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1 use, but failed to infect productively human corneal epithelial cells.
2  EGF-induced migration of immortalized human corneal epithelial cells.
3 ed in bactericidal lysate fractions of human corneal epithelial cells.
4 n/AP-1 through a novel Plk3 pathway in human corneal epithelial cells.
5  Pax6 functions directly to maintain normal, corneal epithelial cells.
6 vation of the JNK signaling pathway in human corneal epithelial cells.
7 osis through the activation of Plk3 in human corneal epithelial cells.
8 alized the cytolytic effects of PLY on human corneal epithelial cells.
9 red to MS1 cells co-cultured with PD-L1(-/-) corneal epithelial cells.
10 rogression of healing of wounds in sheets of corneal epithelial cells.
11 f syndecan-1 ectodomains from the surface of corneal epithelial cells.
12 rease in ROS-positive cells in primary human corneal epithelial cells.
13 elationships that develop between nerves and corneal epithelial cells.
14 s regulated during cell cycle progression in corneal epithelial cells.
15 nse to pathophysiological challenge in human corneal epithelial cells.
16 d corneas and proliferation rate in cultured corneal epithelial cells.
17  and amplified HO-1 gene expression in human corneal epithelial cells.
18  concomitant MLC dephosphorylation in bovine corneal epithelial cells.
19 omotion were characterized in cultured human corneal epithelial cells.
20 stellanii trophozoites to adhere to and kill corneal epithelial cells.
21 ovine peripheral blood mononuclear cells and corneal epithelial cells.
22 t (UVB) is known to cause apoptosis in human corneal epithelial cells.
23 activation in UVB-induced apoptosis in human corneal epithelial cells.
24 ation with, invasion of, and survival within corneal epithelial cells.
25  [3H]thymidine uptake in primary cultures of corneal epithelial cells.
26 sfect guinea pig liver transglutaminase into corneal epithelial cells.
27  is selectively expressed in differentiating corneal epithelial cells.
28 e corneal epithelium in vivo and in cultured corneal epithelial cells.
29 d lipid raft formation in occasional surface corneal epithelial cells.
30 nd typical enhancers active in primary human corneal epithelial cells.
31 enic mouse models and cultured primary human corneal epithelial cells.
32 e a useful additive for ex vivo expansion of corneal epithelial cells.
33 ced in keratinocytes and to a lower level in corneal epithelial cells.
34 anical injury (debridement), and in vitro in corneal epithelial cells.
35 lation were corroborated using primary human corneal epithelial cells.
36 alt and water balance and normal function of corneal epithelial cells.
37 r p15(INK4b) has been localized in migrating corneal epithelial cells.
38 HGF modulates multiple signaling cascades in corneal epithelial cells.
39 ly processed into antimicrobial fragments in corneal epithelial cells.
40 tion of barrier function in stratified human corneal epithelial cells.
41 migrated centripetally to differentiate into corneal epithelial cells.
42 scription complex, resulting in apoptosis of corneal epithelial cells.
43 ibody and NF-kB inhibitor in mouse and human corneal epithelial cells.
44 d chemokine CXCL10 and its receptor CXCR3 in corneal epithelial cells.
45 hibitory and oxidant effects of 4-HNE on the corneal epithelial cells.
46 on infected mouse corneas and human cultured corneal epithelial cells.
47 ells and proliferation of subconfluent human corneal epithelial cells.
48 in primary and telomerase-immortalized human corneal epithelial cells.
49  N-glycan structures in differentiated human corneal epithelial cells.
50 icated that the NC-1059 peptide did not kill corneal epithelial cells.
51                             In human primary corneal epithelial cells, 24,25(OH)2D3 stimulated migrat
52 in (MBP) mediates adhesion of the amoebae to corneal epithelial cells, a key first step in the pathog
53 t of such an effect, and it suggests that in corneal epithelial cells, activation of apoptotic pathwa
54 rane by providing biophysical cues to direct corneal epithelial cell adherence and migration.
55 t role in mediating programmed cell death of corneal epithelial cells after UV irradiation.
56 ancer of chemotherapy, effectively protected corneal epithelial cells against apoptosis by its specif
57  in human tear fluid and that it can protect corneal epithelial cells against bacterial invasion.
