ライフサイエンスコーパス: cord blood

1 -fold higher than children with nondeficient cord blood.

2 activation of mDC from adults than mDC from cord blood.

3 to estimate eosinophil cells proportions in cord blood.

4 small fraction of CpGs, and focused on whole cord blood.

5 s from maternal blood and neonatal umbilical cord blood.

6 vels were assessed at prenatal visits and in cord blood.

7 nd primary mast cells derived from umbilical cord blood.

8 encing (30 AGA, 21 SGA) and also analyzed in cord blood.

9 ethylated in placenta and hypermethylated in cord blood.

10 e were significantly higher in maternal than cord blood.

11 increase in the ratio of %-5mC to %-5hmC in cord blood.

12 n assessed from the mercury concentration in cord blood.

13 natal PFAS with DNA methylation in umbilical cord blood.

14 404 allogeneic HCT recipients, including 125 cord blood, 125 HLA-mismatched, and 154 HLA-matched HCTs

15 to 59 mo with deficient versus nondeficient cord blood 25(OH)D (10.9 vs. 8.7 P = 0.14).

16 Children 12 to 35 mo of age with "deficient" cord blood (25(OH)D <30 nmol/L) had a mean dmft score tw

17 34.5% [31.4-37.9] to 44.2% [42.1-46.3]; and cord blood 36.3% [33.9-39] to 43.7% [40.8-46.8]).

18 first 12 h of life, from high mean levels in cord blood (47.6% (95% CI 43.7-51.5%)) to levels at the

19 At birth, median cord blood %-5mC, %-5hmC, and their ratio were 4.95%, 0.

20 in regulation of imprinting in placenta and cord blood; a lack of correlation of the methylome, tran

21 ons between maternal PFAS concentrations and cord blood adipocytokine concentrations were of small ma

22 t trimester and term maternal plasma, and in cord blood after delivery.

23 lar, further research should examine whether cord blood analyses can be used to identify HIE within a

24 1036 cord blood and 487 heel prick samples were collected fro

25 may be sensitive to cigarette smoke in both cord blood and adult [Formula: see text] cells.

26 We used myeloid DC (mDC) from cord blood and adult blood donors to evaluate whether im

27 Finally, experiments using human cord blood and airway epithelial cells suggested that DE

28 ct biological specimens, including umbilical cord blood and amniotic fluid, to be made available to t

29 y releasing factors that cause DNA damage in cord blood and bone marrow cells, including stem cells.

30 anti-human CD117 mAb, SR-1, inhibits normal cord blood and bone marrow HSCs in vitro.

31 xpansion of phenotypically primitive CD34(+) cord blood and bone-marrow-derived HSPCs.

32 The decreased vitamin B-12 in liver and cord blood and decreased expression and activity of MS i

33 iferation, we compared the miRNA profiles of cord blood and peripheral blood ECFC-derived cells.

34 enotype and DNA methylation (DNAm) data from cord blood and peripheral blood to identify SNPs associa

35 In distributed lag models, both cord blood and placental telomere length were associated

36 In 641 newborns, cord blood and placental telomere length were significan

37 with newborn telomere length as reflected by cord blood and placental telomere length.

38 nalysis, participants with full data on both cord blood and placental telomere lengths were included,

39 In the newborns, cord blood and placental tissue relative telomere length

40 associated with higher leptin levels in both cord blood and post-delivery maternal serum.

41 tion levels at 482,397 CpG loci in umbilical cord blood and retained 394,460 loci after quality contr

42 Cord blood and two further blood samples up to 96 hours

43 xposure during midgestation (weeks 12-25 for cord blood and weeks 15-27 for placenta).

44 ed a single infusion of autologous umbilical cord blood and, as part of their clinical outcome measur

45 red in blood cell samples obtained at birth (cord blood) and ages 1 and 3 years.

