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1  differentiation of the neural crest derived corneal endothelial cell.
2 g-term viability of the neural crest derived corneal endothelial cells.
3 sease involving metaplasia and overgrowth of corneal endothelial cells.
4 lts in replication in nonproliferating human corneal endothelial cells.
5 /gld mice did not induce apoptosis of BALB/c corneal endothelial cells.
6 ike coupling mechanisms play roles in CCE in corneal endothelial cells.
7 taken to investigate the mechanism of CCE in corneal endothelial cells.
8        AQP1 does not contribute to PCO(2) in corneal endothelial cells.
9 hesion molecule, N-cadherin, is expressed by corneal endothelial cells.
10 soforms expressed in primary cultures of rat corneal endothelial cells.
11 ial and possibly other cellular functions in corneal endothelial cells.
12 these cells often fail to differentiate into corneal endothelial cells.
13 a, -eta, -gamma, and -theta were detected in corneal endothelial cells.
14 n important role in Fas ligand expression in corneal endothelial cells.
15 us humor did not promote apoptosis of murine corneal endothelial cells.
16  aqueous humor suppress S-phase entry of rat corneal endothelial cells.
17 n aqueous humor on DNA synthesis in cultured corneal endothelial cells.
18 n corneal epithelial cells, keratocytes, and corneal endothelial cells.
19 tein which inhibits the NK-mediated lysis of corneal endothelial cells.
20 ittle ALDH3 is present in the keratocytes or corneal endothelial cells.
21 y conjunctiva, corneal epithelial cells, and corneal endothelial cells.
22 rbol esters on the function and structure of corneal endothelial cells.
23 port the adhesion and proliferation of human corneal endothelial cells.
24 in or a matrix elaborated by cultured bovine corneal endothelial cells.
25 cell proliferation and cell shape changes in corneal endothelial cells.
26 ized FN and to cultured monolayers of bovine corneal endothelial cells.
27 only a potent mitogen, it is a modulator for corneal endothelial cells.
28 glandin E2 synthesis is increased in injured corneal endothelial cells.
29 ranscriptional control of type I collagen in corneal endothelial cells.
30 ressively impairs vision through the loss of corneal endothelial cells.
31  is a H(+)/NH(3)/water transport protein, of corneal endothelial cells.
32  a highly effective, novel carrier for human corneal endothelial cells.
33  to overcome is the transport and storage of corneal endothelial cells.
34 r integrity in monolayers of cultured bovine corneal endothelial cells.
35 P) was used to transduce cryopreserved human corneal endothelial cells.
36 oxic to cryopreserved human primary cultured corneal endothelial cells.
37 t is undetectable on stromal fibroblasts and corneal endothelial cells.
38                                        Human corneal endothelial cells and Descemet's membrane (HCEC-
39 compensation by replacing the malfunctioning corneal endothelial cells and Descemet's membrane.
40 alpha 2(I) collagen RNA obtained from normal corneal endothelial cells and from modulated corneal end
41 ed for overexpression of E2F2 in transfected corneal endothelial cells and permitted calculation of t
42 ggest that type I collagen is synthesized in corneal endothelial cells and that such undesired expres
43   Two forms of cyclooxygenase are present in corneal endothelial cells, and pharmacologic studies ind
44                       Limbal basal cells and corneal endothelial cells appear to be inhibited in the
45                                              Corneal endothelial cells are arranged as a monolayer on
46 It is possible that in situ human and rabbit corneal endothelial cells are arrested at different poin
47                                        Since corneal endothelial cells are crucial for maintaining co
48                                        Human corneal endothelial cells are derived from neural crest
49                                          The corneal endothelial cells are enlarged and reduced in nu
50                                  Adult human corneal endothelial cells are G1-arrested, even in respo
51                             In neonatal rat, corneal endothelial cells are still entering the cell cy
52       Cryopreserved, primary, cultured human corneal endothelial cells are viable and retain their ab
53 not induce acute significant cytotoxicity in corneal endothelial cells at concentrations up to 100 mi
54 lated onto the matrices elaborated by bovine corneal endothelial cells attached to the culture dish a
55 ssion levels in confluent cultures of bovine corneal endothelial cells (BCECs) affects membrane PCO(2
56 ence of Ca(2+) stores in lysosomes of bovine corneal endothelial cells (BCECs) and examines their int
57 inhibition of CA activity in cultured bovine corneal endothelial cells (BCECs) by dorzolamide, a topi
58 LC4A11 expression in cultured primary bovine corneal endothelial cells (BCECs) was determined by semi
59 xperiments were performed in cultured bovine corneal endothelial cells (BCECs).