58 ese results suggest that ALDH3A1 may protect corneal epithelial cells against oxidative damage not on
59                                Human primary corneal epithelial cells and cell line were cultured in
60 Ac resulted in increased adhesion of applied corneal epithelial cells and corneal fibroblasts.
61 rived exosomes mediate communication between corneal epithelial cells and corneal keratocytes as well
62 ILs), toward two different cell lines, human corneal epithelial cells and Escherichia coli bacterial
63 enzyme that is highly expressed in mammalian corneal epithelial cells and has been shown to protect a
64                     Primary human and rabbit corneal epithelial cells and immortalized human corneal
65 wound healing was further evaluated in human corneal epithelial cells and in the corneas of wild-type
66 l epithelial cell types: insulin-insensitive corneal epithelial cells and insulin-sensitive bronchial
67    Water-soluble proteins were isolated from corneal epithelial cells and keratocytes of several spec
68 duced apoptosis of T and B cells, but not of corneal epithelial cells and liver cells.
69 ting TLR4 signaling through IRF3 in resident corneal epithelial cells and macrophages and thereby mod
70 tions of CD4(+) T cells with MHC class II(+) corneal epithelial cells and macrophages in infected DC-
71                    Overexpression of CTCF in corneal epithelial cells and mouse corneas significantly
72     S. aureus-induced cytokine production by corneal epithelial cells and neutrophils was also signif
73 role of CD14 in regulating LPS activation of corneal epithelial cells and Pseudomonas aeruginosa corn
74 he pro-inflammatory interleukin-8 from human corneal epithelial cells and reverses reductions in tran
75 eba interacts with a mannosylated protein on corneal epithelial cells and stimulates trophozoites to
76 bacterial adherence to the apical surface of corneal epithelial cells and suggest that alteration of
77 perties of cell surface-associated mucins in corneal epithelial cells and suggest that alterations in
78 ll-like receptor 4 (TLR4) internalization in corneal epithelial cells and that blocking with anti-CD1
79                              Fewer layers of corneal epithelial cells and the absence of cytokeratin
80 oxicity on infected mouse and cultured human corneal epithelial cells and the role of PVL and antibod
81  exosome-like vesicles were observed between corneal epithelial cells and the stroma during wound hea
82 0% of the water-soluble protein of the mouse corneal epithelial cells and thus, by analogy with the a
83            Pseudomonas aeruginosa can invade corneal epithelial cells and translocates multilayered c
84 ucts were transiently transfected into human corneal epithelial cells, and activity was assessed in r
85 nstitutively expressed at high levels by the corneal epithelial cells, and at low levels by corneal C
86 estigated in cultured porcine corneas, human corneal epithelial cells, and human corneas using Wester
87  Inflammation induces PD-L1 up-regulation by corneal epithelial cells, and infiltration of significan
88  SSEA4 is highly expressed in differentiated corneal epithelial cells, and SSEA4(-) limbal epithelial
89 for transient transduction of primary rabbit corneal epithelial cells, and the effect on cell cycle p
90 un, which appears to be important to mediate corneal epithelial cell apoptosis after UV irradiation.
91 ed JNK (c-Jun N-terminal kinase) cascade and corneal epithelial cell apoptosis and protection against
92 yte growth factor (HGF) in the prevention of corneal epithelial cell apoptosis and to identify signal
93                                              Corneal epithelial cell apoptosis was increased 17-fold
94 sites; CPE assays were performed with rabbit corneal epithelial cells as host cells; and the expressi
95 ere co-cultured with PD-L1-expressing normal corneal epithelial cells, as compared to MS1 cells co-cu
96 e that stratification and differentiation of corneal epithelial cells, as measured by the capacity to
97 nstrate that factors secreted by MMC-treated corneal epithelial cells attenuate collagen deposition b
98 ata show functional roles for AMPs in normal corneal epithelial cell barrier function against P. aeru
99 hosphorylation in primary cultures of bovine corneal epithelial cells (BCECs).
100 amma caused LDH leakage from sensitive human corneal epithelial cells, but even higher doses of TNF-a
101 dlim2 gene was specifically expressed in the corneal epithelial cells, but not in the corneal stroma
102 llowing HPAI H7N7 virus infection in primary corneal epithelial cells, but not respiratory cells, ide
103  correlated with an increase in infection of corneal epithelial cells by P. aeruginosa.
104 ot required for the colonization of cultured corneal epithelial cells by S. aureus, suggesting that s
105                                              Corneal epithelial cells can adhere and proliferate on t
106           Electrotaxis of dissociated bovine corneal epithelial cells (CECs) on planar quartz require
107 re upregulated in response to wounding in NL corneal epithelial cells (CECs), whereas the latter was
108 roliferation and vertical migration of basal corneal epithelial cells (CECs).