46 hed unrelated, haploidentical, and umbilical cord blood), and compared transplantation outcomes acros

47 (bone marrow, peripheral blood, or umbilical cord blood), and transplantation period (2008-12 and 201

48 specific IgG reactivity profiles in mothers, cord blood, and breast milk were highly correlated.

49 lizing antibodies were measured in maternal, cord blood, and infant sera at 3 and 6 months of age.

50 he presence of SARS-CoV-2 in amniotic fluid, cord blood, and neonatal throat swab samples.

51 t were measured in maternal blood, umbilical cord blood, and placental tissue when available.

52 The CpG targets of ASD meQTLs across cord, blood, and brain tissues are enriched for immune-r

53 We demonstrate that maternally-derived, cord-blood anti-PfSEA-1 antibodies predict decreased ris

54 Children with high cord-blood anti-PfSEA-1 antibody levels had 51.4% fewer

55 y associated with maternal and GPx levels in cord blood as well as maternal TNF-alpha levels were inv

56 at glucocorticoids are lower in maternal and cord blood at delivery in obese pregnancies.

57 regnancy have been described in placenta and cord blood at delivery, in fetal lung, and in buccal epi

58 We collected maternal and cord blood at delivery, measured manganese using inducti

59 enatal lead exposure with DNA methylation in cord blood at epigenome-wide significance level [false d

60 les from mothers during the third trimester, cord blood, breast milk collected 2 months after deliver

61 paired sampling between maternal plasma and cord blood, breastmilk and infant plasma.

62 Cord blood cadmium was analyzed as detectable or undetec

63 RSV antibody levels in cord blood can predict whether an infant will be infecte

64 provided clinical evidence that allogeneic, cord blood CAR-NK cells induce high rates of non-durable

65 ity of RSV and IAV to activate primary human cord blood (CB) and adult blood (AB) myeloid DC (mDC).

66 tor (TCR) alphabeta/CD19-depleted grafts, 43 cord blood (CB) grafts (72% with no serotherapy), 17 CD3

67 Human umbilical cord blood (CB) has attracted much attention as a reserv

68 ted with Fenton BW z-score and the umbilical cord blood (CB) lipidome.

69 For this purpose, we gathered 136 human cord blood (CB) samples.

70 en HSC numbers are limited, as in the use of cord blood (CB).

71 haracterization was performed from umbilical cord blood (CB).

72 ogenitors from peripheral blood (PB) but not cord blood (CB).

73 generation was evaluated in neutrophils from cord blood (CB, n = 11) and adult blood (n = 10) followi

74 that LC and iDDC generated from pluripotent cord blood CD34(+) cell precursors support productive in

75 A small subset of human cord blood CD34(+) cells express endothelial protein C r

76 OXA(+) CD34(+) progenitors, as well as human cord blood CD34(+) cells, give rise to NK cells that exh

77 mental model system was employed using human cord blood CD34(+) hematopoietic stem/progenitor cells.

78 e engineering of primary adult and umbilical cord blood CD34(+) human hematopoietic stem and progenit

79 he offspring of mothers with T1D had reduced cord blood CD4(+) T-cell responses to proinsulin and ins

80 Neonatal and human cord blood CD71(+) cells express arginase II, and this e

81 sthma/allergy and determined the capacity of cord blood cells (n = 151) to produce any of a panel of

82 etic output capabilities in neonatal CD34(+) cord blood cells are determined by a history of external

83 at conventionally defined subsets of CD34(+) cord blood cells are heterogeneous in their functional p

84 IR/HLA genetic incompatibilities and studied cord blood cells at both the phenotypic and functional l

85 and cell-cycle indicators in single CD34(+) cord blood cells before and up to 2 hours after their st

86 It is interesting to note that cord blood cells exhibited extremely low HLA class I exp

87 Cord blood cells from females of HIV-uninfected sex-disc

88 Culturing 5azaD/TSA-expanded cord blood cells in extended cultures reveals that they

89 utputs from >1000 index-sorted CD34(+) human cord blood cells in short-term cultures with and without

90 t human iTregs generated in vitro from naive cord blood cells preferentially recruit Disc large homol

91 (BJ, K562, JK1, and Jurkat), and 480 primary cord blood cells undergoing erythroblast differentiation

92 3/ITD(neg) cells and spared normal umbilical cord blood cells.

