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1 hat underly HSK using a mouse model of HSV-1 corneal infection.
2 ro and to cause disease in a murine model of corneal infection.
3 ns of clodronate-containing liposomes before corneal infection.
4 that resulted in the rapid resolution of the corneal infection.
5 is an important contributor to P. aeruginosa corneal infection.
6 ral spread and promoting host survival after corneal infection.
7 rtant role in restricting HSV-1 growth after corneal infection.
8 increased HSV-1 titers in the TG after HSV-1 corneal infection.
9 nearly completely resistant to P. aeruginosa corneal infection.
10 cific IgA in mucosal secretions and prevents corneal infection.
11 anthamoeba keratitis is a vision-threatening corneal infection.
12 as as virulent as the parental strain during corneal infection.
13 red for the establishment and maintenance of corneal infection.
14 canthamoeba keratitis is a sight-threatening corneal infection.
15 evaluated in a murine model of P. aeruginosa corneal infection.
16  inside epithelial cells during experimental corneal infection.
17 eminal ganglion at various times after HSV-1 corneal infection.
18 ol the replication and spread of HSV-1 after corneal infection.
19 ginosa provokes a painful, sight-threatening corneal infection.
20 cellular and viral factors involved in acute corneal infection.
21 for the treatment of patients suffering from corneal infection.
22  epithelial cells and Pseudomonas aeruginosa corneal infection.
23  mice to experimental Pseudomonas aeruginosa corneal infection.
24 le in pathogenesis and immunity to S. aureus corneal infection.
25 wild-type virus levels after intracranial or corneal infection.
26 f epithelial cells, in Staphylococcus aureus corneal infection.
27 and mBD3 together promote resistance against corneal infection.
28 ost resistance against P. aeruginosa-induced corneal infection.
29 8HR from all tissues at late times following corneal infection.
30  to promote resistance against P. aeruginosa corneal infection.
31 ific virulence factor for C. albicans during corneal infection.
32  potential virulence factor in posttraumatic corneal infection.
33 f Acanthamoeba keratitis (AK), a devastating corneal infection.
34 amoebae produce a painful, sight-threatening corneal infection.
35 sion did not appear to be upregulated during corneal infection.
36 ct lenses and as a treatment for established corneal infections.
37 l wounds and Pseudomonas aeruginosa-mediated corneal infections.
38 mice are resistant to Pseudomonas aeruginosa corneal infections.
39 y, causing respiratory tract, cutaneous, and corneal infections.
40 ved as an essential therapeutic approach for corneal infections, allowing for rapidly eliminating the
41 s suggest that the galU gene is required for corneal infection and for efficient systemic spread foll
42 hage functions, which are switched on during corneal infection and injury.
43            This study explored their role in corneal infection and potential synergy with mBD2, a def
44 s primarily in the corneal epithelium before corneal infection and was also in the corneal stroma aft
45 at evolutionary pressure enhances fitness in corneal infection, and may be a molecular determinant in
46 essing cells during Streptococcus pneumoniae corneal infection, and P2X7R was required for bacterial
47 pread have not been fully examined following corneal infection, and the possible pathologic consequen
48                      The role of SAP6 during corneal infection appears to be associated with the morp
49                       Cosmetic CL users with corneal infections are generally young, female, and wear
50                                              Corneal infections are surprisingly rare, suggesting tha
51 rs that in the murine model of P. aeruginosa corneal infection at challenge doses of bacteria 10-fold
52  aeruginosa keratitis, functioning to reduce corneal infection (bacterial load) and inflammation by n
53             Approximately 7 days after HSV-1 corneal infection, BALB/c mice develop tissue-destructiv
54 mice, Deltavhs remains highly attenuated via corneal infection but is fully virulent via intracranial
55  100 CFU of C. albicans initiated measurable corneal infection, but 1 million or more colony-forming
56 conazole could be a good choice for treating corneal infection by Cladosporium species.
57 R is critical in resistance to P. aeruginosa corneal infection by down-regulating type 1 immunity, an
58  syndecan-1 ectodomains to promote S. aureus corneal infection by inhibiting a key arm of neutrophil
59  5c and 6 were effective in a mouse model of corneal infection by S. aureus and MRSA.
60             We conclude that transient HSV-1 corneal infections cause long-term alterations of the co
61 r the treatment of established P. aeruginosa corneal infections caused by diverse LPS serogroups.
