionality for the devel
224 ariectomized mice with or without sustained, controlled levels of 17-beta-E2 administered by s.c. imp
225 s, we have generated mice having genetically controlled levels of AM mRNA ranging from approximately
229 negatively regulates cytokinin responses by controlling levels of a key family of transcription fact
230 uences antiviral recognition responses, that controlling levels of adaptor molecules is a recurring s
231 B1 plays an important role in development by controlling levels of cell cycle regulators and thereby
233 ing-protein EIPR-1 as proteins important for controlling levels of DCV cargoes in Caenorhabditis eleg
235 ts cargo transport to the Golgi apparatus by controlling levels of phosphatidylinositol 4-phosphate,
236 ght an important new role for cholesterol in controlling levels of proteins, extending the known repe
237 xide dismutase (SOD) enzymes are critical in controlling levels of reactive oxygen species (ROS) that
238 ntial phase independent of Ler by negatively controlling levels of the regulators GrlA and GrlR throu
239 USP42 forms a direct complex with p53 and controls level of ubiquitination during the early phase
240 d nonvesicular cholesterol transport pathway controls levels of accessible cholesterol in the PM, as
243 midase (GBA2) is one of several enzymes that controls levels of glycolipids and whose activity is lin
245 protein, EGLN1/PHD2, is an O(2) sensor that controls levels of the Hypoxia Inducible Factor-alpha (H
247 nts and 291 matched individuals without IBD (controls); levels of tryptophan were measured using high
248 elial cells from 19 patients with DTS and 16 controls, levels of MMP-9 and its regulating cytokine mR
249 d increased expression of IL28 compared with controls; levels of IL28R were increased in the colonic
250 were able to restore M channel activity to a control level or above, while at saturating voltages the
251 ities in mutant membranes were mostly at the control level or modestly reduced, except mutants of Glu
257 GC-A mRNA in GC-A KO podocytes to 1% of the control level, Podo-GC-A KO mice and control littermates
259 diminished elevated mitochondrial ROS to the control level, prevented structural damage to mitochondr
263 e co-treatment restored most fluxes to their control levels, resulting in steatotic lipid accumulatio
264 GABAergic neurons were reduced to 70% of control levels short-term, where they remained in epilep
265 rylation to approximately 70% of the in vivo control level slows ktr and reduces force generation.
267 ubstances to a maximum of only 50-60% of the control level, suggesting that complex protein-protein i
268 of disinhibited synaptic currents to healthy control levels, suggesting a Src kinase- and actin-depen
269 expression reversed these phenotypes back to control levels, suggesting that lap may modulate glutama
270 ated endothelin-converting enzyme to 180% of control levels, suggesting that the Abeta-lowering abili
271 a decrease in neuronal ATP levels (to 48% of control level) that was reversed significantly to 70% of
273 the BLA reduced DAMGO-induced food intake to control levels, this treatment led to exaggerated number
274 one concentration is followed by a return to control level throughout the days of head-fixed training
275 maintain glucose concentrations at sedentary control levels, thus not requiring the counter-regulatio
276 it provides a global chromosome-wide nominal control level to clustering, as opposed to previous meth
277 ast differentiation and invasion reverted to control levels upon activation of PPARgamma and concomit
280 ne in vitro restores hippocampal function to control levels via activation of high-affinity nicotinic
281 Also, normalization of striatal mGluR5 to control levels was associated with reduced craving ("des
282 , a full reversal of mtDNA damage to healthy control levels was observed and correlated with measures
283 ases, 100 controls) and dust (682 cases, 513 controls) levels were treated as natural log-transformed
284 as well as subjects without these disorders (controls); levels were correlated with clinical features
289 rmone/testosterone ratio returned to Vehicle control levels which was not the case in EDS + Sham anim
290 maintained miR-133a expression near vehicle control levels, which coincided with (1) a decrease in c
291 resource allocation toward roots returned to control levels, while allocation to the young leaves inc
292 itic spine turnover in adult ngr1-/- mice to control levels, while an acutely enriched environment in
294 vated RyR2 levels in AD mice are restored to control levels with dantrolene treatment, as are synapti
296 ased after METH exposure, but was reduced to control levels with NACA treatment, as measured by dichl
299 ation within the knee, and were decreased to control levels with the use of a specific inhibitor of T
300 active DA neurons in the VTA of MAM rats to control levels without affecting DA firing in SAL rats,