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1 sure to platelet agonists (thrombin, ADP, or convulxin).
2 of protease-activated receptor 4 peptide and convulxin.
3  responses to the GPVI agonists collagen and convulxin.
4 ype I collagen, collagen-related peptide, or convulxin.
5 R agonists SFLLRN and AYPGKF or GPVI agonist convulxin.
6 ollagen, collagen-related peptide (CRP), and convulxin.
7  high concentrations of thrombin, AYPGKF, or convulxin.
8  FcR gamma-chain and functional responses to convulxin.
9 ponse to high concentrations of collagen and convulxin.
10 e (PMA) and only slightly attenuated that by convulxin.
11 on stimulation with the snake C-type lectin, convulxin.
12 ly marginally inhibit aggregation induced by convulxin.
13 rylation and Ca(++) elevation in response to convulxin.
14 (U46619), and very low doses of thrombin and convulxin.
15 ADP, thromboxane analogue (U46619), TRAP, or convulxin.
16  ligand blotting using the snake venom toxin convulxin.
17      The addition of cathepsin G (425 nm) or convulxin (10 nm) to PRP dramatically reduced the t(50)
18 s strong adhesive and signaling responses to convulxin (a snake venom protein that directly binds GPV
19  Rap1 is robustly activated upon addition of convulxin, a GPVI-specific agonist.
20  in response to type I fibrillar collagen or convulxin, a snake venom protein and known platelet agon
21 erived plasminogen retention on thrombin and convulxin activated human platelets was confirmed by flo
22 ous activation with 2 agonists, thrombin and convulxin, an activator of the collagen receptor glycopr
23 coprotein (GP)VI-selective snake venom toxin convulxin and by collagen.
24                   The GPVI-specific agonists convulxin and collagen-related peptide (CRP) also stimul
25 gregation using the GPVI-selective agonists, convulxin and collagen-related peptide (CRP) as well as
26 to initiate signalling events in response to convulxin and collagen-related peptide.
27 e cluster ligands in a manner reminiscent of convulxin and flavocetin.
28 imulation with the collagen receptor agonist convulxin and thrombin, 68% of platelets from C57BL/6 mi
29 f the total population and is referred to as convulxin and thrombin-induced-FV (COAT-FV) platelets.
30  co-injection of the glycoprotein VI agonist convulxin and was mimicked by glycoprotein VI inhibition
31 e, thrombin, PAR1-agonist, PAR4-agonist, and convulxin) and micro-ELISA arrays were used to quantify
32 )) mice were activated with the GPVI agonist convulxin, and surface expression of P-selectin (a marke
33 gonist thrombin, the glycoprotein VI agonist convulxin, and the cytokine receptor Mpl agonist thrombo
34 ivation induced by adenosine 5'-diphosphate, convulxin, and thrombin; (3) significant increases of pr
35  Notably, aegyptin prevents collagen but not convulxin binding to recombinant glycoprotein VI.
36 osphorylated proteins in response to TPO and convulxin but not by thrombin occurred with a similar ti
37 iate functional responses to the snake venom convulxin by reconstitution of mutant forms of GPVI in R
38 d with a number of agonists (TRAP, thrombin, convulxin, collagen, PMA, thapsigargin, or A23187) and a
39 50 = 6.7 muM), CRP-XL (IC50 = 53.5 muM), and convulxin (CVX) (IC50 = 5.7 muM) mediated platelet aggre
40                                              Convulxin (CVX) is a C-type lectin-like protein from the
41                                              Convulxin (CVX), a C-type snake protein from Crotalus du
42 t reactivity to adenosine diphosphate (ADP), convulxin (CVX), and thrombin receptor activator peptide
43 ide (CRP), and the snake venom C-type lectin convulxin (CVX).
44 23187 in conjunction with either thrombin or convulxin did generate this population.