62                             After HSV type 1 corneal infection, CD4(+) T cells are expanded in the dr
63                          After primary HSV-1 corneal infection, CD8(+) T cells infiltrate the trigemi
64 minal ganglia, and periocular skin following corneal infection compared to that with the ICP34.5-null
65 c1(-/-)) mice significantly resist S. aureus corneal infection compared with wild type (WT) mice that
66  lens wearers have much higher incidences of corneal infections compared with those of non-contact le
67                         Acute pneumonias and corneal infections due to Pseudomonas aeruginosa are typ
68 de range of recent applications that include corneal infections, dystrophies and disease.
69 its and protects BALB/c mice against primary corneal infection equivalent to a vhs- strain (BGS41).
70 eal or s.c. immunization of conidia prior to corneal infection exhibited enhanced fungal killing and
71 nd electron microscopy showed no evidence of corneal infection (experiment II).
72 anism that has the ability to cause blinding corneal infections following trauma and during contact l
73 on with aPA and MIP-133 was determined after corneal infection had been established.
74            By contrast, pigs immunized after corneal infections had been established displayed kerati
75      The involvement of ocular glands during corneal infection has been only briefly described.
76 ions but no well-described case of D. pigrum corneal infection has been reported.
77                                        After corneal infection, herpes simplex virus type 1 (HSV-1) i
78                                    Following corneal infection, HSV-1 viral titers were elevated in t
79        The influence of steroid treatment on corneal infection in a Chinese hamster model of Acantham
80 hat syndecan-1 ectodomain promotes S. aureus corneal infection in an HS-dependent manner.
81 is was examined after Pseudomonas aeruginosa corneal infection in C57BL/6 (B6, susceptible) and BALB/
82 ole of macrophages in Pseudomonas aeruginosa corneal infection in susceptible (cornea perforates), C5
83             D. pigrum is the likely cause of corneal infection in three patients, with effective anti
84 tear fluid can protect against P. aeruginosa corneal infection in vivo and that the mechanism likely
85 monas aeruginosa can cause sight-threatening corneal infections in humans, particularly those who wea
86         Pseudomonas aeruginosa causes severe corneal infections in mice after inoculations with low d
87 a vaccines delivered nasally protect against corneal infections in mice and potentially can be used t
88 for protection against Staphylococcus aureus corneal infections in mice, which are a serious cause of
89 ergillus species of mold are major causes of corneal infections in the United States and worldwide, r
90 tion of these ExsA-regulated proteins toward corneal infections in vivo was investigated.
91 evaluated for clinical score in experimental corneal infection induced by a clinical isolate and a ci
92 )-dominated response following P. aeruginosa corneal infection is associated with genetic susceptibil
93            A very intriguing aspect of HSV-1 corneal infection is that the virus spread is normally r
94                    Its role during bacterial corneal infections is unknown.
95  severity in a murine model of P. aeruginosa corneal infection (keratitis).
96   Acanthamoebae provoke a vision-threatening corneal infection known as Acanthamoeba keratitis (AK).
97 we observed that neutrophilic recruitment to corneal infections limits P. aeruginosa biofilms to the
98 vivo imaging of P. aeruginosa in implant and corneal infection mice models.
99                                         In a corneal infection model, the galU mutants were significa
100                            In a murine HSV-1 corneal infection model, we establish 17-beta estradiol
101 target antigens, of this vaccine in a murine corneal infection model.
102              Fusarium keratitis is a serious corneal infection, most commonly associated with corneal
103                                         Many corneal infections occur secondary to epithelial breache
104                                              Corneal infection of BALB/c mice with herpes simplex vir
105 f immunoinflammatory lesions associated with corneal infection of HSV.
106                                    Following corneal infection of mice with HSV type 2 (HSV-2), we qu
107 hes latency in trigeminal ganglia (TG) after corneal infection of mice.
108 hat influence immune responses to subsequent corneal infection or trauma.