93 to CIDRalpha1 domains were more frequent in cord blood compared with antibodies to CIDRalpha2-6 doma

94 ryptophan, and methionine) were increased in cord blood compared with the maternal samples.

95 sma PFAS concentrations and birth weight and cord blood concentrations of leptin and adiponectin usin

96 e associations between maternal or umbilical cord blood concentrations of perfluorooctanoic acid and

97 n but is clinically relevant as maternal and cord blood cortisol levels are correlated and offspring

98 em cells (HSC) within a single unit of human cord blood currently limits its use as an alternate graf

99 Our results demonstrated that maternal and cord blood cytokines were differentially associated with

100 e, enucleated erythroid cells from umbilical cord blood derived CD34(+) haematopoietic progenitor cel

101 ersulfone nanofiber-expanded human umbilical cord blood-derived CD34(+) cells (henceforth CD34(+) cel

102 Accordingly, the treatment of umbilical cord blood-derived CD34(+) HSPCs with stimulatory EVs-al

103 hairpin RNA knockdown of CLC/Gal-10 in human cord blood-derived CD34(+) progenitor cells impairs eosi

104 1A (LSD1) induces a rapid expansion of human cord blood-derived CD34+ cells and promotes in vitro pro

105 Comparison with expanded cord blood-derived CD4(+)CD25(hi) tTreg and expanded Tef

106 Cs, the endothelial layer consisted of human cord blood-derived endothelial progenitor cells (hCB-EPC

107 ated results are obtained in human umbilical cord blood-derived erythroid progenitor-2 cells, in whic

108 ator of adhesive properties in primary human cord blood-derived hematopoietic stem and progenitor cel

109 lts in a marked expansion of human umbilical cord blood-derived HSPCs following cytokine stimulation.

110 s prolong VEGFR-2 and Akt phosphorylation in cord blood-derived late outgrowth endothelial progenitor

111 Similarly, human cord blood-derived macrophages express IL-27 genes and s

112 ine bone marrow-derived mast cells and human cord blood-derived mast cells.

113 The LAD2 MC line or primary human cord blood-derived MCs (CBMCs) were infected with HRV or

114 e marrow MKs, platelets, and differentiating cord blood-derived MK cultures, and identified MK miR-12

115 adoptive transfer of ex vivo expanded human cord blood-derived NK cells into humanized mice reconsti

116 successfully differentiated human umbilical cord blood-derived progenitor cells into CARiK cells.

117 llowed by independent validation of selected cord blood differentially methylated regions, using bisu

118 Cord blood DNA from singletons was measured using the Me

119 y levels were associated with lower regional cord blood DNA methylation at the Paraoxonase 1 gene (PO

120 ker measured mid-pregnancy, and Illumina450K cord blood DNA methylation from newborns in the Norwegia

121 ions of eosinophils in cord blood, using the cord blood DNA methylation profiles, based on markers of

122 sal effect of maternal vitamin B12 levels on cord blood DNA methylation using the maternal FUT2 genot

123 Umbilical cord blood DNA methylation was evaluated using the Illum

124 sal effect of maternal vitamin B12 levels on cord blood DNA methylation, and a causal effect of vitam

125 aternal prenatal blood mercury levels in 321 cord blood DNA samples and examined the persistence of t

126 Cord blood DNA samples from the infants were interrogate

127 iated with altered prenatal GCs in newborn's cord blood DNA.

128 FDR) <= 0.05), respectively, in the infant's cord blood DNA.

129 /10 matched unrelated donor or 6/6 unrelated cord blood donor.