45                                              Convulxin did not stimulate procoagulant activity in eit
46 ein kinase C further reduced the response to convulxin in pearl platelets demonstrating a direct role
47                                              Convulxin induced surface expression of P-selectin in SL
48 gamma null mice, for which collagen, but not convulxin, induced procoagulant activity (P <.01).
49                                          The convulxin-induced decrease was sensitive to both PKCalph
50                                 In contrast, convulxin-induced dense granule secretion was potentiate
51  shifted the concentration-response curve of convulxin-induced platelet aggregation to the right.
52          In PKC-delta-null murine platelets, convulxin-induced SHIP-1 phosphorylation was inhibited.
53          Consistent with the latter finding, convulxin-induced Syk phosphorylation is significantly a
54 MS16 also inhibits collagen-induced, but not convulxin-induced, platelet cytosolic Ca(2+) mobilizatio
55 her hand, activation of ERK2 by thrombin and convulxin is delayed and also inhibited by the protein k
56 e whether the weak inhibitory action against convulxin is due to release of agonists other than ADP f
57                                 Importantly, convulxin is not able to activate cells transfected with
58 methyl-BAPTA resulted in total inhibition of convulxin-mediated aggregation.
59 e to collagen or GPVI-specific agonists like convulxin or collagen-related peptide (CRP).
60 telets after costimulation with thrombin and convulxin (P <.05).
61  became "coated" after dual stimulation with convulxin plus thrombin (P < .05 vs C57BL/6 platelets).
62 rface following dual-agonist activation with convulxin plus thrombin to produce coated platelets.
63 ithout affecting aggregation induced by ADP, convulxin, PMA, and ristocetin.
64  antibody and by GP VI ligands (collagen and convulxin) reduced binding of biotinylated convulxin to
65 th thrombin plus the glycoprotein VI agonist convulxin resulted in a rapid loss of mitochondrial tran
66  that stimulation of platelets with thrombin/convulxin significantly increased the plasminogen signal
67 as significantly attenuated in thrombin- and convulxin-stimulated platelets from Plg-RKT-/- mice comp
68                     The Arf6-GTP decrease in convulxin-stimulated platelets showed similar requiremen
69 mong the proteins tyrosine phosphorylated on convulxin stimulation in PMA-differentiated HEL cells.
70 cellular Ca2+ in response to the snake venom convulxin that targets GPVI.
71  pairwise combinations of six agonists (ADP, convulxin, thrombin, U46619, iloprost and GSNO used at 0
72 but enhances, caspase-dependent PS exposure; convulxin-/thrombin-induced PS exposure is entirely depe
73 creases of procoagulant platelets induced by convulxin/thrombin and platelet-dependent thrombin gener
74 re induced by the Ca(2+)-mobilizing agonists convulxin/thrombin fully relied on mitochondrial depolar
75 to pairwise combinations of ADP, U46619, and convulxin to activate the P2Y(1)/P2Y(12), TP, and GPVI r
76 d convulxin) reduced binding of biotinylated convulxin to the stimulated platelets.
77 eated plasma to six different agonists (ADP, convulxin, U46619, SFLLRN, AYPGKF and PGE(2)) at three c
78  (BAPTA-AM) abolished aggregation induced by convulxin under all conditions.
79 ation induced by threshold concentrations of convulxin undergoes synergy with ADP acting via the P2Y1
80 uction by dual stimulation with thrombin and convulxin was less than that observed with A23187, indic
81  greater reduction in aggregation induced by convulxin was observed in pearl platelets than in the pr
82             Activation of phospholipase C by convulxin was potentiated by ADP acting through the P2Y1
83 elet aggregation at higher concentrations of convulxin was unaffected by these agents.
84  agonists collagen-related peptide (CRP) and convulxin were significantly inhibited in RhoG-deficient
85 ightly to the right at low concentrations of convulxin, whereas platelet aggregation at higher concen
86 are required for full aggregation induced by convulxin, whereas the response induced by collagen show
87                      Strong agonists such as convulxin, which acts on GPVI, and thrombin cause sustai