109                             First, following corneal infection, OVA257-specific OT-1 CD8 T cells do n
110 , epitheliopathy (P < 0.001), suture-related corneal infections (P < 0.001), and wound dehiscence (P
111  Six hundred and ninety-four CL wearers with corneal infection presented: 204 cosmetic CL (29.4%) and
112                          Cases of CL-related corneal infection presenting over a 12-month period were
113  cytokines and chemokines upregulated during corneal infection provides important information relevan
114  TG can be detected as early as 2 days after corneal infection, reaches peak titers by 3-5 days after
115 neutralization of IL-17 during P. aeruginosa corneal infection reduces neutrophil influx and patholog
116 peptide (VIP), during Pseudomonas aeruginosa corneal infection remains unexplored.
117                        Staphylococcus aureus corneal infection results in extensive inflammation and
118 man species D adenovirus not associated with corneal infection, revealed areas of non-conserved seque
119                                      As with corneal infections, severe or prolonged inflammation jeo
120           Thus, in sharp contrast with other corneal infections such as herpes and Pseudomonas aerugi
121 clear cornea furthest away from the focus of corneal infection suggests that the host response extend
122                                         Upon corneal infection, this mutant replicates transiently bu
123 rophages control resistance to P. aeruginosa corneal infection through regulation of PMN number, bact
124  mouse model of herpes simplex virus (HSV) 1 corneal infection, tissue destruction results from a CD4
125                    In a P. aeruginosa rabbit corneal infection, topical administration of DFO-Ga toge
126  diphtheria toxin treatment 2 d before HSV-1 corneal infection transiently depleted CD11c(+) DCs from
127 eta) production after Pseudomonas aeruginosa corneal infection was examined in susceptible (cornea pe
128 ast, the time to endpoint criteria following corneal infection was greater for the US11-null virus th
129 ophages in the subconjunctival tissue before corneal infection was not accompanied by enhanced virus
130 jor participants in innate immunity to HSV-1 corneal infection was not supported.
131  in susceptibility to Pseudomonas aeruginosa corneal infection was studied in inbred C57Bl/6 (B6) bet
132        Using a murine model of S. pneumoniae corneal infection, we demonstrated a requirement for IL-
133 ma is produced during Pseudomonas aeruginosa corneal infection, we examined the role of this cytokine
134        Using a murine model of P. aeruginosa corneal infection, we show that although infected CD14(-
135         Using a murine model of A. fumigatus corneal infection, we showed that DeltarodA conidia indu
136 ection of L-Cl2MDP 4 and 2 days before HSV-1 corneal infection were found to have ocular virus titers
137 teroids for Corneal Ulcers Trial (SCUT), 500 corneal infections were treated with topical moxifloxaci
138 amoeba significantly reduced the severity of corneal infection which includes infiltration and ulcera
139 a role in the upregulation of MT-MMPs during corneal infection, which in turn can cause the destructi
140 infection with 10(1)(1) CFU S. pneumoniae or corneal infection with 10(5) CFU of S. pneumoniae.
141 , or phosphate-buffered saline (PBS), before corneal infection with 10(5) colony-forming units (CFU)
142                                    Following corneal infection with 2 x 10(6) PFU of HSV-1 strain McK
143 inhibitor also reduced clinical scores after corneal infection with a clinical isolate, KEI-1025, and
144   Eyes inoculated with C. albicans developed corneal infection with a mean clinical score of 8.2+/-0.
145                                              Corneal infection with a thymidine kinase-deleted report
146 helium renders Chinese hamsters resistant to corneal infection with Acanthamoeba castellanii.
147                                              Corneal infection with herpes simplex virus 1 (HSV-1) le
148                                              Corneal infection with herpes simplex virus-1 in immunoc
149 ely susceptible to liver infection following corneal infection with HSV-1.
150 whereas TLR4/5(-/-) mice were susceptible to corneal infection with parent strains.
151 nd chemokine mRNAs that were expressed after corneal infection with Pseudomonas aeruginosa in mice.
152                                              Corneal infection with Pseudomonas aeruginosa leads to a
153                                              Corneal infection with Pseudomonas aeruginosa perforates
154                                              Corneal infection with Pseudomonas aeruginosa results in
155 orphonuclear neutrophil (PMN) function after corneal infection with Pseudomonas aeruginosa.
156 esponses in mice that are protective against corneal infection with virulent HSV-1, but it has the ca
157 hs) protein diminishes primary and recurrent corneal infection with wild-type HSV-1.
158                                              Corneal infections with antibiotic-resistant microorgani
159    Fungal keratitis is the foremost cause of corneal infections worldwide, of which Fusariumspp. is t

 
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