60 ymal transition and preserve the function of corneal endothelial cells both during ex vivo culture an
61 sage (<4) stromal cells and to low levels in corneal endothelial cells but not in corneal epithelial
62 ness of cryopreserved human primary cultured corneal endothelial cells by characterizing their morpho
63  that WNT10B promotes proliferation in human corneal endothelial cells by simultaneously utilizing bo
64 dy we investigated whether tissue engineered corneal endothelial cells can be preserved in hypothermi
65                     Regardless of donor age, corneal endothelial cells can enter and complete the cel
66                              Cultured bovine corneal endothelial cells (CBCECs) were grown to conflue
67  water channel protein(s) in cultured bovine corneal endothelial cells (CBCECs).
68                            We report data on corneal endothelial cell (CEC) count, peripapillary reti
69                                      In vivo corneal endothelial cell (CEC) counts and morphology wer
70       We sought to assess the correlation of corneal endothelial cell (CEC) density to alterations of
71 e current study, we performed in vivo rabbit corneal endothelial cell (CEC) injury via CEC scraping,
72 ated cells with pseudopodia is observed when corneal endothelial cells (CECs) are simultaneously trea
73                                              Corneal endothelial cells (CECs) are terminally differen
74 mation (EMT), in which the contact-inhibited corneal endothelial cells (CECs) become multilayers of s
75 ation, there is an initial, rapid decline in corneal endothelial cells (CECs) following surgery.
76                  However, Wnt5a signaling in corneal endothelial cells (CECs) has not been well chara
77 othelial corneal dystrophy (FECD) and normal corneal endothelial cells (CECs) to oxidative stress, an
78                                              Corneal endothelial cells (CECs) treated with FGF-2 from
79 elial mesenchymal transformation observed in corneal endothelial cells (CECs).
80          FGF-2 is a potent mitogen of rabbit corneal endothelial cells (CECs).
81 ollagen was posttranslationally regulated in corneal endothelial cells (CECs).
82 which is a morphogen and a potent mitogen of corneal endothelial cells (CECs).
83 growth factors for primary cultures of human corneal endothelial cells (CECs).
84 iferative capacity (premature senescence) of corneal endothelial cells (CECs).
85 othelial corneal dystrophy (FECD) and normal corneal endothelial cells (CECs).
86 cilia that subsequently disassemble in adult corneal endothelial cells (CECs).
87                                EDTA released corneal endothelial cell-cell contacts in a dose- and ti
88 for the establishment and expansion of human corneal endothelial cells (CEnC) has provided a source o
89                 The functional competence of corneal endothelial cells (CEnCs) is critical for surviv
90                 Here, isolated primary human corneal endothelial cells (CEnCs) propagated using a dua
91                                              Corneal endothelial cells (CEnCs) regulate corneal hydra
92 plantation mainly relies on the integrity of corneal endothelial cells (CEnCs), which maintain tissue
93 owed by ciliary body, lens epithelial cells, corneal endothelial cells, conjunctiva, retina, and corn
94                                        Human corneal endothelial cells could be reliably scored for t
95 heral anterior synechiae, visual acuity, and corneal endothelial cell count (CECC).
96 atients who were unsuitable for FLACS, whose corneal endothelial cell count (ECC) was <1,500 cells/mm
97                                     The mean corneal endothelial cell count (ECD) values were 2692.82
98  0.05; 95% CI, -0.01 to 0.12; P = 0.10), and corneal endothelial cell count (WMD, 73.39; 95% CI, -6.2
99 ly received PIOL implantation and have a low corneal endothelial cell count.
100 rm safety, particularly in eyes with reduced corneal endothelial cell counts from prior intraocular s
101                                              Corneal endothelial cell counts were lower in eyes that
102                       The corneal thickness, corneal endothelial cell counts, specular microscopy par
103 pare preoperative and postoperative BCVA and corneal endothelial cell counts.
