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コーパス検索結果 (1語後でソート)

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1 ys after coronary angioplasty or directional coronary atherectomy.
2 ion abnormalities associated with rotational coronary atherectomy.
3 independent risk factor for restenosis after coronary atherectomy.
4 tational atherectomy and adjunct directional coronary atherectomy.
5  and to 145 +/- 87 degrees after directional coronary atherectomy.
6 tational atherectomy and adjunct directional coronary atherectomy.
7 rences in the mechanisms by which rotational coronary atherectomy and coronary angioplasty produce th
8 on abnormalities are common after rotational coronary atherectomy and have a longer duration than tho
9 n provided, which includes PTCA, directional coronary atherectomy and high speed rotational atherecto
10 sluminal coronary angioplasty or directional coronary atherectomy and whose peak CK levels did not ex
11                                   Adjunctive coronary atherectomy (CA) can be utilized in treating se
12 ates from 16 patients undergoing directional coronary atherectomy (DCA) and control samples from the
13      Previous clinical trials of directional coronary atherectomy (DCA) have failed to show significa
14 estenosis in patients treated by directional coronary atherectomy (DCA) in the Coronary Angioplasty V
15       CAVEAT-I demonstrated that directional coronary atherectomy (DCA) resulted in higher rates of e
16                                  Directional coronary atherectomy (DCA) specimens from 63 lesions wer
17                         Previous directional coronary atherectomy (DCA) trials have shown no signific
18 s to assess serial changes after directional coronary atherectomy (DCA).
19 e mechanisms of restenosis after directional coronary atherectomy (DCA).
20       We recruited 89 patients who underwent coronary atherectomy for de novo atherosclerosis (n=55)
21 ined from 20 patients undergoing directional coronary atherectomy for stable angina were analyzed for
22  consecutive patients undergoing directional coronary atherectomy for symptomatic coronary artery dis
23 mflex artery) retrieved by using directional coronary atherectomy from 25 patients at 0.5 to 23 (mean
24              Samples acquired by directional coronary atherectomy from 25 patients with type 2 diabet
25 rimary lesions that develop restenosis after coronary atherectomy have more macrophages and smooth mu
26 tween rotational atherectomy and directional coronary atherectomy in the treatment of calcific lesion
27 domized trials of balloon angioplasty versus coronary atherectomy, laser angioplasty, or cutting ball
28                    After adjunct directional coronary atherectomy, lumen area increased even more to
29 elevation was the performance of directional coronary atherectomy (odds ratio, 4.1; P < .0001), follo
30                      VSMCs were derived from coronary atherectomies or from normal coronary arteries
31 ansluminal coronary angioplasty, directional coronary atherectomy, rotational atherectomy, or excimer
32                                    All human coronary atherectomy samples stained positive for Ang II
33                                A total of 47 coronary atherectomy specimens from patients with diabet
34                                              Coronary atherectomy specimens were definitely positive
35                                     Eighteen coronary atherectomy specimens with restenosis after PTC
36  from patients with DM were compared with 18 coronary atherectomy specimens with restenosis after PTC
37  with rotational atherectomy and directional coronary atherectomy than in those treated with rotation
38 t wall motion abnormalities after rotational coronary atherectomy, the prevalence and duration of the
39                 Macrophages are increased in coronary atherectomy tissue from primary lesions that de
40  quantified on trichrome-stained sections of coronary atherectomy tissue.
41         Using both cultured human CASMCs and coronary atherectomy tissues, we studied the roles of os
42                                  Directional coronary atherectomy was associated with a larger acute
43 mens from 90 symptomatic patients undergoing coronary atherectomy were tested for the presence of Chl
44 c plaques in patients undergoing directional coronary atherectomy with a variety of control specimens