130 d, mismatched unrelated, haploidentical, and cord blood donors.

131 Unrelated non-cord-blood donors and recipients were typed at the allel

132 d be measured in spots of wild-type adult or cord blood dried onto filter paper at levels significant

133 vely correlated with several plasma-treated, cord blood ECFC proliferative capacity parameters in the

134 Cord blood ECFC proliferative capacity was significantly

135 The miR-193a-3p mimic reduced cord blood ECFC-derived cell proliferation, migration an

136 HMGB1 silencing in cord blood ECFC-derived cells confirmed its role in regu

137 reporter assays of miR-193a-3p mimic treated cord blood ECFC-derived cells, we identified 2 novel miR

138 ative capacity parameters were obtained from cord blood ECFCs treated with participant plasma.

139 PON1 was determined in cord blood for 474 infants.

140 We examined prenatal and cord blood for 76 and 57 mother/infant pairs, respective

141 et production, and for the improved usage of cord blood for transplantation research and therapy.

142 Cord blood formate was influenced by fetal genotype, bei

143 text]) in an EPIC (Illumina) array study of cord blood [Formula: see text] cells ([Formula: see text

144 oke exposure in pregnancy is associated with cord blood [Formula: see text] DNA methylation in key re

145 ro exposure to maternal tobacco smoke on the cord blood [Formula: see text] DNA methylome.

146 ison, antitoxin IgG and NAb were measured in cord blood from 50 mothers unrelated to study infants.

147 m samples before and after immunisation, and cord blood from a subset of women and infants.

148 after vaccination and at both deliveries, in cord blood from both siblings, and in infants before and

149 T cells were isolated from wild-type mice or cord blood from healthy donors and submitted to polarizi

150 and fetal priming to tetanus vaccination in cord blood from human United States and Kenyan neonates.

151 We confirmed the decreased vitamin B-12 in cord blood from NTD pregnancies.

152 We replicated the main findings in cord blood from pregnancies of 41 NTD fetuses compared w

153 bolism) were hypermethylated in placenta and cord blood from SGA newborns, whereas GPR120 (related to

154 ssociations between maternal urinary TCS and cord blood FT3 as well as maternal blood FT4 concentrati

155 t positive correlations between maternal and cord blood glucocorticoid levels, increased maternal BMI

156 d maternal BMI was not associated with lower cord blood glucocorticoid levels.

157 ing outcomes: newborn weight, adiposity, and cord blood glucose, insulin, lipids, and leptin.

158 children with deficient vitamin D levels in cord blood had a dmft score at 12 to 35 mo 2-fold higher

159 explored whether specific types of folate in cord blood have differential association with ASD.

160 CD4+ and CD8+ T cells from cord blood, healthy young children, and adults were chal

161 ng the dominant-negative ETS1 p27 isoform in cord blood hematopoietic progenitor cells, we show that

162 ut of CLC/Gal-10 expression in human CD34(+) cord blood hematopoietic progenitors differentiated to e

163 istent with CPI203-mediated reprogramming of cord blood hematopoietic stem and progenitor cells.

164 bitor CPI203 enhanced the expansion of human cord blood HSCs without losing cell viability in vitro.

165 re-compatible marker of UM171-expanded human cord blood HSCs.

166 eduction in non-relapse mortality, except in cord blood HSCT recipients, who had a lower relapse inci

167 gress among recipients of haploidentical and cord blood HSCT.

168 Rationale: Human umbilical cord blood (hUCB) contains diverse populations of stem/p

169 Human umbilical cord blood (hUCB)-derived hematopoietic stem cells (HSCs

170 nt EBV (Delta3A) and wild-type (WT) EBV in a cord blood-humanized (CBH) mouse model.

