戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 adpoles to pentylenetetrazole (PTZ), a known convulsant.
2 -CCM is a GABAA receptor inverse agonist and convulsant.
3  during the age when CRF is most potent as a convulsant.
4 l activity elicited by the administration of convulsants.
5  are anaesthetics but a few unexpectedly are convulsants.
6 lity and reduce seizure threshold to further convulsants.
7  currents and decreased binding of the caged-convulsant [(35)S]TBPS to GABA(A) receptors.
8 x and allosterically modulate the binding of convulsant ([35S]t-butylbicyclophosphorothionate, [35S]T
9                                          The convulsant 4-aminopyridine (4AP) facilitates the synchro
10                                          The convulsant 4-aminopyridine was used to induce interictal
11 odulates seizure-like bursts elicited by the convulsant 4-aminopyridine.
12 re administered subconvulsant (25 mg/kg) and convulsant (60 mg/kg) doses of PTZ during a week, and br
13                  It is unknown whether these convulsants act as negative allosteric modulators by bin
14 tory sites are not enantioselective, and the convulsant action of S-mTFD-MPPB results from its neglig
15 ll pharmacological profile suggests that the convulsant actions of PS are mediated predominantly via
16                                    A lack of convulsant activity coupled with neuroprotective effects
17 thereby inhibiting cannabinoid-mediated anti-convulsant activity in mice.
18 the low affinity site, but without potential convulsant activity, inherent to agents which block GABA
19 say and also inhibited delta agonist-induced convulsant activity.
20 etermine the functional consequences of anti-convulsant administration directed against seizure activ
21 nula was placed in the lateral ventricle for convulsant agent administration (20 microg of NMDA), whi
22 tylenetetrazol (PTZ), which is a widely used convulsant agent, on rat brain.
23  study was to examine the effect of the anti-convulsant agent, riluzole, on high-affinity glutamate u
24 izure phenotype of DKO mice at high doses of convulsant agents and advanced stages of seizures.
25 d DMCM, a negative allosteric modulator with convulsant and anxiogenic properties.
26 nyl) barbituric acid (S-mTFD-MPPB) acts as a convulsant and GABAAR inhibitor.
27 arbiturates have been discovered that act as convulsants and inhibitors of gamma-aminobutyric acid ty
28 es (e.g. endosulfan and fipronil) are potent convulsants and insecticides that block the GABA-gated c
29 , including trycyclic anti-depressants, anti-convulsants, and opiates.
30 tigated the effect of carbamazepine, an anti-convulsant, as it is known to have membrane stabilising
31 (S-mTFD-MPPB), a photoreactive analog of the convulsant barbiturate S-MPPB, inhibits alpha1beta3gamma
32 e compared how glyphosate and Roundup affect convulsant behavior in C. elegans and found that glyphos
33                In mice the S-enantiomer is a convulsant, but the R-enantiomer is an anticonvulsant.
34 electroencephalographic seizures produced by convulsant compounds, and these effects were associated
35  DM-CocE administered to rats 10 min after a convulsant dose of cocaine alters the normal metabolism
36 er maze before and after administration of a convulsant dose of pilocarpine at P34.
37 nsity of the seizure response to intravenous convulsant doses of cocaine and lidocaine individually (
38  Bicuculline injected intracortically in non-convulsant doses produced a stereotypy indistinguishable
39 lts from the present study suggest that anti-convulsant drug administration directed against subclini
40                                         Anti-convulsant drug administration or recurrent seizures can
41 ctional recovery than seizures alone or anti-convulsant drug treatment after seizure activity has occ
42                                 Certain anti-convulsant drugs, as well as convulsive seizures impede
43 flammatory drugs, paracetamol, opioids, anti-convulsant drugs, skeletal muscle relaxants, or corticos
44 es reported in immature zebrafish exposed to convulsant drugs.
45 suggest that alpha4betadelta GABARs are anti-convulsant during adolescence.
46 er, MK-906, along with P, prevented this pro-convulsant effect of P withdrawal, suggesting that the G
47 ic acid in the brain contributes to the anti-convulsant effect of the medium chain triglyceride ketog
48 BA in the hippocampus, and result in an anti-convulsant effect with this peptide.
49 These data are consistent with T having anti-convulsant effects and that 5alpha-reduced metabolites m
50 this report, we test the hypothesis that pro-convulsant effects are produced by withdrawal from the G
51 ich are likely to contribute to the additive convulsant effects noted when these drugs are abused in
52 +)-deficient mice were more sensitive to the convulsant effects of a peripheral injection of NMDA (10
53                    However, tolerance to the convulsant effects of CRF develops rapidly.
54 lly regulated CRF1 receptor in mediating the convulsant effects of CRF in the developing brain.
55 und that +/T mice were more sensitive to the convulsant effects of nicotine compared with their wild-
56 nt of epilepsy, as well as the pro- and anti-convulsant effects of the newer anesthetic agents.
57 ent which may be functionally related to the convulsant effects of this excitotoxin.
58 rimarily upon a differential response to the convulsant effects of various drugs.
59 ith in vivo anesthetic, anxiolytic, and anti-convulsant effects.
60 on of food intake, reward behaviour and anti-convulsant effects.