109 ll-infection" in vivo models, cultured human corneal epithelial cells, contact lens-wearing animal mo
110 rane mucin N-glycans in differentiated human corneal epithelial cells contain primarily complex-type
111 ly that IFN-gamma-induced MD-2 expression by corneal epithelial cells contributes to the host respons
112 nd after PA exposure and in serum-free human corneal epithelial cell culture (hTCEpi).
113       Expression was studied in normal human corneal epithelial cell cultures and hINV promoter trans
114 orneal epithelium, their expression in human corneal epithelial cell cultures, and the effect of ultr
115 lutaminase enzymatic activity is involved in corneal epithelial cell death after UVB and appears to p
116 sorders, especially blindness as a result of corneal epithelial cell deficiency.
117 giogenesis, and cancer, we hypothesized that corneal epithelial cell-derived exosomes may gain access
118 studying the molecular mechanisms regulating corneal epithelial cell differentiation and desquamation
119 fies a role for KLF7 as a KLF4 antagonist in corneal epithelial cell differentiation, and explains ho
120 ulation of involucrin gene expression during corneal epithelial cell differentiation.
121  in neutrophils and significant increases in corneal epithelial cell division and migration.
122 notion that Pax6 plays a role in controlling corneal epithelial cell dynamics in vivo.
123 ctopic expression of PREX1 in cultured human corneal epithelial cells enhanced their rate of wound he
124                                      Primary corneal epithelial cells, established from donor limbal
125 e maintained on a high-fat diet, or cultured corneal epithelial cells exposed to high glucose, which
126 ation and division, and during wound closure corneal epithelial cells express intercellular adhesion
127                         Additionally, murine corneal epithelial cell expressed the catecholamine-synt
128 est a central role of the WNT7A-PAX6 axis in corneal epithelial cell fate determination, and point to
129            In murine models, LRIG1 regulated corneal epithelial cell fate during wound repair.
130 c-Jun/AP-1 transcriptional complex and human corneal epithelial cell fate.
131 idement wounds and in vitro in primary human corneal epithelial cells following a linear scratch woun
132 ntly, B2088 did not show any cytotoxicity to corneal epithelial cells for at least 96 h continuous ex
133 e surface ectoderm, and prospective lens and corneal epithelial cells formed a multilayered mass of c
134                            Transformed human corneal epithelial cells from a patient homozygous for D
135 reduced proliferation in primary cultures of corneal epithelial cells from AQP3-null mice.
136                                              Corneal epithelial cells from bitransgenic Krt12Cre/+/RO
137       Using FACS analysis, 60% to 70% of the corneal epithelial cells from Krt12Cre/+/ROSAEGFP mice w
138       Microdissection was used to remove the corneal epithelial cells from the button.
139 cratch wound assay using primary cultures of corneal epithelial cells from wild-type versus AQP3-null
140 ation resulted in a complete loss of in vivo corneal epithelial cell gene expression.
141                                              Corneal epithelial cells have large stores of glycogen,
142 4-HNE suppressed the cell viability of human corneal epithelial cells (HCE) in a concentration depend
143                 Primary mouse LGAC and human corneal epithelial cells (HCE-T cells) exposed to interf
144 ty in THCE cells, an SV40-immortalized human corneal epithelial cell (HCEC) line, and primary HCECs w
145 o investigate whether LL-37 stimulates human corneal epithelial cell (HCEC) migration, proliferation,
146 t NaNO2 (0.1 muM to 100 muM) increased human corneal epithelial cell (HCEC) viability and migration.
147 his study was designed to determine in human corneal epithelial cells (HCEC) whether the balance betw
148 ility of the dendrimer was assessed in human corneal epithelial cells (HCECs) and across isolated bov
149 ssay and immunohistochemistry study of human corneal epithelial cells (HCECs) and human keratocytes (
150 ion using an in vitro culture model of human corneal epithelial cells (HCECs) exposed to hyperosmotic
151  In this study, it was determined that human corneal epithelial cells (HCECs) express asialogangliosi
152                                Primary human corneal epithelial cells (HCECs) from limbal explants we
153 us studies have shown that wounding of human corneal epithelial cells (HCECs) results in the release
154 uction of proinflammatory mediators by human corneal epithelial cells (HCECs) stimulated by a fungal
155 eviously demonstrated that wounding of human corneal epithelial cells (HCECs) transactivates epiderma
156                           In contrast, human corneal epithelial cells (HCECs) transfected with MyD88
157                                        Human corneal epithelial cells (HCECs) were stimulated with LL
158 )) were able to bind to the surface of human corneal epithelial cells (HCECs) with similar efficiency
159  of eritoran tetrasodium on stimulated human corneal epithelial cells (HCECs), macrophages, and neutr
160 uction of proinflammatory cytokines in human corneal epithelial cells (HCECs).
