ライフサイエンスコーパス: corneal injury

1 model of photorefractive keratectomy-induced corneal injury.

2 mplications of MSC-mediated tissue repair in corneal injury.

3 inhibition may be an effective treatment for corneal injury.

4 eal infection, most commonly associated with corneal injury.

5 ween proteoglycans and growth factors during corneal injury.

6 small and reached significance I clay after corneal injury.

7 directly related to area and depth of acute corneal injury.

8 des re-epithelialization after some types of corneal injury.

9 pithelial wound healing following mechanical corneal injury.

10 in a mouse model of diabetes with or without corneal injury.

11 which is significantly upregulated following corneal injury.

12 mitigate preventable causes of perioperative corneal injury.

13 caused by Rhizomucor miehei after traumatic corneal injury.

14 expansion of the stem/eTA cell cluster after corneal injury.

15 tilt position may reduce the risk of severe corneal injury.

16 ically to treat severe inflammation-mediated corneal injuries.

17 nd its impact on inflammation and healing of corneal injuries.

18 mising targets for regenerative therapies of corneal injuries.

19 f animal models of myocardial infarction and corneal injury/allogenic transplant.

20 % of traumas and were mainly responsible for corneal injuries and hyphema (30.9% and 27%, respectivel

21 Visually significant corneal injuries and subsequent scarring collectively re

22 eal neovascularization in mice 3 weeks after corneal injury and 1 week after intrastromal injection o

23 This study utilized a mouse model of corneal injury and fibrosis to determine whether macroph

24 After corneal injury and inflammation, PAF may be an important

25 SM induced corneal injury and opacity from 401 in-house rabbit corn

26 s reliable and uniform grading of SM-induced corneal injury and opacity levels in affected eyes.

27 sification model for an objective grading of corneal injury and opacity levels in live rabbits follow

28 It localises macrophages to the site of corneal injury and recruits neutrophils in lipopolysacch

29 Surprisingly, corneal injury and treatment with PEDF + DHA induced tra

30 nd protein were induced to high levels after corneal injury and were temporally and spatially correla

31 their contributions to tissue repair during corneal injury are undefined.

32 These findings provide novel evidence that corneal injury causes significant mobilization of endoge

33 revious reports indicated that pregnancy and corneal injury (CI) trigger alterations of lacrimal glan

34 Following corneal injury, coordinated cellular and protein interac

35 veal an increased incidence in perioperative corneal injuries during the COVID era and should alert c

36 eals an increased incidence in perioperative corneal injuries during the COVID era and should alert c

37 amine if there were increased peri-operative corneal injuries during the COVID pandemic compared to o

38 ular surface epithelial cells in response to corneal injury, epithelial cells undergoing DNA synthesi

39 All corneal injury event reports were aggregated for the tim

40 Comparison to known incidence of corneal injuries from the same institution in the pre CO

41 and chronic (corneal suture placement model) corneal injury, here we identified distinct functions of

42 nal study based on the hospital reporting of corneal injuries in the peri-operative time-period durin

43 Corneal injury in HO-2 null mice leads to impaired heali

44 Corneal injury in mice was induced by thermal cauterizat

45 Following the induction of corneal injury in mice, a 2-fold increase in the frequen

46 Furthermore, they strongly suggest that corneal injury induced by toxicants can be treated using

47 limination of myofibroblasts after repair of corneal injury is essential for the maintenance of corne

48 Corneal injury may ultimately lead to a scar by way of c

49 An automated method to grade corneal injury minimizes diagnostic errors and enhances

50 d upon transplantation onto eyes in a rabbit corneal injury model, these reprogrammed cells are able

51 Using two corneal injury models, we examined the temporal and spat

52 of rSEA was validated in articular joint and corneal injury models.

53 In addition, in the setting of corneal injury, MSC administration showed significant an

54 When severe retinal and corneal injury occur together, a temporary keratoprosthe

55 present study, we investigated the impact of corneal injury on the homeostasis of endogenous MSCs, an

56 t of induction of ocular pathology following corneal injury or wearing of contaminated contact lenses

57 Corneal injury produces changes in PAF-R mRNA expression

58 Corneal injury rates (in lieu of total number of events)

59 Using Poisson regression, corneal injury rates were significantly higher after Mar

60 We were aware of several adverse corneal injury reports during COVID and as a result did

61 Moreover, environmental exposure to corneal injury significantly increases growth of both bl

62 topical hepatocyte growth factor (HGF) after corneal injury suppresses the development of stromal sca

63 Using a mouse model of perforating corneal injury that causes acute and severe fluid accumu

64 potential ocular drug delivery platform for corneal injury treatment.

65 Wound healing after corneal injury typically involves fibrosis, with transfo

66 IRB approved retrospective review to see if corneal injuries were more common during that period.

67 In addition, miR-210 is decreased after corneal injury while EphA2 is increased.

68 In vivo CM revealed corneal injury with slight irritants to be restricted to