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1 cells and corneal epithelial cells regulates corneal angiogenesis.
2 F-induced (5%-57%) and VEGF-induced (3%-66%) corneal angiogenesis.
3 ts as a broad-spectrum negative regulator of corneal angiogenesis.
4 lly responsible for suppressing inflammatory corneal angiogenesis.
5 migration are initial events in PAF-promoted corneal angiogenesis.
6 the lack of TSP-1,2 and both TSPs on induced-corneal angiogenesis.
7 s TSP-1 and/or -2 resulted in no spontaneous corneal angiogenesis.
8 sly implanted Matrigel plugs, as well as rat corneal angiogenesis.
9 ration, and fibroblast growth factor-induced corneal angiogenesis.
10 , and basic fibroblast growth factor-induced corneal angiogenesis.
11 tic ring assay and a model of suture-induced corneal angiogenesis.
12  the function of PD-L1 in Treg modulation of corneal angiogenesis.
13 by Tregs plays a pivotal role in suppressing corneal angiogenesis, acting through a contact-dependent
14 ctival injection of the morpholino-inhibited corneal angiogenesis and lymphangiogenesis, and suppress
15 th a selective inhibitor was shown to reduce corneal angiogenesis and SK severity.
16    We show that FOXC1 mutations also lead to corneal angiogenesis, and that mice homozygous for eithe
17            The molecules involved in causing corneal angiogenesis are multiple and include the vascul
18                    Endothelial migration and corneal angiogenesis are similarly inhibited by a TXA2 r
19 cularization, as well as models with induced corneal angiogenesis, are widely used to investigate the
20                               An in vivo rat corneal angiogenesis assay demonstrated neo-vessel forma
21                                            A corneal angiogenesis assay was performed by implanting n
22 ll migration and tube formation, and in vivo corneal angiogenesis assays, HRGP inhibited the antiangi
23 clude that activin A stimulates inflammatory corneal angiogenesis by increasing VEGF levels through a
24 exposure of the eyes to radiation suppressed corneal angiogenesis comparably to whole-body exposure.
25 tion of the following in injured corneas: i) corneal angiogenesis; ii) corneal fibrosis; iii) infiltr
26                                 IL-6 induced corneal angiogenesis in a dose-dependent manner, with 35
27 ds and Eph receptors in vitro and in vivo in corneal angiogenesis in a mouse model.
28 e compound necessary to inhibit VEGF-induced corneal angiogenesis in BALB/c mice.
29          Analyses of the relative extents of corneal angiogenesis in these same animals, induced by b
30  aortic ring assay and promoted bFGF-induced corneal angiogenesis in vivo in a corneal pocket assay.
31 lary endothelial cell migration in vitro and corneal angiogenesis in vivo, and apparently act via bin
32 ular endothelial cells in vitro and inhibits corneal angiogenesis in vivo.
33 ar network formation in vitro and stimulates corneal angiogenesis in vivo.
34 bits endothelial cell migration in vitro and corneal angiogenesis in vivo.
35 t loci (QTLs), which influence the extent of corneal angiogenesis induced by vascular endothelial gro
36  topical and systemic cyclosporin A (CsA) on corneal angiogenesis induced by xenotransplantation or b
37 Understanding the roles of Eph and ephrin in corneal angiogenesis may provide a therapeutic intervent
38 nhibition observed at 100 mg/kg) and the rat corneal angiogenesis model (ED(50) = 16.3 mg/kg).
39 and this finding was confirmed by an in vivo corneal angiogenesis model and an ex vivo aortic ring as
40                          In a suture-induced corneal angiogenesis model, we observed a significantly
41 to vascular endothelial growth factor in the corneal angiogenesis model.
42  and lymphangiogenesis in the suture-induced corneal angiogenesis model.
43 nt for the increase in inflammation-mediated corneal angiogenesis observed in sdc1(-/-) mice.
44 SP-2, helps to suppress inflammation-induced corneal angiogenesis postnatally, implying that angiogen
45               In vivo, in the mouse model of corneal angiogenesis, Shh is expressed by MCs of newly f
46 st phase III study on a topical inhibitor of corneal angiogenesis showed that aganirsen eye drops sig
47  all TSP(-/-) mice had significantly greater corneal angiogenesis than those of control mice (P < 0.0
48 ings identify a role for FoxC1 in inhibiting corneal angiogenesis, thereby maintaining corneal transp
49                                              Corneal angiogenesis was provoked by either xenotranspla
50 r supporting a role for MMP-9 in HSV-induced corneal angiogenesis was the observation that inhibition
51  area and density and extent of bFGF-induced corneal angiogenesis were determined in C57BL/6J, BALB/c
52 dependent reduction in growth factor-induced corneal angiogenesis, while PEG sTNFRI did not.
53                               By stimulating corneal angiogenesis with an alkali burn in Tie2-GFP flu