161  expression by PAF was investigated in human corneal epithelial cells (HCECs).
162  SiNPs on ocular surface cells such as human corneal epithelial cells (HCECs).
163 rt a novel study of the 3D printing of human corneal epithelial cells (HCECs)/collagen/gelatin/algina
164 l extracts were obtained from cultured human corneal epithelial cells (HCEpCs) as a source of ZEB1 pr
165 ternative splicing study, immortalized human corneal epithelial cells (HCET) harboring doxycycline-in
166 rried out with telomerase-immortalized human corneal epithelial cells (HCLE).
167       However, the effect was muted in human corneal epithelial cells (HCLEs), which did not accumula
168 ggesting virulence strategies affecting live corneal epithelial cell health are selected for among ke
169 mine whether the ability to adversely affect corneal epithelial cell health is a factor common to Pse
170 er, our work defines regulatory enhancers in corneal epithelial cells, highlights global gene-regulat
171 y and angiogenesis, metastatic melanoma, and corneal epithelial cells; however, less is known about h
172  examine the ion transport mechanisms in the corneal epithelial cells; however, this technique has be
173                     CTCF activities in human corneal epithelial cells immortalized by telomerase (HTC
174 , production, and activity of MMP-9 by human corneal epithelial cells in a concentration-dependent fa
175 ophozoites of A. castellanii bound avidly to corneal epithelial cells in a mannose-inhibitable manner
176 hanced cell proliferation of wild-type mouse corneal epithelial cells in an organ culture.
177 xhibited an excessive desquamation of apical corneal epithelial cells in association with an increase
178 gnificantly decreased expression of PD-L1 by corneal epithelial cells in DED and significantly increa
179 s are the first to probe the roles played by corneal epithelial cells in reducing collagen deposition
180 gulation of human beta-defensin 2 (hBD-2) by corneal epithelial cells in response to P. aeruginosa an
181                                              Corneal epithelial cells in two-dimensional or organ cul
182 an tear fluid could protect individual human corneal epithelial cells in vitro against invasion by an
183                Pseudomonas aeruginosa enters corneal epithelial cells in vitro via membrane microdoma
184  protease activity and cytotoxicity to human corneal epithelial cells in vitro was determined.
185 it moderately) reduced electrotaxis of human corneal epithelial cells in vitro, but did not affect th
186 o keratocytes both in vitro and in vivo, and corneal epithelial cells in vitro, but it is uncertain i
187 te the effects of rapamycin on primary human corneal epithelial cells in vitro.
188 y suppress hypoxia-induced VEGF secretion by corneal epithelial cells in vitro.
189           EphA2 was immunolocalized to human corneal epithelial cells in vivo and in vitro.
190                                              Corneal epithelial cells in vivo did not stain for CFTR
191 vitro, but it is uncertain if MSC can assume corneal epithelial cells in vivo.
192 pithelial sheets (ex vivo) and primary human corneal epithelial cells (in vitro).
193 pressing a dominant-negative mutant in human corneal epithelial cells induced mesenchymal features.
194 otype was determined in vitro by quantifying corneal epithelial cell invasion by gentamicin survival
195                                PA binding to corneal epithelial cells is a prerequisite for infection
196 tion of TGF-beta2 protein by cultured rabbit corneal epithelial cells is reduced by plating on a base
197 , -3.4, -4.2, and -4.3, demonstrated that in corneal epithelial cells Kv3.4 channel was highly expres
198             To examine this further, a human corneal epithelial cell line (HCE) lacking endogenous AL
199                                   In a human corneal epithelial cell line (HCEC), 1,25(OH)2D3 increas
200 s on the gene expression of MT isoforms in a corneal epithelial cell line (HCEsv).
201 orneal basal epithelial cells and in a human corneal epithelial cell line (HTCE).
