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1 s were found to bias the spectrum of genomic copy number abnormalities.
2  to detect allelic imbalance and chromosomal copy number abnormalities.
3 al mRNA synthesis, but no mtDNA deletions or copy number abnormalities.
4 r laboratory for array CGH and found to have copy-number abnormalities.
5                    The frequency of specific copy number abnormalities [+4, del(6q23.3-6q25.3), +12,
6 rofiles were correlated with genome-wide DNA copy number abnormalities and clinical and outcome featu
7 molecular level, myeloma is characterized by copy number abnormalities and recurrent translocations i
8 tions/deletions, chromosomal translocations, copy number abnormalities, and zygosity changes in a sin
9                   We identify two loci where copy number abnormalities are correlated with poor survi
10 he rate of diagnosis of patients with genome copy number abnormalities as the cause of developmental
11 rom 45 AA and 196 EA MM patients for somatic copy number abnormalities associated with poor outcome.
12  Plots of BAC end-sequences density identify copy number abnormalities at high resolution.
13   In this study, we compared the genome-wide copy number abnormalities between 29 human and 10 canine
14  mutations by PCR-capillary sequencing, EGFR copy number abnormalities by fluorescence and chromogeni
15 arch studies to identify clinically relevant copy number abnormality (CNA) profiles.
16                     The most commonly shared copy number abnormalities (CNAs) in all types were losse
17  a Wnt pathway signature and those harboring copy number abnormalities (CNAs) of potential prognostic
18                                              Copy number abnormalities (CNAs) represent an important
19                                              Copy number abnormalities (CNAs) such as somatically-acq
20 precursor ALL, we evaluated gene expression, copy number abnormalities (CNAs), and DNA methylation.
21  genomic hybridization (aCGH) to investigate copy number abnormalities (CNAs), one of the most promin
22    Multiple myeloma (MM) is characterized by copy number abnormalities (CNAs), some of which influenc
23 pically showed different patterns of genomic copy number abnormalities (CNAs), with the CNAs acquired
24                                The number of copy number abnormalities does not correlate with the la
25 arative genomic hybridization summarizes DNA copy number abnormalities in a tumor by mapping them to
26            To determine genomewide miRNA DNA copy number abnormalities in cancer, 283 known human miR
27 erstanding of common genomic and chromosomal copy number abnormalities in ccRCC, including chromosome
28 describes analyses of associations of genome copy number abnormalities in ovarian cancers with clinic
29  genetic lesions affecting the same genes or copy number abnormality in different subclones.
30 yses have identified a high frequency of DNA copy-number abnormalities in ALL, but the prognostic imp
31        Finally, we identified high frequency copy number abnormalities of Dicer1, Argonaute2, and oth
32  Subsequent analysis pinpoints suspected DNA copy number abnormalities of specific samples and chromo
33                                              Copy number abnormalities play an important role in pati
34               Thus, detection and mapping of copy number abnormalities provide an approach for associ
35                                     Frequent copy number abnormalities targeting cancer-associated ge
36                                            A copy-number abnormality was identified as a predictor of
37                                        Using copy-number abnormalities, we identified a predictor of
38                                          DNA copy number abnormalities were identified using microarr
39                       More than 50 recurring copy-number abnormalities were identified, most commonly
40                      We found two classes of copy number abnormalities: whole chromosome and partial