171 nduces B cell lymphomas in a newly developed cord blood-humanized mouse model that allows EBV-infecte

172 of B cells in vitro In this study, we used a cord blood-humanized mouse model to compare the phenotyp

173 We recently used a newly developed cord blood-humanized mouse model to show that EBV can co

174 ity of EBV to induce B cell lymphomas in the cord blood-humanized mouse model, although the simultane

175 ns/infectious exposures had higher levels of cord blood IFN-gamma, IL-7, sTNFR1, and sTNFR2 compared

176 Cord-blood IgG concentration >=1.04 and >=1.53ug/ml were

177 In infants born >=34 weeks gestational age, cord-blood IgG geometric mean concentrations (GMC) were

178 zed mice reconstituted with autologous human cord blood immune cells.

179 to determine the concentration of formate in cord blood in comparison with maternal blood taken earli

180 ions, detection of tetanus-specific MBC from cord blood, indicative of fetal priming with tetanus vac

181 ti-capsular IgG was measured on maternal and cord blood/infant sera by multiplex Luminex assay; and t

182 Moreover, resting cord blood KIR3DL1 NK cells exhibited a basal alloreacti

183 a: see text] (DDE), hexachlorobenzene (HCB), cord blood lead (Pb), and maternal hair mercury (Hg).

184 metals in infants, the observed increases in cord blood lead and cadmium require further exploration.

185 ernal lead and with significant increases in cord blood lead and cadmium.

186 00, 16,800, 28,000 IU) vs. placebo, neonatal cord blood lead levels were 8.5% (95% CI: - 3.5, 22), 16

187 as early as the second trimester with fetal cord blood leptin and stronger association beginning as

188 with 8.8% (95% CI, -14.1% to -3.1%) shorter cord blood leukocyte telomeres and 13.2% (95% CI, -19.3%

189 Cord blood levels of free triiodothyronine (FT3), FT4, T

190 Overall, there was no correlation between cord blood levels of hydroxychloroquine and the neonatal

191 We examined maternal and neonatal cord blood levels of lead, cadmium, manganese, and mercu

192 With adjustment for potential confounders, cord blood log(FT3)pmol/L concentration was 0.11 lower i

193 hat lympho-myeloid progenitor populations in cord blood - lymphoid-primed multi-potential progenitors

194 The increased formate concentrations in cord blood may support the increased activity of one-car

195 tivation and chemokine receptor switching in cord blood mDC, RSV did not.

196 We collected cord blood, measured antibodies to Plasmodium falciparum

197 lead, and manganese on cognitive score when cord blood metals concentrations were all above the 60th

198 We examined cord blood micronutrient biomarkers in relation to anten

199 ania, we evaluated the presence and level of cord blood MMc in offspring of women with and without PM

200 ith motor improvement, after human umbilical cord blood mononuclear cell (hUCBC) infusion.

201 Cord blood mononuclear cell-derived IL-1beta levels were

202 very Assay chip to survey DNA methylation in cord blood mononuclear cells from 36 children (18 nonast

203 uction in CpG methylation of the INS gene in cord blood mononuclear cells from offspring with a susce

204 imulation, and an enhanced IL-10 response of cord blood mononuclear cells to dexamethasone treatment

205 pectrum proinflammatory cytokine response of cord blood mononuclear cells to innate and mitogenic sti

206 ood mononuclear cells and neonatal umbilical cord blood mononuclear cells were collected and cryopres

207 y was to determine the mRNA transcriptome of cord blood mononuclear leukocytes from term neonates to

208 n Rural Environments (PASTURE) birth cohort (cord blood [n = 836], 1 year [n = 734], 4.5 years [n = 7

209 Amniotic fluid, cord blood, neonatal throat swab, and breastmilk samples

210 including haploidentical NK cells, umbilical cord blood NK cells, stem cell-derived NK cells, NK cell

211 epigenome-wide DNA methylation in umbilical cord blood nucleated cells in Project Viva, a prospectiv

212 explored sex-specific DNA methylation in the cord blood of 39 females and 32 males born at term and w

213 (LOI) of IGF2 and H19 genes in placentas and cord blood of 90 mother-child dyads in association with