202 lly expressing miR-31 in an undifferentiated corneal epithelial cell line promotes differentiation an
203                                     A rabbit corneal epithelial cell line SIRC was used to establish
204                                      A human corneal epithelial cell line was used.
205 iated apoptosis in a telomerase-immortalized corneal epithelial cell line.
206 of cell proliferation in Pax6-overexpressing corneal epithelial cell lines and primary cell culture i
207 iated with no contact lens protection showed corneal epithelial cell loss plus lens epithelial cell s
208 ones were observed for the usually prominent corneal epithelial cell marker aquaporin 5 (AQP5), a wat
209 rogenitor cell line (TKE2) and mature murine corneal epithelial cells (MCE).
210                         The effects of CS on corneal epithelial cell migration and associated signali
211 in vivo to evaluate their ability to promote corneal epithelial cell migration and corneal wound heal
212 icient mice results from distinct defects in corneal epithelial cell migration and proliferation.
213                            Tbeta4 stimulated corneal epithelial cell migration in the presence of SSW
214 cies of bacteria inhibited human and porcine corneal epithelial cell migration in vitro and ex vivo.
215             The authors found EGF stimulated corneal epithelial cell migration in wound healing by en
216                                              Corneal epithelial cell migration was measured by using
217                             EphA2 attenuates corneal epithelial cell migration when stimulated by eph
218 ared with EGF, is a more potent activator of corneal epithelial cell migration.
219 nitude of growth factor-induced increases in corneal epithelial cell migration.
220 synthetic genes as being required to inhibit corneal epithelial cell migration.
221 EphA2 receptor and ephrin-A1 ligand in human corneal epithelial cell migration.
222 059 enhances drug permeation across cultured corneal epithelial cell monolayers by transiently affect
223 cted cells and, subsequently, promoted human corneal epithelial cell motility, migration, and wound h
224 aNp63alpha is the dominant active isoform in corneal epithelial cell nuclei.
225                       The limbal and central corneal epithelial cells of 6-week-old rats were isolate
226 6 and Oct1 are both present in the nuclei of corneal epithelial cells of the 6-week-old mouse.
227 ocytes but slightly reduced the migration of corneal epithelial cells on amnion ex vivo.
228 w BM formed by ex vivo expanded human limbal corneal epithelial cells on iAM deposits much faster and
229 the effect of overexpressing NFkappaB p50 in corneal epithelial cells on the promotion of wound heali
230 epithelium (in vivo) and in cultured primary corneal epithelial cells (PCECs) (in vitro).
231 d loss of tight junction bearing superficial corneal epithelial cells, perhaps by proteolytic cleavag
232 orneal wound-healing model and primary human corneal epithelial cells (pHCE).
233 ave demonstrated the in vitro induction of a corneal epithelial cell phenotype from pluripotent stem
234 ed decrease in CTCF activities mediate human corneal epithelial cell proliferation and apoptosis, res
235                                              Corneal epithelial cell proliferation and differentiatio
236 ing factor (CTCF) plays an important role in corneal epithelial cell proliferation by suppressing the
237  effects of 1,25(OH)2D3 and 24R,25(OH)2D3 on corneal epithelial cell proliferation, migration, and on
238 ulated, consistent with the increased Klf4CN corneal epithelial cell proliferation.
239 h factors play important roles in regulating corneal epithelial cell proliferation/differentiation du
240                                       Rabbit corneal epithelial cells (RCECs) in culture were arreste
241 PD-L1 in both vascular endothelial cells and corneal epithelial cells regulates corneal angiogenesis.
242 ude that 24R,25(OH)2D3 is likely involved in corneal epithelial cell regulation independent of 1,25(O
243 c effect of epidermal growth factor (EGF) in corneal epithelial cells required suppression of PAX6 ac
244 s were assessed in porcine and primary human corneal epithelial cells, respectively.
245                                        Human corneal epithelial cells respond rapidly following injur
246         Furthermore, H7N7 virus infection of corneal epithelial cells resulted in enhanced and signif
247  cell transplantation using cultivated human corneal epithelial cell sheets has been used successfull
248 igation into the quality of cultivated human corneal epithelial cell sheets using time-lapse imaging
249 d regenerative potential of cultivated human corneal epithelial cell sheets.
250 ether with in-vitro experiments of confluent corneal epithelial cell sheets.