104 enhanced the proliferative response of human corneal endothelial cell cultures to endothelial cell gr
105 el that utilizes ultraviolet light to induce corneal endothelial cell damage resulting in decreased C
106 l to modulate alloimmunity and/or to prevent corneal endothelial cell death.
107 talloproteinase activity in confluent bovine corneal endothelial cells decreased the level of endothe
108  diabetic rats demonstrated cataracts, lower corneal endothelial cell densities, altered aqueous meta
109  to measure central corneal thickness (CCT), corneal endothelial cell density (CECD) along with cell
110 emergent adverse events (TEAEs), and central corneal endothelial cell density (CECD) were evaluated t
111 reatment-emergent adverse events (TEAEs) and corneal endothelial cell density (CECD).
112                                   Changes in corneal endothelial cell density (ECD) and central corne
113                                              Corneal endothelial cell density (ECD) loss after glauco
114 n the subjects who underwent trabeculectomy, corneal endothelial cell density (ECD) significantly dec
115 l endothelial cell density and correlates of corneal endothelial cell density among adults attending
116              To assess the prevalence of low corneal endothelial cell density and correlates of corne
117                     In this study we compare corneal endothelial cell density and morphometry measure
118            Mean (+/-SE) percentage change in corneal endothelial cell density from baseline at month
119                               Measurement of corneal endothelial cell density is important both for c
120                                          The corneal endothelial cell density was 2400 cells/mm(2) in
121                       Cataract formation and corneal endothelial cell density were assessed using mic
122  segment, including crystalline lens health, corneal endothelial cell density, aqueous humor metaboli
123                                              Corneal endothelial cell density, endothelial cell viabi
124                                     When the corneal endothelial cells density (ECD) drops, the HCEC
125                                        Human corneal endothelial cell-Descemet's membrane (HCEC-DM) c
126  grafts did not recover clarity in vivo, and corneal endothelial cells did not proliferate in organ c
127                 Type I collagen synthesis in corneal endothelial cells does not correlate with steady
128 ly of bicarbonate transporters, give rise to corneal endothelial cell dystrophies.
129                                              Corneal endothelial cells express IRK1 (Kir2.1) inwardly
130        All corneal and limbal epithelial and corneal endothelial cells express protein kinases, p33cd
131                        That limbal basal and corneal endothelial cells express receptors for TGF-beta
132 ct intraocular tumors, 5) CD4(+) T cells and corneal endothelial cells express TRAIL and induce apopt
133                       Limbal basal cells and corneal endothelial cells expressed mRNA and protein for
134                      Both CD4(+) T cells and corneal endothelial cells expressed TRAIL and induced ap
135 reatments for reversible blindness caused by corneal endothelial cell failure involve replacing the f
136                                              Corneal endothelial cells from both mice and humans disp
137                  These results indicate that corneal endothelial cells from both the central and peri
138                                              Corneal endothelial cells from FECD patients harbor a po
139 late a significant proliferative response in corneal endothelial cells from old individuals.
140 stance (rho) was measured in cultured bovine corneal endothelial cells grown on permeable substrates
141             The propagation and expansion of corneal endothelial cells has been widely reported.
142   The use of substratum elaborated by bovine corneal endothelial cells has proved useful in the prepa
143 ction, no definitive effects of TNF alpha on corneal endothelial cells have been reported.
144                                        Human corneal endothelial cells have long telomeres throughout
145 es of SiNPs (50, 100, and 150 nm) in a human corneal endothelial cell (HCEC) line, B4G12.
146                                        Human corneal endothelial cell (HCEC) proliferation is control
147  (SB) on the morphology and density of human corneal endothelial cells (HCEC).
148 ative DNA damage increases with age in human corneal endothelial cells (HCECs) and contributes to the
149                                        Human corneal endothelial cells (HCECs) are considered to be n
150                                        Human corneal endothelial cells (HCECs) have limited prolifera
151 e of intracameral injection of MXF for human corneal endothelial cells (HCECs) is still debatable.
152 lation, preservation, and expansion of human corneal endothelial cells (HCECs).
153 ression vector in human keratocytes (HK) and corneal endothelial cells (HCEnC).