214 cing PLZF+ CD4+ T cells were enriched in the cord blood of infants with gastroschisis, a natural mode

215 pecific effector T cells was assessed in the cord blood of infants with localized and systemic inflam

216 experiment 1, Klotho levels were measured in cord blood of preterm infants who were enrolled in a lon

217 ose-associated methylation signatures in the cord blood of the infant appeared to be attenuated by th

218 nsplant from a HLA-mismatched donor and from cord blood, older age, and duration of severe neutropeni

219 s for detection of multiple viruses included cord blood or HLA-mismatched HCT, myeloablative conditio

220 Other BMT options include unrelated cord blood or mismatched family donors.

221 uch as from the bone marrow, human umbilical cord blood, or skin in vitro.

222 nts, and greater IgG compared with unrelated cord blood (P = .005).

223 vitro generation of red cells from adult and cord blood progenitors do not yet provide a sustainable

224 source (n = 839; 84%); peripheral blood and cord blood progenitors were used in 73 (7%) and 88 (9%)

225 uring each trimester of pregnancy and in the cord blood, providing unambiguous assurance of drug expo

226 In cord blood, qPCR, TESA-blot, and micromethod sensitiviti

227 excellent regardless of donor type, even in cord blood recipients (90%).

228 llergy-promoting IL-4Ralpha mutation; and hu cord blood-reconstituted immunodeficient, hu cytokine-se

229 ed huFcepsilonRIalpha mice, as well as human cord blood-reconstituted reNSGS mice, which are immune-d

230 , adjusting for estimated cell count using a cord blood reference, sample plate, maternal sociodemogr

231 Cord blood regulatory T-cell populations were measured a

232 e ranges (IQR)] of manganese in maternal and cord blood, respectively, were 24.0 (19.5-29.7) and 43.1

233 118-239 maternal blood specimens and 100-201 cord blood samples analyzed.

234 so identified shared clonotypes in umbilical cord blood samples and all adult repertoires.

235 ferences in TRB diversity between HEU and HU cord blood samples and provides evidence that maternal H

236 ecific gene expression of IGF2, but 32.4% of cord blood samples displayed LOI of IGF2 and 10.8% showe

237 y (12-16 wk), at delivery (37-42 wk), and in cord blood samples from 215 mothers, of a prospective co

238 trimesters of pregnancy by using a subset of cord blood samples from a randomized, double-blind, plac

239 cell receptor beta chain (TRB) expressed in cord blood samples from HEU infants was determined using

240 Cord blood samples from neonates born to mothers supplem

241 formate concentration was ~60% higher in the cord blood samples than in mothers' plasma.

242 ng was performed with a subset of placentas, cord blood samples, and buccal samples collected during

243 esting myeloid cells and CD4(+) T cells from cord blood samples, as well as in response to lipopolysa

244 ed allergens in mothers during pregnancy, in cord blood samples, in breast milk, and in infants in th

245 ith PON1 expression in an independent set of cord blood samples.

246 n the mother's plasma and breast milk and in cord blood seemed to protect against allergic sensitizat

247 chain reaction in leukocytes extracted from cord blood shortly after birth.

248 ntribute to inferior platelet recovery after cord blood stem cell transplantation and may underlie in

249 blood-derived stem cells (2; 4%), umbilical cord blood stem cells (2; 4%), allogenic bone marrow-der

250 fter they are engrafted with human umbilical cord blood stem cells.

251 show that naive-like MAIT cells in umbilical cord blood switch to a central/effector memory-like prof

252 Also, we found that cord blood T cells, mainly CD4(+) T cells, obtained from

253 o 4-fold higher levels of circulating EDA in cord blood than in adult sera.