251 6- or CTCF-specific small interfering RNA in corneal epithelial cells significantly promoted or atten
252 trix metalloproteinase (MMP)-9 is induced in corneal epithelial cells stimulated with platelet-activa
253             Epidermal growth factor (EGF) in corneal epithelial cells stimulates proliferation by ind
254 opening and provide new probes for exploring corneal epithelial cell stratification, development, and
255 , which can be explained by the fragility of corneal epithelial cells that did not produce Krt12 beca
256 reater desquamation of differentiated apical corneal epithelial cells that expressed the tight juncti
257 ols confirmed PA14 cytotoxicity toward these corneal epithelial cells that was absent with exoU mutan
258 ndings could be replicated in cultured human corneal epithelial cells that were treated with active M
259 s constitutively expressed at high levels by corneal epithelial cells, the authors studied the expres
260 ied epithelia), the centripetal migration of corneal epithelial cells, the exclusive location of slow
261 GFR-3), which is synthesized and secreted by corneal epithelial cells; they show that sVEGFR-3 modula
262                                           In corneal epithelial cells, this suggests that inhibition
263              HAdV-D19p is not infectious for corneal epithelial cells, thus explaining the lack of an
264       SF, a novel biomaterial, could support corneal epithelial cells to proliferate, differentiate,
265 targeting of EphA2 restricted the ability of corneal epithelial cells to seal linear scratch wounds i
266 ntiation and (3) function redundantly in the corneal epithelial cells to suppress proliferation.
267   Wound closure rates were measured in human corneal epithelial cells transfected with an NFkappaB ac
268                                        Human corneal epithelial cells, transfected with plasmids enco
269 evels of BiP/GRP78, sXBP1 and GRP94 in human corneal epithelial cells treated with TNFalpha.
270 ypes, and by in vitro studies we showed that corneal epithelial cell tropism can be predicted by the
271                   P. aeruginosa enters human corneal epithelial cells via lipid rafts containing CFTR
272                     Osmotic stress decreased corneal epithelial cell viability, which was due in part
273                                 Apoptosis of corneal epithelial cells was determined by caspase-3 and
274 y sulfated keratan sulfate in cultured human corneal epithelial cells was dramatically reduced when e
275                  Cytotoxicity on corneas and corneal epithelial cells was evaluated by LDH assays.
276  traversal of airlifted, multilayered, human corneal epithelial cells was quantified in vitro up to 8
277                Hypoxia induced VEGF by human corneal epithelial cells was sequestered by both Flt23K
278 ty of aPA and MIP-133 to induce cytolysis of corneal epithelial cells was tested in vitro.
279                             Rabbit and human corneal epithelial cells were cultured in DMEM/F12 mediu
280                                Primary human corneal epithelial cells were cultured to confluence.
281                                              Corneal epithelial cells were exposed to a soluble ephri
282                   Exosomes secreted by mouse corneal epithelial cells were found to fuse to keratocyt
283                                       Rabbit corneal epithelial cells were grown to multilayers with
284                    Primary cultures of human corneal epithelial cells were infected with a retroviral
285                                      TSLP in corneal epithelial cells were largely induced in a conce
286               To assay cell migration, human corneal epithelial cells were plated inside a cylinder a
287 nd closure and cell migration rates of human corneal epithelial cells were significantly suppressed a
288  epithelial (hTCEpi) cells and primary human corneal epithelial cells were tested for their ability t
289 e second experiment, primary cultured bovine corneal epithelial cells were transiently stressed with
290 zation of HIF-2alpha protein was detected in corneal epithelial cells, whereas HIF-1alpha protein sta
291         Accordingly, we determined, in human corneal epithelial cells, whether or not (i) CCE is depe
292 mportant sheddases of syndecan-1 shedding in corneal epithelial cells, which are natural targets of H
293   Thus, TRPC4 is a component of SOC in human corneal epithelial cells whose activation by EGF is requ
294                             Treatment of the corneal epithelial cells with 12-O-tetradecanoylphorbol-
295 termined that pretreatment of cultured human corneal epithelial cells with flagellin isolated from th
296           We found that stimulation of human corneal epithelial cells with hypoxic stress suppressed
297                 Potent transfection of human corneal epithelial cells with siRNA-ProSilic(R) formulat
298  vivo (topically in male mice) and in vitro (corneal epithelial cell wound healing).
299 ential role in growth factor-regulated human corneal epithelial cell wound healing.
300                        In VDR wildtype mouse corneal epithelial cells (WT), 1,25(OH)2D3 increased CYP

 
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