154                                        Human corneal endothelial cells (HCEnCs) are responsible for m
155                                        Human corneal endothelial cells (HCEnCs) are terminally differ
156 CD), mitochondrial and oxidative stresses in corneal endothelial cells (HCEnCs) contribute to cell de
157  and COL4A3 mRNA expression in primary human corneal endothelial cells (HCEnCs) was assayed in both P
158                                        Human corneal endothelial cells (HCEnCs) were exposed to vario
159  corneal fibroblasts, and immortalized human corneal endothelial cells (HCEnCs), and in vivo intralam
160                           In media for human corneal endothelial cells, immature cells differentiated
161 ase (MAPK) signaling pathway was examined in corneal endothelial cells in 1 patient.
162 correlates with areas of accentuated loss of corneal endothelial cells in advanced Fuchs endothelial
163 microscopy showed moderate to severe loss of corneal endothelial cells in all patients.
164                          Accelerated loss of corneal endothelial cells in Fuchs' dystrophy is multifa
165                                              Corneal endothelial cells in humans do not replicate to
166 compare the morphological characteristics of corneal endothelial cells in type 2 diabetic patients an
167 specific DTH and induced apoptosis of BALB/c corneal endothelial cells in vitro.
168 roteins in donor corneas suggests that human corneal endothelial cells in vivo have not exited the ce
169  the relative proliferative status of rabbit corneal endothelial cells in vivo.
170 ry mechanism of matrix protein production by corneal endothelial cells in which miR-199B hypermethyla
171  in G(0)-phase synchronized subconfluent rat corneal endothelial cells incubated for 24 hours in 10%
172 ption factor E2F2 in nonproliferating rabbit corneal endothelial cells induces cell cycle progression
173 emove corneal epithelial cells, conjunctiva, corneal endothelial cells, iris, ciliary body, lens epit
174                   Homeostatic maintenance of corneal endothelial cells is essential for maintenance o
175 we show that gene transfer to nonreplicating corneal endothelial cells is feasible using recombinant
176 PK signaling pathway was demonstrated in the corneal endothelial cells isolated from the NF-1 eyes.
177 humor suppress S-phase entry in cultured rat corneal endothelial cells, it is not known whether TGF-b
178                                              Corneal endothelial cell layer analysis was performed wi
179 nd binding of RAC1 and beta-catenin in human corneal endothelial cells, leading to the activation of
180 n the eye, we employed an immortalized mouse corneal endothelial cell line (C3H3) that constitutively
181 the effects of alpha-MSH on a cultured human corneal endothelial cell line (HCEnC-21T) exposed to hyd
182                                      A human corneal endothelial cell line and primary human corneal
183  studies, immortalized normal and FECD human corneal endothelial cell lines (HCECi and FECDi, respect
184                     Cataract surgery induces corneal endothelial cell loss (ECL).
185 t surgery is known to lead to some degree of corneal endothelial cell loss (ECL).
186 ent studies have demonstrated a reduction in corneal endothelial cell loss after phacoemulsification
187     No difference was found in postoperative corneal endothelial cell loss between groups.
188 operative and perioperative risk factors for corneal endothelial cell loss during cataract surgery, a
189 re and describes in detail how the degree of corneal endothelial cell loss is influenced by specific
190                                              Corneal endothelial cell loss remains a well known, unde
191 erative and postoperative complications, and corneal endothelial cell loss were assessed at 6 months
192               Although clinical outcomes and corneal endothelial cell loss were similar in both group
193 al and inflammatory TEAEs of interest (e.g., corneal endothelial cell loss, iritis) was higher with b
194 ons of 30% and 33.3% over one year with mild corneal endothelial cell loss, which is consistent with
195 rative MMC was not associated with long-term corneal endothelial cell loss.
196  to elucidate the factors that predispose to corneal endothelial cell loss.