254 tem and progenitor cells isolated from human cord blood that may better recapitulate the behavior of

255 ve on data obtained from both heel prick and cord blood, the latter being a more feasible option in l

256 as heterozygous for H19 and 37 placentas and cord blood tissues heterozygous for IGF2 and H19 methyla

257 smatched anti-CD19 CAR-NK cells derived from cord blood to 11 patients with relapsed or refractory CD

258 reactivity is favored after double umbilical cord blood transplantation (dUCBT) in which cord blood (

259 atching who received a single unit umbilical cord blood transplantation for non-malignant diseases re

260 n of clinically effective therapies, such as cord blood transplantation, has been impeded because of

261 In cord blood transplantation, optimal unit selection requi

262 ion for non-malignant diseases-for umbilical cord blood transplantation.

263 immune response on outcomes after unrelated cord blood transplantations (CBTs).

264 iving combination haploidentical single-unit cord blood transplants, we have added 4 Gy TBI to the wi

265 cg09093485, cg09637273) were associated with cord blood triglycerides and birth weight, respectively

266 rtoire is private and initially unfocused in cord blood, typically becoming strongly focused on a few

267 stage acute leukemia, and received umbilical cord blood (UCB) allografts.

268 rand breaks, PFG, and apoptosis in umbilical cord blood (UCB) cells including CD34+ hematopoietic ste

269 tem and progenitor cells within an umbilical cord blood (UCB) graft shortens the time to hematopoieti

270 Umbilical cord blood (UCB) has had considerable impact in pediatri

271 Umbilical cord blood (UCB) is a promising source of stem cells to

272 ge as to whether FLG expression in umbilical cord blood (UCB) is associated with eczema development a

273 We recently reported that umbilical cord blood (UCB) monocytes from babies born to obese mot

274 cord blood transplantation (dUCBT) in which cord blood (UCB) units and patients are often HLA class

275 We evaluated the impact of recipient and cord blood unit (CBU) genetic polymorphisms related to i

276 erformed not in complete remission or from a cord-blood unit, a relapse after transplant, or chronic

277 e standard for selecting unrelated umbilical cord blood units for transplantation for non-malignant d

278 ice in that selection of unrelated umbilical cord blood units for transplantation for non-malignant d

279 g algorithms that provide lists of donor and cord blood units ordered in terms of the likelihood of a

280 or optimal selection of unrelated donors and cord blood units.

281 t (BBC), we assessed the association between cord blood unmetabolized folic acid (UMFA), 5-methyl tet

282 Metals were measured in cord blood using inductively coupled plasma-mass spectro

283 el to estimate proportions of eosinophils in cord blood, using the cord blood DNA methylation profile

284 ia (CMH, 8% O(2)) on the integrity of spinal cord blood vessels and the responses of neighboring glia

285 Spinal cord blood vessels of CMH-treated mice showed reduced ex

286 triggered transient vascular leak in spinal cord blood vessels, particularly in white matter, which

287 y enhancing the barrier properties of spinal cord blood vessels.

288 race, mode of delivery, breast-feeding, and cord blood vitamin D levels are associated with infant g

289 Cord blood vitamin D was linked to increased Lachnobacte

290 DNA methylation in umbilical cord blood was associated with maternal serum PFAS conce

291 RSV-A IC80 titers in cord blood were associated with decreased episodes of pn

292 Placenta, venous and arterial cord blood were collected obtaining a wide range of FA e

293 Additionally, BPA and TNF-alpha levels in cord blood were inversely associated with maternal and G

294 stem cell transplantation, frozen samples of cord blood were thawed and the purity of viable nucleate

295 of samples (93.2% of heel prick and 89.9% of cord blood) were collected from term infants.

296 s included measurement of DNA methylation in cord blood, where the functional significance of such ch

297 Cord blood, which has high levels of hemoglobin, iron an

298 It was not possible to estimate FA FSR in cord blood with this oral bolus dose approach.

299 ed in human serum as well as in maternal and cord blood within 30s.

300 e bone marrow, adipose tissue, and umbilical cord blood without altering their ex vivo characteristic