61 s cognition in animals without anxiogenic or convulsant effects.
62 ced in otherwise normal rats by the volatile convulsant flurothyl decrease the accuracy of locating a
63 ssy fiber sprouting is neither pro- nor anti-convulsant; however, there are caveats.
64 eptors expressed in Xenopus oocytes and were convulsant in mice after parenteral administration.
65 )-251D and aPTX (+)-267A proved to be potent convulsants in mice, with (+)-267A being approximately 5
66  However, they reduce thresholds to chemical convulsants in vivo and electrical stimulation in vitro,
67 reover, intervention after birth reduces the convulsant-induced seizure threshold to a level similar
68 ensitivity of naive mice to several chemical convulsant-induced seizures was also correlated with EtO
69 can be instigated in experimental animals by convulsant-induced status epilepticus (SE).
70 riform cortex" ("area tempestas") from which convulsants initiate temporal lobe seizures, and blockad
71 se in sensitivity to seizures induced by the convulsant kainic acid.
72                                            A convulsant, kainic acid, increased bursts only in the sp
73 The application of drugs targeting different convulsant mechanisms (4-Aminopyridine, Pentylenetetrazo
74 ent, less severe and more responsive to anti-convulsant medication.
75 anesthetics, non-anesthetics, and anesthetic/convulsants on tubulin dynamics.
76 ippocampal-dependent memory task-without the convulsant or proconvulsant activity associated with non
77 r to measuring seizure susceptibility to the convulsant pentylenetetrazol (PTZ), given via timed tail
78  a subthreshold dose (40 mg/kg) of the chemo-convulsant pentylenetetrazol and events recorded over 1
79 e showed an elevated seizure response to the convulsant pentylenetetrazol compared with sham mice, as
80 tin levels and delayed the onset of chemical convulsant pentylenetetrazole-induced generalized convul
81 bupivacaine (IC50 = 68 microM), and the anti-convulsant phenytoin ( approximately 50% inhibition at 2
82                   The central nervous system convulsant picrotoxin (PTX) inhibits GABA(A) and glutama
83 d bilobalide) have structures similar to the convulsant picrotoxin, a GABA(A) receptor antagonist, so
84 f many pharmacological agents, including the convulsant picrotoxin.
85 entally, a single SE episode, induced by the convulsant pilocarpine, initiates the development of an
86 pticus; SE) were induced by injection of the convulsant pilocarpine.
87 mg/kg failed to induce seizures or alter the convulsant potencies of pentylenetetrazol and NMDA.
88  of PS (50 nmol) significantly increased the convulsant potencies of systemically administered pentyl
89                  This study investigated the convulsant potency of cocaine and lidocaine alone and in
90                      It does not exhibit the convulsant, proconvulsant, or anxiogenic activity associ
91 azole-induced seizures, consistent with anti-convulsant properties and possible anxiolytic activity.
92 A receptors substantially contributed to the convulsant properties of acetylene analog 9.
93 campus, and that this action may confer anti-convulsant properties on agonists acting at the BB(2) re
94 alone, further suggesting that SNC80 has pro-convulsant properties.
95 SR mice than in WSP mice, suggesting an anti-convulsant role for receptor activation.
96                   The in vivo actions of the convulsant S-enantiomer are accounted for by its interac
97                                          The convulsant S-enantiomer binds solely at an inhibitory si
98 ognitive disorders because of anxiogenic and convulsant side effects.
99  the GABAAR transmembrane domain or to known convulsant sites in the ion channel or extracellular dom
100     To apply the same strategy to locate the convulsant sites requires the creation and mechanistic c
101 toxication) or seizure activity (produced by convulsants) slowed diffusion by >10-fold and created de
102                         One hour before each convulsant, Sprague Dawley rats received either saline (
103 activation of glutamate receptors, and other convulsant stimuli, the specific molecular steps require
104  mutant (Dbh -/-) mouse using four different convulsant stimuli: 2,2,2-trifluroethyl ether (flurothyl
105                          It is the target of convulsants such as picrotoxin and is mutated in some fo
106 ce exhibiting greater sensitivity in various convulsant tests compared to C57 mice.
107 ntifies the binding site of the highly toxic convulsant tetramethylenedisulfotetramine (TETS), which
108       4-Aminopyridine (4-AP) is a well known convulsant that enhances the release of both excitatory
109 sulfotetramine (TETS) is a so-called "caged" convulsant that is responsible for thousands of accident
110     Further work is warranted regarding anti-convulsants that dampen post-traumatic seizures as a rou
111  long-term enhanced susceptibility to limbic convulsants that lasted to adulthood.
112                       However, after the pro-convulsant, the test mice (n = 9) had increased numbers
113  kinase, which may be the basis for new anti-convulsant therapies.
114 ures nor increased seizure susceptibility to convulsant were observed when Lgi1 was deleted in parval
115              In mice, S-mTFD-MPPB acted as a convulsant, whereas R-mTFD-MPPB acted as an anticonvulsa
116  are anaesthetics but unexpectedly a few are convulsants whose mechanism of action is poorly understo
117                                          Pro-convulsant withdrawal properties have been reported for

 
Page Top