197 utive expression of MHC class I molecules on corneal endothelial cells makes them potential targets f
198                 No significant disruption to corneal endothelial cell morphologic features, increased
199 luorophotometry, [protein]Ac by Lowry assay, corneal endothelial cell morphology by specular micropho
200           Ex vivo culture or regeneration of corneal endothelial cells often is subjected to gradual
201  the coculture of lens epithelial cells with corneal endothelial cells on the proliferation of rabbit
202 h either Ia- spleen cells, Ia+ spleen cells, corneal endothelial cells, or corneal epithelial cells f
203                                              Corneal endothelial cell origin was confirmed by morphol
204                                        Human corneal endothelial cells plated onto the matrices elabo
205 ally with regard to decreased keratocyte and corneal endothelial cell populations noted months to yea
206 tly knocked down mutant COL8A2 expression in corneal endothelial cells, prevented endothelial cell lo
207 ous studies from this laboratory showed that corneal endothelial cell proliferation ceases in neonata
208                                              Corneal endothelial cell proliferation under our culture
209                                              Corneal endothelial cell proliferation under our culture
210 ermine the role of protein kinase C (PKC) in corneal endothelial cell proliferation.
211 -epsilon activity, is important in promoting corneal endothelial cell proliferation.
212 GF-2 stimulates cell proliferation of rabbit corneal endothelial cells (rCECs) by degrading the cycli
213 .37; 95% CI, -11.88 to -0.86; P = 0.02), and corneal endothelial cell reduction (WMD, -55.43; 95% CI,
214                 Descemet's membrane supports corneal endothelial cell regeneration in rabbits after e
215 ranscription factor E2F2 in nonmitotic human corneal endothelial cells results in short-term expressi
216  AC priming with Ia- NZB spleen cells or NZB corneal endothelial cells results in the permanent accep
217                       Human primary cultured corneal endothelial cells retain their phenotypic proper
218 neal endothelial cell line and primary human corneal endothelial cells retained their characteristic
219                                              Corneal endothelial cells showed intense labeling for ra
220 arkedly increased the tolerizing activity of corneal endothelial cells, so that a single dose of chol
221 , low mitogenic culture conditions preserved corneal endothelial cell state identity better than cult
222 atinocyte growth factors, and coculture with corneal endothelial cells stimulated proliferation of ra
223                      Evidence indicates that corneal endothelial cells synthesize mRNA for TGF-beta1
224 ing for the RIII protein was more intense in corneal endothelial cells than in limbal basal cells.
225 ive neurons and into p75(NTR)-positive human corneal endothelial cells that exhibited transendothelia
226 demonstrated that EMAP is a novel protein in corneal endothelial cells that is capable of inducing pr
227 corneal endothelial cells and from modulated corneal endothelial cells that predominantly produce typ
228                          The capacity of pig corneal endothelial cells to proliferate in vivo was ass
229                                        Human corneal endothelial cells, transformed with human papill
230 DM) helps maintain phenotype and function of corneal endothelial cells under physiological conditions
231 we found that during ex vivo culture, bovine corneal endothelial cells underwent endothelial-mesenchy
232 antagonist, vMIP II, was introduced into the corneal endothelial cells using a non-viral vector consi
233 examined the feasibility of gene transfer to corneal endothelial cells using replication-defective re
234 hether Alamar blue could be used to evaluate corneal endothelial cell viability in vitro.
235 ton fraction of the normal and the modulated corneal endothelial cells was immunoprecipitated with PL
236                          Apoptosis of BALB/c corneal endothelial cells was mediated by double negativ
237 lagen RNA structures of normal and modulated corneal endothelial cells were analyzed by S1 nuclease p
238                                       Bovine corneal endothelial cells were cultured to confluence.
239        Human corneal Descemet's membrane and corneal endothelial cells were digested with collagenase
240                   Primary cultures of bovine corneal endothelial cells were established on membrane-p
241                                          Rat corneal endothelial cells were grown in explant culture
242                                              Corneal endothelial cells were morphologically normal; h
243                                        Human corneal endothelial cells were plated onto tissue cultur
244                                    Images of corneal endothelial cells were recorded by using specula
245 oliferation studies, primary cultures of rat corneal endothelial cells were serum-starved for 48 hour
246                                     Infected corneal endothelial cells were swollen and partly detach
247    We observed LOXHD1 mRNA in cultured human corneal endothelial cells, whereas antibody staining of
248 he anterior chamber of the eye is lined with corneal endothelial cells, which are terminally differen
249                                              Corneal endothelial cells, which line the back of the co
250 eta3 induced myofibroblast transformation of corneal endothelial cells, with formation of stress fibe
251 as found to be almost entirely restricted to corneal endothelial cells, with scattered expression in

 
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